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Environmental and Molecular Mutagenesis, ISSN 0893-6692, 08/2018, Volume 59, Issue 7, pp. 653 - 667
Interleukin 1 beta ( IL‐1β ) and Tumor necrosis factor alpha ( TNF‐α ) are key inflammatory cytokines whose polymorphisms have been correlated with increased... 
single‐nucleotide polymorphisms | pylori | cytokines | pro‐inflammatory | inflammation | H. pylori | single-nucleotide polymorphisms | pro-inflammatory | METAANALYSIS | RECEPTOR | CHINESE POPULATION | ENVIRONMENTAL SCIENCES | CARCINOGENESIS | HELICOBACTER-PYLORI INFECTION | INCREASED RISK | KOREAN POPULATION | GENETICS & HEREDITY | PROMOTER REGION | TOXICOLOGY | TNF-ALPHA | Stomach Neoplasms - genetics | Genetic Predisposition to Disease | Gene Frequency | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | India - epidemiology | Helicobacter Infections - complications | Male | Interleukin-1beta - genetics | Case-Control Studies | Stomach Neoplasms - microbiology | Alleles | Survival Analysis | Adult | Female | Stomach Neoplasms - epidemiology | Helicobacter pylori - isolation & purification | Polymorphism, Single Nucleotide | Oncology, Experimental | Genes | Disease susceptibility | Research | Stomach cancer | Risk factors | Necrosis | Interleukins | Tumor necrosis factor | Analysis | Genetic research | Genetic aspects | Rankings | Single nucleotide polymorphisms | Cancer | Cytokines | Interleukin | Data processing | Inflammation | Single-nucleotide polymorphism | Tumor necrosis factor-α | Risk analysis | Survival | Rank tests | Survival analysis | Interleukin 1 | Tumor necrosis factor-TNF | Survivability | Gastric cancer | Genotypes | Tumors | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Chromosomal proteins | Mass spectrometry | Analysis | Index Medicus | Abridged Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 03/2013, Volume 38, Issue 3, pp. 541 - 554
Elevated concentrations of tumor necrosis factor-α (TNF-α) are detected in pathologies characterized by chronic inflammation. Whether TNF-α plays a role in... 
RECRUITMENT | KILLER-CELL | IMMUNE SUPPRESSION | IMMUNOSUPPRESSION | DOWN-REGULATION | TNF-ALPHA | ACCUMULATION | IMMUNOLOGY | CANCER | T-CELLS | PROGRESSION | Calgranulin B - metabolism | Calgranulin A - metabolism | Tumor Necrosis Factor-alpha - genetics | Immunoblotting | Calgranulin A - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Cell Differentiation - genetics | Inflammation - metabolism | Flow Cytometry | Etanercept | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Calgranulin B - immunology | Gene Expression - immunology | Tumor Necrosis Factor-alpha - immunology | Immunoglobulin G - pharmacology | Killer Cells, Natural - immunology | Myeloid Cells - drug effects | Receptors, Antigen, T-Cell - immunology | Calgranulin B - genetics | Mitogen-Activated Protein Kinases - immunology | Calgranulin A - immunology | Receptors, Antigen, T-Cell - metabolism | Mice, Inbred C57BL | Cells, Cultured | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Cell Differentiation - immunology | Animals | Receptors, Tumor Necrosis Factor | Cell Differentiation - drug effects | Inflammation - genetics | Myeloid Cells - metabolism | Mitogen-Activated Protein Kinases - genetics | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Receptors, Antigen, T-Cell - genetics | Killer Cells, Natural - metabolism | Chronic Disease | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 10/2015, Volume 88, Issue 4, pp. 722 - 733
Monocyte/macrophage recruitment correlates strongly with the progression of diabetic nephropathy. Tumor necrosis factor-α (TNF-α) is produced by... 
