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1. Full Text Cracking the BAFF code
by Schneider, Pascal and Mackay, Fabienne
Nature Reviews Immunology, ISSN 1474-1733, 07/2009, Volume 9, Issue 7, pp. 491 - 502
The tumour necrosis factor (TNF) family members B cell activating factor (BAFF) and APRIL (a proliferation-inducing ligand) are crucial survival factors for... 
B-LYMPHOCYTE STIMULATOR | TUMOR-NECROSIS-FACTOR | CELL-ACTIVATING FACTOR | PROLIFERATION-INDUCING LIGAND | COMMON VARIABLE IMMUNODEFICIENCY | LUPUS-LIKE DISEASE | T-CELL | IMMUNOLOGY | MARGINAL-ZONE | ADAPTIVE IMMUNE-RESPONSES | TNF FAMILY | B-Cell Maturation Antigen - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - immunology | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - immunology | Humans | Toll-Like Receptors - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - metabolism | Transmembrane Activator and CAML Interactor Protein - metabolism | Signal Transduction - immunology | Lymphocyte Activation - immunology | Animals | B-Cell Activating Factor - metabolism | B-Cell Maturation Antigen - metabolism | B-Lymphocyte Subsets - metabolism | Autoimmunity - immunology | Immune Tolerance - immunology | B-Cell Activating Factor - immunology | B-Lymphocyte Subsets - immunology | Ligands | Toll-Like Receptors - metabolism | Transmembrane Activator and CAML Interactor Protein - immunology | Autoimmunity | Physiological aspects | B cells | Research | Tumor necrosis factor | Risk factors
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2. Full Text TNF receptor superfamily member 13b ( TNFRSF13B ) hemizygosity reveals transmembrane activator and CAML interactor haploinsufficiency at later stages of B-cell development
by Romberg, Neil, MD and Virdee, Manmeet, BS and Chamberlain, Nicolas, BS and Oe, Tyler, BA and Schickel, Jean-Nicolas, PhD and Perkins, Tiffany, MD and Cantaert, Tineke, PhD and Rachid, Rima, MD and Rosengren, Sally, MD and Palazzo, Regina, MD and Geha, Raif, MD, PhD and Cunningham-Rundles, Charlotte, MD, PhD and Meffre, Eric, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 5, pp. 1315 - 1325
Background Heterozygous C104R or A181E TNF receptor superfamily member 13b ( TNFRSF13B ) mutations impair removal of autoreactive B cells, weaken B-cell... 
Allergy and Immunology | B-cell tolerance | Smith-Magenis syndrome | transmembrane activator and CAML interactor | TNFRSF13B | common variable immune deficiency | T-FOLLICULAR HELPER | BAFF | TOLERANCE | COMMON VARIABLE IMMUNODEFICIENCY | IMMUNOLOGY | DEFICIENCY | ALLERGY | LIGAND | TACI MUTATIONS | EXPRESSION | APRIL | Autoimmunity | Common Variable Immunodeficiency - immunology | Humans | Middle Aged | Male | Mutation, Missense - genetics | Haploinsufficiency | Hemizygote | T-Lymphocytes, Regulatory - immunology | Transmembrane Activator and CAML Interactor Protein - metabolism | Young Adult | Transmembrane Activator and CAML Interactor Protein - genetics | Lymphocyte Activation - genetics | B-Lymphocytes - immunology | Smith-Magenis Syndrome - immunology | Adolescent | Aged, 80 and over | Adult | Female | Immunologic Memory | Aged | Child | Antibody Formation - genetics | Broadway theater | Medical colleges | Tumor necrosis factor | Analysis | Immunodeficiency | B cells | Studies | Medical research | Flow cytometry | Cell growth | Genes | Ear diseases | Ligands | Mutation
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3. Full Text Primary B Cell Immunodeficiencies: Comparisons and Contrasts
by Conley, Mary Ellen and Dobbs, A. Kerry and Farmer, Dana M and Kilic, Sebnem and Paris, Kenneth and Grigoriadou, Sofia and Coustan-Smith, Elaine and Howard, Vanessa and Campana, Dario
Annual Review of Immunology, ISSN 0732-0582, 2009, Volume 27, Issue 1, pp. 199 - 227
Sophisticated genetic tools have made possible the identification of the genes responsible for most well-described immunodeficiencies in the past 15 years.... 
