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1. Full Text Transforming Growth Factor Beta3 Promotes Tendon Differentiation of Equine Embryo-Derived Stem Cells
by Barsby, Tom and Guest, Debbie
Tissue Engineering Part A, ISSN 1937-3341, 10/2013, Volume 19, Issue 19-20, pp. 2156 - 2165
Tendon injuries occur frequently in horses and have a poor capacity to regenerate, which leads to high re-injury rates. Equine embryo-derived stem cells (ESCs)... 
Original Articles | TRANSCRIPTION FACTOR SCLERAXIS | TGF-BETA | CONVERSION | BONE-MARROW | FLEXOR TENDON | FIBROBLASTS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | GENE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MECHANICAL FORCE | HEALING IN-VITRO | EXPRESSION | Immunohistochemistry | Animals | Transforming Growth Factor beta3 - genetics | Embryo, Mammalian - cytology | Polymerase Chain Reaction | Horses | Stem Cells - cytology | Cell Differentiation - physiology | Transforming Growth Factor beta3 - metabolism | Tissue engineering | Injuries | Stem cells | Tendons
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2. Full Text Glycoprotein A repetitions predominant (GARP) positively regulates transforming growth factor (TGF) β3 and is essential for mouse palatogenesis
by Wu, Bill X and Li, Anqi and Lei, Liming and Kaneko, Satoshi and Wallace, Caroline and Li, Xue and Li, Zihai and O'Neill, Luke
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 44, pp. 18091 - 18097
Glycoprotein A repetitions predominant (GARP) (encoded by the gene) plays important roles in cell-surface docking and activation of TGFβ. However, GARP's role... 
Phosphorylation | Transforming Growth Factor beta3 - genetics | Humans | Protein Multimerization | Palate - pathology | Recombinant Fusion Proteins - metabolism | Transforming Growth Factor beta3 - metabolism | Embryo, Mammalian - metabolism | Gene Expression Regulation, Developmental | Transforming Growth Factor beta3 - chemistry | HEK293 Cells | Female | Membrane Proteins - metabolism | Animals, Newborn | Palate - metabolism | Embryo, Mammalian - pathology | Signal Transduction | Membrane Proteins - genetics | Smad2 Protein - metabolism | Cleft Palate - embryology | Recombinant Fusion Proteins - chemistry | Embryo, Mammalian - abnormalities | Palate - embryology | Gene Knock-In Techniques | Mice, Knockout | Organogenesis | Protein Transport | Pregnancy | Animals | Membrane Proteins - chemistry | Palate - abnormalities | Cleft Palate - pathology | Heterozygote | Cleft Palate - metabolism | Protein Processing, Post-Translational | Transforming Growth Factor beta3 - agonists | Apoptosis | development | GARP, palatogenesis | cell signaling | TGFβ | apoptosis | embryo | Developmental Biology
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3. Full Text Residues of Alpha Helix H3 Determine Distinctive Features of Transforming Growth Factor β3
by Nayeem, Shahid M and Oteri, Francesco and Baaden, Marc and Deep, Shashank
The Journal of Physical Chemistry B, ISSN 1520-6106, 06/2017, Volume 121, Issue 22, pp. 5483 - 5498
Transforming growth factors (TGF-βs) are proteins that regulate cell growth by binding to their receptors. In contrast to transforming growth factor (TGF) β1,... 
Protein Conformation, alpha-Helical | Transforming Growth Factor beta3 - genetics | Transforming Growth Factor beta3 - chemistry | Molecular Dynamics Simulation | Index Medicus | Life Sciences
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4. Role of TGF-β3 in the regulation of immune responses
by Okamura, Tomohisa and Okamura, Tomohisa and Morita, Kaoru and Morita, Kaoru and Iwasaki, Yukiko and Iwasaki, Yukiko and Inoue, Mariko and Inoue, Mariko and Komai, Toshihiko and Komai, Toshihiko and Fujio, Keishi and Fujio, Keishi and Yamamoto, Kazuhiko and Yamamoto, Kazuhiko
Clinical and experimental rheumatology, ISSN 0392-856X, 2015, Volume 33, Issue 4, pp. S63 - S69
Transforming growth factor-betas (TGF-βs) are multifunctional cytokines that have been implicated in the regulation of a broad range of biological processes,... 
