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The New England Journal of Medicine, ISSN 0028-4793, 07/2014, Volume 371, Issue 1, pp. 42 - 49
In this cohort study involving 6403 children at increased risk for celiac disease on the basis of HLA haplotype, the risks of celiac disease autoimmunity and... 
ANTIBODIES | MEDICINE, GENERAL & INTERNAL | GLUTEN | AUTOIMMUNITY | GENE DOSAGE | SUSCEPTIBILITY | TEDDY | CHILDHOOD | CHILDREN | Autoimmune Diseases - epidemiology | United States - epidemiology | Genetic Predisposition to Disease | Prospective Studies | Celiac Disease - genetics | Humans | Kaplan-Meier Estimate | Child, Preschool | Europe - epidemiology | Infant | Male | Risk | Transglutaminases - immunology | Autoimmune Diseases - genetics | Homozygote | HLA-DR4 Antigen - genetics | HLA-DR3 Antigen - genetics | HLA-DQ Antigens - genetics | Antibodies - blood | Female | Celiac Disease - epidemiology | Infant, Newborn | Haplotypes | Viral antibodies | Celiac disease | Histocompatibility antigens | Analysis | HLA histocompatibility antigens | Antibodies | Children | Health aspects | Risk factors | Diabetes mellitus | Heterozygotes | Environmental factors | Small intestine | Transglutaminase 2 | Children & youth | Homozygotes | Genotype & phenotype | Biopsy | Histocompatibility antigen HLA | Autoimmune diseases | Age | Genotypes | Immune system | Index Medicus | Abridged Index Medicus | Autoimmune Diseases: epidemiology | Celiac Disease: genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Europe: epidemiology | Antibodies: blood | HLA-DQ Antigens: genetics | HLA-DR3 Antigen: genetics | Klinisk medicin | Transglutaminases: immunology | United States: epidemiology | HLA-DR4 Antigen: genetics | Clinical Medicine | Endokrinologi och diabetes | Autoimmune Diseases: genetics | Celiac Disease: epidemiology | Endocrinology and Diabetes
Journal Article
Biochimica et Biophysica Acta - Gene Regulatory Mechanisms, ISSN 1874-9399, 08/2016, Volume 1859, Issue 8, pp. 964 - 974
Transglutaminase 2 (TGM2) is a ubiquitously expressed multifunctional protein, which participates in various biological processes including thymocyte... 
ChIP-seq | T cells | Enhancer RNA | Transglutaminase 2 | Gene regulation | Apoptosis | CELLS | SIGNALING PATHWAYS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESPONSE ELEMENT | GENE PROMOTER | HUMAN GENOME | BIOPHYSICS | RETINOID-X-RECEPTOR | BINDING-SITES | ENHANCER RNAS | TISSUE TRANSGLUTAMINASE | Thymocytes - metabolism | E1A-Associated p300 Protein - genetics | Transglutaminases - genetics | Apoptosis - genetics | E1A-Associated p300 Protein - metabolism | Phosphoproteins - metabolism | GTP-Binding Proteins - genetics | Receptors, Retinoic Acid - genetics | Smad4 Protein - genetics | Transcription, Genetic | Membrane Proteins - metabolism | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | Repressor Proteins - metabolism | Tretinoin - pharmacology | Promoter Regions, Genetic | Signal Transduction | Membrane Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Repressor Proteins - genetics | Receptors, Retinoic Acid - metabolism | Phosphoproteins - genetics | Smad4 Protein - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Transforming Growth Factor beta - pharmacology | Animals | Histones - genetics | Enhancer Elements, Genetic | CCCTC-Binding Factor | Transglutaminases - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Thymocytes - cytology | Thymocytes - drug effects | Bucladesine - pharmacology | Mice | Histones - metabolism | Primary Cell Culture | GTP-Binding Proteins - metabolism | Analysis | Genomics | Bone morphogenetic proteins | Genetic aspects | Cyclic adenylic acid | Genetic transcription | Retinoids | Transforming growth factors | Protein binding
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 51, pp. 40212 - 40229
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 863 - 875
Accumulation of unwanted/misfolded proteins in aggregates has been observed in airways of patients with cystic fibrosis (CF), a life-threatening genetic... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | BECLIN 1 | DOWN-REGULATION | ENDOPLASMIC-RETICULUM | MICE | GENE-TRANSFER | TISSUE TRANSGLUTAMINASE | NEURODEGENERATIVE DISEASES | CELL-DEATH | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Sequestosome-1 Protein | Humans | Transglutaminases - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Young Adult | Cystamine - therapeutic use | Inflammation - metabolism | Autophagy - genetics | Nasal Polyps - metabolism | Mice, Inbred CFTR | Respiratory Mucosa - cytology | Cystic Fibrosis - metabolism | Membrane Proteins - genetics | Mice, Transgenic | Protein Transport - genetics | Apoptosis Regulatory Proteins - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Models, Biological | Cystic Fibrosis - genetics | Transglutaminases - metabolism | Adolescent | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | GTP-Binding Proteins | Cystic Fibrosis - drug therapy | Protein Binding - physiology | Microtubule-Associated Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Salicylates - pharmacology | Epithelial Sodium Channels - genetics | Apoptosis Regulatory Proteins - genetics | Adult | Cystamine - pharmacology | Membrane Proteins - metabolism | Acetylcysteine - therapeutic use | Beclin-1 | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cell Line | Small Ubiquitin-Related Modifier Proteins - metabolism | Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Mice, Inbred Strains | Nasal Polyps - drug therapy | Antioxidants - therapeutic use | Animals | Acetylcysteine - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Organometallic Compounds - pharmacology | Complications and side effects | Active oxygen | Gene mutations | Lung diseases | Physiological aspects | Cystic fibrosis | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article
BioEssays, ISSN 0265-9247, 05/2016, Volume 38, Issue 5, pp. 427 - 439
We propose that inhibition of GRINA and its interacting partners by the 33‐mer gliadin fragment provokes phenotypes that are found in extraintestinal... 
