Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (4662) 4662
Book Review (741) 741
Publication (518) 518
Newsletter (117) 117
Book Chapter (52) 52
Conference Proceeding (27) 27
Book / eBook (6) 6
Dissertation (6) 6
Magazine Article (3) 3
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (4041) 4041
humans (2975) 2975
animals (1562) 1562
tissue transglutaminase (1446) 1446
transglutaminases - metabolism (1298) 1298
male (1176) 1176
transglutaminases - genetics (1168) 1168
female (1120) 1120
transglutaminase (1112) 1112
celiac disease (942) 942
biochemistry & molecular biology (901) 901
proteins (749) 749
mice (741) 741
adult (634) 634
transglutaminases (624) 624
celiac disease - immunology (584) 584
cell biology (583) 583
transglutaminases - immunology (559) 559
expression (513) 513
research (508) 508
molecular sequence data (507) 507
gluten (503) 503
middle aged (491) 491
amino acid sequence (478) 478
cross-linking (466) 466
celiac disease - diagnosis (462) 462
enzymes (457) 457
gastroenterology & hepatology (449) 449
child (445) 445
gtp-binding proteins - metabolism (445) 445
analysis (440) 440
prevalence (433) 433
diagnosis (425) 425
immunology (423) 423
adolescent (419) 419
antibodies (395) 395
transglutaminase 2 (381) 381
apoptosis (378) 378
gtp-binding proteins - genetics (368) 368
identification (361) 361
cells, cultured (349) 349
gene expression (347) 347
child, preschool (336) 336
aged (322) 322
children (320) 320
biopsy (313) 313
autoantibodies (296) 296
abridged index medicus (294) 294
protein (287) 287
base sequence (282) 282
genetic aspects (282) 282
peptides (281) 281
mutation (280) 280
cells (279) 279
dermatology (276) 276
physiological aspects (274) 274
biochemistry (272) 272
celiac disease - genetics (272) 272
autoantibodies - blood (268) 268
celiac-disease (268) 268
activation (266) 266
autoimmunity (265) 265
gluten-free diet (265) 265
transglutaminases - chemistry (258) 258
gene (240) 240
article (239) 239
gliadin (236) 236
rats (236) 236
celiac disease - epidemiology (235) 235
infant (235) 235
oncology (235) 235
gliadin - immunology (227) 227
celiac disease - pathology (224) 224
research article (224) 224
multidisciplinary sciences (222) 222
differentiation (221) 221
cell line (219) 219
gtp-binding proteins - immunology (217) 217
coeliac disease (215) 215
risk factors (214) 214
disease (213) 213
medicine (212) 212
food science & technology (209) 209
immunoglobulin a - blood (209) 209
immunohistochemistry (209) 209
health aspects (205) 205
biotechnology & applied microbiology (200) 200
pediatrics (195) 195
genes (193) 193
rna, messenger - metabolism (193) 193
celiac disease - complications (190) 190
life sciences (190) 190
microbial transglutaminase (190) 190
genetics & heredity (186) 186
signal transduction (185) 185
medical research (179) 179
protein binding (177) 177
young adult (176) 176
cell line, tumor (174) 174
transglutaminases - biosynthesis (173) 173
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (4732) 4732
Chinese (20) 20
French (16) 16
Spanish (12) 12
German (10) 10
Japanese (8) 8
Portuguese (4) 4
Italian (3) 3
Swedish (3) 3
Czech (1) 1
Dutch (1) 1
Hebrew (1) 1
Persian (1) 1
Polish (1) 1
Russian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Risk of Pediatric Celiac Disease According to HLA Haplotype and Country
by Liu, Edwin and Lee, Hye-Seung and Aronsson, Carin A and Hagopian, William A and Koletzko, Sibylle and Rewers, Marian J and Eisenbarth, George S and Bingley, Polly J and Bonifacio, Ezio and Simell, Ville and Agardh, Daniel and TEDDY Study Grp and TEDDY Study Group and Lund University and Diabetes och celiaki and Diabetes and Celiac Unit and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
The New England Journal of Medicine, ISSN 0028-4793, 07/2014, Volume 371, Issue 1, pp. 42 - 49
In this cohort study involving 6403 children at increased risk for celiac disease on the basis of HLA haplotype, the risks of celiac disease autoimmunity and... 
