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Circulation, ISSN 0009-7322, 08/2017, Volume 136, Issue 9, pp. 834 - 848
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 04/2011, Volume 589, Issue 7, pp. 1725 - 1740
Non‐technical summary  Following a myocardial infarction, cardiac muscle becomes irreversibly damaged and over time this may lead to heart failure. Strategies... 
POLY(ADP-RIBOSE) POLYMERASE | HEPATOCELLULAR-CARCINOMA | PHYSIOLOGY | ENDOTHELIAL-CELL PROLIFERATION | EPH RECEPTORS | HEART-FAILURE | GROWTH-FACTOR | CARDIAC TROPONIN-I | NF-KAPPA-B | STIMULATES MIGRATION | NEUROSCIENCES | RECEPTOR TYROSINE KINASE | Neovascularization, Physiologic - drug effects | Male | Mice, 129 Strain | Immunoglobulin Fc Fragments - administration & dosage | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Tissue Distribution | Myocardial Infarction - pathology | Myocardium - metabolism | Receptors, Eph Family - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Recombinant Fusion Proteins - administration & dosage | Disease Models, Animal | Ephrin-A1 - administration & dosage | Gene Expression | Receptors, Eph Family - genetics | RNA, Messenger - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Ephrin-A1 - genetics | Transcription Factors - metabolism | Poly(ADP-ribose) Polymerases - metabolism | Animals | Myocardial Infarction - drug therapy | Ephrin-A1 - pharmacokinetics | Transcription Factor RelA - metabolism | Recombinant Fusion Proteins - pharmacokinetics | Recombinant Fusion Proteins - genetics | Mice | Poly (ADP-Ribose) Polymerase-1 | Immunoglobulin Fc Fragments - genetics | Troponin I - metabolism | Heart failure | Tyrosine | Physiological aspects | Gene expression | Heart attack | Cardiovascular
Journal Article
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2010, Volume 50, Issue 3, pp. 487 - 499
Journal Article
Cardiovascular Research, ISSN 0008-6363, 05/2018, Volume 114, Issue 6, pp. 830 - 845
Abstract Aims Phosphodiesterase 2 A (Pde2A), a cAMP-hydrolysing enzyme, is essential for mouse development; however, the cause of Pde2A knockout embryonic... 
Tbx genes | Icer | Pde2A | CHD | CARDIAC & CARDIOVASCULAR SYSTEMS | PULMONARY ARTERIAL-HYPERTENSION | CONDUCTION SYSTEM-DEVELOPMENT | OUTFLOW TRACT | T-BOX GENE | CARDIAC CHAMBERS | MICE LACKING | TBX2 | DISEASE | REPRESSOR | EXPRESSION | MEF2 Transcription Factors - genetics | Cell Proliferation | LIM-Homeodomain Proteins - metabolism | Cyclic AMP Response Element Modulator - metabolism | Transcription Factor AP-2 - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 2 - genetics | Morphogenesis | Myocytes, Cardiac - enzymology | Heart Defects, Congenital - genetics | Gene Expression Regulation, Developmental | Troponin I - genetics | Heart Defects, Congenital - enzymology | Cell Differentiation | Fetal Heart - abnormalities | Cyclic AMP - metabolism | Genetic Predisposition to Disease | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Heart Defects, Congenital - pathology | Fetal Heart - enzymology | Transcription Factors - genetics | Gestational Age | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Mice, Knockout | Cyclic Nucleotide Phosphodiesterases, Type 2 - deficiency | Transcription Factors - metabolism | Myocytes, Cardiac - pathology | LIM-Homeodomain Proteins - genetics | Phenotype | Transcription Factor AP-2 - genetics | Animals | Cyclic AMP Response Element Modulator - genetics | MEF2 Transcription Factors - metabolism | Troponin I - metabolism | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2014, Volume 111, Issue 8, pp. 3182 - 3187
Previous studies have demonstrated that hydrogen sulfide (H2S) protects against multiple cardiovascular disease states in a similar manner as nitric oxide... 
Heart | Sulfides | Oxidative stress | Phosphorylation | Hydrogen | Nitrites | Liver | Oxides | Physical trauma | Bioavailability | Myocardial infarction | Cystathionase | Nitrite | Cth | eNOS uncoupling | INDUCED HEART-FAILURE | MOUSE HEART | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | myocardial infarction | cystathionase | H2S | AKT | PROTECTS | ISCHEMIA-REPERFUSION INJURY | CYTOCHROME-C-OXIDASE | nitrite | MYOCARDIAL ISCHEMIA | IN-VIVO | Hydrogen Sulfide - metabolism | Immunohistochemistry | Hydrogen Sulfide - pharmacology | Oxidative Stress - physiology | Alanine Transaminase - blood | Chromatography, High Pressure Liquid | Cytoprotection - physiology | Oxygen Consumption - physiology | Blotting, Western | Mice, Knockout | Myocardial Reperfusion Injury - metabolism | Animals | Analysis of Variance | Aspartate Aminotransferases - blood | Cytoprotection - drug effects | Signal Transduction - physiology | Mice | Cystathionine gamma-Lyase - genetics | Myocardial Reperfusion Injury - drug therapy | Nitric Oxide Synthase Type III - metabolism | Mitochondria - physiology | Nitric Oxide - metabolism | Troponin I - metabolism | Hydrogen sulfide | Prevention | Cell research | Nitric oxide | Physiological aspects | Cell physiology | Research | Cardiovascular diseases | Enzymes | Signal transduction | Cardiovascular disease | Rodents | Biological Sciences
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 05/2015, Volume 285, Issue 1, pp. 51 - 60
Safety pharmacology studies that evaluate new drug entities for potential cardiac liability remain a critical component of drug development. Current studies... 
Cardiomyocytes | Preclinical | Cardiotoxicity | Drug evaluation | In vitro | Stem cells | RISK | MECHANISMS | TOXICITY | PROLONGATION | PREDICTION | PATHOGENESIS | HEART | INHIBITION | THERAPY | SUNITINIB | PHARMACOLOGY & PHARMACY | TOXICOLOGY | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Humans | Structure-Activity Relationship | Dose-Response Relationship, Drug | Time Factors | Toxicity Tests - methods | Antineoplastic Agents - pharmacology | Molecular Structure | Antineoplastic Agents - classification | Heart Diseases - chemically induced | Induced Pluripotent Stem Cells - metabolism | Biomarkers - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Survival - drug effects | Reproducibility of Results | Induced Pluripotent Stem Cells - drug effects | Risk Assessment | Cells, Cultured | Myocytes, Cardiac - pathology | High-Throughput Screening Assays | Myocytes, Cardiac - drug effects | Lipid Metabolism - drug effects | Myocytes, Cardiac - metabolism | Oxidative Stress - drug effects | Troponin I - metabolism | Heart Diseases - pathology | Drugs | Heart cells | Index Medicus | HUMAN POPULATIONS | SAFETY | VIABILITY | LIABILITIES | 60 APPLIED LIFE SCIENCES | OXYGEN | TESTING | RISK ASSESSMENT | IN VITRO | PHARMACOLOGY | DRUGS | SCREENING | STEM CELLS | SECRETION | DEPTH | HAZARDS | LIPIDS
Journal Article
Circulation: Heart Failure, ISSN 1941-3289, 09/2009, Volume 2, Issue 5, pp. 472 - 481
Journal Article