regulatory T cells | CD4 | LAG3 | Autoimmune disease | Egr-2 | TGF-β1 | TGF-β2 | CD25 | TGF-β3 | Humoral immunity | Inflammation Mediators - immunology | Signal Transduction | Transforming Growth Factor beta3 - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Inflammation - immunology | Transforming Growth Factor beta3 - metabolism | Transforming Growth Factor beta2 - immunology | Transforming Growth Factor beta1 - immunology | Inflammation - metabolism | Animals | Immune System - metabolism | Transforming Growth Factor beta3 - chemistry | Inflammation Mediators - metabolism | Transforming Growth Factor beta3 - immunology | Immune System - immunology | Inflammation - genetics | Inflammation - prevention & control
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5. Full Text Novel RNA-binding protein P311 binds Eukaryotic translation initiation factor 3 subunit B (eIF3b) to promote translation of transforming growth factor β1-3 (TGF-β1-3)
by Yue, Michael M and Lv, Kaosheng and Meredith, Stephen C and Martindale, Jennifer L and Gorospe, Myriam and Schuger, Lucia
Journal of Biological Chemistry, ISSN 0021-9258, 12/2014, Volume 289, Issue 49, pp. 33971 - 33983
P311, a conserved 8-kDa intracellular protein expressed in brain, smooth muscle, regenerating tissues, and malignant glioblastomas, represents the first... 
NIH 3T3 Cells | Oncogene Proteins - genetics | Protein Biosynthesis | Luciferases - metabolism | Transforming Growth Factor beta3 - genetics | Eukaryotic Initiation Factor-3 - chemistry | Transforming Growth Factor beta2 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Molecular Sequence Data | Male | Transforming Growth Factor beta3 - metabolism | Luciferases - genetics | Transforming Growth Factor beta1 - chemistry | Nerve Tissue Proteins - chemistry | Transforming Growth Factor beta2 - chemistry | Transforming Growth Factor beta3 - chemistry | Escherichia coli - metabolism | Eukaryotic Initiation Factor-3 - genetics | Binding Sites | Genes, Reporter | Recombinant Proteins - metabolism | Amino Acid Sequence | Oncogene Proteins - chemistry | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Oncogene Proteins - metabolism | Rats | Recombinant Proteins - chemistry | Eukaryotic Initiation Factor-3 - metabolism | Recombinant Proteins - genetics | Transforming Growth Factor beta1 - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Sequence Alignment | Animals | 5' Untranslated Regions | Escherichia coli - genetics | Protein Binding | Mice | Transforming Growth Factor beta2 - genetics | Transforming Growth Factor β (TGF-β) | Translation Initiation Factor | mRNA | RNA-binding Protein | Protein-Protein Interaction
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6. Full Text Transforming Growth Factor β Drives Hemogenic Endothelium Programming and the Transition to Hematopoietic Stem Cells
by Monteiro, Rui and Pinheiro, Philip and Joseph, Nicola and Peterkin, Tessa and Koth, Jana and Repapi, Emmanouela and Bonkhofer, Florian and Kirmizitas, Arif and Patient, Roger
Developmental Cell, ISSN 1534-5807, 08/2016, Volume 38, Issue 4, pp. 358 - 370
Hematopoietic stem cells (HSCs) are self-renewing multipotent stem cells that generate mature blood lineages throughout life. They, together with hematopoietic... 
