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extracellular matrix proteins - metabolism (65) 65
gene (65) 65
mice, inbred c57bl (65) 65
rodents (64) 64
abridged index medicus (63) 63
differentiation (63) 63
proliferation (63) 63
transforming growth factor-beta (63) 63
gene expression regulation (62) 62
up-regulation (62) 62
smad (60) 60
signal transduction - physiology (59) 59
corneal dystrophies, hereditary - genetics (58) 58
genetic aspects (58) 58
rna, messenger - metabolism (58) 58
smad3 protein - metabolism (58) 58
dna-binding proteins - metabolism (57) 57
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Journal Article
Investigative ophthalmology & visual science, ISSN 0146-0404, 03/2019, Volume 60, Issue 4, pp. 978 - 989
Journal Article
Structure, ISSN 0969-2126, 11/2017, Volume 25, Issue 11, pp. 1740 - 1750.e2
A major cause of visual impairment, corneal dystrophies result from accumulation of protein deposits in the cornea. One of the proteins involved is... 
extracellular matrix protein | cysteine-rich CROPT domain | multi-domain protein | pathological mutants | crystal structure | corneal dystrophy | fasciclin FAS1 domain | GENOTYPE-PHENOTYPE CORRELATIONS | KERATO-EPITHELIN MUTATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID DEPOSITS | STRUCTURE ALIGNMENT | CELL BIOLOGY | BIOPHYSICS | STRUCTURE VALIDATION | MACROMOLECULAR CRYSTALLOGRAPHY | CHINESE PATIENTS | BIGH3 GENE-MUTATIONS | STRUCTURE PREDICTION | GRANULAR DYSTROPHY | Protein Aggregates | Humans | Integrins - chemistry | Corneal Dystrophies, Hereditary - genetics | Integrins - genetics | Crystallography, X-Ray | Transforming Growth Factor beta - chemistry | Integrins - metabolism | Corneal Dystrophies, Hereditary - pathology | Cloning, Molecular | HEK293 Cells | Protein Interaction Domains and Motifs | Binding Sites | Protein Aggregation, Pathological - genetics | Extracellular Matrix Proteins - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Protein Conformation, alpha-Helical | Extracellular Matrix Proteins - chemistry | Gene Expression | Genetic Vectors - chemistry | Extracellular Matrix Proteins - genetics | Genetic Vectors - metabolism | Models, Molecular | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Corneal Dystrophies, Hereditary - metabolism | Sequence Homology, Amino Acid | Sequence Alignment | Transforming Growth Factor beta - genetics | Protein Conformation, beta-Strand | Protein Binding | Mutation | Transforming Growth Factor beta - metabolism | Protein Aggregation, Pathological - metabolism | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 03/2016, Volume 6, Issue 1, pp. 23836 - 23836
Journal Article
Autophagy, ISSN 1554-8627, 12/2012, Volume 8, Issue 12, pp. 1782 - 1797
Granular corneal dystrophy type 2 (GCD2) is an autosomal dominant disease characterized by a progressive age-dependent extracellular accumulation of... 
SQSTM1/p62 | TGFBI | defective autophagy | ubiquitination | LC3/MAP1LC3B | rapamycin | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Ubiquitination | Rapamycin | Defective autophagy | APOPTOSIS | KERATO-EPITHELIN MUTATIONS | AGGREGATE-PRONE PROTEINS | NEURODEGENERATION | ALPHA-SYNUCLEIN | CELL-DEATH | MITOCHONDRIAL FISSION | CELL BIOLOGY | BETA-IG-H3 | DISEASE | DEGRADATION | Humans | Middle Aged | Caspase 3 - metabolism | Male | Fibroblasts - ultrastructure | Autophagy - drug effects | Young Adult | Proteolysis - drug effects | Cornea - drug effects | Lysosomes - metabolism | Corneal Dystrophies, Hereditary - pathology | Adult | Female | Cornea - pathology | Cell Death - drug effects | Child | Extracellular Matrix Proteins - metabolism | Membrane Fusion - drug effects | Fibroblasts - metabolism | Phagosomes - drug effects | Biomarkers - metabolism | Lysosomes - drug effects | Cell Survival - drug effects | Intracellular Space - drug effects | Phagosomes - metabolism | Mutant Proteins - metabolism | Corneal Dystrophies, Hereditary - metabolism | Enzyme Activation - drug effects | Fibroblasts - pathology | Sirolimus - pharmacology | Cornea - metabolism | Intracellular Space - metabolism | Kinetics | Transforming Growth Factor beta - metabolism | Index Medicus | MAP1LC3B | Basic Research Paper | LC3 | SQSTM1 | p62
Journal Article
Journal Article
Journal Article
International Journal of Oncology, ISSN 1019-6439, 10/2011, Volume 39, Issue 4, pp. 1001 - 1009
As an uncommon cancer, mesothelioma is very hard to treat with a low average survival rate owing to its usual late detection and being highly invasive. The... 
Extracellular matrix | Mouse model | Metastasis | TGFBI | Adhesion-mediated signaling | adhesion-mediated signaling | mouse model | BRONCHIAL EPITHELIAL-CELLS | MALIGNANT PLEURAL MESOTHELIOMA | TUMORIGENIC PHENOTYPE | BETAIG-H3 GENE | MTOR | PATHOGENESIS | LUNG-CANCER | ONCOLOGY | metastasis | BETA-IG-H3 | extracellular matrix | ASBESTOS | Asbestos - toxicity | Cyclin D1 - metabolism | RNA, Small Interfering - genetics | Mesothelioma - pathology | Oncogene Proteins v-mos - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Gene Knockdown Techniques - methods | DNA-Binding Proteins - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Neoplasms, Mesothelial - pathology | Proto-Oncogene Proteins c-akt - metabolism | Extracellular Matrix Proteins - metabolism | Neoplasms, Mesothelial - genetics | Cell Line | Cell Growth Processes - drug effects | Extracellular Matrix Proteins - antagonists & inhibitors | Extracellular Matrix Proteins - genetics | Epidermal Growth Factor - metabolism | Mesothelioma - genetics | Disease Progression | Transcription Factors - metabolism | Mesothelioma - metabolism | Transforming Growth Factor beta - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Protein Biosynthesis - drug effects | Neoplasms, Mesothelial - metabolism | Mutation | Cell Cycle - drug effects | RNA, Small Interfering - administration & dosage | Transforming Growth Factor beta - metabolism | Mesothelioma - etiology
Journal Article
Journal Article
Journal Article
Chinese Journal of Clinical Oncology, ISSN 1000-8179, 01/2012, Volume 39, Issue 2, pp. 77 - 79
Journal Article