gluten ataxia | osteopenia | cleft lip and palate | thyroid | celiac disease | dermatitis herpetiformis | GRINA | Celiac disease | Gluten ataxia | Dermatitis herpetiformis | Osteopenia | Cleft lip and palate | Thyroid | BAX INHIBITOR MOTIF | SENSORINEURAL HEARING-LOSS | BONE-MINERAL DENSITY | CEREBRAL CALCIFICATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLEFT-LIP | TRANSGLUTAMINASE ANTIBODIES | GLUTEN-FREE DIET | PROTEIN-INTERACTION NETWORKS | RESTLESS LEGS SYNDROME | SMALL-INTESTINAL MUCOSA | BIOLOGY | Coenzyme A Ligases - genetics | Humans | Protein Multimerization | Receptors, N-Methyl-D-Aspartate - metabolism | Bone Diseases, Metabolic - pathology | Cleft Palate - genetics | Coenzyme A Ligases - metabolism | Receptors, N-Methyl-D-Aspartate - genetics | Thyroiditis - pathology | Gliadin - metabolism | Thyroiditis - genetics | Cleft Lip - pathology | Membrane Proteins - metabolism | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Ataxia - genetics | Ataxia - metabolism | Ataxia - pathology | Amino Acid Sequence | Celiac Disease - chemically induced | Gliadin - genetics | Celiac Disease - genetics | Signal Transduction | Membrane Proteins - genetics | Thyroiditis - metabolism | Gene Expression Regulation | Dermatitis Herpetiformis - metabolism | Cleft Lip - metabolism | Dermatitis Herpetiformis - pathology | Bone Diseases, Metabolic - metabolism | Proteins - genetics | Sequence Homology, Amino Acid | Celiac Disease - pathology | Cleft Lip - genetics | Animals | Proteins - metabolism | Cleft Palate - pathology | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Glutens - adverse effects | Cleft Palate - metabolism | Models, Genetic | Bone Diseases, Metabolic - genetics | Adaptor Proteins, Signal Transducing - metabolism | Dermatitis Herpetiformis - genetics | Celiac Disease - metabolism | Cleft lip | Research | Binding proteins | Neuroglia | Restless legs syndrome | Hypotheses | Rodents | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 3, pp. e0152476 - e0152476
Objectives There are significant geographical differences in the prevalence and incidence of celiac disease that cannot be explained by HLA alone. More than 40... 
RHEUMATOID-ARTHRITIS | POPULATION | MULTIPLE COMMON | METAANALYSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | TRANSGLUTAMINASE ANTIBODIES | PADI4 | RISK VARIANTS | GENOME-WIDE ASSOCIATION | CHILDREN | Pediatrics | Prospective Studies | Follow-Up Studies | United States | Autoantibodies - blood | Humans | Child, Preschool | Transglutaminases - genetics | Family Health | Infant | Male | HLA Antigens - genetics | GTP-Binding Proteins - genetics | Transglutaminases - immunology | Sweden | Celiac Disease - immunology | Female | Child | Infant, Newborn | Celiac Disease - genetics | GTP-Binding Proteins - immunology | Europe | Proportional Hazards Models | Diabetes Mellitus, Type 1 - genetics | Genotype | International Cooperation | Disease Progression | Biopsy | Adolescent | Heterozygote | Polymorphism, Single Nucleotide | Celiac disease | Analysis | Development and progression | Genetic aspects | Disease susceptibility | Research | Prevalence studies (Epidemiology) | Haplotypes | Neonates | Genomics | Genes | Genomes | Diabetes mellitus (insulin dependent) | Single-nucleotide polymorphism | Kinases | Epidemiology | Small intestine | CTLA-4 protein | Genetic analysis | Interleukin 1 | Population | Children | Bioinformatics | Gluten | Genotypes | Public health | Autoantibodies | Diabetes mellitus | Health risks | Principal components analysis | Data processing | Hazards | Loci | Transglutaminase 2 | Survival analysis | Rheumatoid arthritis | Histocompatibility antigen HLA | Diabetes | Autoimmune diseases | Health risk assessment | CCR9 protein | Index Medicus | Medicinsk genetik | Basic Medicine | Medical Genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Movement Disorders, ISSN 0885-3185, 03/2012, Volume 27, Issue 3, pp. 442 - 446
Journal Article