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Forensic Science International, ISSN 0379-0738, 2012, Volume 226, Issue 1, pp. 54 - 61
Abstract The aim of this study was to investigate the usefulness of postmortem biochemical investigations in the diagnosis of fatal hypothermia. 10 cases of... 
Pathology | Ketone bodies | Postmortem biochemistry | Blood | Hypothermia | Biochemical markers | C-REACTIVE PROTEIN | MEDICINE, LEGAL | FATTY DEGENERATION | ACCIDENTAL HYPOTHERMIA | MEDICOLEGAL AUTOPSY | CREATINE-KINASE MB | CORTICOTROPIN-RELEASING FACTOR | POSTMORTEM CHEMISTRY UPDATE | PERICARDIAL FLUID | CARDIAC TROPONIN-I | ACUTE COLD STRESS | Calcium - metabolism | Humans | Middle Aged | Hydrocortisone - metabolism | Male | Pericardium - metabolism | Case-Control Studies | Young Adult | Fatty Acids, Nonesterified - metabolism | 2-Propanol - metabolism | C-Reactive Protein - metabolism | Human Growth Hormone - metabolism | Vitreous Body - metabolism | Hypothermia - metabolism | Hypothermia - mortality | Adult | Female | Forensic Pathology | Hypothermia - diagnosis | Calcitonin - metabolism | Adrenocorticotropic Hormone - metabolism | Biomarkers - metabolism | Calcitonin Gene-Related Peptide | Acetone - metabolism | Magnesium - metabolism | Thyrotropin - metabolism | Protein Precursors - metabolism | Glucose - metabolism | Aged | Hydroxybutyrates - metabolism | Ketones - metabolism | Postmortem Changes | Epinephrine - metabolism | Diagnosis | Biological markers | Analysis | Legal medicine | Fluid | Histology | Forensic pathology | Glucose | Nitrogen | Studies | Rodents | Pituitary gland | Fatalities | Growth hormones | Females | Age
Journal Article
Journal Article
International Journal of Legal Medicine, ISSN 0937-9827, 3/2013, Volume 127, Issue 2, pp. 267 - 276
Journal Article
Cardiovascular Research, ISSN 0008-6363, 8/2011, Volume 91, Issue 3, pp. 402 - 411
Journal Article
Differentiation, ISSN 0301-4681, 03/2012, Volume 83, Issue 3, pp. 106 - 115
Journal Article
FEBS Letters, ISSN 0014-5793, 05/2014, Volume 588, Issue 10, pp. 1935 - 1941
Androgen reduces fat mass, although the underlying mechanisms are unknown. Here, we examined the effect of testosterone on heat production and mitochondrial... 
Testosterone | Mitochondrial biogenesis | Skeletal muscle | Androgen receptor deficient mice | ANDROGEN RECEPTOR | ACTIVATED RECEPTOR-GAMMA | ADIPOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INSULIN SENSITIVITY | CELL BIOLOGY | OBESITY | BIOPHYSICS | MASS | MEN | LATE-ONSET | HYPOGONADISM | EXPRESSION | Myoglobin - metabolism | Troponin - genetics | Mitochondria, Muscle - metabolism | Myoglobin - genetics | Body Weight - drug effects | Male | Muscle, Skeletal - metabolism | Receptors, Androgen - deficiency | Cytochromes c - genetics | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Weight Loss - drug effects | Muscle, Skeletal - cytology | Testosterone - pharmacology | Nuclear Respiratory Factor 1 - genetics | Testosterone - metabolism | DNA, Mitochondrial - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Nuclear Respiratory Factor 1 - metabolism | Mitochondria, Muscle - genetics | Troponin - metabolism | Cell Line | DNA, Mitochondrial - metabolism | Cytochromes c - metabolism | Mice, Inbred C57BL | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Transcription Factors - metabolism | Eating - drug effects | Animals | Receptors, Androgen - genetics | Androgens - pharmacology | Muscle Fibers, Skeletal - cytology | Mice | Androgens - metabolism | Blood Glucose - metabolism | Energy Metabolism - drug effects | Muscles | Biosynthesis | Body weight | Weight loss
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e108024
The protective role of etanercept in myocardial ischemia/reperfusion is not well understood. The aim of this study was to investigate whether etanercept... 
