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Molecular Cell, ISSN 1097-2765, 2006, Volume 21, Issue 1, pp. 51 - 64
Members of the ING family of tumor suppressors regulate cell cycle progression, apoptosis, and DNA repair as important cofactors of p53. ING1 and ING3 are... 
MYST FAMILY | YEAST | TRANSCRIPTIONAL ACTIVATION | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | REPLICATION ORIGIN | HISTONE ACETYLTRANSFERASE COMPLEX | DEACETYLASE COMPLEX | PHD-FINGER | CANCER METASTASIS SUPPRESSOR-1 | CELL BIOLOGY | Chromatin - metabolism | Trans-Activators - classification | Acetyltransferases - metabolism | Homeodomain Proteins - metabolism | Humans | Multiprotein Complexes | Histone Acetyltransferases - genetics | Molecular Sequence Data | Cell Cycle Proteins - classification | Intracellular Signaling Peptides and Proteins - metabolism | Acetyltransferases - genetics | Receptors, Cytoplasmic and Nuclear - classification | Protein Subunits - metabolism | RNA Interference | Histone Acetyltransferases - metabolism | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Nuclear Proteins - classification | Acetylation | Lysine Acetyltransferase 5 | Nuclear Proteins - genetics | Genes, Tumor Suppressor | Intracellular Signaling Peptides and Proteins - genetics | Repressor Proteins - metabolism | Protein Subunits - genetics | Amino Acid Sequence | Gene Expression | Tumor Suppressor Proteins - classification | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Homeodomain Proteins - classification | Repressor Proteins - genetics | DNA Replication | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | Intracellular Signaling Peptides and Proteins - classification | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Sequence Alignment | Cell Cycle - physiology | Inhibitor of Growth Protein 1 | Trans-Activators - metabolism | Histones - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | DNA replication | Chromatin | Oncology, Experimental | Genomics | DNA | Development and progression | Research | Universities and colleges | Tumor proteins | Acetates | Tumors | Cancer | Index Medicus | Life Sciences
Journal Article
Journal Article
Developmental Cell, ISSN 1534-5807, 02/2010, Volume 18, Issue 2, pp. 288 - 299
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2002, Volume 4, Issue 9, pp. 699 - 704
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 11/2008, Volume 8, Issue 11, pp. 865 - 873
The von Hippel-Lindau disease is caused by inactivating germline mutations of the VHL tumour suppressor gene and is associated with an increased risk of a... 
RNA-POLYMERASE-II | HYDROXYLASE DOMAIN PROTEIN-2 | ONCOLOGY | HYPOXIA-INDUCIBLE-FACTOR | PROLINE HYDROXYLATION | RENAL-CELL-CARCINOMA | VASCULAR TUMORS | HIF-ALPHA | GENE-PRODUCT | PROLYL HYDROXYLASE | FACTOR-ALPHA
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 09/2018, Volume 38, Issue 17
RASSF6 is a member of the tumor suppressor Ras association domain family (RASSF) proteins. RASSF6 is frequently suppressed in human cancers, and its low... 
RB1 | Cell cycle arrest | Tumor suppressor | RASSF6 | Apoptosis | PROMOTER HYPERMETHYLATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | tumor suppressor | PROLIFERATION | HIPPO PATHWAY REGULATION | cell cycle arrest | CANCER | FAMILY | P53 | CELL BIOLOGY | E2F | EXPRESSION | Genomic Instability | Humans | Tumor Protein p73 - genetics | Apoptosis - genetics | Tumor Protein p73 - metabolism | Tumor Suppressor Protein p53 - genetics | Gene Knockdown Techniques | Genes, p53 | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cell Cycle Checkpoints - genetics | E2F1 Transcription Factor - antagonists & inhibitors | Retinoblastoma Binding Proteins - genetics | Retinoblastoma Binding Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Genes, Retinoblastoma | HCT116 Cells | Retinoblastoma Binding Proteins - deficiency | Tumor Suppressor Protein p53 - metabolism | Ubiquitin-Protein Ligases - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Cell Cycle Checkpoints - physiology | E2F1 Transcription Factor - metabolism | Monomeric GTP-Binding Proteins - genetics | Tumor Suppressor Protein p53 - deficiency | Monomeric GTP-Binding Proteins - deficiency | Monomeric GTP-Binding Proteins - metabolism | Models, Biological | DNA Repair | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Ubiquitin-Protein Ligases - deficiency | Apoptosis - physiology | HeLa Cells | Tumor Suppressor Protein p14ARF - genetics | E2F1 Transcription Factor - genetics | Tumor Suppressor Protein p14ARF - metabolism | Ubiquitin-Protein Ligases - genetics | Index Medicus
Journal Article
Journal Article
2012, Springer theses : recognizing outstanding Ph.D. research, ISBN 4431541349
Web Resource
Cell, ISSN 0092-8674, 2005, Volume 123, Issue 2, pp. 207 - 218
The Mixed-Lineage Leukemia (MLL) protein is a histone methyltransferase that is mutated in clinically and biologically distinctive subsets of acute leukemia.... 
MYELOID PROGENITORS | MUTANT MICE | DROSOPHILA-TRITHORAX | HISTONE METHYLTRANSFERASE COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEMATOLOGICAL MALIGNANCIES | MULTIPLE ENDOCRINE NEOPLASIA | LINEAGE-LEUKEMIA GENE | ONCOPROTEIN MLL | FACTOR JUND | HEMATOPOIETIC PROGENITORS | CELL BIOLOGY | Consensus Sequence | Myeloid-Lymphoid Leukemia Protein - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Proto-Oncogene Proteins - chemistry | Myeloid-Lymphoid Leukemia Protein - chemistry | Myeloid Progenitor Cells | Recombinant Fusion Proteins - metabolism | Flow Cytometry | Chromatin Immunoprecipitation | Retroviridae - genetics | Tumor Suppressor Proteins - genetics | Conserved Sequence | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Cell Line | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Bone Marrow Cells - cytology | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Recombinant Fusion Proteins - chemistry | Blotting, Western | Homeodomain Proteins - genetics | Precipitin Tests | Amino Acid Motifs | Sequence Homology, Amino Acid | Leukemia, Myeloid - etiology | Animals | Cell Transformation, Neoplastic | Models, Biological | Myeloid-Lymphoid Leukemia Protein - genetics | Cell Line, Tumor | Mice | Genetic research | Tumor suppressor genes | Research | Protein binding | Physiological aspects | Histones | Methyltransferases | Gene expression | Leukemia | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2017, Volume 127, Issue 6, pp. 2081 - 2090
Journal Article