ANTIBODIES | MEDICINE, GENERAL & INTERNAL | GLUTEN | AUTOIMMUNITY | GENE DOSAGE | SUSCEPTIBILITY | TEDDY | CHILDHOOD | CHILDREN | Autoimmune Diseases - epidemiology | United States - epidemiology | Genetic Predisposition to Disease | Prospective Studies | Celiac Disease - genetics | Humans | Kaplan-Meier Estimate | Child, Preschool | Europe - epidemiology | Infant | Male | Risk | Transglutaminases - immunology | Autoimmune Diseases - genetics | Homozygote | HLA-DR4 Antigen - genetics | HLA-DR3 Antigen - genetics | HLA-DQ Antigens - genetics | Antibodies - blood | Female | Celiac Disease - epidemiology | Infant, Newborn | Haplotypes | Viral antibodies | Celiac disease | Histocompatibility antigens | Analysis | HLA histocompatibility antigens | Antibodies | Children | Health aspects | Risk factors | Diabetes mellitus | Heterozygotes | Environmental factors | Small intestine | Transglutaminase 2 | Children & youth | Homozygotes | Genotype & phenotype | Biopsy | Histocompatibility antigen HLA | Autoimmune diseases | Age | Genotypes | Immune system | Index Medicus | Abridged Index Medicus | Autoimmune Diseases: epidemiology | Celiac Disease: genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Europe: epidemiology | Antibodies: blood | HLA-DQ Antigens: genetics | HLA-DR3 Antigen: genetics | Klinisk medicin | Transglutaminases: immunology | United States: epidemiology | HLA-DR4 Antigen: genetics | Clinical Medicine | Endokrinologi och diabetes | Autoimmune Diseases: genetics | Celiac Disease: epidemiology | Endocrinology and Diabetes
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text The development of a mRNA multiplex RT-PCR assay for the definitive identification of body fluids
by Fleming, Rachel I and Harbison, SallyAnn
Forensic Science International: Genetics, ISSN 1872-4973, 2009, Volume 4, Issue 4, pp. 244 - 256
Abstract With current methodology, DNA profiling can identify an individual from a sample of biological material but it does not reveal what body fluid or... 
Pathology | RT-PCR multiplex | Forensic DNA | mRNA profiling | MEDICINE, LEGAL | PROSTATE-SPECIFIC ANTIGEN | SALIVA STAINS | SEMEN | DNA | GENETICS & HEREDITY | SAMPLES | EXPRESSION | BLOOD | Glucose-6-Phosphatase - genetics | Salivary Proteins and Peptides - genetics | RNA, Messenger - isolation & purification | DNA Fingerprinting | Humans | Cats - genetics | Transglutaminases - genetics | Male | Swine - genetics | Blood | Rabbits - genetics | Histatins - genetics | Chickens - genetics | Electrophoresis | Female | Transcriptional Elongation Factors - genetics | Dogs - genetics | Mice - genetics | Semen | RNA, Messenger - genetics | Glycophorin - genetics | Ubiquitin-Conjugating Enzymes - genetics | Reverse Transcriptase Polymerase Chain Reaction | DNA - genetics | DNA - isolation & purification | Animals | Matrix Metalloproteinase 11 - genetics | Protamines - genetics | Guinea Pigs - genetics | Saliva | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text Transcriptional control of transglutaminase 2 expression in mouse apoptotic thymocytes
by Sándor, Katalin and Daniel, Bence and Kiss, Bea and Kovács, Fruzsina and Szondy, Zsuzsa
Biochimica et Biophysica Acta - Gene Regulatory Mechanisms, ISSN 1874-9399, 08/2016, Volume 1859, Issue 8, pp. 964 - 974
Transglutaminase 2 (TGM2) is a ubiquitously expressed multifunctional protein, which participates in various biological processes including thymocyte... 