EHT | TGFβ | hematopoietic stem cell | notch | jag1a | zebrafish | ZEBRAFISH EMBRYOS | VASCULAR DEVELOPMENT | TRANSGENIC ZEBRAFISH | TGF-BETA | PATHWAY | IN-VIVO | GENE-EXPRESSION | NOTCH | DEVELOPMENTAL BIOLOGY | DORSAL AORTA | AORTIC ENDOTHELIUM | CELL BIOLOGY | Zebrafish Proteins - biosynthesis | Morpholinos - genetics | Signal Transduction | Transforming Growth Factor beta3 - genetics | Animals, Genetically Modified | Transforming Growth Factor beta2 - metabolism | Zebrafish Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Jagged-1 Protein - biosynthesis | Transforming Growth Factor beta1 - genetics | Vascular Endothelial Growth Factor A - metabolism | Zebrafish - embryology | Notochord - embryology | Transforming Growth Factor beta3 - metabolism | Jagged-1 Protein - genetics | Animals | Multipotent Stem Cells - cytology | Hematopoietic Stem Cells - cytology | Epithelial-Mesenchymal Transition | Cell Differentiation | Transforming Growth Factor beta2 - genetics | Zebrafish Proteins - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Endothelium, Vascular - embryology
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7. Full Text Biological Role and Therapeutic Targeting of TGF-β 3 in Glioblastoma
by Seystahl, Katharina and Papachristodoulou, Alexandros and Burghardt, Isabel and Schneider, Hannah and Hasenbach, Kathy and Janicot, Michel and Roth, Patrick and Weller, Michael
Molecular cancer therapeutics, ISSN 1535-7163, 06/2017, Volume 16, Issue 6, pp. 1177 - 1186
Transforming growth factor (TGF)-β contributes to the malignant phenotype of glioblastoma by promoting invasiveness and angiogenesis and creating an... 
Phosphorylation | Prognosis | Transforming Growth Factor beta3 - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | RNA, Messenger - metabolism | Transforming Growth Factor beta3 - metabolism | Heterografts | Glioblastoma - genetics | Smad2 Protein - genetics | Glioblastoma - metabolism | Disease Models, Animal | Gene Expression | Signal Transduction | RNA, Messenger - genetics | Gene Silencing | Smad2 Protein - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Transforming Growth Factor beta1 - genetics | Animals | Oligonucleotides, Antisense - genetics | Protein Isoforms | Cell Line, Tumor | Mice | Transforming Growth Factor beta2 - genetics | Glioblastoma - drug therapy | Glioblastoma - mortality
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8. Full Text Effect of TGFβ1, TGFβ3 and keratinocyte conditioned media on functional characteristics of dermal fibroblasts derived from reparative (Balb/c) and regenerative (Foxn1 deficient; nude) mouse models
by Bukowska, Joanna and Kopcewicz, Marta and Kur-Piotrowska, Anna and Szostek-Mioduchowska, Anna Z and Walendzik, Katarzyna and Gawronska-Kozak, Barbara
Cell and Tissue Research, ISSN 0302-766X, 10/2018, Volume 374, Issue 1, pp. 149 - 163
Skin injuries in mammals are healed through repair or regeneration. Our previous studies demonstrated that deficient expression of the transcription factor... 
Human Genetics | Dermal fibroblasts | Biomedicine | Wound healing | Proteomics | TGFβ | Molecular Medicine | Skin | Foxn1 | Transforming Growth Factor beta3 - pharmacology | Skin - cytology | Transforming Growth Factor beta3 - genetics | Skin - metabolism | Keratinocytes - cytology | Transforming Growth Factor beta1 - genetics | Culture Media, Conditioned | Animals | Keratinocytes - metabolism | Mice, Nude | Fibroblasts - cytology | Mice | Mice, Inbred BALB C | Transforming Growth Factor beta3 - biosynthesis | Forkhead Transcription Factors - deficiency | Transforming Growth Factor beta1 - biosynthesis | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Fibroblasts - metabolism | Phenotypes | Collagen (type I) | Animal models | Transforming growth factor | Migration | Homeostasis | Smooth muscle | Epidermis | Seeding | Contraction | Actin | Fibroblasts | Regular
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9. Full Text Transforming growth factor beta family: Insight into the role of growth factors in regulation of fracture healing biology and potential clinical applications
by Poniatowski, Lukasz A and Wojdasiewicz, Piotr and Gasik, Robert and Szukiewicz, Dariusz
Mediators of Inflammation, ISSN 0962-9351, 2015, Volume 2015, pp. 137823 - 17
The transforming growth factor beta (TGF-beta) family forms a group of three isoforms, TGF-beta 1, TGF-beta 2, and TGF-beta 3, with their structure formed by... 
