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Circulation research, ISSN 0009-7330, 07/2011, Volume 109, Issue 3, pp. e14 - e26
.... Despite the lack of angiogenesis, the expression of vascular endothelial growth factor A (VEGF) was shown to be upregulated in SSc skin and circulation... 
antiangiogenic agents | systemic sclerosis | peripheral vascular diseases | vascular endothelial growth factor A | angiogenesis | scleroderma | Phosphorylation - physiology | Gene Expression - drug effects | Humans | Transforming Growth Factor beta1 - metabolism | Culture Media, Conditioned - pharmacology | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | RNA-Binding Proteins | Phosphoproteins - metabolism | Vascular Endothelial Growth Factor A - genetics | Gene Expression - physiology | Scleroderma, Systemic - physiopathology | Dermis - blood supply | Neovascularization, Pathologic - physiopathology | Phosphorylation - drug effects | Nuclear Proteins - genetics | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Transforming Growth Factor beta1 - pharmacology | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Ischemia - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Alternative Splicing - physiology | Nuclear Proteins - metabolism | Transforming Growth Factor beta1 - genetics | Ischemia - physiopathology | Phosphoproteins - genetics | Vascular Endothelial Growth Factor B - pharmacology | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Endothelial Cells - cytology | Serine-Arginine Splicing Factors | Neovascularization, Pathologic - genetics | Signal Transduction - physiology | Vascular Endothelial Growth Factor B - genetics | Neovascularization, Pathologic - metabolism | Index Medicus
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 08/2006, Volume 177, Issue 3, pp. 1872 - 1878
.... Inhibition of the alternative pathway by factor B ARNA resulted in decreased levels of membrane attack complex and angiogenic factors-vascular endothelial growth factor and TGF-beta 2... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Complement C5 - genetics | Complement Factor H - antagonists & inhibitors | Complement C1q - biosynthesis | Vascular Endothelial Growth Factor A - biosynthesis | Injections, Intravenous | Complement Membrane Attack Complex - metabolism | Male | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Transforming Growth Factor beta - biosynthesis | Complement Factor B - genetics | Lasers | Transforming Growth Factor beta - antagonists & inhibitors | Complement Pathway, Alternative - genetics | Transforming Growth Factor beta2 | Complement Factor B - physiology | Complement Factor H - physiology | Choroidal Neovascularization - immunology | Mice, Inbred C57BL | Up-Regulation - genetics | Choroidal Neovascularization - prevention & control | Complement Factor H - biosynthesis | Complement C1q - genetics | Complement C5 - deficiency | Complement C4 - genetics | Complement Factor B - biosynthesis | Complement C4 - deficiency | Down-Regulation - genetics | Mice, Knockout | Animals | Up-Regulation - immunology | Complement C1q - antagonists & inhibitors | Down-Regulation - immunology | Complement Pathway, Alternative - immunology | Mice | RNA, Small Interfering - administration & dosage | Choroidal Neovascularization - genetics | Complement Factor B - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 05/2018, Volume 557, Issue 7707, pp. 724 - 728
Microglia and astrocytes modulate inflammation and neurodegeneration in the central nervous system (CNS)(1-3). Microglia modulate pro-inflammatory and... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | Microglia - metabolism | Central Nervous System - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Astrocytes - pathology | Central Nervous System - pathology | Vascular Endothelial Growth Factor B - biosynthesis | Tryptophan - metabolism | Lipopolysaccharide Receptors - metabolism | Inflammation - metabolism | Tryptophan - deficiency | Microglia - pathology | Receptors, Aryl Hydrocarbon - metabolism | Female | Transforming Growth Factor alpha - metabolism | Central Nervous System - microbiology | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - microbiology | Multiple Sclerosis - metabolism | Transforming Growth Factor alpha - biosynthesis | Symbiosis | Inflammation - microbiology | Encephalomyelitis, Autoimmune, Experimental - pathology | ErbB Receptors - metabolism | Mice, Inbred C57BL | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Animals | Vascular Endothelial Growth Factor B - metabolism | Encephalomyelitis, Autoimmune, Experimental - prevention & control | Multiple Sclerosis - pathology | Inflammation - prevention & control | Mice | Astrocytes - metabolism | Physiological aspects | Microbiological research | Metabolites | Transforming growth factors | Astrocytes | Vascular endothelial growth factor | Regulators | Multiple sclerosis | Transcription | Central nervous system | Hydrocarbons | Nervous system | CD14 antigen | Recruitment | Angiogenesis | Signal transduction | Neurotoxicity | Microorganisms | Neurodegeneration | Aromatic compounds | Bone marrow | ErbB-1 protein | Tryptophan | Inflammation | Gene expression | Experimental allergic encephalomyelitis | Microglia | Neurological diseases | Diet | Lymphocytes B | Flora | Spinal cord injuries | Alzheimers disease | Index Medicus | Life Sciences | Immunology
Journal Article
Journal Article
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 02/2018, Volume 58, Issue 2, pp. 194 - 207
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90353 - e90353
Journal Article
PloS one, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54001 - e54001
... of extracellular matrix (ECM) proteins [1], [2]. It has been demonstrated that transforming growth factor-beta1 (TGF-beta1) is the most important growth factor in inducing... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Kidney - pathology | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Smad3 Protein - metabolism | Ureteral Obstruction - metabolism | Leukocytes - immunology | Kidney - metabolism | Receptor, Epidermal Growth Factor - metabolism | Benzamides - therapeutic use | Benzamides - pharmacology | Phosphorylation - drug effects | Ureteral Obstruction - drug therapy | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Pyridines - therapeutic use | Collagen Type I - metabolism | Kidney - drug effects | Histone Deacetylases - metabolism | Fibroblasts - pathology | Fibronectins - metabolism | Ureteral Obstruction - immunology | Gene Expression Regulation - drug effects | Acetylation - drug effects | Animals | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Fibroblasts - drug effects | Ureteral Obstruction - pathology | Fibrosis | Histone Deacetylase Inhibitors - pharmacology | Leukocytes - drug effects | Cell Proliferation - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Mice | Pyridines - pharmacology | Histones - metabolism | Fibronectins | Muscle proteins | Growth factors | Collagen | Analysis | Drugs | Histone deacetylase | Phosphorylation | Smad protein | Animal models | Collagen (type I) | Nephrology | Transcription | Syngeneic grafts | Transforming growth factor-b | Smooth muscle | Activation | Kinases | Fibronectin | Medical schools | Epidermal growth factor | Actin | Rodents | Cell cycle | Fibroblasts | Ureter | Inhibition | Kidneys | Transforming growth factor-b1 | Epidermal growth factor receptors | Pulmonary arteries | Fibrils | Muscles | Medicine | Signaling | Hospitals | Kidney diseases | Cancer | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/1995, Volume 92, Issue 21, pp. 9570 - 9574
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 04/2019, Volume 69, Issue 4, pp. 1375 - 1378
... TGF‐β transforming growth factor beta TGFBR TGF‐β receptor TIC tumor‐initiating cell Hepatocellular carcinoma (HCC), accounting for 85%‐90% of all primary liver... 
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Hepatocytes | Humans | Receptors, Transforming Growth Factor beta | Liver Neoplasms | Carcinogenesis | Transforming Growth Factors | Transforming Growth Factor beta | Liver cancer | Transforming growth factor-b | Index Medicus
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 03/2019, Volume 50, Issue 3, pp. 645 - 654.e6
Journal Article