ChIP-seq | T cells | Enhancer RNA | Transglutaminase 2 | Gene regulation | Apoptosis | CELLS | SIGNALING PATHWAYS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESPONSE ELEMENT | GENE PROMOTER | HUMAN GENOME | BIOPHYSICS | RETINOID-X-RECEPTOR | BINDING-SITES | ENHANCER RNAS | TISSUE TRANSGLUTAMINASE | Thymocytes - metabolism | E1A-Associated p300 Protein - genetics | Transglutaminases - genetics | Apoptosis - genetics | E1A-Associated p300 Protein - metabolism | Phosphoproteins - metabolism | GTP-Binding Proteins - genetics | Receptors, Retinoic Acid - genetics | Smad4 Protein - genetics | Transcription, Genetic | Membrane Proteins - metabolism | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | Repressor Proteins - metabolism | Tretinoin - pharmacology | Promoter Regions, Genetic | Signal Transduction | Membrane Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Repressor Proteins - genetics | Receptors, Retinoic Acid - metabolism | Phosphoproteins - genetics | Smad4 Protein - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Transforming Growth Factor beta - pharmacology | Animals | Histones - genetics | Enhancer Elements, Genetic | CCCTC-Binding Factor | Transglutaminases - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Thymocytes - cytology | Thymocytes - drug effects | Bucladesine - pharmacology | Mice | Histones - metabolism | Primary Cell Culture | GTP-Binding Proteins - metabolism | Analysis | Genomics | Bone morphogenetic proteins | Genetic aspects | Cyclic adenylic acid | Genetic transcription | Retinoids | Transforming growth factors | Protein binding
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text RGD-independent cell adhesion via a tissue transglutaminase-fibronectin matrix promotes fibronectin fibril deposition and requires syndecan-4/2 and α5β1 integrin co-signaling
by Wang, Zhuo and Collighan, Russell J and Gross, Stephane R and Danen, Erik H. J and Orend, Gertraud and Telci, Dilek and Griffin, Martin
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 51, pp. 40212 - 40229
Fibronectin (FN) deposition mediated by fibroblasts is an important process in matrix remodeling and wound healing. By monitoring the deposition of soluble... 
RNA, Small Interfering - genetics | Integrin alpha5beta1 - genetics | Cricetulus | Protein Kinase C-alpha - metabolism | Humans | Protein Kinase C-alpha - genetics | Syndecan-4 - genetics | Actins - metabolism | Extracellular Matrix - metabolism | Transglutaminases - genetics | GTP-Binding Proteins - genetics | Heparitin Sulfate - metabolism | Actins - genetics | Extracellular Matrix - genetics | rho-Associated Kinases - metabolism | Mice, Mutant Strains | Syndecan-2 - genetics | CHO Cells | Syndecan-2 - metabolism | Cell Adhesion - genetics | Cricetinae | Syndecan-4 - metabolism | Cytoskeleton - genetics | rho-Associated Kinases - genetics | Cell Adhesion - drug effects | Fibronectins - metabolism | Animals | Signal Transduction - drug effects | Heparitin Sulfate - genetics | Transglutaminases - metabolism | Cytoskeleton - metabolism | Signal Transduction - physiology | Fibronectins - genetics | Mice | Oligopeptides - pharmacology | Integrin alpha5beta1 - metabolism | GTP-Binding Proteins - metabolism | Index Medicus | Fibronectin Deposition | Integrin | Extracellular Matrix | Signal Transduction | Cell Adhesion | Fibronectin | Syndecan | Cell Biology | Tissue Transglutaminase
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text Defective CFTR induces aggresome formation and lung inflammation in cystic fibrosis through ROS-mediated autophagy inhibition
by Luciani, Alessandro and Villella, Valeria Rachela and Brunetti-Pierri, Nicola and Masliah, Eliezer and Spencer, Brian and Pulze, Laura and Ballabio, Andrea and Gavina, Manuela and Medina, Diego and Raia, Valeria and Esposito, Speranza and Guido, Stefano and Quaratino, Sonia and Giardino, Ida and Pettoello-Mantovani, Massimo and D'Apolito, Maria and Settembre, Carmine and Maiuri, Luigi
Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 863 - 875
Accumulation of unwanted/misfolded proteins in aggregates has been observed in airways of patients with cystic fibrosis (CF), a life-threatening genetic... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | BECLIN 1 | DOWN-REGULATION | ENDOPLASMIC-RETICULUM | MICE | GENE-TRANSFER | TISSUE TRANSGLUTAMINASE | NEURODEGENERATIVE DISEASES | CELL-DEATH | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Sequestosome-1 Protein | Humans | Transglutaminases - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Young Adult | Cystamine - therapeutic use | Inflammation - metabolism | Autophagy - genetics | Nasal Polyps - metabolism | Mice, Inbred CFTR | Respiratory Mucosa - cytology | Cystic Fibrosis - metabolism | Membrane Proteins - genetics | Mice, Transgenic | Protein Transport - genetics | Apoptosis Regulatory Proteins - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Models, Biological | Cystic Fibrosis - genetics | Transglutaminases - metabolism | Adolescent | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | GTP-Binding Proteins | Cystic Fibrosis - drug therapy | Protein Binding - physiology | Microtubule-Associated Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Salicylates - pharmacology | Epithelial Sodium Channels - genetics | Apoptosis Regulatory Proteins - genetics | Adult | Cystamine - pharmacology | Membrane Proteins - metabolism | Acetylcysteine - therapeutic use | Beclin-1 | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cell Line | Small Ubiquitin-Related Modifier Proteins - metabolism | Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Mice, Inbred Strains | Nasal Polyps - drug therapy | Antioxidants - therapeutic use | Animals | Acetylcysteine - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Organometallic Compounds - pharmacology | Complications and side effects | Active oxygen | Gene mutations | Lung diseases | Physiological aspects | Cystic fibrosis | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text Extraintestinal manifestations of celiac disease: 33‐mer gliadin binding to glutamate receptor GRINA as a new explanation
by Garcia‐Quintanilla, Albert and Miranzo‐Navarro, Domingo
BioEssays, ISSN 0265-9247, 05/2016, Volume 38, Issue 5, pp. 427 - 439
We propose that inhibition of GRINA and its interacting partners by the 33‐mer gliadin fragment provokes phenotypes that are found in extraintestinal... 
gluten ataxia | osteopenia | cleft lip and palate | thyroid | celiac disease | dermatitis herpetiformis | GRINA | Celiac disease | Gluten ataxia | Dermatitis herpetiformis | Osteopenia | Cleft lip and palate | Thyroid | BAX INHIBITOR MOTIF | SENSORINEURAL HEARING-LOSS | BONE-MINERAL DENSITY | CEREBRAL CALCIFICATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLEFT-LIP | TRANSGLUTAMINASE ANTIBODIES | GLUTEN-FREE DIET | PROTEIN-INTERACTION NETWORKS | RESTLESS LEGS SYNDROME | SMALL-INTESTINAL MUCOSA | BIOLOGY | Coenzyme A Ligases - genetics | Humans | Protein Multimerization | Receptors, N-Methyl-D-Aspartate - metabolism | Bone Diseases, Metabolic - pathology | Cleft Palate - genetics | Coenzyme A Ligases - metabolism | Receptors, N-Methyl-D-Aspartate - genetics | Thyroiditis - pathology | Gliadin - metabolism | Thyroiditis - genetics | Cleft Lip - pathology | Membrane Proteins - metabolism | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Ataxia - genetics | Ataxia - metabolism | Ataxia - pathology | Amino Acid Sequence | Celiac Disease - chemically induced | Gliadin - genetics | Celiac Disease - genetics | Signal Transduction | Membrane Proteins - genetics | Thyroiditis - metabolism | Gene Expression Regulation | Dermatitis Herpetiformis - metabolism | Cleft Lip - metabolism | Dermatitis Herpetiformis - pathology | Bone Diseases, Metabolic - metabolism | Proteins - genetics | Sequence Homology, Amino Acid | Celiac Disease - pathology | Cleft Lip - genetics | Animals | Proteins - metabolism | Cleft Palate - pathology | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Glutens - adverse effects | Cleft Palate - metabolism | Models, Genetic | Bone Diseases, Metabolic - genetics | Adaptor Proteins, Signal Transducing - metabolism | Dermatitis Herpetiformis - genetics | Celiac Disease - metabolism | Cleft lip | Research | Binding proteins | Neuroglia | Restless legs syndrome | Hypotheses | Rodents | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Identification of Non-HLA Genes Associated with Celiac Disease and Country-Specific Differences in a Large, International Pediatric Cohort
by Sharma, Ashok and Liu, Xiang and Hadley, David and Hagopian, William and Liu, Edwin and Chen, Wei-Min and Onengut-Gumuscu, Suna and Simell, Ville and Rewers, Marian and Ziegler, Anette-G and Lernmark, Åke and Simell, Olli and Toppari, Jorma and Krischer, Jeffrey P and Akolkar, Beena and Rich, Stephen S and Agardh, Daniel and She, Jin-Xiong and TEDDY Study Grp and TEDDY Study Group and Lund University and Diabetes and Celiac Unit and Institutionen för kliniska vetenskaper, Malmö and Diabetes och celiaki and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden and Department of Clinical Sciences, Malmö
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 3, pp. e0152476 - e0152476
Objectives There are significant geographical differences in the prevalence and incidence of celiac disease that cannot be explained by HLA alone. More than 40... 
RHEUMATOID-ARTHRITIS | POPULATION | MULTIPLE COMMON | METAANALYSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | TRANSGLUTAMINASE ANTIBODIES | PADI4 | RISK VARIANTS | GENOME-WIDE ASSOCIATION | CHILDREN | Pediatrics | Prospective Studies | Follow-Up Studies | United States | Autoantibodies - blood | Humans | Child, Preschool | Transglutaminases - genetics | Family Health | Infant | Male | HLA Antigens - genetics | GTP-Binding Proteins - genetics | Transglutaminases - immunology | Sweden | Celiac Disease - immunology | Female | Child | Infant, Newborn | Celiac Disease - genetics | GTP-Binding Proteins - immunology | Europe | Proportional Hazards Models | Diabetes Mellitus, Type 1 - genetics | Genotype | International Cooperation | Disease Progression | Biopsy | Adolescent | Heterozygote | Polymorphism, Single Nucleotide | Celiac disease | Analysis | Development and progression | Genetic aspects | Disease susceptibility | Research | Prevalence studies (Epidemiology) | Haplotypes | Neonates | Genomics | Genes | Genomes | Diabetes mellitus (insulin dependent) | Single-nucleotide polymorphism | Kinases | Epidemiology | Small intestine | CTLA-4 protein | Genetic analysis | Interleukin 1 | Population | Children | Bioinformatics | Gluten | Genotypes | Public health | Autoantibodies | Diabetes mellitus | Health risks | Principal components analysis | Data processing | Hazards | Loci | Transglutaminase 2 | Survival analysis | Rheumatoid arthritis | Histocompatibility antigen HLA | Diabetes | Autoimmune diseases | Health risk assessment | CCR9 protein | Index Medicus | Medicinsk genetik | Basic Medicine | Medical Genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Mutations in rare ataxia genes are uncommon causes of sporadic cerebellar ataxia
by Fogel, Brent L and Lee, Ji Yong and Lane, Jessica and Wahnich, Amanda and Chan, Sandy and Huang, Alden and Osborn, Greg E and Klein, Eric and Mamah, Catherine and Perlman, Susan and Geschwind, Daniel H and Coppola, Giovanni
Movement Disorders, ISSN 0885-3185, 03/2012, Volume 27, Issue 3, pp. 442 - 446
Background: Sporadic-onset ataxia is common in a tertiary care setting but a significant percentage remains unidentified despite extensive evaluation. Rare... 