SIGNAL-TRANSDUCTION | PLATELET-RICH PLASMA | IGF-I | ACTIVATED PROTEIN-KINASE | BONE-MINERAL DENSITY | I RECEPTOR | BINDING-PROTEIN | OSTEOBLAST-LIKE CELLS | IMMUNOLOGY | LATENT TGF-BETA | SOFT-TISSUE INJURY | CELL BIOLOGY | Transforming Growth Factor beta3 - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Transforming Growth Factor beta1 - genetics | Transforming Growth Factor beta3 - metabolism | Cell Differentiation - genetics | Animals | Transforming Growth Factor beta - genetics | Fracture Healing - physiology | Wound Healing - physiology | Fracture Healing - genetics | Transforming Growth Factor beta2 - genetics | Wound Healing - genetics | Cell Differentiation - physiology | Transforming Growth Factor beta - metabolism | Medical research | Fractures | Physiological aspects | Medicine, Experimental | Healing | Research | Transforming growth factors | Health aspects | Polypeptides | Cytokines | Rheumatology | Proteins | Signal transduction | Osteoporosis | Musculoskeletal system | Immunology | Ligands | Clinical medicine | Chromosomes | Growth factors | Review
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10. Full Text Generation of mice with a conditional allele for the transforming growth factor beta3 gene
by Doetschman, Thomas and Georgieva, Teodora and Li, Hongqi and Reed, Thomas D and Grisham, Christina and Friel, Jacqueline and Estabrook, Mark A and Gard, Connie and Sanford, L.P and Azhar, Mohamad
genesis, ISSN 1526-954X, 01/2012, Volume 50, Issue 1, pp. 59 - 66
The transforming growth factor beta (TGFβ) pathway is involved in embryonic development and several inherited and acquired human diseases. The gene for TGFβ3... 
wound healing | neuromuscular | autoimmunity | transforming growth factor beta | craniofacial | cancer | cardiovascular | Autoimmunity | Neuromuscular | Wound healing | Cardiovascular | Craniofacial | Transforming growth factor beta | Cancer | TRANSFORMING-GROWTH-FACTOR-BETA-3 | FIBROSIS | TGF-BETA-3 | TGF-BETA | CARDIOMYOPATHY | DEVELOPMENTAL BIOLOGY | DISRUPTION | GENETICS & HEREDITY | MUTATIONS | EXPRESSION | GROWTH-FACTOR-BETA-1 | ASSOCIATION | Transforming Growth Factor beta3 - genetics | Exons | Mice, Inbred C57BL | Cleft Palate - embryology | Male | Sequence Analysis, DNA | Transforming Growth Factor beta3 - metabolism | Mice, Knockout | Phenotype | Animals | Receptors, Transforming Growth Factor beta - metabolism | Gene Expression Regulation, Developmental | Alleles | Female | Mice | Real-Time Polymerase Chain Reaction
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11. Full Text Survivin-TGFB3-TIMP1 Gene Therapy Via Lentivirus Vector Slows the Course of Intervertebral Disc Degeneration in an In Vivo Rabbit Model
by Yue, Bin and Lin, YaZhou and Ma, XueXiao and Xiang, HongFei and Qiu, ChenSheng and Zhang, JianHua and Li, LongYang and Chen, BoHua
SPINE, ISSN 0362-2436, 06/2016, Volume 41, Issue 11, pp. 926 - 934
STUDY DESIGN.The ability of lentivirus vector (LV) survivin-transforming growth factor beta 3 (TGFB3)-tissue inhibitor of metalloproteinases 1 (TIMP1) on... 
