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1. Full Text Unique Genetic and Survival Characteristics of Invasive Mucinous Adenocarcinoma of the Lung
by Shim, Hyo Sup and Kenudson, Mari and Zheng, Zongli and Liebers, Matthew and Cha, Yoon Jin and Hoang Ho, Quan and Onozato, Maristela and Phi Le, Long and Heist, Rebecca S and Iafrate, A. John
Journal of Thoracic Oncology, ISSN 1556-0864, 08/2015, Volume 10, Issue 8, pp. 1156 - 1162
Invasive mucinous adenocarcinoma is a unique histologic subtype of lung cancer, and our knowledge of its genetic and clinical characteristics is rapidly... 
Adenocarcinoma | Mucinous | Mutation | Targeted therapy | Lung | Gene fusion | GEFITINIB | SENSITIVITY | RESPIRATORY SOCIETY CLASSIFICATION | REARRANGEMENTS | MUTATIONAL ANALYSIS | CANCER GENOMICS | INHIBITION | ONCOLOGY | RESPIRATORY SYSTEM | CARCINOMAS | INTERNATIONAL ASSOCIATION | FUSIONS | Receptor, Epidermal Growth Factor - genetics | Tropomyosin - genetics | Receptor, ErbB-2 - genetics | Adenocarcinoma, Mucinous - pathology | Lung Neoplasms - mortality | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Vesicle-Associated Membrane Protein 2 - genetics | Neuregulin-1 - genetics | Tumor Suppressor Protein p53 - genetics | DNA Mutational Analysis | Oncogene Fusion | Aged, 80 and over | Adult | Female | DNA, Neoplasm - analysis | Antigens, Differentiation, B-Lymphocyte - genetics | Lung Neoplasms - genetics | Neoplasm Invasiveness | Phosphatidylinositol 3-Kinases - genetics | Adenocarcinoma, Mucinous - mortality | Disease-Free Survival | Class I Phosphatidylinositol 3-Kinases | Oncogene Proteins, Fusion - genetics | Proto-Oncogene Proteins B-raf - genetics | Receptor, trkA - genetics | Neoplasm Recurrence, Local - genetics | Aged | Adenocarcinoma, Mucinous - genetics | Histocompatibility Antigens Class II - genetics | Index Medicus
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2. Full Text Recurrent BRAF Gene Fusions in a Subset of Pediatric Spindle Cell Sarcomas: Expanding the Genetic Spectrum of Tumors With Overlapping Features With Infantile Fibrosarcoma
by Kao, Yu-Chien and Fletcher, Christopher D.M and Alaggio, Rita and Wexler, Leonard and Zhang, Lei and Sung, Yun-Shao and Orhan, Dicle and Chang, Wei-Chin and Swanson, David and Dickson, Brendan C and Antonescu, Cristina R
The American Journal of Surgical Pathology, ISSN 0147-5185, 01/2018, Volume 42, Issue 1, pp. 28 - 38
Infantile fibrosarcomas (IFS) represent a distinct group of soft tissue tumors occurring in patients under 2 years of age and most commonly involving the... 
NTRK1 | BRAF | infantile fibrosarcoma | NTRK3 | fibrosarcoma | fusions | SURGERY | ACTIVATION | TISSUE | CONGENITAL MESOBLASTIC NEPHROMA | PATHOLOGY | THERAPY | RAF | Tropomyosin - genetics | Microtubule-Associated Proteins - genetics | Proto-Oncogene Proteins c-ets - genetics | Soft Tissue Neoplasms - diagnosis | Humans | Child, Preschool | Infant | Male | Fibrosarcoma - pathology | Receptor, trkC - genetics | Serine Endopeptidases - genetics | Cell Cycle Proteins - genetics | Female | Sarcoma - diagnosis | Sarcoma - genetics | Child | Infant, Newborn | Soft Tissue Neoplasms - genetics | Fibrosarcoma - diagnosis | Septins - genetics | Repressor Proteins - genetics | Gene Fusion | Sarcoma - pathology | Fibrosarcoma - genetics | Proto-Oncogene Proteins B-raf - genetics | Adolescent | Soft Tissue Neoplasms - pathology | Receptor, trkA - genetics | Biomarkers, Tumor - genetics | Index Medicus
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3. Full Text Diagnostic yield, interpretation, and clinical utility of mutation screening of sarcomere encoding genes in Danish hypertrophic cardiomyopathy patients and relatives
by Andersen, Paal Skytt and Havndrup, Ole and Hougs, Lotte and Sørensen, Karina M and Jensen, Morten and Larsen, Lars Allan and Hedley, Paula and Thomsen, Alex Rojas Bie and Moolman-Smook, Johanna and Christiansen, Michael and Bundgaard, Henning
Human Mutation, ISSN 1059-7794, 03/2009, Volume 30, Issue 3, pp. 363 - 370
The American Heart Association (ANA) recommends family screening for hypertrophic cardiomyopathy (HCM). We assessed the outcome of family screening combining... 
