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Journal of Experimental Medicine, ISSN 0022-1007, 01/2009, Volume 206, Issue 1, pp. 249 - 258
Psoriasis is a type I interferon-driven T cell-mediated disease characterized by the recruitment of plasmacytoid dendritic cells (pDC) into the skin. The... 
PATHOGENESIS | MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | DERMATITIS | INFLAMMATION | LYMPH-NODES | IN-VIVO | CYTOKINE | IMMUNOLOGY | PRECURSORS | T-CELLS | PROTEASES | CD8-Positive T-Lymphocytes - cytology | Neutrophils - cytology | Membrane Glycoproteins - metabolism | Skin - metabolism | Humans - metabolism | Antigens, CD - metabolism | Chemotaxis, Leukocyte - physiology | Lectins, C-Type - metabolism | Chemotaxis, Leukocyte - drug effects | Psoriasis - pathology | Psoriasis - metabolism | CD8-Positive T-Lymphocytes - metabolism | Receptors, Chemokine - genetics | Neutrophils - metabolism | Antibodies, Monoclonal - immunology | Fibroblasts - metabolism | Tretinoin - pharmacology | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Chemokines - genetics | Blotting, Western | Intercellular Adhesion Molecule-1 - metabolism | Calcitriol - pharmacology | Fibroblasts - drug effects | Chemokines - metabolism | Fibroblasts - cytology | Receptors, Immunologic - genetics | Chemokine CXCL10 - metabolism | Dermatitis, Atopic - genetics | Gene Expression - drug effects | Culture Media, Conditioned - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Lectins, C-Type - genetics | Psoriasis - genetics | Adult | Dendritic Cells - metabolism | Skin - pathology | Intercellular Signaling Peptides and Proteins | Receptors, Chemokine - metabolism | Cells, Cultured | Dermatitis, Atopic - pathology | Receptors, Chemokine - immunology | Membrane Glycoproteins - genetics | Chemokine CXCL10 - genetics | CD8-Positive T-Lymphocytes - drug effects | Dendritic Cells - cytology | Dermatitis, Atopic - metabolism | Receptors, Immunologic - metabolism | Index Medicus
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 06/2009, Volume 23, Issue 6, pp. 1643 - 1654
Late-onset Alzheimer's disease is often connected with nutritional misbalance, such as enhanced cholesterol intake, deficiency in polyunsaturated fatty acids,... 
Shedding | Vitamin A | Nuclear receptors | Alzheimer's disease | Amyloid precursor protein | vitamin A | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID-PRECURSOR-PROTEIN | shedding | X-RECEPTOR | BETA-PEPTIDE | NEUROBLASTOMA-CELLS | HIPPOCAMPAL-NEURONS | VITAMIN-A | CELL BIOLOGY | nuclear receptors | RESPONSE ELEMENT | amyloid precursor protein | BIOLOGY | LONG-TERM POTENTIATION | EXPRESSION | Amyloid Precursor Protein Secretases - genetics | Up-Regulation | Tretinoin - chemistry | Humans | Male | PPAR gamma - metabolism | Receptors, Calcitriol - genetics | Receptors, Retinoic Acid - genetics | DNA-Binding Proteins - metabolism | Keratolytic Agents - metabolism | Receptors, Thyroid Hormone - genetics | Tretinoin - metabolism | Liver X Receptors | Acitretin - metabolism | Amyloid beta-Protein Precursor - metabolism | Membrane Proteins - metabolism | Molecular Structure | Orphan Nuclear Receptors | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | PPAR gamma - genetics | Cell Line | Promoter Regions, Genetic | Membrane Proteins - genetics | Gene Expression Regulation | ADAM10 Protein | Mice, Transgenic | Receptors, Retinoic Acid - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Receptors, Calcitriol - metabolism | DNA-Binding Proteins - genetics | Acitretin - chemistry | Receptors, Thyroid Hormone - metabolism | ADAM Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Mice | ADAM Proteins - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
PLoS Biology, ISSN 1545-7885, 2006, Volume 4, Issue 8, pp. 1438 - 1452
The link between extrinsic signaling, progenitor cell specification and neuronal subtype identity is central to the developmental organization of the... 
