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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2005, Volume 102, Issue 21, pp. 7671 - 7676
Resolvin E1 (RvE1; 5S, 12R, 18R-trihydroxyeicosapentaenoic acid) is an antiinflammatory lipid mediator derived from omega-3 fatty acid eicosapentaenoic acid... 
Biological Sciences | Inflammatory bowel diseases | Antiinflammatories | Lipids | Inflammation | Histology | Mice | Colitis | Leukocytes | Neutrophil infiltration | Vehicles | Antiinflammation | Omega-3 pufa | Resolvins | Aspirin-triggered lipid mediators | OXIDATION | OMEGA-3-FATTY-ACIDS | MULTIDISCIPLINARY SCIENCES | RESOLUTION | RECEPTOR | aspirin-triggered lipid mediators | RESPONSES | antiinflammation | NECROSIS-FACTOR | INFLAMMATORY-BOWEL-DISEASE | HAPTEN-INDUCED COLITIS | POLYUNSATURATED FATTY-ACIDS | omega-3 PUFA | resolvins | MICE | Tumor Necrosis Factor-alpha - metabolism | Immunoglobulin G - blood | Eicosapentaenoic Acid - analogs & derivatives | Nitric Oxide Synthase Type II | Protein Subunits - metabolism | Eicosapentaenoic Acid - pharmacology | Aspirin - metabolism | Female | Aspirin - therapeutic use | Colitis, Ulcerative - prevention & control | Enzyme-Linked Immunosorbent Assay | Eicosapentaenoic Acid - metabolism | Interleukin-12 - metabolism | DNA Primers | Reverse Transcriptase Polymerase Chain Reaction | Gene Expression Regulation - drug effects | Animals | Colitis, Ulcerative - chemically induced | Leukocytes - drug effects | Eicosapentaenoic Acid - therapeutic use | Mice, Inbred BALB C | Nitric Oxide Synthase - metabolism | Trinitrobenzenesulfonic Acid - toxicity | Interleukin-12 Subunit p40 | Peroxidase - metabolism | Research | Anti-inflammatory drugs | Omega-3 fatty acids | Index Medicus
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 11/2007, Volume 22, Issue 11, pp. 1859 - 1865
Background and Aim:  α‐Lipoic acid (ALA) has been shown to combat oxidative stress by quenching a variety of reactive oxygen species. It is involved in the... 
colitis | rat | α‐lipoic acid | trinitrobenzenesulfonic acid (TNBS) | ileitis | α-lipoic acid | Colitis | Ileitis | Rat | Trinitrobenzenesulfonic acid (TNBS) | LIPOATE | ACTIVATION | alpha-lipoic acid | COLON | MODEL | BOWEL-DISEASE | REACTIVE OXYGEN | METABOLISM | GLUTATHIONE | ANTIOXIDANT | GASTROENTEROLOGY & HEPATOLOGY | Ileum - pathology | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Ileitis - chemically induced | Colon - drug effects | Ileum - metabolism | Colitis - pathology | Trinitrobenzenesulfonic Acid | Ileitis - pathology | Ileum - drug effects | Colitis - chemically induced | Lipid Peroxidation - drug effects | Anti-Inflammatory Agents - therapeutic use | Colitis - drug therapy | Ileitis - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Malondialdehyde - metabolism | Ileitis - drug therapy | Neutrophils - enzymology | Anti-Inflammatory Agents - pharmacology | Colon - pathology | Neutrophils - drug effects | Rats | Thioctic Acid - pharmacology | Antioxidants - pharmacology | Rats, Sprague-Dawley | Colon - metabolism | Antioxidants - therapeutic use | Gastrointestinal Agents - pharmacology | Animals | Colitis - metabolism | Gastrointestinal Agents - therapeutic use | Oxidative Stress - drug effects | Peroxidase - metabolism | Thioctic Acid - therapeutic use | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2012, Volume 487, Issue 7408, pp. 477 - 481
Malnutrition affects up to one billion people in the world and is a major cause of mortality. In many cases, malnutrition is associated with diarrhoea and... 