MYBPC3 | Cardiomyopathy | TNNC1 | TPM1 | ACTC | CSRP3 | HCM | TNNT2 | MYL3 | TCAP | TNNI3 | Hypertrophy | MYH7 | BINDING-PROTEIN-C | hypertrophy | MUSCLE LIM PROTEIN | MYOSIN HEAVY-CHAIN | MESSENGER-RNA | CARDIAC TROPONIN-T | FAMILIES | GENETICS & HEREDITY | MISSENSE MUTATION | SPECTRUM | cardiomyopathy | SEPTAL HYPERTROPHY | ALPHA-TROPOMYOSIN | Tropomyosin - genetics | Connectin | Genetic Testing | Myosin Light Chains - genetics | Humans | Middle Aged | Myosin Heavy Chains - genetics | Male | Actins - genetics | Sarcomeres - pathology | Young Adult | DNA Mutational Analysis | Troponin I - genetics | Adult | Female | Troponin T - genetics | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease - genetics | LIM Domain Proteins | Sarcomeres - metabolism | Cardiac Myosins - genetics | Cardiomyopathy, Hypertrophic - diagnosis | Troponin C - genetics | Muscle Proteins - genetics | Carrier Proteins - genetics | Denmark | Family | Aged | Mutation | Index Medicus
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4. Full Text Rare Functional Variant in TM2D3 is Associated with Late-Onset Alzheimer's Disease
by Jakobsdottir, Margret and Lee, Sven and Bis, Joshua and Chouraki, Vincent and Li-Kroeger, D and Yamamoto, S and Grove, Megan and Naj, Adam and Vronskaya, M and Salazar, J.L and DeStefano, Anita and Brody, Jennifer and Smith, Albert Vernon and Amin, Najaf and Sims, Rebecca and Ibrahim-Verbaas, Carla and Choi, Seung-Hoan and Satizabal, Claudia L and Lopez, Oscar L and Beiser, Alexa and Ikram, Kamran and Garcia, M and Hayward, Caroline and Varga, Tibor V and Ripatti, Samuli and Franks, Paul and Hallmans, Göran and Rolandsson, Olov and Jansson, J.-H and Porteous, David J and Salomaa, Veikko and Eiriksdottir, Gudny and Rice, Kenneth and Bellen, H.J and Levy, Daniel and Uitterlinden, Ané and Emilsson, Valur and Rotter, J.I and Aspelund, Thor and Ódonnell, Christopher and Fitzpatrick, Annette and Launer, Lenore and Hofman, Albert and Wang, Liang and Williams, Julie and Schellenberg, Gerard D and Boerwinkle, Eric and Psaty, Bruce and Seshai, Sudha and Shulman, Lee and Gudnason, Vilmundur and Duijn, Cornelia and Cohorts Heart Aging Res Genomic Ep and Genetic Environm Risk Alzheimer's and Cohorts for Heart and Aging Research in Genomic Epidemiology consortium and Alzheimer’s Disease Genetic Consortium and Genetic and Environmental Risk in Alzheimer’s Disease consortium and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Enheten för biobanksforskning and Allmänmedicin and Umeå universitet
PL o S Genetics (Online), ISSN 1553-7390, 10/2016, Volume 12, Issue 10, pp. e1006327 - e1006327
textabstractWe performed an exome-wide association analysis in 1393 late-onset Alzheimer’s disease (LOAD) cases and 8141 controls from the CHARGE consortium.... 