ANTERIOR NEUROECTODERM | RETINOIC ACID SYNTHESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | SPINAL-CORD | CHICK-EMBRYO | T-BOX GENE | DISTINCT ROLES | POSITIONAL IDENTITY | INDUCTIVE SIGNALS | PARAXIAL MESODERM | MOUSE EMBRYO | Spinal Cord - metabolism | Homeodomain Proteins - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Wnt Proteins - metabolism | Central Nervous System - embryology | Rhombencephalon - cytology | Fibroblast Growth Factors - metabolism | Wnt Proteins - genetics | Tretinoin - metabolism | Gene Expression Regulation, Developmental | Motor Neurons - cytology | Spinal Cord - cytology | Genes, Homeobox | Rhombencephalon - embryology | Avian Proteins - metabolism | Signal Transduction | Chick Embryo | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | T-Box Domain Proteins - metabolism | Motor Neurons - metabolism | Spinal Cord - embryology | Animals | Rhombencephalon - metabolism | Avian Proteins - genetics | Index Medicus | Development | Neuroscience | Chicken | Cell Biology | Basic Medicine | Gene Expression Regulation; Developmental | T-Box Domain Proteins/genetics/metabolism | Medical and Health Sciences | Medicin och hälsovetenskap | Central Nervous System/embryology | Cell and Molecular Biology | Spinal Cord/cytology/embryology/metabolism | Medicinska och farmaceutiska grundvetenskaper | Fibroblast Growth Factors/metabolism | Motor Neurons/cytology/metabolism | Stem Cells/cytology/metabolism | Wnt Proteins/genetics/metabolism | Avian Proteins/genetics/metabolism | Cell- och molekylärbiologi | Rhombencephalon/cytology/embryology/metabolism | Genes; Homeobox | Homeodomain Proteins/genetics/metabolism | Tretinoin/metabolism | Neurons | Spinal cord | Rodents
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 04/2018, Volume 46, Issue 1, pp. 187 - 202
Background/Aims: All-trans retinoic acid (ATRA) has protective effects against obesity and metabolic syndrome. We here aimed to gain further insight into the... 
Myokines | Obesity | AMPK | Muscle | PPARβ/δ | Nuclear receptors | Intramyocellular lipids | Retinoids/Vitamin A | Fatty acid/oxidation | PPAR beta/delta | PHYSIOLOGY | LIPID-METABOLISM | VITAMIN-A SUPPLEMENTATION | ACTIVATED RECEPTOR | CELL BIOLOGY | INSULIN-RESISTANCE | Retinoids/vitamin A | GENE-EXPRESSION | LEPTIN EXPRESSION | UNCOUPLING PROTEIN-3 | MICE | ADIPOSE-TISSUE | WHITE ADIPOCYTES | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Male | Muscle, Skeletal - metabolism | Muscle, Skeletal - cytology | Fibroblast Growth Factors - metabolism | Liver - drug effects | PPAR-beta - agonists | Muscle, Skeletal - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Fatty Acids - metabolism | Interleukin-6 - metabolism | Retinoid X Receptors - metabolism | Tretinoin - pharmacology | Neuropeptides - analysis | Cell Line | Enzyme-Linked Immunosorbent Assay | PPAR-beta - metabolism | Liver - metabolism | Neuropeptides - metabolism | Fibronectins - blood | Fibronectins - metabolism | Triglycerides - metabolism | Animals | Retinoid X Receptors - agonists | PPAR delta - agonists | Lipid Metabolism - drug effects | Glucose - metabolism | Fibronectins - genetics | Mice | Muscle, Skeletal - pathology | PPAR delta - metabolism | Liver | Homeostasis | Cytotoxicity | Lipids | Adipocytes | Glucose | Metabolism | Kinases | Fatty acids | Insulin | Proteins | Musculoskeletal system | Vitamin A | Antibiotics | Energy | Rodents | Oxidation | Diabetes | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2005, Volume 280, Issue 22, pp. 21453 - 21462
In most neurodegenerative disorders, including multiple sclerosis, Parkinson disease, and Alzheimer disease, a massive neuronal cell death occurs as a... 