Btk | Common variable immunodeficiency | TACI | X-linked agammaglobulinemia | Hyper-IgM syndrome | DISEASE GENE SH2D1A | INDUCED CYTIDINE DEAMINASE | common variable immunodeficiency | MAJOR HISTOCOMPATIBILITY COMPLEX | IMMUNOLOGY | hyper-IgM syndrome | BRUTONS TYROSINE KINASE | ANTIBODY-DEFICIENCY SYNDROME | CLASS-SWITCH RECOMBINATION | AUTOSOMAL RECESSIVE FORM | Inducible T-Cell Co-Stimulator Protein | CD79 Antigens - immunology | Protein-Tyrosine Kinases - metabolism | Humans | Precursor Cells, B-Lymphoid - metabolism | Precursor Cells, B-Lymphoid - immunology | Antigens, CD19 - genetics | Protein-Tyrosine Kinases - immunology | Transmembrane Activator and CAML Interactor Protein - metabolism | Protein-Tyrosine Kinases - genetics | Adaptor Proteins, Signal Transducing - immunology | Immunologic Deficiency Syndromes - immunology | B-Lymphocytes - metabolism | Antigens, CD19 - immunology | Antigens, Differentiation, T-Lymphocyte - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | CD79 Antigens - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | Animals | B-Lymphocytes - immunology | Adaptor Proteins, Signal Transducing - genetics | Antigens, Differentiation, T-Lymphocyte - genetics | Immunologic Deficiency Syndromes - genetics | Antigens, CD19 - metabolism | CD79 Antigens - genetics | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Transmembrane Activator and CAML Interactor Protein - immunology | Genetic aspects | B cells | Properties | Identification and classification | Immunodeficiency | Immunological research
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4. Full Text Signaling by the tumor necrosis factor receptor superfamily in B‐cell biology and disease
by Rickert, Robert C and Jellusova, Julia and Miletic, Ana V
Immunological Reviews, ISSN 0105-2896, 11/2011, Volume 244, Issue 1, pp. 115 - 133
Members of the tumor necrosis factor receptor superfamily (TNFRSF) participate prominently in B‐cell maturation and function. In particular, B‐cell activating... 
BAFF | B cell | CD40 | BCMA | NF‐κB | TACI | NF-κB | FORKHEAD TRANSCRIPTION FACTOR | SYSTEMIC-LUPUS-ERYTHEMATOSUS | HUMORAL IMMUNE-RESPONSES | COMMON VARIABLE IMMUNODEFICIENCY | IMMUNOLOGY | KINASE-C-DELTA | GERMINAL CENTER FORMATION | NF-?B | ACTIVATING FACTOR-RECEPTOR | NON-HODGKIN-LYMPHOMA | X-LINKED IMMUNODEFICIENCY | NF-KAPPA-B | Cell Proliferation | Humans | NF-kappa B - immunology | NF-kappa B - metabolism | Transmembrane Activator and CAML Interactor Protein - metabolism | Signal Transduction - immunology | CD40 Antigens - metabolism | B-Cell Activating Factor - immunology | B-Lymphocyte Subsets - immunology | CD40 Antigens - genetics | B-Cell Activation Factor Receptor - genetics | B-Lymphocytes - metabolism | B-Cell Activation Factor Receptor - immunology | B-Cell Maturation Antigen - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - immunology | Gene Expression | Mice, Transgenic | Cell Survival - immunology | Immunity, Innate | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | B-Cell Activation Factor Receptor - metabolism | Animals | B-Cell Activating Factor - metabolism | B-Cell Maturation Antigen - metabolism | B-Lymphocytes - immunology | B-Lymphocyte Subsets - metabolism | NF-kappa B - genetics | B-Cell Activating Factor - genetics | Neoplasms - immunology | Autoimmune Diseases | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice | B-Cell Maturation Antigen - genetics | Transmembrane Activator and CAML Interactor Protein - immunology | CD40 Antigens - immunology | Autoimmunity | Antigens | Tumor necrosis factor | Tumors | Necrosis
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5. Full Text TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts
by Bossen, Claudia and Cachero, Teresa G and Tardivel, Aubry and Ingold, Karine and Willen, Laure and Dobles, Max and Scott, Martin L and Maquelin, Aris and Belnoue, Elodie and Siegrist, Claire-Anne and Chevrier, Stéphane and Acha-Orbea, Hans and Leung, Helen and Mackay, Fabienne and Tschopp, Jürg and Schneider, Pascal
Blood, ISSN 0006-4971, 2008, Volume 111, Issue 3, pp. 1004 - 1012
The cytokine BAFF binds to the receptors TACI, BCMA, and BAFF-R on B cells, whereas APRIL binds to TACI and BCMA only. The signaling properties of soluble... 
