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Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 22, pp. 13875 - 13887
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e48897
Background: Interstitial cells of Cajal (ICC) have been identified in urinary bladder of several species, but their presence in mice remains uncertain.... 
MERLIN | PROGENITORS | GAP-JUNCTIONS | KIT-POSITIVE CELLS | URINARY-BLADDER | MULTIDISCIPLINARY SCIENCES | SMOOTH-MUSCLE-CELLS | GUINEA-PIG | IDENTIFICATION | GASTROINTESTINAL STROMAL TUMORS | CONNEXIN EXPRESSION | Tryptases - metabolism | Anoctamin-1 | Antigens, CD34 - analysis | Antigens, CD34 - metabolism | Vimentin - metabolism | Neurons - cytology | Mesoderm - cytology | Interstitial Cells of Cajal - cytology | Myofibroblasts - metabolism | Interstitial Cells of Cajal - metabolism | Desmin - analysis | Adenosine Triphosphatases - analysis | Urinary Bladder - cytology | Neurons - metabolism | Neurofibromin 2 - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Biomarkers - metabolism | Connexin 43 - analysis | Receptor, Platelet-Derived Growth Factor beta - analysis | Chloride Channels - analysis | Neurofibromin 2 - analysis | Connexin 43 - metabolism | Urinary Bladder - metabolism | Mice, Inbred C57BL | Muscle Cells - metabolism | Adenosine Triphosphatases - metabolism | Tryptases - analysis | Muscle Cells - cytology | Chloride Channels - metabolism | Myofibroblasts - cytology | Vimentin - analysis | Animals | Desmin - metabolism | Mesoderm - metabolism | Mice | Intermediate filament proteins | Platelet-derived growth factor | Stem cells | Vimentin | Mesenchyme | Laboratories | Neurofibromin 2 | Bladder | Interstitial cells | Neurofibromin 1 | Smooth muscle | Connexin 43 | Kinases | Urinary bladder | Rodents | Fibroblasts | Colon | Localization | CD34 antigen | Markers | Muscles | Interstitial cells of Cajal | c-Kit protein | Desmin | Signaling | Tryptase | Mutation | Position (location) | Neurotransmission
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 4, pp. 1425 - 1434
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 07/2011, Volume 184, Issue 1, pp. 116 - 123
Journal Article
Journal Article
Journal of Immunological Methods, ISSN 0022-1759, 10/2012, Volume 384, Issue 1-2, pp. 25 - 32
Nasal allergen challenge can be used to assess the clinical and immunological aspects of rhinitis due to inhalant allergens. We aimed to develop a reproducible... 
Grass pollen | Nasal cytokines | Biomarker | Nasal allergen challenge | Seasonal allergic rhinitis | Nasal allergen provocation | CELLS | COLONY-STIMULATING FACTOR | PROVOCATION | ANTIGEN CHALLENGE | BIOCHEMICAL RESEARCH METHODS | CYTOKINE LEVELS | IL-5 | IMMUNOLOGY | RHINITIS | SECRETIONS | IMMUNOTHERAPY | INFLAMMATORY MEDIATORS | Tryptases - metabolism | Pollen - metabolism | Rhinitis, Allergic, Seasonal - metabolism | Humans | Middle Aged | Interleukin-13 - immunology | Male | Interferon-gamma - metabolism | Pollen - immunology | Rhinitis, Allergic, Seasonal - diagnosis | Eosinophil Cationic Protein - immunology | Rhinitis, Allergic, Seasonal - immunology | Young Adult | Allergens - immunology | Time Factors | Sensitivity and Specificity | Hypersensitivity, Immediate - immunology | Interleukin-10 - metabolism | Adult | Female | Hypersensitivity, Immediate - diagnosis | Tryptases - immunology | Phleum - metabolism | Nasal Provocation Tests - methods | Reproducibility of Results | Interleukin-5 - metabolism | Interleukin-4 - metabolism | Eosinophil Cationic Protein - metabolism | Interleukin-13 - metabolism | Phleum - immunology | Allergens - metabolism | Interleukin-5 - immunology | Interleukin-4 - immunology | Hypersensitivity, Immediate - metabolism | Interferon-gamma - immunology | Interleukin-10 - immunology | Allergy | Allergens | Allergic reaction
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 03/2010, Volume 298, Issue 3, pp. G352 - G363
Smith F, Clark JE, Overman BL, Tozel CC, Huang JH, Rivier JE, Blisklager AT, Moeser AJ. Early weaning stress impairs development of mucosal barrier function in... 
Weaning age | Mast cells | Corticotropin releasing factor | GASTROINTESTINAL PERMEABILITY | SIGNALING PATHWAYS | PHYSIOLOGY | ION-TRANSPORT | POTASSIUM CHANNELS | mast cells | NIPPOSTRONGYLUS-BRASILIENSIS | weaning age | CORTICOTROPIN-RELEASING-FACTOR | NEONATAL MATERNAL SEPARATION | corticotropin releasing factor | GASTROENTEROLOGY & HEPATOLOGY | RECTAL DISTENSION | IRRITABLE-BOWEL-SYNDROME | MAST-CELL TRYPTASE | Potassium - metabolism | Tryptases - metabolism | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Jejunum - pathology | Receptors, Corticotropin-Releasing Hormone - metabolism | Weaning | Cell Count | Male | Receptors, Glucocorticoid - metabolism | Intestinal Mucosa - drug effects | Jejunum - metabolism | Jejunum - drug effects | Protease Inhibitors - pharmacology | Cromolyn Sodium - pharmacology | Jejunum - physiopathology | Mannitol - metabolism | Mast Cells - metabolism | Female | Hydrocortisone - blood | Potassium Channel Blockers - pharmacology | Animals, Newborn | Stress, Physiological - physiology | Chlorides - pharmacology | Electrophysiological Phenomena - physiology | Corticotropin-Releasing Hormone - blood | Electric Impedance | Permeability | Mast Cells - drug effects | Corticotropin-Releasing Hormone - metabolism | Barium Compounds - pharmacology | Corticotropin-Releasing Hormone - pharmacology | Animals | Receptors, Corticotropin-Releasing Hormone - antagonists & inhibitors | Mast Cells - pathology | Inulin - metabolism | Ion Transport - physiology | Sus scrofa | Electrophysiological Phenomena - drug effects | Intestinal Mucosa - pathology | Hormone receptors | Gastrointestinal diseases | Colorectal diseases | Physiological aspects | Research | Stress (Psychology) | Risk factors | Mucosal Biology
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 10/2016, Volume 434, Issue C, pp. 69 - 80
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2004, Volume 24, Issue 18, pp. 4300 - 4312
Inflammatory proteases (mast cell tryptase and trypsins) cleave protease-activated receptor 2 (PAR(2)) on spinal afferent neurons and cause persistent... 
Protein kinase C | Inflammation | TRPV1 | Substance P | Hyperalgesia | Protease-activated receptors | protease-activated receptors | THERMAL HYPERALGESIA | ROOT GANGLION NEURONS | PAIN PATHWAY | RAT SENSORY NEURONS | NEUROSCIENCES | inflammation | SIGNALING PATHWAY | KINASE-C ACTIVATION | MYENTERIC NEURONS | MOLECULAR-CLONING | hyperalgesia | substance P | THROMBIN RECEPTOR | protein kinase C | MAST-CELL TRYPTASE | Enzyme Activators - pharmacology | Hyperalgesia - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Receptors, Drug - genetics | Humans | Receptor, PAR-2 - metabolism | Type C Phospholipases - metabolism | Male | Neurons - cytology | RNA, Messenger - metabolism | Receptors, Drug - agonists | Type C Phospholipases - antagonists & inhibitors | Kidney - metabolism | Protein Kinase C - metabolism | Neurons - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neurons - drug effects | Receptor, PAR-2 - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Kidney - cytology | Protein Kinase C - antagonists & inhibitors | Rats, Sprague-Dawley | Receptors, Drug - metabolism | Mice, Knockout | Animals | Receptor, PAR-2 - agonists | Substance P - metabolism | Mice | Cellular | Molecular
Journal Article
2007, Advances in Immunology, ISBN 9780123737083, Volume 95, 89
Book Chapter
Respiratory Research, ISSN 1465-9921, 12/2011, Volume 12, Issue 1, pp. 1 - 16
Many studies have found that smoking reduces lung function, but the relationship between cigarette smoke and allergic asthma has not been clearly elucidated,... 
Medicine & Public Health | Pneumology/Respiratory System | GROWTH-FACTOR-BETA | ACTIVATION | TGF-BETA | INDUCED AIRWAY INFLAMMATION | SIGNALING PATHWAY | SENSITIZATION | RESPIRATORY SYSTEM | MAP KINASE | MICE | KAPPA-B | EXPOSURE | Immunohistochemistry | Tryptases - metabolism | Asthma - metabolism | Phosphorylation | Bronchial Hyperreactivity - immunology | Smoking - immunology | Bronchial Provocation Tests | Bronchoalveolar Lavage Fluid - immunology | NF-kappa B - metabolism | Smad3 Protein - metabolism | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | Antigen-Antibody Reactions | Bronchoconstriction | Plasminogen Activator Inhibitor 1 - metabolism | RNA Interference | Time Factors | Bronchial Hyperreactivity - physiopathology | Asthma - immunology | Mast Cells - metabolism | Bronchial Hyperreactivity - metabolism | Female | Lung - metabolism | Disease Models, Animal | Mast Cells - immunology | Ovalbumin | Asthma - physiopathology | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Signal Transduction | Immunoglobulin E - metabolism | Plethysmography, Whole Body | Cells, Cultured | Smad2 Protein - metabolism | Smad Proteins - genetics | Lung - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Asthma - genetics | Blotting, Western | Collagen - metabolism | Animals | Mice | Mice, Inbred BALB C | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Lung - immunology | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Smad proteins | Influence | Mast cells | Genetic aspects | Research | Health aspects | Risk factors | Asthma | Smoking | Antigens | RNA | Collagen | Immunoglobulin E | Bone morphogenetic proteins | Transforming growth factors | Enzyme-linked immunosorbent assay
Journal Article