CEREBELLAR GRANULE NEURONS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MICROGLIA | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | 15-DEOXY-DELTA(12,14)-PROSTAGLANDIN J | PPAR-GAMMA | EXPRESSION | ASTROCYTES | BRAIN | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Models, Chemical | Cyclooxygenase 2 | Lipopolysaccharides - metabolism | Nitrites - metabolism | Glycogen Synthase Kinase 3 beta | PPAR gamma - metabolism | Nitric Oxide Synthase Type II | Brain - metabolism | Dose-Response Relationship, Drug | Staurosporine - metabolism | Transfection | Time Factors | Tretinoin - metabolism | Inflammation - drug therapy | Lithium Chloride - pharmacology | Cell Death | Neurons - metabolism | Neurons - drug effects | Thiazolidinediones - pharmacology | Interleukin-6 - metabolism | Cell Line | Anti-Inflammatory Agents - pharmacology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Neurodegenerative Diseases - metabolism | Nitrites - chemistry | Hippocampus - metabolism | Microscopy, Confocal | Animals | Cell-Free System | Prostaglandin-Endoperoxide Synthases - metabolism | Anilides - pharmacology | Neuroglia - metabolism | Glutamic Acid - metabolism | Mice | Nitric Oxide Synthase - metabolism | In Vitro Techniques | Microscopy, Fluorescence | Apoptosis | Astrocytes - metabolism | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2016, Volume 291, Issue 21, pp. 11216 - 11229
Journal Article
Blood, ISSN 0006-4971, 03/2010, Volume 115, Issue 10, pp. 1958 - 1968
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 5, pp. 1278 - 1288.e2
Background & Aims Reduced generation of all-trans retinoic acid (RA) by CD103+ intestinal dendritic cells (DCs) is linked to intestinal inflammation in mice.... 
Gastroenterology and Hepatology | Regulation | Inflammatory Bowel Disease | Immune Response | Cytokine | Keywords | TGF-BETA | LAMINA PROPRIA | RECEPTOR-ALPHA | BOWEL-DISEASE | RESPONSES | REGULATORY T-CELL | CD103(+) DENDRITIC CELLS | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | TH17 | Crohn Disease - genetics | Up-Regulation | Ileum - pathology | Intestinal Mucosa - metabolism | Dendritic Cells - immunology | Humans | Retinal Dehydrogenase - metabolism | Crohn Disease - metabolism | Ileum - metabolism | Colon - immunology | Ileum - immunology | Case-Control Studies | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Tretinoin - metabolism | Intestinal Mucosa - immunology | Receptors, Retinoic Acid - immunology | Aldehyde Dehydrogenase - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Macrophages - pathology | Signal Transduction | Colon - pathology | Cells, Cultured | Aldehyde Dehydrogenase - genetics | Receptors, Retinoic Acid - metabolism | Integrin alpha Chains - metabolism | Retinal Dehydrogenase - genetics | Crohn Disease - immunology | Colon - metabolism | Gene Expression Regulation, Enzymologic | Macrophages - metabolism | Phenotype | Crohn Disease - pathology | Retinoic Acid Receptor alpha | Intestinal Mucosa - pathology | Medical colleges | Genetic aspects | Dendritic cells | Macrophages | Gastrointestinal diseases | Tretinoin | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2016, Volume 213, Issue 1, pp. 53 - 73
Protective immunoglobulin A (IgA) responses to oral antigens are usually orchestrated by gut dendritic cells (DCs). Here, we show that lung CD103(+) and... 