albuminuria | inflammation | diabetic nephropathy | CELLS | SODIUM RETENTION | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RELEASE | NEPHROPATHY | URINARY | UROLOGY & NEPHROLOGY | CHEMOKINE | EXPRESSION | TNF RECEPTORS 1 | Tumor Necrosis Factor-alpha - metabolism | Kidney - pathology | Blood Urea Nitrogen | Tumor Necrosis Factor-alpha - genetics | Diabetes Mellitus, Experimental - genetics | Macrophages, Peritoneal - pathology | Male | Receptors, Tumor Necrosis Factor, Type I - metabolism | Tumor Necrosis Factor-alpha - deficiency | CD11b Antigen - genetics | Kidney - metabolism | Receptors, Tumor Necrosis Factor, Type II - metabolism | Inflammation Mediators - metabolism | Mice, Inbred DBA | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Inflammation Mediators - antagonists & inhibitors | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Macrophages, Peritoneal - drug effects | Diabetic Nephropathies - pathology | Genetic Predisposition to Disease | Kidney - drug effects | Signal Transduction | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Antibodies, Neutralizing - pharmacology | Diabetic Nephropathies - genetics | Biomarkers - blood | Albuminuria - prevention & control | Chemotaxis | Mice, Knockout | Albuminuria - genetics | Phenotype | Animals | Creatinine - blood | CD11b Antigen - metabolism | Macrophages, Peritoneal - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 44, pp. 31715 - 31727
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2011, Volume 286, Issue 52, pp. 44326 - 44335
Endogenous glucocorticoids (GCs) and inflammatory cytokines contribute to the age-associated loss of bone mass and strength, but the molecular mechanisms... 
TRANSCRIPTIONAL ACTIVITY | REACTIVE OXYGEN | BONE-MINERAL DENSITY | PROTEASOMAL DEGRADATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FORKHEAD-BOX-O | GENE-EXPRESSION | EXTENDS LIFE-SPAN | T-CELL-FACTOR | BETA-CATENIN | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Shc Signaling Adaptor Proteins - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Oxidative Stress - physiology | Tumor Necrosis Factor-alpha - genetics | Wnt Signaling Pathway - physiology | Male | Wnt Proteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Dexamethasone - pharmacology | MAP Kinase Kinase 4 - metabolism | Wnt Proteins - genetics | Forkhead Transcription Factors - metabolism | Female | Osteoblasts - cytology | Proto-Oncogene Proteins c-akt - metabolism | Cell Differentiation - physiology | Forkhead Transcription Factors - genetics | Prednisolone - pharmacology | beta Catenin - metabolism | beta Catenin - genetics | Mice, Knockout | Animals | Shc Signaling Adaptor Proteins - genetics | Src Homology 2 Domain-Containing, Transforming Protein 1 | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Glucocorticoids - pharmacology | MAP Kinase Kinase 4 - genetics | Cell Proliferation - drug effects | Mice | Apoptosis - physiology | Oxidative Stress - drug effects | Osteoblasts - metabolism | Index Medicus | Wnt Signaling | FoxO | Glucocorticoids | Oxidative Stress | Tumor Necrosis Factor (TNF) | Osteoblasts | Cell Biology
Journal Article
Experimental and Therapeutic Medicine, ISSN 1792-0981, 07/2016, Volume 12, Issue 1, pp. 323 - 328
The present study aimed to investigate the regulatory mechanisms underlying sepsis progression in patients with tumor necrosis factor (TNF)-alpha genetic... 
Sepsis | Ubiquitination | MicroRNA | Tumor necrosis factor-α variation | Microarray data | tumor necrosis factor-alpha variation | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | CYCLIN-E | PROTEIN | microarray data | FUS | CANCER | ubiquitination | SEPTIC SHOCK | microRNA | TARGETS | TNF-ALPHA | EXPRESSION | sepsis | TRAUMA PATIENTS
Journal Article
Cell Metabolism, ISSN 1550-4131, 2006, Volume 4, Issue 6, pp. 465 - 474
Journal Article