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Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23524
Aberrant activation of the Wnt/beta-catenin pathway is critical for the initiation and progression of most colon cancers. This activation provokes the... 
STEM-CELLS | APC | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GROWTH | PHENOTYPE | E-CADHERIN | DIFFERENTIATION | BETA-CATENIN | WNT | EXPRESSION | Steroid Hydroxylases - metabolism | Colonic Neoplasms - genetics | Humans | Protein Transport - drug effects | Wnt Proteins - metabolism | Receptors, Calcitriol - genetics | Colonic Neoplasms - metabolism | Cell Nucleus - metabolism | RNA Interference | Cell Membrane - metabolism | Intestinal Neoplasms - genetics | Steroid Hydroxylases - genetics | Adenomatous Polyposis Coli Protein - genetics | Receptors, Calcitriol - deficiency | Signal Transduction | HCT116 Cells | Intestinal Neoplasms - metabolism | Vitamin D3 24-Hydroxylase | Tumor Burden | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | Intestinal Neoplasms - pathology | Mice, Knockout | Gene Expression Regulation - drug effects | HT29 Cells | Animals | Calcitriol - pharmacology | Colonic Neoplasms - pathology | Cell Line, Tumor | Mice | Mutation | Phosphorylation | Transcription factors | Carcinoma | Wnt protein | Laboratories | Genes | Colorectal cancer | Calcitriol | Oncology | Genomes | Activation | Kinases | Small intestine | Nuclei | Accumulation | Proteins | Colon cancer | Vitamin D | Colon | Relocation | Phenotypes | Nutrient deficiency | Baldness | Breeding | Gene expression | Adenomatous polyposis coli | Signaling | Alleles | Stem cells | Ligands | β-Catenin | Vitamin D receptors | Aberration | Tumors | Cancer
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2018, Volume 378, Issue 22, pp. 2093 - 2104
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2017, Volume 377, Issue 25, pp. 2500 - 2501
Journal Article
Journal Article
Journal Article
Journal Article
Cell stem cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 603 - 615
Recent evidence suggests that a subpopulation of cancer cells, cancer stem cells (CSCs), is responsible for tumor growth in colorectal cancer. However,... 
STEMCELL | CELLCYCLE | BREAST-CANCER | INITIATING CELLS | DIPEPTIDYL-PEPTIDASE-IV | CARCINOMA CELLS | EXTRACELLULAR-MATRIX | ACUTE MYELOID-LEUKEMIA | E-CADHERIN | IDENTIFICATION | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Gene Expression Profiling | Colorectal Neoplasms - diagnosis | Organoplatinum Compounds - pharmacology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Colorectal Neoplasms - drug therapy | Liver Neoplasms - physiopathology | Neoplastic Stem Cells - pathology | Tumor Burden - genetics | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Carcinoma - drug therapy | Liver Neoplasms - genetics | Dipeptidyl Peptidase 4 - biosynthesis | Liver Neoplasms - drug therapy | Carcinoma - diagnosis | Dipeptidyl Peptidase 4 - genetics | Mice, SCID | Carcinoma - physiopathology | Disease Progression | Colorectal Neoplasms - physiopathology | Drug Resistance, Neoplasm - genetics | Animals | Liver Neoplasms - diagnosis | Tumor Burden - drug effects | Liver Neoplasms - metabolism | Carcinoma - genetics | Biomarkers, Tumor - genetics | Fluorouracil - pharmacology | Mice | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Neoplasm Invasiveness - genetics | Cell Migration Assays | Biomarkers, Tumor - biosynthesis
Journal Article
Cancer, ISSN 0008-543X, 2017, Volume 123, Issue 16, pp. 3150 - 3158
Journal Article