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PloS one, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0179610 - e0179610
Objective To assess the prognostic roles of BAP1, PBRM1, pS6, PTEN, TGase2, PD-L1, CA9, PSMA, and Ki-67 tissue biomarkers in localized renal cell carcinoma... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Immunohistochemistry | Prognosis | Tissue Array Analysis | Humans | Middle Aged | Ki-67 Antigen - metabolism | Male | Kidney Neoplasms - metabolism | Porphyrins - metabolism | Neoplasm Recurrence, Local - mortality | Neoplasm Recurrence, Local - pathology | Glutamate Carboxypeptidase II - metabolism | Neoplasm Grading | Antigens, Neoplasm - metabolism | Antigens, Surface - metabolism | Biomarkers, Tumor - metabolism | Ubiquitin Thiolesterase - metabolism | Adult | Female | Carbonic Anhydrase IX - metabolism | Retrospective Studies | Tumor Suppressor Proteins - metabolism | Neoplasm Recurrence, Local - metabolism | Carcinoma, Renal Cell - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Survival Rate | Kidney Neoplasms - mortality | Transcription Factors - metabolism | Carcinoma, Renal Cell - metabolism | Carcinoma, Renal Cell - mortality | Disease-Free Survival | B7-H1 Antigen - metabolism | Transglutaminases - metabolism | Sulfonamides - metabolism | Kidney Neoplasms - pathology | Aged | Neoplasm Staging | GTP-Binding Proteins - metabolism | Genetic aspects | Carcinoma, Renal cell | Research | Biological markers | Biometrics | Metastasis | Kinases | Epidemiology | Cancer therapies | Urology | Proteins | Nephrectomy | Quality | Growth factors | Age | Antigens | Markers | Hazards | Patients | Survival | Disease prevention | Hospitals | Medical prognosis | PD-L1 protein | Biomarkers | Death | Mutation | Diabetes | Prostate cancer | PTEN protein | Clear cell-type renal cell carcinoma | Kidney transplantation | Tumors | Index Medicus
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 8, pp. 2640 - 2649
Transglutaminase 2 (TG2) is a ubiquitously expressed, intracellular as well as extracellular protein with multiple modes of post-translational regulation,... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Enzymes, Immobilized - metabolism | Oxidants - pharmacology | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Glutathione - metabolism | Protein Disulfide-Isomerases - metabolism | GTP-Binding Proteins - antagonists & inhibitors | Humans | Membrane Glycoproteins - chemistry | Oxidants - metabolism | Extracellular Matrix - metabolism | Transglutaminases - genetics | Protein Disulfide-Isomerases - antagonists & inhibitors | Protein Transport - drug effects | GTP-Binding Proteins - genetics | Thioredoxins - genetics | Oxidoreductases Acting on Sulfur Group Donors - chemistry | Protein Processing, Post-Translational - drug effects | RNA Interference | Biocatalysis - drug effects | Human Umbilical Vein Endothelial Cells - cytology | Thioredoxins - metabolism | Peptide Fragments - genetics | Transglutaminases - antagonists & inhibitors | Transglutaminases - chemistry | Allosteric Regulation - drug effects | Oxidoreductases Acting on Sulfur Group Donors - metabolism | Recombinant Proteins - metabolism | Peptide Fragments - metabolism | GTP-Binding Proteins - chemistry | Oxidation-Reduction | Extracellular Matrix - drug effects | Cells, Cultured | Hydrogen Peroxide - pharmacology | Recombinant Proteins - chemistry | Membrane Glycoproteins - genetics | Extracellular Matrix - enzymology | Peptide Fragments - chemistry | Protein Disulfide-Isomerases - genetics | Human Umbilical Vein Endothelial Cells - enzymology | Cystine - metabolism | Transglutaminases - metabolism | Oxidoreductases Acting on Sulfur Group Donors - genetics | Enzymes, Immobilized - antagonists & inhibitors | GTP-Binding Proteins - metabolism | Index Medicus | Editors' Picks | post-translational modification (PTM) | PDIA3 | transglutaminase | thioredoxin | disulfide | oxidation-reduction (redox) | ERp57 | redox switch | celiac disease | transglutaminase 2 | protein disulfide isomerase family A member 3
Journal Article
International journal of cancer, ISSN 0020-7136, 06/2014, Volume 134, Issue 12, pp. 2798 - 2807
Aberrant glucose metabolism characterized by high levels of glycolysis, even in the presence of oxygen, is an important hallmark of cancer... 