intervertebral disc degeneration | animal experimentation | survivin | TIMP1 | genetic therapy | TGFB3 | NUCLEUS PULPOSUS | TGF-BETA | FUSION | LOW-BACK-PAIN | CULTURE | CLINICAL NEUROLOGY | PUNCTURE | INJECTION | INHIBITOR | ORTHOPEDICS | EXPRESSION | Rabbits | Inhibitor of Apoptosis Proteins - biosynthesis | Genetic Vectors - administration & dosage | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Transforming Growth Factor beta3 - genetics | Inhibitor of Apoptosis Proteins - genetics | Inhibitor of Apoptosis Proteins - administration & dosage | Genetic Vectors - genetics | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - therapy | Tissue Inhibitor of Metalloproteinase-1 - genetics | Tissue Inhibitor of Metalloproteinase-1 - administration & dosage | Animals | Female | Lentivirus - genetics | Transforming Growth Factor beta3 - biosynthesis | Transforming Growth Factor beta3 - administration & dosage | Disease Models, Animal | Genetic Therapy - methods | Intervertebral Disc Degeneration - genetics
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12. Full Text Regenerating Cartilages by Engineered ASCs: Prolonged TGF-β3/BMP-6 Expression Improved Articular Cartilage Formation and Restored Zonal Structure
by Lu, Chia-Hsin and Yeh, Tsung-Szu and Yeh, Chia-Lin and Fang, Yu-Hua Dean and Sung, Li-Yu and Lin, Shih-Yeh and Yen, Tzu-Chen and Chang, Yu-Han and Hu, Yu-Chen
Molecular Therapy, ISSN 1525-0016, 01/2014, Volume 22, Issue 1, pp. 186 - 195
Adipose-derived stem cells (ASCs) hold promise for cartilage regeneration but their chondrogenesis potential is inferior. Here, we used a baculovirus (BV)... 
Rabbits | Gene Expression | Transduction, Genetic | Transforming Growth Factor beta3 - genetics | Chondrogenesis - genetics | Adipose Tissue - cytology | Male | Cartilage - metabolism | Stem Cells - metabolism | Transforming Growth Factor beta3 - metabolism | Genetic Vectors - genetics | Tissue Scaffolds | Biomechanical Phenomena | Animals | Bone Morphogenetic Protein 6 - metabolism | Bone Morphogenetic Protein 6 - genetics | Cartilage - cytology | Gene Order | Guided Tissue Regeneration | Tissue Engineering | Original
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13. Full Text MicroRNAs miR-26a, miR-26b, and miR-29b accelerate osteogenic differentiation of unrestricted somatic stem cells from human cord blood
by Trompeter, Hans-Ingo and Dreesen, Janine and Hermann, Eugenie and Iwaniuk, Katharina M and Hafner, Markus and Renwick, Neil and Tuschl, Thomas and Wernet, Peter
BMC Genomics, ISSN 1471-2164, 02/2013, Volume 14, Issue 1, pp. 111 - 111
Background: MicroRNAs are a population of short non-coding RNAs with widespread negative regulatory impact on mRNA translation. Unrestricted somatic stem cells... 