UNITED-STATES | HAPLOTYPE RECONSTRUCTION | COMMON VARIANTS | IDENTIFIES VARIANTS | PROTEIN | EARLY NEUROGENESIS | GENETICS & HEREDITY | MUTATIONS | DROSOPHILA-MELANOGASTER DEVELOPMENT | GENOME-WIDE ASSOCIATION | APOLIPOPROTEIN-E | Tropomyosin - genetics | Genome-Wide Association Study | Membrane Proteins - genetics | Genomics | Humans | European Continental Ancestry Group | Male | Receptors, Notch - genetics | Drosophila melanogaster - genetics | Iceland | Alzheimer Disease - pathology | Exome - genetics | Phenotype | Amyloid beta-Protein Precursor - genetics | Animals | Apolipoproteins E - genetics | Age of Onset | Alleles | Female | Aged | Drosophila Proteins - genetics | Mutation | Alzheimer Disease - genetics | Intracellular Signaling Peptides and Proteins - genetics | Genetic aspects | Genetic variation | Health aspects | Alzheimer's disease | Consortia | Amino acids | Analysis | Drosophila | Heart | Medical research | Neurosciences | Physicians | Colleges & universities | Epidemiology | Medicine | Neurology | Hospitals | Genetics | Clinical medicine | Alzheimers disease | Public health | Index Medicus | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
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5. Full Text A systematic screen for morphological abnormalities during fission yeast sexual reproduction identifies a mechanism of actin aster formation for cell fusion
by Dudin, Omaya and Merlini, Laura and Bendezú, Felipe O and Groux, Raphaël and Vincenzetti, Vincent and Martin, Sophie G
PLoS Genetics, ISSN 1553-7390, 04/2017, Volume 13, Issue 4, pp. e1006721 - e1006721
In non-motile fungi, sexual reproduction relies on strong morphogenetic changes in response to pheromone signaling. We report here on a systematic screen for... 
CYTOPLASMIC DYNEIN | CYTOKINESIS | TROPOMYOSIN FUNCTION | SPINDLE POLES | POLARITY | GENE | GROWTH | GENETICS & HEREDITY | MYOSIN-V | CAPPING PROTEIN | SCHIZOSACCHAROMYCES-POMBE | Sequence Deletion | Actin Cytoskeleton - metabolism | Myosin Type V - metabolism | Schizosaccharomyces pombe Proteins - genetics | Cytoskeleton - genetics | Reproduction - genetics | Amino Acid Sequence - genetics | Myosins - genetics | Actins - genetics | Schizosaccharomyces - genetics | Phenotype | Myosin Type V - genetics | Cell Cycle Proteins - genetics | Cytoskeleton - metabolism | Protein Binding | Actin Cytoskeleton - genetics | Physiological aspects | Genetic aspects | Research | Yeast fungi | Sexual reproduction | Actin | Polarization | Yeast | Tropomyosin | Secretion | Cell division | Pheromones | Cell fusion | Visual perception | Coalescing | Mating | Fungi | Signal transduction | Clonal deletion | Filaments | Sporulation | Morphology | Myosin | Cell adhesion | Localization | Index Medicus
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6. Full Text MicroRNA-21 targets tumor suppressor genes in invasion and metastasis
by Shuomin Zhu Hailong Wu Fangting Wu Daotai Nie Shijie Sheng Yin-Yuan Mo
细胞研究：英文版, ISSN 1001-0602, 2008, Volume 18, Issue 3, pp. 350 - 359
MicroRNAs （miRNAs） are a class of naturally occurring small non-coding RNAs that target protein-coding mRNAs at the post-transcriptional level. Our previous... 