ISCHEMIA-REPERFUSION INJURY | APOPTOSIS | TUMOR-NECROSIS-FACTOR | INFARCTION | MULTIDISCIPLINARY SCIENCES | MONOCLONAL-ANTIBODY | CHEMOKINE | INHIBITOR | ARTHRITIS | DAMAGE | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Lactate Dehydrogenases - metabolism | Apoptosis - drug effects | Creatine Kinase, MB Form - metabolism | Tumor Necrosis Factor-alpha - blood | Ventricular Dysfunction - drug therapy | Male | Troponin I - blood | Inflammation - metabolism | Myocardial Reperfusion Injury - pathology | Etanercept | Inflammation - drug therapy | Myocardial Infarction - pathology | Immunoglobulin G - pharmacology | Myocardial Reperfusion Injury - drug therapy | Myocardial Reperfusion Injury - genetics | Neutrophil Infiltration - drug effects | Superoxide Dismutase - metabolism | Disease Models, Animal | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Rats | Toll-Like Receptor 4 - genetics | Receptors, Tumor Necrosis Factor - administration & dosage | Myocardial Infarction - metabolism | Immunoglobulin G - administration & dosage | Toll-Like Receptor 4 - metabolism | Myocardial Reperfusion Injury - physiopathology | Gene Expression Regulation - drug effects | Myocardial Reperfusion Injury - metabolism | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Creatine Kinase, MB Form - blood | Myocytes, Cardiac - metabolism | Oxidative Stress - drug effects | Troponin I - metabolism | Myocardial infarction | Bioengineering | Heart | Oxidative stress | Critical care | Superoxide dismutase | Myocardial ischemia | Arthritis | Antioxidants | Reperfusion | Ischemia | Rodents | Tumor necrosis factor-TNF | Peroxidase | Injuries | Glutathione | Creatinine | Glutathione peroxidase | Psoriasis | Cytokines | Neutrophils | Cardiomyocytes | Lactate dehydrogenase | Inflammation | Tumor necrosis factor-α | Gene expression | Malondialdehyde | L-Lactate dehydrogenase | Digital imaging | TNF inhibitors | Infarction | Lactic acid | Laboratory animals | Apoptosis
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1310 - 1322
Coagulation is a host defense system that limits the spread of pathogens. Coagulation proteases, such as thrombin, also activate cells by cleaving PARs. In... 
TOLL-LIKE RECEPTOR-3 | MEDICINE, RESEARCH & EXPERIMENTAL | NATURAL-KILLER-CELLS | ISCHEMIA/REPERFUSION INJURY | PROTEASE-ACTIVATED RECEPTORS | VIRUS-INFECTION | IN-VIVO | ENDOTHELIAL-CELLS | FACTOR MESSENGER-RNA | INTERFERON-BETA | TISSUE FACTOR EXPRESSION | Receptor, PAR-1 - metabolism | Humans | Male | Troponin I - blood | STAT1 Transcription Factor - metabolism | Myocardium - metabolism | HEK293 Cells | Interferon-beta - genetics | Influenza A virus - immunology | Myocarditis - immunology | Influenza A virus - physiology | Orthomyxoviridae Infections - metabolism | Coxsackievirus Infections - metabolism | Liver - metabolism | Mice, Inbred C57BL | Enterovirus - physiology | Myocarditis - blood | Immunity, Innate | STAT1 Transcription Factor - genetics | Toll-Like Receptor 3 - metabolism | Receptor, PAR-1 - genetics | Mice, Knockout | Myocarditis - virology | Animals | Interferon-beta - metabolism | Thromboplastin - genetics | Enterovirus - immunology | Mice | HeLa Cells | Thrombin - metabolism | Thromboplastin - metabolism | Fibrin - metabolism | Chemokine CXCL10 - metabolism | Coxsackievirus Infections - immunology | Orthomyxoviridae Infections - immunology | Receptor, PAR-1 - physiology | Virus diseases | Cell receptors | Immune response | Physiological aspects | Development and progression | Genetic aspects | Research
Journal Article