HIF‐1α | metabolism | NF‐κB | drug resistance | metastasis | EMT | NF-κB | HIF-1α | Life Sciences & Biomedicine | Oncology | Science & Technology | Up-Regulation | Epithelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | Transglutaminases - genetics | Mammary Glands, Human - metabolism | GTP-Binding Proteins - genetics | Promoter Regions, Genetic - genetics | Oxygen - metabolism | Breast Neoplasms - metabolism | Signal Transduction - immunology | Cell Respiration - genetics | MCF-7 Cells | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Lactic Acid - biosynthesis | Female | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Down-Regulation | Transglutaminases - biosynthesis | Epithelial Cells - pathology | Mammary Glands, Human - pathology | Inflammation - immunology | Mitochondria - metabolism | Breast Neoplasms - genetics | Transcription Factor RelA - metabolism | Transcription Factor RelA - biosynthesis | Transglutaminases - metabolism | Cell Line, Tumor | Glucose - metabolism | Glycolysis | GTP-Binding Proteins - biosynthesis | RNA, Small Interfering | GTP-Binding Proteins - metabolism | Lactates | Glucose metabolism | Physiological aspects | Mitochondrial DNA | Glucose | Drug resistance | Dextrose | Rodents | Protein expression | Breast cancer | Metastasis | Metabolism | Cells | Index Medicus
Journal Article
Human molecular genetics, ISSN 1460-2083, 08/2014, Volume 23, Issue 15, pp. 4064 - 4076
iRHOM2 is a highly conserved, catalytically inactive member of the Rhomboid family, which has recently been shown to regulate the maturation of the... 
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | ADAM17 Protein | RNA, Small Interfering - genetics | Keratoderma, Palmoplantar - genetics | Staphylococcus aureus - physiology | Esophageal Neoplasms - microbiology | Humans | ErbB Receptors - genetics | Staphylococcal Skin Infections - metabolism | Transglutaminases - genetics | Male | Esophageal Neoplasms - pathology | Desmosomes - pathology | Epidermis - microbiology | Keratoderma, Palmoplantar - metabolism | Keratoderma, Palmoplantar - pathology | Esophageal Neoplasms - metabolism | Female | Keratinocytes - microbiology | Desmosomes - metabolism | Epidermis - metabolism | Epidermis - pathology | ADAM Proteins - antagonists & inhibitors | ErbB Receptors - metabolism | Signal Transduction | Epidermal Growth Factor - genetics | Gene Expression Regulation | Epidermal Growth Factor - metabolism | Staphylococcal Skin Infections - pathology | Carrier Proteins - genetics | ADAM Proteins - metabolism | Keratinocytes - pathology | Carrier Proteins - metabolism | Esophageal Neoplasms - genetics | Keratinocytes - metabolism | Transglutaminases - metabolism | Mutation | ADAM Proteins - genetics | Keratoderma, Palmoplantar - microbiology | Staphylococcal Skin Infections - microbiology | Cytokines - biosynthesis | RNA, Small Interfering - metabolism | Staphylococcal Skin Infections - genetics | Index Medicus
Journal Article
American journal of physiology. Lung cellular and molecular physiology, ISSN 1040-0605, 11/2017, Volume 313, Issue 5, pp. L752 - L762
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 12/2010, Volume 285, Issue 51, pp. 40212 - 40229
Journal Article
Cell death & disease, ISSN 2041-4889, 07/2017, Volume 8, Issue 7, pp. e2931 - e2931
Filaggrin (FLG) mutation is a well-confirmed genetic aberration in atopic dermatitis (AD). Genome-wide association studies on AD have revealed other... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Humans | Transglutaminases - genetics | DNA-Binding Proteins - metabolism | Interleukin-4 - pharmacology | Cell Nucleus - metabolism | RNA Interference | Intermediate Filament Proteins - genetics | Receptors, Aryl Hydrocarbon - metabolism | Membrane Proteins - metabolism | Cell Culture Techniques | Cell Line | DNA-Binding Proteins - antagonists & inhibitors | Protein Precursors - genetics | Membrane Proteins - genetics | Receptors, Aryl Hydrocarbon - genetics | Transcription Factors - antagonists & inhibitors | Dermatitis, Atopic - pathology | Keratinocytes - cytology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Tars - pharmacology | Protein Precursors - metabolism | Receptors, Aryl Hydrocarbon - agonists | Transcription Factors - metabolism | Up-Regulation - drug effects | Keratinocytes - metabolism | Transglutaminases - metabolism | Carbazoles - pharmacology | Dermatitis, Atopic - metabolism | Intermediate Filament Proteins - metabolism | RNA, Small Interfering - metabolism | Tar | Hydrocarbons | Keratinocytes | Genomes | Filaggrin | Gene expression | Dermatitis | Carbazole | Nuclear transport | Interleukin 4 | Cellular biology | Atopic dermatitis | Ligands | Skin | Receptor mechanisms | Mutation | Index Medicus | Original
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 39, pp. 64109 - 64123
Several members of the Poly(ADP-ribose) polymerase (PARP) family are essential regulators of genome integrity, actively prospected as drug targets for cancer... 