miRNA | 细胞侵入 | MDA-MB-231 | 肿瘤发生 | Gene silencing | Cell invasion | Maspin | Mir-21 | MiRNA | PDCD4 | Post-transcriptional regulation | Tumorigenesis | Metastasis | post-transcriptional regulation | gene silencing | mir-21 | CELL-DEATH | CELL BIOLOGY | BREAST-CANCER | metastasis | MICRO-RNA | RT-PCR | PROSTATE-CANCER | GROWTH | TROPOMYOSIN-1 | cell invasion | tumorigenesis | maspin | EXPRESSION | Neoplasm Transplantation | RNA-Binding Proteins - genetics | Tropomyosin - genetics | Humans | Lung Neoplasms - metabolism | Male | Breast Neoplasms - metabolism | RNA, Messenger - biosynthesis | RNA-Binding Proteins - biosynthesis | Serpins - biosynthesis | Neoplasm Metastasis | Lung Neoplasms - secondary | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Female | Gene Expression Regulation, Neoplastic - genetics | Oncogenes | Apoptosis Regulatory Proteins - biosynthesis | Lung Neoplasms - genetics | Serpins - genetics | Neoplasm Invasiveness | MicroRNAs - biosynthesis | Tropomyosin - biosynthesis | Animals | Breast Neoplasms - genetics | Mice, Nude | Cell Line, Tumor | Mice | MicroRNAs - genetics | Tumor Suppressor Proteins - biosynthesis | Index Medicus
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7. Full Text Drosophila as a Model for the Identification of Genes causing Adult Human Heart Disease
by Matthew J. Wolf and Hubert Amrein and Joseph A. Izatt and Michael A. Choma and Mary C. Reedy and Howard A. Rockman
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2006, Volume 103, Issue 5, pp. 1394 - 1399
Drosophila melanogaster genetics provides the advantage of molecularly defined P-element insertions and deletions that span the entire genome. Although... 
Phenotypes | Dilated cardiomyopathy | Drosophila | Medical genetics | Cardiomyopathies | Adults | Cardiac output | Genetic mutation | Human genetics | Genetic screening | Optical coherence tomography | Cardiomyopathy | CARDIAC-FUNCTION | SMOOTH-MUSCLE | FLIGHT-MUSCLE | optical coherence tomography | MULTIDISCIPLINARY SCIENCES | MICE | MECHANISMS | SPECIFICATION | cardiomyopathy | MELANOGASTER | EXPRESSION | Tropomyosin - genetics | Cardiomyopathy, Dilated - pathology | Drosophila melanogaster - physiology | Heart - physiology | Male | Drosophila melanogaster - genetics | Time Factors | Tomography | Female | Sarcoplasmic Reticulum - metabolism | Transgenes | Disease Models, Animal | Myocardium - ultrastructure | Cardiomyopathy, Dilated - genetics | Wings, Animal | Microscopy, Electron | Homozygote | Point Mutation | Animals | Models, Biological | Models, Genetic | Drosophila Proteins - genetics | Mutation | DNA, Complementary - metabolism | Troponin I - metabolism | Heart Diseases - genetics | Crosses, Genetic | Heart | Analysis | Physiological aspects | Genetic aspects | Research | Diagnosis | Heart diseases | Optical tomography | Genetics | Cardiovascular disease | Insects | Comparative analysis | Measurement techniques | Index Medicus | Biological Sciences
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8. Full Text Genetics and Classifications
by Hall, Judith G and Kimber, Eva and van Bosse, Harold J P and Sahlgrenska akademin and Institute of Clinical Sciences, Section for the Health of Women and Children, Department of Pediatrics and Göteborgs universitet and Gothenburg University and Institutionen för kliniska vetenskaper, sektionen för kvinnors och barns hälsa, Avdelningen för pediatrik and Sahlgrenska Academy
Journal of Pediatric Orthopaedics, ISSN 0271-6798, 07/2017, Volume 37 Suppl 1, Issue 5, pp. S4 - S8
Arthrogryposis multiplex congenital (AMC) is a descriptive term for a group of conditions that all share the characteristic of congenital contractures. There... 