B-LYMPHOCYTE STIMULATOR | MEDICINE, RESEARCH & EXPERIMENTAL | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PEYERS PATCH | IMMUNOGLOBULIN-A | RETINOIC ACID | INFLUENZA-VIRUS INFECTION | HEAT-LABILE ENTEROTOXIN | NECROSIS-FACTOR FAMILY | ESCHERICHIA-COLI | IMMUNOLOGY | T-CELLS | Receptors, CCR - metabolism | CD24 Antigen - metabolism | Dendritic Cells - immunology | Adaptor Proteins, Vesicular Transport - genetics | Immunoglobulin A - genetics | Integrins - genetics | Adaptor Proteins, Vesicular Transport - metabolism | Integrins - metabolism | Cell Movement - genetics | Antigens, CD - metabolism | Gastrointestinal Tract - immunology | Microbiota | Immunoglobulin Class Switching - drug effects | B-Lymphocytes - metabolism | Dendritic Cells - metabolism | Macrophages, Alveolar - immunology | Tretinoin - pharmacology | Gene Expression | Receptors, CCR - genetics | Myeloid Differentiation Factor 88 - genetics | Gastrointestinal Tract - microbiology | Integrin alpha Chains - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Gastrointestinal Tract - metabolism | B-Lymphocytes - drug effects | Transforming Growth Factor beta - pharmacology | Animals | B-Cell Activating Factor - metabolism | B-Lymphocytes - immunology | B-Cell Activating Factor - genetics | Immunoglobulin A - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice | Macrophages, Alveolar - metabolism | Immunoglobulin Class Switching - genetics | Myeloid Differentiation Factor 88 - metabolism | Lung - immunology | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 2008, Volume 7, Issue 3, pp. 258 - 268
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 44, pp. 18886 - 18891
The proper function of the bone morphogenic protein (BMP) pathway during embryonic development and organ maintenance requires its communication with other... 
Receptors | Phosphorylation | Plasmids | Neurons | Ubiquitins | Antibodies | Gene expression regulation | Embryos | Progenitor cells | Cells | PATHWAYS | NERVOUS-SYSTEM | TRANSCRIPTIONAL ACTIVATION | TGF-BETA | BMP | MULTIDISCIPLINARY SCIENCES | E3 UBIQUITIN LIGASES | RECEPTORS | CELL-TYPES | DORSAL SPINAL-CORD | FAMILY | Phosphorylation - physiology | Mitogen-Activated Protein Kinase Kinases - genetics | Cell Proliferation | Stem Cells - metabolism | Wnt Proteins - metabolism | Embryo, Mammalian - metabolism | Receptors, Retinoic Acid - genetics | Mitogen-Activated Protein Kinase Kinases - metabolism | Bone Morphogenetic Proteins - metabolism | Wnt Proteins - genetics | Tretinoin - metabolism | Smad1 Protein - genetics | Cell Cycle Proteins - genetics | Ubiquitination - physiology | Nuclear Proteins - genetics | Bone Morphogenetic Proteins - genetics | Cell Differentiation - physiology | Epidermal Growth Factor - genetics | Cell Cycle Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | Embryonic Development - physiology | Epidermal Growth Factor - metabolism | Nuclear Proteins - metabolism | Receptors, Retinoic Acid - metabolism | Glycogen Synthase Kinase 3 - metabolism | Chick Embryo | Proteasome Endopeptidase Complex - genetics | Animals | Glycogen Synthase Kinase 3 - genetics | Embryo, Mammalian - cytology | Cell Line, Tumor | Smad1 Protein - metabolism | Signal Transduction - physiology | Mice | Proteasome Endopeptidase Complex - metabolism | Neural Tube - embryology | Ubiquitin-Protein Ligases - genetics | Physiological aspects | Bone morphogenetic proteins | Research | Properties | Proteolysis | Tretinoin | Proteins | Signal transduction | Birds | T cell receptors | Gene expression | Index Medicus | Cell proliferation | Fibroblast growth factor | Pattern formation | Smad protein | Transcription | Wnt protein | Retinoic acid receptors | proteasomes | MAP kinase | Embryogenesis | Neural tube | ubiquitination | GADD45 protein | Neural stem cells | Differentiation | Retinoic acid | Ubiquitin-protein ligase | Communication | Biological Sciences
Journal Article