TGFβ | EMT | Poly(ADP-ribose) polymerase 3 (PARP3) | ROS | Stem cells | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Reactive Oxygen Species - metabolism | Cadherins - metabolism | Neoplastic Stem Cells - drug effects | CD24 Antigen - metabolism | Humans | Gene Expression Regulation, Neoplastic | Transglutaminases - genetics | Drug Resistance, Neoplasm | Epithelial-Mesenchymal Transition - drug effects | GTP-Binding Proteins - genetics | SOXB1 Transcription Factors - metabolism | Cell Self Renewal | Breast Neoplasms - enzymology | Snail Family Transcription Factors - genetics | Transfection | RNA Interference | Time Factors | Hyaluronan Receptors - metabolism | Cell Cycle Proteins - genetics | Neoplastic Stem Cells - pathology | Female | Cadherins - genetics | Spheroids, Cellular | A549 Cells | Snail Family Transcription Factors - metabolism | Signal Transduction | Cell Cycle Proteins - metabolism | Etoposide - pharmacology | Mammary Glands, Human - pathology | Topoisomerase II Inhibitors - pharmacology | Breast Neoplasms - drug therapy | Hep G2 Cells | Phenotype | Poly(ADP-ribose) Polymerases - metabolism | Breast Neoplasms - genetics | Poly(ADP-ribose) Polymerases - genetics | Breast Neoplasms - pathology | Octamer Transcription Factor-3 - metabolism | Transglutaminases - metabolism | Mammary Glands, Human - enzymology | Neoplastic Stem Cells - enzymology | Transforming Growth Factor beta - metabolism | Cell Movement | GTP-Binding Proteins - metabolism | Index Medicus | Octamer Transcription Factor-3 | Biotechnology | Poly(ADP-ribose) Polymerases | Reactive Oxygen Species | Breast Neoplasms | Life Sciences | Mammary Glands, Human | Topoisomerase II Inhibitors | Epithelial-Mesenchymal Transition | SOXB1 Transcription Factors | Snail Family Transcription Factors | Transforming Growth Factor beta | Hyaluronan Receptors | Etoposide | Neoplastic Stem Cells | CD24 Antigen | Cadherins | Transglutaminases | GTP-Binding Proteins | Cell Cycle Proteins | Antigens, CD
Journal Article
eLife, ISSN 2050-084X, 05/2018, Volume 7
In the central nervous system (CNS), myelin formation and repair are regulated by oligodendrocyte (OL) lineage cells, which sense and integrate signals from... 
Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Oligodendroglia - metabolism | Receptors, G-Protein-Coupled - metabolism | Demyelinating Diseases - genetics | GTP-Binding Proteins - deficiency | Humans | Transglutaminases - genetics | Male | Demyelinating Diseases - metabolism | GTP-Binding Proteins - genetics | Neurogenesis - genetics | CX3C Chemokine Receptor 1 - metabolism | CX3C Chemokine Receptor 1 - genetics | Gene Expression Regulation, Developmental | Female | Cell Differentiation | Oligodendroglia - cytology | Prosencephalon - metabolism | Demyelinating Diseases - pathology | Oligodendrocyte Precursor Cells - metabolism | Cell Lineage - genetics | Microglia - cytology | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Cerebellum - metabolism | Transglutaminases - deficiency | Mice, Knockout | Laminin - genetics | Animals | Oligodendrocyte Precursor Cells - cytology | Prosencephalon - cytology | Receptor, Platelet-Derived Growth Factor alpha - genetics | Cerebellum - cytology | Mice | Receptors, G-Protein-Coupled - genetics | Laminin - metabolism | Remyelination - genetics | Dendritic cells | Physiological aspects | Brain | Neurosciences | Animal models | Central nervous system | Cell division | Glial cells | Glial stem cells | Transglutaminase 2 | Microglia | Medicine | Proteins | Signal transduction | Myelination | Laminin | Hospitals | Demyelination | Ligands | Extracellular matrix | Software | Index Medicus
Journal Article