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Nature Reviews Rheumatology, ISSN 1759-4790, 04/2017, Volume 13, Issue 4, pp. 217 - 233
TNF blockers are highly efficacious at dampening inflammation and reducing symptoms in rheumatic diseases such as rheumatoid arthritis, psoriatic arthritis and... 
LIGAND 1A TL1A | COLLAGEN-INDUCED ARTHRITIS | FIBROBLAST-LIKE SYNOVIOCYTES | TUMOR-NECROSIS-FACTOR | SYSTEMIC-LUPUS-ERYTHEMATOSUS | LYMPHOTOXIN-BETA-RECEPTOR | SYNOVIAL FIBROBLASTS | CD4(+) T-CELLS | CD40 LIGAND | MONOCLONAL-ANTIBODY | RHEUMATOLOGY | Fas Ligand Protein - metabolism | Rheumatic Diseases - drug therapy | OX40 Ligand - metabolism | CD27 Ligand - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Lymphotoxin-alpha - metabolism | Molecular Targeted Therapy | 4-1BB Ligand - antagonists & inhibitors | TNF-Related Apoptosis-Inducing Ligand - antagonists & inhibitors | CD40 Ligand - metabolism | CD27 Ligand - metabolism | Cell Death | Rheumatic Diseases - immunology | Signal Transduction | Lymphocyte Activation | Tumor Necrosis Factors - metabolism | 4-1BB Ligand - metabolism | Immune Tolerance | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | TNF-Related Apoptosis-Inducing Ligand - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Tumor Necrosis Factors - immunology | OX40 Ligand - antagonists & inhibitors | Cytokine TWEAK | Fas Ligand Protein - antagonists & inhibitors | Animals | Tumor Necrosis Factor Ligand Superfamily Member 15 - antagonists & inhibitors | T-Lymphocytes - immunology | Tumor Necrosis Factor Ligand Superfamily Member 14 - antagonists & inhibitors | CD40 Ligand - antagonists & inhibitors | Lymphotoxin-alpha - antagonists & inhibitors | Tumor Necrosis Factors - antagonists & inhibitors | Care and treatment | Cytokines | Tumor necrosis factor | Rheumatic diseases | Development and progression | Genetic aspects | Health aspects
Journal Article
The Journal of Pathology, ISSN 0022-3417, 11/2015, Volume 237, Issue 3, pp. 307 - 318
Lymphangiogenesis is essential in embryonic development but is rare in adults. It occurs, however, in many disease conditions including cancers. Vascular... 
Tnfsf15 | lymphangiogenesis | lymphatic endothelial cell | DR3 | Vegfr3 | APOPTOSIS | ANGIOGENESIS | PATHOLOGY | ONCOLOGY | ROLES | INFLAMMATION | GROWTH-FACTOR RECEPTOR-3 | TNF-ALPHA | DIFFERENTIATION | INHIBITOR | NF-KAPPA-B | T-CELLS | Up-Regulation | Cell Proliferation | Receptors, Tumor Necrosis Factor, Member 25 - metabolism | NF-kappa B - metabolism | Dose-Response Relationship, Drug | Lymphatic Vessels - drug effects | Lymphatic Vessels - metabolism | Transfection | RNA Interference | Time Factors | Lymph - metabolism | Receptors, Tumor Necrosis Factor, Member 25 - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - genetics | Lymphatic Vessels - cytology | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Injections, Intraperitoneal | Mice, Transgenic | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Recombinant Proteins - administration & dosage | Lymphangiogenesis - drug effects | Tumor Necrosis Factor Ligand Superfamily Member 15 - administration & dosage | Animals | Vascular Endothelial Growth Factor C - metabolism | Mice, Inbred BALB C | Cell Movement | Endothelial Cells - drug effects | Embryonic development | Analysis | Genes | Genetic engineering | Gene expression | Vascular endothelial growth factor | Tumors | Necrosis | Endothelium
Journal Article
Immunological Reviews, ISSN 0105-2896, 11/2011, Volume 244, Issue 1, pp. 188 - 196
DR3 (TNFRSF25) is a member of the tumor necrosis factor receptor (TNFR) superfamily expressed primarily on lymphocytes and is a receptor for the TNF family... 
DR3 | tumor necrosis factor family cytokines | asthma | allergic disease | autoimmune disease | inflammatory bowel disease | TL1A | Inflammatory bowel disease | Allergic disease | Autoimmune disease | Tumor necrosis factor family cytokines | Asthma | NKT CELLS | APOPTOSIS | IFN-GAMMA PRODUCTION | SUSCEPTIBILITY | CYTOKINE | IMMUNOLOGY | DOMAIN-CONTAINING RECEPTOR | T-CELL RESPONSES | TNFRSF25 | EXPRESSION | DEATH-DOMAIN | Autoimmunity | Asthma - metabolism | Cell Proliferation | Receptors, Tumor Necrosis Factor, Member 25 - immunology | Humans | Inflammatory Bowel Diseases - immunology | Receptors, Tumor Necrosis Factor, Member 25 - metabolism | Inflammatory Bowel Diseases - metabolism | TNF Receptor-Associated Death Domain Protein - genetics | Signal Transduction - immunology | T-Lymphocytes - metabolism | Asthma - immunology | Gene Expression - immunology | Killer Cells, Natural - immunology | Receptors, Tumor Necrosis Factor, Member 25 - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - genetics | TNF Receptor-Associated Death Domain Protein - metabolism | Cytokines - immunology | Lymphocyte Activation | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Immunity, Innate | Tumor Necrosis Factor Ligand Superfamily Member 15 - immunology | Mice, Knockout | Animals | T-Lymphocytes - immunology | Mice | Killer Cells, Natural - metabolism | TNF Receptor-Associated Death Domain Protein - immunology | Cytokines - biosynthesis | Inflammation | T cells | Tumor necrosis factor | Gastrointestinal diseases | Hyperplasia | TNF-family cytokines
Journal Article
Journal Article
Structure, ISSN 0969-2126, 2011, Volume 19, Issue 2, pp. 162 - 171
Decoy Receptor 3 (DcR3), a secreted member of the Tumor Necrosis Factor (TNF) receptor superfamily, neutralizes three different TNF ligands: FasL, LIGHT, and... 
NECROSIS-FACTOR RECEPTOR | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | TNF SUPERFAMILY | T-CELL | FACTOR-ALPHA | TL1A | CELL BIOLOGY | BIOPHYSICS | LIGHT-MEDIATED APOPTOSIS | FACTOR SUPERFAMILY | LIGAND | Fas Ligand Protein - metabolism | Receptors, Tumor Necrosis Factor, Member 6b - metabolism | Humans | Molecular Conformation | Molecular Sequence Data | Crystallography, X-Ray | Fas Ligand Protein - immunology | Signal Transduction - immunology | Receptors, Tumor Necrosis Factor, Member 6b - immunology | Cloning, Molecular | Receptors, Tumor Necrosis Factor, Member 6b - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - genetics | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Amino Acid Sequence | Mutagenesis, Site-Directed | Tumor Necrosis Factor Ligand Superfamily Member 14 - immunology | Cells, Cultured | Crystallization | Recombinant Proteins - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 15 - immunology | Animals | Drosophila melanogaster | Index Medicus | Decoys | Stoichiometry | Receptors | Ligands | Strategy | Biochemistry | Invariants | Crystal structure | ATOMS | CRYSTAL STRUCTURE | MUTAGENESIS | MATERIALS SCIENCE | SPECIFICITY | AFFINITY
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92743
Atsttrin, a progranulin (PGRN)-derived molecule composed of three TNFR-binding domains of PGRN, binds to TNF receptors (TNFR) and is therapeutic against... 
IMMUNITY | DEATH RECEPTOR | NECROSIS-FACTOR TNF | MULTIDISCIPLINARY SCIENCES | MOUSE | SUPERFAMILY | THERAPIES | CYTOKINE | T-CELL | GROWTH-FACTOR | FAMILY | Multigene Family | Recombinant Fusion Proteins - pharmacology | Body Weight - drug effects | Dextran Sulfate - adverse effects | Receptors, Tumor Necrosis Factor, Member 25 - metabolism | Colitis - pathology | Hemorrhage - prevention & control | Recombinant Fusion Proteins - metabolism | Dose-Response Relationship, Drug | Colitis - chemically induced | Protein Binding - drug effects | Protein Interaction Domains and Motifs | Receptors, Tumor Necrosis Factor, Member 25 - genetics | Disease Models, Animal | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Cell Line | Cysteine - chemistry | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Macrophages - metabolism | Animals | Tumor Necrosis Factor Ligand Superfamily Member 15 - antagonists & inhibitors | Colitis - metabolism | Receptors, Tumor Necrosis Factor, Member 25 - chemistry | Mice | Tumor necrosis factor receptors | Pediatrics | Yeast | Arthritis | Proteins | Receptors | Biomedical materials | Rodents | Dextran sulfate | Tumor necrosis factor-TNF | Biocompatibility | Inhibition | Cytokines | Cloning | Inflammation | Dextran | Osteoclastogenesis | Hospitals | Sodium | Tumor necrosis factor | Rheumatoid arthritis | Ligands | Sulfate | Colitis | Alzheimers disease | Autoimmune diseases
Journal Article
FEBS Letters, ISSN 0014-5793, 04/2017, Volume 591, Issue 8, pp. 1187 - 1195
Exploiting regulatory T cells (Tregs) to control aberrant immune reactions is a promising therapeutic approach, but is hampered by their relative paucity. In... 
TNFRSF25 | DR3 | TNFSF15 | Treg | TL1A | ACTIVATION | IFN-GAMMA PRODUCTION | TNF-LIKE LIGAND | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALLERGIC LUNG INFLAMMATION | PERIPHERAL-BLOOD | VERSUS-HOST-DISEASE | CELL BIOLOGY | BIOPHYSICS | EXPRESSION | MARROW TRANSPLANTATION | T-Lymphocytes, Regulatory - metabolism | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Receptors, Tumor Necrosis Factor, Member 25 - metabolism | Receptors, Tumor Necrosis Factor, Member 25 - agonists | Recombinant Fusion Proteins - metabolism | T-Lymphocytes, Regulatory - immunology | Protein Isoforms - metabolism | Protein Processing, Post-Translational - drug effects | Protein Isoforms - chemistry | Receptors, Tumor Necrosis Factor, Type II - metabolism | T-Lymphocytes, Regulatory - cytology | Protein Interaction Domains and Motifs | Tumor Necrosis Factor Ligand Superfamily Member 15 - chemistry | Phosphorylation - drug effects | Receptors, Tumor Necrosis Factor, Member 25 - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - genetics | Immunosuppressive Agents - pharmacology | Peptide Fragments - genetics | Peptide Fragments - metabolism | Cells, Cultured | Recombinant Fusion Proteins - chemistry | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Receptors, Tumor Necrosis Factor, Type II - genetics | Sirolimus - pharmacology | NF-kappa B p52 Subunit - metabolism | Up-Regulation - drug effects | Peptide Fragments - chemistry | T-Lymphocytes, Regulatory - drug effects | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Receptors, Tumor Necrosis Factor, Member 25 - chemistry | Ligands | Cell Proliferation - drug effects | NF-kappa B p52 Subunit - agonists | Protein Isoforms - genetics
Journal Article