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Blood, ISSN 0006-4971, 03/2012, Volume 119, Issue 10, pp. 2368 - 2375
Diseases that cause hemolysis or myonecrosis lead to the leakage of large amounts of heme proteins. Free heme has proinflammatory and cytotoxic effects. Heme... 
INDUCED CELL-DEATH | OXIDATIVE STRESS | SEVERE MALARIA | NEUTROPHIL MIGRATION | INDUCED APOPTOSIS | RECEPTOR-INTERACTING PROTEIN | OXYGEN SPECIES GENERATION | TOLL-LIKE RECEPTORS | KINASE RIP | HEMATOLOGY | NF-KAPPA-B | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | NIH 3T3 Cells | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Necrosis | Dose-Response Relationship, Drug | Heme - pharmacology | Heme Oxygenase-1 - genetics | Indoles - pharmacology | Membrane Proteins - metabolism | Tumor Necrosis Factors - pharmacology | Cell Survival - drug effects | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Tumor Necrosis Factors - metabolism | Toll-Like Receptor 4 - genetics | Imidazoles - pharmacology | Macrophages - cytology | Receptors, Tumor Necrosis Factor, Type I - genetics | Toll-Like Receptor 4 - metabolism | Blotting, Western | Mice, Knockout | Macrophages - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Macrophages - drug effects | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Myeloid Differentiation Factor 88 - metabolism | Index Medicus | Abridged Index Medicus | Red Cells, Iron, and Erythropoiesis | Phagocytes, Granulocytes, and Myelopoiesis
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 95 - 99
Receptor interacting protein kinase 1 (RIPK1) has an essential role in the signalling triggered by death receptors and pattern recognition receptors(1,2).... 
UBIQUITINATION | NECROPTOSIS | MEDIATED RECOMBINATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | KINASE | MICE | TNF-ALPHA | CRE-LOXP | NECROSIS-FACTOR RECEPTOR-1 | CELL-DEATH | Intestines - drug effects | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Epithelial Cells - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Caspase 8 - metabolism | Male | NF-kappa B - metabolism | Intestines - metabolism | Organoids - enzymology | Necrosis | Caspase 8 - genetics | Receptors, Tumor Necrosis Factor, Type I - deficiency | Inflammation - metabolism | Gene Deletion | Organoids - metabolism | Female | Homeostasis - drug effects | Epithelial Cells - cytology | Tumor Necrosis Factors - pharmacology | Cell Survival - drug effects | Epithelium - drug effects | Epithelium - pathology | Intestines - pathology | Epithelium - metabolism | Organoids - cytology | Epithelial Cells - pathology | Mice, Knockout | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Myeloid Differentiation Factor 88 - deficiency | Organoids - drug effects | Survival Analysis | Anti-Bacterial Agents - pharmacology | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Intestines - cytology | Protein research | Physiological aspects | Homeostasis | Research | Epithelium | Protein kinases | Apoptosis | Proteins | Genotype & phenotype | Antibiotics | Transgenic animals | Rodents | Ligands | Colon | Kinases | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 04/2011, Volume 18, Issue 4, pp. 656 - 665
Journal Article
Gastroenterology, ISSN 0016-5085, 2017, Volume 152, Issue 4, pp. 867 - 879
Journal Article
Journal Article
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 11/2012, Volume 82, Issue 5, pp. 948 - 957
Journal Article
Nature, ISSN 0028-0836, 03/2007, Volume 446, Issue 7135, pp. 557 - 561
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2009, Volume 29, Issue 12, pp. 2061 - 2068
OBJECTIVE—Tumor necrosis factor–like weak inducer of apoptosis (TWEAK) is a member of the tumor necrosis factor superfamily of cytokines. TWEAK binds and... 
Inflammation | TWEAK | Hyperlipidemia | Kidney | Atherosclerosis | atherosclerosis | hyperlipidemia | ATHEROSCLEROTIC PLAQUES | INJURY | OXIDIZED LDL | APOLIPOPROTEIN-E | kidney | GLOMERULAR MESANGIAL CELLS | ENDOTHELIAL-CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | NF-KAPPA-B | MULTIFUNCTIONAL CYTOKINE | Apolipoproteins E - deficiency | Gene Expression - drug effects | Kidney - pathology | Blood Vessels - pathology | Humans | Male | NF-kappa B - metabolism | Blood Vessels - physiopathology | Atherosclerosis - etiology | Inflammation Mediators - metabolism | Hyperlipidemias - physiopathology | Tumor Necrosis Factors - pharmacology | Cytokines - genetics | Kidney - physiopathology | Macrophages - physiology | Atherosclerosis - pathology | Kidney - drug effects | Macrophages - pathology | Atherosclerosis - physiopathology | Blood Vessels - injuries | Lipoproteins, LDL - pharmacology | Recombinant Proteins - pharmacology | Kidney - injuries | Tumor Necrosis Factors - immunology | Mice, Knockout | Diet, Atherogenic | Tumor Necrosis Factors - physiology | Cytokine TWEAK | Animals | Antibodies, Monoclonal - administration & dosage | Apolipoproteins E - genetics | Blood Vessels - drug effects | Hyperlipidemias - pathology | Macrophages - drug effects | Mice | Tumor Necrosis Factors - antagonists & inhibitors | Index Medicus
Journal Article