MAMMALIAN TARGET | RAPAMYCIN | CARBOXYPEPTIDASE | CROHNS-DISEASE | COLITIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MICE | HARTNUP DISORDER | BLOOD-PRESSURE | ANGIOTENSIN-CONVERTING-ENZYME | Dextran Sulfate | TOR Serine-Threonine Kinases - metabolism | Homeostasis | Malnutrition - metabolism | Colitis - pathology | Male | Trinitrobenzenesulfonic Acid | Tryptophan - metabolism | Malnutrition - complications | Dietary Proteins - metabolism | Peptidyl-Dipeptidase A - genetics | Dietary Proteins - pharmacology | Gene Deletion | Peptidyl-Dipeptidase A - metabolism | Female | Colitis - drug therapy | Diarrhea - complications | Peptidyl-Dipeptidase A - deficiency | Genetic Predisposition to Disease | Biocatalysis | Intestines - pathology | Niacinamide - metabolism | Niacinamide - therapeutic use | Renin-Angiotensin System - physiology | Immunity, Innate | Colitis - etiology | Animals | Colitis - microbiology | Intestines - microbiology | Models, Biological | Germ-Free Life | Mice | Tryptophan - pharmacology | Niacinamide - pharmacology | Metagenome | Tryptophan - therapeutic use | Intestines | Angiotensin converting enzyme | Physiological aspects | Amino acids | Development and progression | Inflammation | Health aspects | Microbial colonies | Inflammatory bowel disease | Genotype & phenotype | Peptides | Rodents | Bone marrow | Ecology | Colon | Malnutrition | Index Medicus
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 3/2017, Volume 62, Issue 3, pp. 639 - 651
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6251 - 6261
Journal Article
Molecules, ISSN 1420-3049, 06/2017, Volume 22, Issue 6, p. 904
Drugs such as mesalamine (5-ASA) are currently recommended for the treatment of inflammatory bowel disease (IBD). To reduce the frequency of their... 
Inflammatory bowel disease | Remission | Mucosal healing | Colitis | Hyaluronic acid | ACTIVATION | colitis | ANGIOGENESIS | INVOLVEMENT | CHEMISTRY, ORGANIC | INTERLEUKIN-6 | inflammatory bowel disease | hyaluronic acid | TELMISARTAN | DEGRADATION-PRODUCTS | remission | PPAR-GAMMA | EXPRESSION | CD44 | mucosal healing | TNBS-INDUCED COLITIS | Inflammatory Bowel Diseases - drug therapy | Intestinal Mucosa - metabolism | Cytokines - metabolism | Trinitrobenzenesulfonic Acid - administration & dosage | Rats | Hyaluronic Acid - pharmacology | Male | Inflammatory Bowel Diseases - metabolism | Intestinal Mucosa - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Trinitrobenzenesulfonic Acid - adverse effects | Mesalamine - pharmacology | Animals | Protective Agents - pharmacology | Inflammatory Bowel Diseases - pathology | Cyclooxygenase 2 - metabolism | Inflammation Mediators - metabolism | Inflammatory Bowel Diseases - chemically induced | Disease Models, Animal | Intestinal Mucosa - pathology | Peroxidase - metabolism | Inflammatory bowel diseases | Wound healing | Mucosa | Medical treatment | Prostaglandin endoperoxide synthase | Injury prevention | Acids | Intestine | Ulcers | Rodents | Animal tissues | Vascular tissue | Peroxidase | Combined treatment | Colon | Cyclooxygenase-2 | Injuries | Index Medicus
Journal Article
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 2011, Volume 82, Issue 7, pp. 737 - 745
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 08/2013, Volume 28, Issue 8, pp. 1330 - 1338
Journal Article
Journal Article
Pharmacological Research, ISSN 1043-6618, 09/2012, Volume 66, Issue 3, pp. 235 - 242
Dietary polyphenols present in (pomegranate), such as ellagitannins and ellagic acid (EA) have shown to exert anti-inflammatory and antioxidant properties.... 
COX-2 | TNF-α | TNBS-induced colitis | Pomegranate | Ellagic acid | TNF-alpha | PROTEIN-KINASE | ULCERATIVE-COLITIS | INDUCED COLITIS | BOWEL-DISEASE | MURINE MODEL | ACTIVATED-RECEPTOR-GAMMA | PHARMACOLOGY & PHARMACY | PUNICA-GRANATUM L | POLYPHENOL | NF-KAPPA-B | EXPRESSION | Intestinal Mucosa - metabolism | Rats, Wistar | Colon - drug effects | Polyphenols - pharmacology | Crohn Disease - metabolism | Colitis - pathology | Male | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Intestinal Mucosa - drug effects | Trinitrobenzenesulfonic Acid - adverse effects | MAP Kinase Kinase 4 - metabolism | Colitis - chemically induced | Colitis - drug therapy | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Disease Models, Animal | Punicaceae - chemistry | Ellagic Acid - pharmacology | Interferon-alpha - metabolism | NF-KappaB Inhibitor alpha | Anti-Inflammatory Agents - pharmacology | Colon - pathology | Rats | Colon - metabolism | Animals | Anti-Inflammatory Agents - chemistry | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Crohn Disease - pathology | Crohn Disease - drug therapy | Plant Extracts - pharmacokinetics | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Dietary Supplements | Intestinal Mucosa - pathology | Nitric Oxide Synthase Type II - metabolism | Peroxidase - metabolism | Nitrogen oxide | Gastrointestinal diseases | Nitric oxide | Dietary supplements | Sulfates | Phosphotransferases | Ulcerative colitis | Index Medicus | Animal models | Inflammatory bowel diseases | MAP kinase | Nitric-oxide synthase | Nuclear transport | Western blotting | Antioxidants | NF- Kappa B protein | c-Jun amino-terminal kinase | Polyphenols | Models | Colitis | Cyclooxygenase-2 | ellagic acid | Injuries
Journal Article