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Kidney international, ISSN 0085-2538, 2015, Volume 88, Issue 4, pp. 722 - 733
.... Tumor necrosis factor-α (TNF-α) is produced by monocytes/macrophages but the direct role of TNF-α... 
albuminuria | inflammation | diabetic nephropathy | CELLS | SODIUM RETENTION | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RELEASE | NEPHROPATHY | URINARY | UROLOGY & NEPHROLOGY | CHEMOKINE | EXPRESSION | TNF RECEPTORS 1 | Tumor Necrosis Factor-alpha - metabolism | Kidney - pathology | Blood Urea Nitrogen | Tumor Necrosis Factor-alpha - genetics | Diabetes Mellitus, Experimental - genetics | Macrophages, Peritoneal - pathology | Male | Receptors, Tumor Necrosis Factor, Type I - metabolism | Tumor Necrosis Factor-alpha - deficiency | CD11b Antigen - genetics | Kidney - metabolism | Receptors, Tumor Necrosis Factor, Type II - metabolism | Inflammation Mediators - metabolism | Mice, Inbred DBA | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Inflammation Mediators - antagonists & inhibitors | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Macrophages, Peritoneal - drug effects | Diabetic Nephropathies - pathology | Genetic Predisposition to Disease | Kidney - drug effects | Signal Transduction | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Antibodies, Neutralizing - pharmacology | Diabetic Nephropathies - genetics | Biomarkers - blood | Albuminuria - prevention & control | Chemotaxis | Mice, Knockout | Albuminuria - genetics | Phenotype | Animals | Creatinine - blood | CD11b Antigen - metabolism | Macrophages, Peritoneal - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Cell death and differentiation, ISSN 1476-5403, 2014, Volume 21, Issue 10, pp. 1511 - 1521
...) and RIPK3 by receptors such as tumor necrosis factor (TNF) receptor-1. Structural studies indicate that activation of RIPK3 by RIPK1 involves the formation of oligomers via interactions of the RIP homotypic interaction motif (RHIM... 
APOPTOSIS | RIPOPTOSOME | MLKL | MACROPHAGES | DOWNSTREAM | BIOCHEMISTRY & MOLECULAR BIOLOGY | MIXED LINEAGE KINASE | PROGRAMMED NECROSIS | DOMAIN-LIKE | CLEAVAGE | CELL-DEATH | CELL BIOLOGY | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
Journal Article
Journal Article
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2014, Volume 204, Issue 2, pp. 231 - 245
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 9, p. e0138372
The functional crosstalk between angiotensin II (Ang II) and tumor necrosis factor (TNF... 
CTGF EXPRESSION | NADPH OXIDASE | INDUCED HYPERTENSION | XANTHINE-OXIDASE | NAD(P)H OXIDASE | CARDIAC RESTRICTED OVEREXPRESSION | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | GENE-EXPRESSION | BLOOD-PRESSURE | TRANSGENIC MICE | Tumor Necrosis Factor-alpha - metabolism | MAP Kinase Signaling System - physiology | Oxidative Stress - physiology | Collagen Type III - metabolism | NADPH Oxidases - metabolism | Male | NF-kappa B - metabolism | Fibrosis - metabolism | RNA, Messenger - metabolism | Myocardium - metabolism | Blood Pressure - drug effects | Blood Pressure - physiology | p38 Mitogen-Activated Protein Kinases - metabolism | Heart - physiopathology | Angiotensin II - pharmacology | Collagen Type I - metabolism | Ventricular Remodeling - physiology | Hypertension - physiopathology | Hypertension - metabolism | Animals | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Receptor, Angiotensin, Type 1 - metabolism | Heart - drug effects | Signal Transduction - physiology | Mice | Fibrosis - pathology | Oxidative Stress - drug effects | Transforming Growth Factor beta - metabolism | Ventricular Remodeling - drug effects | Connective Tissue Growth Factor - metabolism | Cardiomegaly - metabolism | Oxidases | Hypertension | Heart | Oxidative stress | RNA | Heart enlargement | Transforming growth factors | Necrosis | Tumor necrosis factor | Collagen | Angiotensin | Bone morphogenetic proteins | Tumors | Veterinary colleges | Reactive oxygen species | Collagen (type I) | Crosstalk | Body weight | Collagens | mRNA | Kinases | NAD(P)H oxidase | Infusion | Signal transduction | Etanercept | Tumor necrosis factor-TNF | Blood pressure | Angiotensin II | Heart diseases | NF-κB protein | Oxygen | Collagen (type III) | MAP kinase | Connective tissue growth factor | JNK protein | Tumor necrosis factor-α | Gene expression | Signaling | Nitric oxide | Fibrosis | Ventricle | Hypertrophy
Journal Article