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Journal Article
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 03/2010, Volume 518, Issue 5, pp. 687 - 698
Transient receptor potential ankyrin 1 (TRPA1), responding to noxious cold and pungent compounds, is implicated in the mediation of nociception, but little is... 
synapse | TRPA1 | trigeminal | primary afferent | Synapse | Primary afferent | Trigeminal | TRPA 1 | CORD | A-DELTA-FIBERS | ION-CHANNEL TRPA1 | NOCICEPTIVE RESPONSES | NEUROSCIENCES | PAIN | ZOOLOGY | SUBNUCLEUS INTERPOLARIS | COLD HYPERSENSITIVITY | SUBSTANTIA-GELATINOSA | NEURONS | NEUROPATHIC RATS | Immunohistochemistry | Trigeminal Nerve - metabolism | Posterior Horn Cells - metabolism | Synaptic Vesicles - metabolism | Calcium Channels - metabolism | Trigeminal Caudal Nucleus - metabolism | Dendrites - ultrastructure | Nerve Fibers, Unmyelinated - ultrastructure | Presynaptic Terminals - ultrastructure | Pain - metabolism | Microscopy, Immunoelectron | Posterior Horn Cells - cytology | Trigeminal Caudal Nucleus - cytology | Axons - ultrastructure | Sensory Receptor Cells - metabolism | Trigeminal Nuclei - metabolism | Afferent Pathways - metabolism | Biomarkers - metabolism | Dendrites - metabolism | Trigeminal Ganglion - cytology | Trigeminal Nuclei - cytology | Synaptic Vesicles - ultrastructure | TRPC Cation Channels | Axons - metabolism | Rats | Trigeminal Ganglion - metabolism | Nociceptors - cytology | Afferent Pathways - cytology | Rats, Sprague-Dawley | Animals | Sensory Receptor Cells - cytology | Nerve Fibers, Unmyelinated - metabolism | Pain - physiopathology | TRPA1 Cation Channel | Brain Mapping | Presynaptic Terminals - metabolism | Ankyrins | Substance P - metabolism | Nociceptors - metabolism | Rhizotomy | Plant Lectins - metabolism | Trigeminal Nerve - cytology | Index Medicus
Journal Article
Journal Article
Pain, ISSN 0304-3959, 2006, Volume 121, Issue 3, pp. 219 - 231
Glial activation is known to contribute to pain hypersensitivity following spinal sensory nerve injury. In this study, we investigated mechanisms by which... 
OX-42 | Medullary dorsal horn | Mental nerve | c-Fos | Minocycline | p38 mitogen-activated protein kinase | Glia | Inferior alveolar nerve | Tactile hypersensitivity | GFAP | MECHANICAL ALLODYNIA | DIFFERENTIAL EXPRESSION | mental nerve | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | tactile hypersensitivity | FOS-LIKE IMMUNOREACTIVITY | PATHOLOGICAL PAIN | glia | NEUROSCIENCES | CLINICAL NEUROLOGY | medullary dorsal horn | CONSTRICTION INJURY | NEUROPATHIC PAIN | minocycline | NUCLEUS CAUDALIS | inferior alveolar nerve | ANESTHESIOLOGY | INFRAORBITAL NERVE | Immunohistochemistry | Microglia - metabolism | Trigeminal Nerve Injuries | Wallerian Degeneration - pathology | Minocycline - pharmacology | Trigeminal Caudal Nucleus - metabolism | Male | CD11b Antigen | Gliosis - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Trigeminal Caudal Nucleus - cytology | Up-Regulation - physiology | Neurons, Afferent - pathology | Trigeminal Nerve Diseases - metabolism | Trigeminal Nerve - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Trigeminal Nerve Diseases - complications | Gliosis - etiology | Disease Models, Animal | Astrocytes - cytology | Biomarkers - metabolism | Microglia - cytology | Trigeminal Nerve Diseases - physiopathology | Hyperalgesia - metabolism | Trigeminal Ganglion - pathology | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Rats | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Animals | Trigeminal Ganglion - physiopathology | Hyperalgesia - etiology | Enzyme Activation - physiology | Astrocytes - metabolism | Antigen-antibody reactions | Allergy | Neurosciences | Analysis | Allergic reaction | Intermediate filament proteins | Protein kinases | Index Medicus
Journal Article
Journal Article
Peptides, ISSN 0196-9781, 02/2012, Volume 33, Issue 2, pp. 307 - 316
► We examined the PACAP-27/38-LI in the activated trigeminovascular system. ► Blood plasma, nerve tissues and liquor were analyzed in two rat models. ►... 
Electrical and chemical stimulation | Trigeminal ganglion | Trigeminal nucleus caudalis | Mass spectrometry | Nitroglycerol | Blood plasma | GENE-RELATED PEPTIDE | INDUCED NEURONAL ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR MESSENGER-RNA | SPINAL-CORD | ELECTRICAL-STIMULATION | VASOACTIVE INTESTINAL POLYPEPTIDE | CAUDAL TRIGEMINAL NUCLEUS | ENDOCRINOLOGY & METABOLISM | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | URINARY-BLADDER PATHWAYS | CEREBRAL DURA-MATER | Immunohistochemistry | Electric Stimulation | Spinal Cord - drug effects | Spinal Cord - metabolism | Vasodilator Agents - pharmacology | Vasomotor System - metabolism | Protein Isoforms - cerebrospinal fluid | Trigeminal Caudal Nucleus - metabolism | Vasomotor System - drug effects | Rats | Trigeminal Ganglion - metabolism | Trigeminal Caudal Nucleus - drug effects | Trigeminal Ganglion - drug effects | Nitroglycerin - pharmacology | Rats, Sprague-Dawley | Protein Isoforms - blood | Animals | Protein Isoforms - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - blood | Pituitary Adenylate Cyclase-Activating Polypeptide - cerebrospinal fluid | Trigeminal Ganglion - physiology | Peptides | Neurosciences | Universities and colleges | Headache | Alterations | Spinal cord | Elevated | Neurons | Stimulation | Activation | Ganglia | Index Medicus
Journal Article