TUMOR-NECROSIS-FACTOR | CRYSTAL-STRUCTURE | MARGINAL ZONE | BONE-MARROW | TNF FAMILY-MEMBER | COMMON VARIABLE IMMUNODEFICIENCY | LYMPHOCYTE STIMULATOR | RECEPTOR | T-CELL | HEMATOLOGY | CUTTING EDGE | Up-Regulation | Cell Proliferation | Spleen - immunology | Immunoglobulins - immunology | Humans | Molecular Sequence Data | Transmembrane Activator and CAML Interactor Protein - metabolism | Antibodies - immunology | Transmembrane Activator and CAML Interactor Protein - deficiency | Immunoglobulins - biosynthesis | B-Lymphocytes - metabolism | B-Cell Activation Factor Receptor - immunology | Amino Acid Sequence | Cell Line | Antibody Formation - immunology | B-Lymphocytes - cytology | Signal Transduction | Lymphocyte Activation | B-Cell Activating Factor - chemistry | Mice, Transgenic | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Mice, Knockout | Transmembrane Activator and CAML Interactor Protein - genetics | B-Cell Activation Factor Receptor - metabolism | Sequence Alignment | Animals | B-Cell Activating Factor - metabolism | B-Lymphocytes - immunology | Histocompatibility Antigens Class II - immunology | B-Cell Activating Factor - genetics | Ligands | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice
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6. Full Text TACI-Deficient Macrophages Protect Mice Against Metaflammation and Obesity-Induced Dysregulation of Glucose Homeostasis
by Liu, Lunhua and Inouye, Karen Etsuko and Allman, Windy Rose and Coleman, Adam Steven and Siddiqui, Shafiuddin and Hotamisligil, Gökhan Siddik and Akkoyunlu, Mustafa and Akkoyunlu, Mustafa
Diabetes, ISSN 0012-1797, 08/2018, Volume 67, Issue 8, pp. 1589 - 1603
Transmembrane activator and calcium modulator and cyclophilin ligand interactor (TACI) is a receptor for the TNF superfamily cytokines, B cell-activating... 
B-CELLS | METABOLIC DISEASE | BAFF | ALTERNATIVELY ACTIVATED MACROPHAGES | ENERGY-EXPENDITURE | ADIPOSE-TISSUE INFLAMMATION | ENDOCRINOLOGY & METABOLISM | TNF-ALPHA | RECEPTOR TYROSINE KINASE | T-CELLS | INDUCED INSULIN-RESISTANCE | Macrophages - transplantation | Macrophages, Peritoneal - pathology | Diet, High-Fat - adverse effects | Macrophages, Peritoneal - transplantation | Glucose Intolerance - immunology | Transmembrane Activator and CAML Interactor Protein - metabolism | Glucose Intolerance - prevention & control | Adiposity | RNA Interference | Glucose Intolerance - etiology | Inflammation Mediators - metabolism | Female | Weight Gain | Macrophages - immunology | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | Transmembrane Activator and CAML Interactor Protein - chemistry | Immunotherapy, Adoptive | Macrophages - pathology | Cells, Cultured | Gene Expression Regulation | Inflammation Mediators - blood | Insulin Resistance | Macrophages, Peritoneal - immunology | Obesity - physiopathology | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Transmembrane Activator and CAML Interactor Protein - genetics | Macrophages - metabolism | Animals | Obesity - therapy | Mice | Intra-Abdominal Fat - pathology | Transmembrane Activator and CAML Interactor Protein - antagonists & inhibitors | Macrophages, Peritoneal - metabolism | Type 2 diabetes | Obesity | Complications and side effects | Epidemics | Rattus | Rats | Research | Macrophages | Risk factors | 0501 | Immunology and Transplantation
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7. Full Text Transmembrane activator and calcium-modulating cyclophilin ligand interactor mutations in common variable immunodeficiency: Clinical and immunologic outcomes in heterozygotes
by Zhang, Li and Radigan, Lin and Salzer, Ulrich, MD and Behrens, Timothy W., MD and Grimbacher, Bodo, MD and Diaz, George, MD, PhD and Bussel, James, MD and Cunningham-Rundles, Charlotte, MD, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2007, Volume 120, Issue 5, pp. 1178 - 1185
Background Mutations in the gene coding for transmembrane activator and calcium-modulating cyclophilin ligand interactor (TACI) have been identified in common... 