MicroRNA target identification | Osteogenic differentiation | Cord blood stem cells | MicroRNA expression | MicroRNA function | TRANSCRIPTION FACTORS | OSTEOBLAST DIFFERENTIATION | TGF-BETA | MEDIATED DOWN-REGULATION | RUNX2 | LINEAGE COMMITMENT | CDK6 | PATHWAY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | IN-VITRO DIFFERENTIATION | SKELETAL DEVELOPMENT | Up-Regulation | Adult Stem Cells - cytology | Tumor Suppressor Proteins - metabolism | Histone Deacetylases - genetics | Transforming Growth Factor beta3 - genetics | Humans | Computational Biology | Cyclin-Dependent Kinase 6 - genetics | Repressor Proteins - genetics | Histone Deacetylases - metabolism | Cyclin-Dependent Kinase 6 - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Transcriptome - genetics | Fetal Blood - cytology | Fetal Blood - metabolism | Transforming Growth Factor beta3 - metabolism | Cell Differentiation - genetics | Adult Stem Cells - metabolism | Tumor Suppressor Proteins - genetics | MicroRNAs - genetics | Intracellular Signaling Peptides and Proteins - genetics | Osteogenesis - genetics | Repressor Proteins - metabolism | Physiological aspects | Genetic aspects | MicroRNA | Research | Cell differentiation | Stem cells | Hematopoietic stem cells | Proteins | Cell cycle | Genetic engineering | Software | Molecular biology | Experiments | Milk
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14. Full Text ATP-citrate lyase is essential for high glucose-induced histone hyperacetylation and fibrogenic gene upregulation in mesangial cells
by Deb, DK and Chen, YY and Sun, J and Wang, YL and Li, YC
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, ISSN 1931-857X, 08/2017, Volume 313, Issue 2, pp. F423 - F429
The goal of this study was to address the role of ATP-citrate lyase (ACL), an enzyme that converts citrate to acetyl-CoA, in high glucose (HG)-induced histone... 
PHYSIOLOGY | TGF-BETA | glomerulosclerosis | CHROMATIN | ACETYLATION | PHOSPHORYLATION | TRANSCRIPTION | DIABETIC-NEPHROPATHY | epigenetic regulation | hyperglycemia | METABOLISM | DISEASE | UROLOGY & NEPHROLOGY | GROWTH-FACTOR | EXPRESSION | histone acetylation | ATP-citrate lyase | Up-Regulation | Diabetic Nephropathies - enzymology | Transforming Growth Factor beta3 - genetics | Transforming Growth Factor beta1 - metabolism | Transforming Growth Factor beta3 - metabolism | Mesangial Cells - drug effects | Transfection | Protein Processing, Post-Translational - drug effects | RNA Interference | Time Factors | Mesangial Cells - enzymology | Glucose - toxicity | Epigenesis, Genetic - drug effects | Acetylation | Active Transport, Cell Nucleus | ATP Citrate (pro-S)-Lyase - metabolism | Collagen Type IV - metabolism | Diabetic Nephropathies - pathology | ATP Citrate (pro-S)-Lyase - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Acetyl Coenzyme A - metabolism | Fibronectins - metabolism | Animals | Histones - genetics | Fibrosis | Mesangial Cells - pathology | Collagen Type IV - genetics | Citric Acid - metabolism | Connective Tissue Growth Factor - genetics | Fibronectins - genetics | Mice | Cell Line, Transformed | Connective Tissue Growth Factor - metabolism
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15. Full Text TGF‐β3‐induced miR‐494 inhibits macrophage polarization via suppressing PGE2 secretion in mesenchymal stem cells
by Zhao, Guangfeng and Miao, Huishuang and Li, Xiujun and Chen, Shiwen and Hu, Yali and Wang, Zhiqun and Hou, Yayi
FEBS Letters, ISSN 0014-5793, 06/2016, Volume 590, Issue 11, pp. 1602 - 1613
Abnormal macrophage polarization at the maternal–fetal interface may contribute to the development of Preeclampsia (PE). The reason why macrophage polarization... 
mesenchymal stem cell | macrophage polarization | preeclampsia | TGF‐β3 | miR‐494 | Mesenchymal stem cell | TGF-β3 | Macrophage polarization | MiR-494 | Preeclampsia | miR-494 | Dinoprostone - secretion | Macrophages - physiology | Macrophage Activation - genetics | Decidua - immunology | Transforming Growth Factor beta3 - genetics | Humans | Cells, Cultured | Gene Expression Regulation | Decidua - pathology | Mesenchymal Stromal Cells - secretion | Pre-Eclampsia - pathology | Decidua - metabolism | Pregnancy | Young Adult | Cell Polarity - genetics | Maternal-Fetal Relations | Transforming Growth Factor beta3 - physiology | Adult | Female | MicroRNAs - genetics | MicroRNAs - physiology | Pre-Eclampsia - immunology
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