Amyoplasia | Arthrogryposis multiplex congenital | Arthrogryposis | Distal arthrogryposis | Congenital contracture syndromes | ETIOLOGY | CAUSE DISTAL ARTHROGRYPOSIS | MULTIPLE CONGENITAL CONTRACTURES | DIAGNOSTIC-APPROACH | arthrogryposis multiplex congenital | PREVALENCE | MYOPATHY | BETA-TROPOMYOSIN MUTATION | arthrogryposis | MYH3 | COMMON | PEDIATRICS | distal arthrogryposis | ORTHOPEDICS | congenital contracture syndromes | amyoplasia | Age Factors | Humans | Child, Preschool | Infant | Male | Arthrogryposis - classification | Arthrogryposis - genetics | Syndrome | Arthrogryposis - diagnosis | Arthrogryposis - therapy | Ultrasonography, Prenatal | Adult | Female | Index Medicus | Pediatrics | Pediatrik
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9. Full Text Prevalence and Distribution of Sarcomeric Gene Mutations in Japanese Patients With Familial Hypertrophic Cardiomyopathy
by Otsuka, Haruna and Arimura, Takuro and Abe, Tadaaki and Kawai, Hiroya and Aizawa, Yoshiyasu and Kubo, Toru and Kitaoka, Hiroaki and Nakamura, Hiroshi and Nakamura, Kazufumi and Okamoto, Hiroshi and Ichida, Fukiko and Ayusawa, Mamoru and Nunoda, Shinichi and Isobe, Mitsuaki and Matsuzaki, Masunori and Doi, Yoshinori L and Fukuda, Keiichi and Sasaoka, Taishi and Izumi, Toru and Ashizawa, Naoto and Kimura, Akinori
Circulation Journal, ISSN 1346-9843, 2012, Volume 76, Issue 2, pp. 453 - 461
Background: Hypertrophic cardiomyopathy (HCM), which is inherited as an autosomal dominant trait, is the most prevalent hereditary cardiac disease. Although... 
Hypertrophic cardiomyopathy | Genetics | Genes | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOLOGY | HEAVY-CHAIN GENE | PREVENTION | CLASSIFICATION | BINDING PROTEIN-C | ART. NO. E59 | CARDIAC TROPONIN-T | MISSENSE MUTATION | MOLECULAR-BASIS | ALPHA-TROPOMYOSIN | Geography | Troponin T - genetics | Japan - epidemiology | Tropomyosin - genetics | Myosin Light Chains - genetics | Prevalence | Cardiomyopathy, Hypertrophic, Familial - genetics | MAP Kinase Kinase Kinases - genetics | Cardiac Myosins - genetics | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Myosin Heavy Chains - genetics | Male | Cardiomyopathy, Hypertrophic, Familial - ethnology | Actins - genetics | Young Adult | Carrier Proteins - genetics | Pedigree | Sarcomeres - genetics | Adult | Female | Aged | Asian Continental Ancestry Group - statistics & numerical data | Index Medicus
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10. Full Text Hypertonic stress induces rapid and widespread protein damage in C. elegans
by Burkewitz, Kris and Choe, Keith and Strange, Kevin
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2011, Volume 301, Issue 3, pp. C566 - C576
Burkewitz K, Choe K, Strange K. Hypertonic stress induces rapid and widespread protein damage in C. elegans. Am J Physiol Cell Physiol 301: C566-C576, 2011.... 
Macromolecular crowding | Caenorhabditis elegans | Protein aggregation | Organic osmolytes | Polyglutamine | Proteostasis | Cell volume | Kidney | cell volume | TREHALOSE | HOMEOSTASIS | PHYSIOLOGY | OSMOTIC-STRESS | INSULIN-SIGNALING PATHWAY | proteostasis | polyglutamine | CELL BIOLOGY | kidney | SHOCK | CAENORHABDITIS-ELEGANS | CHAPERONES | macromolecular crowding | protein aggregation | POLYGLUTAMINE DISEASES | MOUSE MODEL | AGGREGATION | organic osmolytes | Protein Denaturation - drug effects | Protein Kinases - genetics | RNA, Double-Stranded - administration & dosage | Muscle Cells - drug effects | Peptides - genetics | Cell Nucleus - metabolism | Peptides - metabolism | Stress, Physiological - drug effects | alpha-Synuclein - genetics | Inclusion Bodies - metabolism | Caenorhabditis elegans - metabolism | Muscle Cells - pathology | Membrane Proteins - genetics | Proteoglycans - metabolism | Saline Solution, Hypertonic - pharmacology | Particle Size | Insulin - metabolism | Signal Transduction - drug effects | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Pharynx - drug effects | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Proteoglycans - genetics | Protein Kinases - metabolism | ras Proteins - genetics | Temperature | Transcription Factors - deficiency | Caenorhabditis elegans Proteins - metabolism | ras Proteins - metabolism | Recombinant Fusion Proteins - metabolism | Protein Folding - drug effects | Receptor, Insulin - genetics | Membrane Proteins - metabolism | Tropomyosin - metabolism | Acclimatization - physiology | Movement - drug effects | Animals, Genetically Modified | Bacterial Proteins - genetics | Muscle Cells - metabolism | Genes, Reporter - genetics | Pharynx - metabolism | Transcription Factors - genetics | Receptor, Insulin - antagonists & inhibitors | Animals | Caenorhabditis elegans - drug effects | Caenorhabditis elegans Proteins - antagonists & inhibitors | RNA, Double-Stranded - pharmacology | Bacterial Proteins - metabolism | Myofibrils - metabolism | alpha-Synuclein - metabolism | Luminescent Proteins - metabolism | Cellular proteins | Genetic aspects | Stress (Physiology) | Properties | Health aspects | Hypertonic solutions | Proteins | Survival analysis | Mutation | Gene expression | Cells | Index Medicus | Membrane Transporters, Ion Channels and Pumps
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11. Full Text Actin Dynamics Affect Mitochondrial Quality Control and Aging in Budding Yeast
by Higuchi, Ryo and Vevea, Jason D and Swayne, Theresa C and Chojnowski, Robert and Hill, Vanessa and Boldogh, Istvan R and Pon, Liza A
Current Biology, ISSN 0960-9822, 12/2013, Volume 23, Issue 23, pp. 2417 - 2422
Actin cables of budding yeast are bundles of F-actin that extend from the bud tip or neck to the mother cell tip, serve as tracks for bidirectional cargo... 
LIFE-SPAN | CELLS | MYOSIN | MOTHER | BIOCHEMISTRY & MOLECULAR BIOLOGY | AGE ASYMMETRY | 2 DIFFERENT MECHANISMS | INHERITANCE | SACCHAROMYCES-CEREVISIAE | RETROGRADE FLOW | MOTILITY | CELL BIOLOGY | Tropomyosin - genetics | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Saccharomyces cerevisiae - genetics | Asymmetric Cell Division | Cell Survival - genetics | Myosin Heavy Chains - genetics | Mitochondria - pathology | Saccharomyces cerevisiae Proteins - genetics | Cytokinesis | Silent Information Regulator Proteins, Saccharomyces cerevisiae - genetics | Actins - genetics | Cell Lineage | Silent Information Regulator Proteins, Saccharomyces cerevisiae - biosynthesis | Sirtuin 2 - genetics | Aging - genetics | Biological Transport | Gene Deletion | Sirtuin 2 - biosynthesis | Mitochondria - physiology | Actin | Analysis | Myosin | Quality control | Polymerization | Mitochondrial DNA | Muscle proteins | Cells | Index Medicus
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12. Full Text Recurrent NTRK1 Gene Fusions Define a Novel Subset of Locally Aggressive Lipofibromatosis-like Neural Tumors
by Agaram, Narasimhan P and Zhang, Lei and Sung, Yun-Shao and Chen, Chun-Liang and Chung, Catherine T and Antonescu, Cristina R and Fletcher, Christopher DM
The American Journal of Surgical Pathology, ISSN 0147-5185, 10/2016, Volume 40, Issue 10, pp. 1407 - 1416
The family of pediatric fibroblastic and myofibroblastic proliferations encompasses a wide spectrum of pathologic entities with overlapping morphologies and... 
NTRK1 | Lipofibromatosis | TPM3 | LMNA | TPR | Neural | SURGERY | neural | CONGENITAL INSENSITIVITY | TISSUE | TRK PROTOONCOGENE PRODUCT | PATHOLOGY | REARRANGEMENTS | CANCER | DISCOVERY | NERVE GROWTH-FACTOR | ONCOGENE | MODULAR FRAMEWORK | MUTATION | lipofibromatosis | Neoplasms, Complex and Mixed - genetics | Tropomyosin - genetics | Follow-Up Studies | Humans | Lipoma - genetics | Child, Preschool | Male | Lipoma - pathology | Nuclear Pore Complex Proteins - genetics | Young Adult | Adult | Female | Child | Soft Tissue Neoplasms - genetics | Gene Fusion | In Situ Hybridization, Fluorescence | Proto-Oncogene Proteins - genetics | Fibroma - genetics | Reverse Transcriptase Polymerase Chain Reaction | Fibroma - pathology | Adolescent | Neoplasms, Complex and Mixed - pathology | Soft Tissue Neoplasms - pathology | Receptor, trkA - genetics | Biomarkers, Tumor - genetics | Index Medicus
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