Allergy and Immunology | B cell | IgA | TACI | IgG | immune thrombocytopenia purpura | Common variable immune deficiency | splenectomy | BAFF | common variable immune deficiency | RECEPTOR | IMMUNOLOGY | SELECTIVE IGA DEFICIENCY | IDENTIFICATION | B-CELLS | ALLERGY | BLYS | BINDING | T-CELLS | APRIL | Prognosis | Purpura, Thrombocytopenic, Idiopathic - genetics | Lymphocyte Activation | Humans | Middle Aged | Autoimmunity - genetics | Common Variable Immunodeficiency - genetics | Male | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Common Variable Immunodeficiency - diagnosis | Transmembrane Activator and CAML Interactor Protein - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | B-Lymphocytes - immunology | Hyperplasia - genetics | DNA Mutational Analysis | Pedigree | Adolescent | Aged, 80 and over | Adult | Female | Heterozygote | Aged | Mutation | Cyclophilin | Immunological deficiency syndromes | Genetic aspects | Pneumonia | Calcium | Ear diseases | Arthritis | Defects | Inflammatory bowel disease | Genotype & phenotype | Cell growth | Ligands | Genetic testing | Deoxyribonucleic acid--DNA | Immune system
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8. Full Text Cutting edge: BAFF promotes autoantibody production via TACI-dependent activation of transitional b cells
by Jacobs, Holly M and Thouvenel, Christopher D and Leach, Sarah and Arkatkar, Tanvi and Metzler, Genita and Scharping, Nicole E and Kolhatkar, Nikita S and Rawlings, David J and Jackson, Shaun W
Journal of Immunology, ISSN 0022-1767, 05/2016, Volume 196, Issue 9, pp. 3525 - 3531
Mice overexpressing B cell activating factor of the TNF family (BAFF) develop systemic autoimmunity characterized by class-switched anti-nuclear Abs.... 
Autoimmunity | Lymphocyte Activation | Mice, Transgenic | Precursor Cells, B-Lymphoid - immunology | Autoantibodies - biosynthesis | Transmembrane Activator and CAML Interactor Protein - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | Autoantibodies - immunology | Animals | B-Cell Activating Factor - metabolism | B-Cell Activating Factor - genetics | B-Lymphocyte Subsets - immunology | Mice | Precursor Cells, B-Lymphoid - physiology
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9. Full Text TACI‐dependent APRIL signaling maintains autoreactive B cells in a mouse model of systemic lupus erythematosus
by Tran, Ngoc Lan and Schneider, Pascal and Santiago‐Raber, Marie‐Laure
European Journal of Immunology, ISSN 0014-2980, 04/2017, Volume 47, Issue 4, pp. 713 - 723
Autoantibodies contribute to the development of systemic lupus erythematosus (SLE). APRIL (a proliferation‐inducing ligand), a member of the TNF superfamily,... 
Systemic lupus erythematosus (SLE) | TACI | mouse model | B‐cell maturation antigen (BCMA) | APRIL | B-cell maturation antigen (BCMA) | TNF RECEPTOR | BAFF | ACTIVATION | PROLIFERATION-INDUCING LIGAND | BONE-MARROW | DEFICIENT MICE | IMMUNOLOGY | DISEASE-ACTIVITY | LYMPHOCYTE STIMULATOR LEVELS | MURINE LUPUS | EXPRESSION | Autoantibodies - metabolism | Disease Susceptibility | Humans | Mice, Inbred C57BL | Cells, Cultured | Male | Signal Transduction - genetics | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Mice, Knockout | Transmembrane Activator and CAML Interactor Protein - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | Animals | B-Cell Maturation Antigen - metabolism | B-Lymphocytes - immunology | Mice, Mutant Strains | Autoantigens - immunology | Lupus Erythematosus, Systemic - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice | B-Cell Maturation Antigen - genetics | Disease Models, Animal | Lupus | Autoimmunity | Autoantibodies | Systemic lupus erythematosus | Analysis | Mortality | B cells | Rodents
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10. Full Text Cutting edge: BAFF overexpression reduces atherosclerosis via TACI-dependent B cell activation
by Jackson, Shaun W and Scharping, Nicole E and Jacobs, Holly M and Wang, Shari and Chait, Alan and Rawlings, David J
Journal of Immunology, ISSN 0022-1767, 12/2016, Volume 197, Issue 12, pp. 4529 - 4534
Patients with systemic lupus erythematosus exhibit accelerated atherosclerosis, a chronic inflammatory disease of the arterial wall. The impact of B cells in... 
ANTIBODIES | IMMUNITY | SYSTEMIC-LUPUS-ERYTHEMATOSUS | RISK-FACTORS | MOUSE | CARDIOVASCULAR-DISEASE | RECEPTOR | BLYS | IMMUNOLOGY | NATURAL IGM | LYMPHOCYTES | Atherosclerosis - immunology | Lymphocyte Activation | Autoantibodies - blood | Humans | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Mice, Knockout | Transmembrane Activator and CAML Interactor Protein - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | Animals | B-Cell Activating Factor - metabolism | B-Lymphocytes - immunology | B-Cell Activating Factor - genetics | Lupus Erythematosus, Systemic - immunology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Immunoglobulin Class Switching
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