dominant genetic conditions | recessive genetic conditions | cerebellar ataxia | copy number variation | spinocerebellar ataxia | Recessive genetic conditions | Dominant genetic conditions | Copy number variation | Cerebellar ataxia | Spinocerebellar ataxia | DEPRESSION | PLACEBO | MOTOR FUNCTION | CLINICAL NEUROLOGY | AMOXAPINE | IMIPRAMINE | ARSACS | SENATAXIN | DISEASE RATING-SCALE | HUNTINGTONS-DISEASE | PARKINSONS-DISEASE | Genetic Predisposition to Disease - genetics | Genetic Testing | Protein Precursors - genetics | Lamin Type B - genetics | Humans | Middle Aged | Transglutaminases - genetics | Male | Mutation - genetics | Nerve Tissue Proteins - genetics | Enkephalins - genetics | Heat-Shock Proteins - genetics | Magnetic Resonance Imaging | Phenotype | Cerebellar Ataxia - genetics | Databases, Bibliographic - statistics & numerical data | Adult | Female | RNA Helicases - genetics | Aged | Nuclear Proteins - genetics | Index Medicus | Spinocerebellar Ataxia | Dominant Genetic Conditions | Recessive Genetic Conditions | Cerebellar Ataxia | Copy Number Variation
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Potentially Functional Single Nucleotide Polymorphisms in the Core Nucleotide Excision Repair Genes and Risk of Squamous Cell Carcinoma of the Head and Neck
by Jiaze An and Zhensheng Liu and Zhibin Hu and Guojun Li and Li-E Wang and Erich M. Sturgis and Adel K. El-Naggar and Margaret R. Spitz and Qingyi Wei
Cancer Epidemiology Biomarkers & Prevention, ISSN 1055-9965, 08/2007, Volume 16, Issue 8, pp. 1633 - 1638
Susceptibility to cancer has been associated with DNA repair capacity, a global reflection of all functional variants, most of which are relatively rare. Among... 
molecular epidemiology | genetic variant | genetic susceptibility | DNA repair | nucleotide excision repair | Laryngeal Neoplasms - genetics | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Age Factors | Genes, Recessive - genetics | Carcinoma, Squamous Cell - genetics | Humans | Middle Aged | Risk Factors | Transglutaminases - genetics | Genotype | Male | DNA Repair - genetics | DNA-Binding Proteins - genetics | Case-Control Studies | Sex Factors | Polymorphism, Single Nucleotide - genetics | Female | Head and Neck Neoplasms - genetics | Valine - genetics | Aged | Genetic Variation - genetics | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Genetic Disruption of the Copulatory Plug in Mice Leads to Severely Reduced Fertility
by Dean, Matthew D
PLoS Genetics, ISSN 1553-7390, 01/2013, Volume 9, Issue 1, pp. e1003185 - e1003185
Seminal fluid proteins affect fertility at multiple stages in reproduction. In many species, a male's ejaculate coagulates to form a copulatory plug. Although... 
GOLDEN-HAMSTERS | SEMINAL FLUID PROTEIN | ENZYMATIC COAGULATION | HYBRID MALE-STERILITY | FACILITATING REPRODUCTIVE SUCCESS | GENETICS & HEREDITY | GLANDULAR STRUCTURE | MESOCRICETUS-AURATUS | LARGE-SCALE | GUINEA-PIG | SPERM COMPETITION | Sperm Count | Oocytes - growth & development | Sperm Motility - genetics | Oocytes - metabolism | Semen | Ejaculation - genetics | Reproduction - genetics | Transglutaminases - genetics | Male | Fertility - physiology | Mice, Knockout | Copulation - physiology | Fertility - genetics | Animals | Transglutaminases - physiology | Female | Mice | Fertility | Ejaculation | Physiological aspects | Genetic aspects | Transglutaminases | Research | Gene expression | Health aspects | Proteins | Motility | Sperm | Rodents | Fluid | Genetic engineering | Males | Females | Experiments | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights