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Kidney international, ISSN 0085-2538, 2015, Volume 88, Issue 4, pp. 722 - 733
.... Tumor necrosis factor-α (TNF-α) is produced by monocytes/macrophages but the direct role of TNF-α... 
albuminuria | inflammation | diabetic nephropathy | CELLS | SODIUM RETENTION | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RELEASE | NEPHROPATHY | URINARY | UROLOGY & NEPHROLOGY | CHEMOKINE | EXPRESSION | TNF RECEPTORS 1 | Tumor Necrosis Factor-alpha - metabolism | Kidney - pathology | Blood Urea Nitrogen | Tumor Necrosis Factor-alpha - genetics | Diabetes Mellitus, Experimental - genetics | Macrophages, Peritoneal - pathology | Male | Receptors, Tumor Necrosis Factor, Type I - metabolism | Tumor Necrosis Factor-alpha - deficiency | CD11b Antigen - genetics | Kidney - metabolism | Receptors, Tumor Necrosis Factor, Type II - metabolism | Inflammation Mediators - metabolism | Mice, Inbred DBA | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Inflammation Mediators - antagonists & inhibitors | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Macrophages, Peritoneal - drug effects | Diabetic Nephropathies - pathology | Genetic Predisposition to Disease | Kidney - drug effects | Signal Transduction | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Antibodies, Neutralizing - pharmacology | Diabetic Nephropathies - genetics | Biomarkers - blood | Albuminuria - prevention & control | Chemotaxis | Mice, Knockout | Albuminuria - genetics | Phenotype | Animals | Creatinine - blood | CD11b Antigen - metabolism | Macrophages, Peritoneal - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Journal Article
Journal Article
Cell death and differentiation, ISSN 1476-5403, 2014, Volume 21, Issue 10, pp. 1511 - 1521
...) and RIPK3 by receptors such as tumor necrosis factor (TNF) receptor-1. Structural studies indicate that activation of RIPK3 by RIPK1 involves the formation of oligomers via interactions of the RIP homotypic interaction motif (RHIM... 
APOPTOSIS | RIPOPTOSOME | MLKL | MACROPHAGES | DOWNSTREAM | BIOCHEMISTRY & MOLECULAR BIOLOGY | MIXED LINEAGE KINASE | PROGRAMMED NECROSIS | DOMAIN-LIKE | CLEAVAGE | CELL-DEATH | CELL BIOLOGY | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
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Journal Article
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