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2009, ISBN 9780199236695, 196
Book
British Journal of Pharmacology, ISSN 0007-1188, 12/2013, Volume 170, Issue 7, pp. 1293 - 1307
The neuropeptide calcitonin gene‐related peptide (CGRP) is reported to play an important role in migraine. It is expressed throughout the trigeminovascular... 
trigeminal ganglia | calcitonin gene‐related peptide | migraine | Amylin | G‐protein coupled receptor | GPCR | CGRP | trigeminovascular system | G-protein coupled receptor | calcitonin gene-related peptide | AMYLOID POLYPEPTIDE AMYLIN | SPINAL TRIGEMINAL NUCLEUS | GENE-RELATED-PEPTIDE | CYCLIC-AMP FORMATION | CALCITONIN-RECEPTOR | SK-N-MC | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | SMOOTH-MUSCLE-CELLS | SUBSTANCE-P | ACTIVITY-MODIFYING PROTEIN-1 | Central Nervous System Agents - therapeutic use | Trigeminal Nerve - metabolism | Trigeminal Nucleus, Spinal - metabolism | Receptors, Calcitonin - metabolism | Humans | Vasodilation | Migraine Disorders - metabolism | Calcitonin Gene-Related Peptide - antagonists & inhibitors | Migraine Disorders - physiopathology | Trigeminal Nerve - physiopathology | Receptors, Islet Amyloid Polypeptide - metabolism | Calcitonin Gene-Related Peptide - metabolism | Trigeminal Nuclei - metabolism | Trigeminal Nuclei - physiopathology | Signal Transduction | Receptor Activity-Modifying Protein 1 - metabolism | Trigeminal Ganglion - metabolism | Adrenomedullin - metabolism | Migraine Disorders - drug therapy | Trigeminal Nerve - drug effects | Animals | Trigeminal Nuclei - drug effects | Cerebral Arteries - innervation | Trigeminal Ganglion - physiopathology | Trigeminal Nucleus, Spinal - physiopathology | Peptides | Physiological aspects | Migraine | Signal transduction | Themed Section | Neuropeptides
Journal Article
Pain, ISSN 0304-3959, 06/2009, Volume 143, Issue 3, pp. 186 - 191
Journal Article
Journal Article
European Journal of Neurology, ISSN 1351-5101, 10/2008, Volume 15, Issue 10, pp. 1013 - 1028
Journal Article
Journal Article
Brain Research Bulletin, ISSN 0361-9230, 09/2017, Volume 134, pp. 72 - 78
Journal Article
Molecular brain, ISSN 1756-6606, 2012, Volume 5, Issue 1, pp. 44 - 44
Background: Trigeminal neuropathic pain attacks can be excruciating for patients, even after being lightly touched. Although there are rodent trigeminal nerve... 
ACTIVATION | RAT | SENSATION LIKE | SPINAL MICROGLIA | TACTILE ALLODYNIA | P38 MAPK | Inflammation | Trigeminal ganglia | Hypersensitivy | NEUROSCIENCES | Chromic gut suture | Nerve compression | P2X7 RECEPTOR | Tic douloureux | Infraorbital nerve | CHRONIC CONSTRICTION INJURY | Trigeminal nucleus | Mice | Orofacial neuropathic pain | Mechanical allodynia | Inflammation - pathology | Neurons - pathology | Neuralgia - complications | Tetrazoles - pharmacology | Minocycline - pharmacology | Male | Purinergic P2X Receptor Antagonists - pharmacology | Trigeminal Nucleus, Spinal - drug effects | Neuralgia - pathology | Nerve Compression Syndromes - complications | Trigeminal Ganglion - drug effects | Hyperalgesia - pathology | Inflammation - complications | Microglia - pathology | Behavior, Animal - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Neurons - drug effects | Orbit - pathology | Disease Models, Animal | Trigeminal Nucleus, Spinal - pathology | Trigeminal Neuralgia - complications | Microglia - drug effects | Trigeminal Ganglion - pathology | Hyperalgesia - complications | Mice, Inbred C57BL | Trigeminal Nerve - pathology | Imidazoles - pharmacology | Orbit - drug effects | Trigeminal Nerve - drug effects | Animals | Nerve Compression Syndromes - pathology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Trigeminal Neuralgia - pathology | Facial Pain - complications | Pyridines - pharmacology | Orbit - innervation | Facial Pain - pathology | Studies | Pain | Anatomy & physiology | Rodents | Surgery | Edema | Animal models | Compression | Pain perception | Hypersensitivity | Neuropathy | Macrophages | Trauma | Maxilla | Mimicry | Trigeminal nerve | Nerves | Sutures | Bone | Immunoreactivity | Spinal trigeminal nucleus | Trigeminal ganglion | Digestive tract
Journal Article
Pain, ISSN 0304-3959, 2006, Volume 121, Issue 3, pp. 219 - 231
Glial activation is known to contribute to pain hypersensitivity following spinal sensory nerve injury. In this study, we investigated mechanisms by which... 
OX-42 | Medullary dorsal horn | Mental nerve | c-Fos | Minocycline | p38 mitogen-activated protein kinase | Glia | Inferior alveolar nerve | Tactile hypersensitivity | GFAP | MECHANICAL ALLODYNIA | DIFFERENTIAL EXPRESSION | mental nerve | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | tactile hypersensitivity | FOS-LIKE IMMUNOREACTIVITY | PATHOLOGICAL PAIN | glia | NEUROSCIENCES | CLINICAL NEUROLOGY | medullary dorsal horn | CONSTRICTION INJURY | NEUROPATHIC PAIN | minocycline | NUCLEUS CAUDALIS | inferior alveolar nerve | ANESTHESIOLOGY | INFRAORBITAL NERVE | Immunohistochemistry | Microglia - metabolism | Trigeminal Nerve Injuries | Wallerian Degeneration - pathology | Minocycline - pharmacology | Trigeminal Caudal Nucleus - metabolism | Male | CD11b Antigen | Gliosis - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Trigeminal Caudal Nucleus - cytology | Up-Regulation - physiology | Neurons, Afferent - pathology | Trigeminal Nerve Diseases - metabolism | Trigeminal Nerve - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Trigeminal Nerve Diseases - complications | Gliosis - etiology | Disease Models, Animal | Astrocytes - cytology | Biomarkers - metabolism | Microglia - cytology | Trigeminal Nerve Diseases - physiopathology | Hyperalgesia - metabolism | Trigeminal Ganglion - pathology | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Rats | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Animals | Trigeminal Ganglion - physiopathology | Hyperalgesia - etiology | Enzyme Activation - physiology | Astrocytes - metabolism | Antigen-antibody reactions | Allergy | Neurosciences | Analysis | Allergic reaction | Intermediate filament proteins | Protein kinases
Journal Article