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细胞研究:英文版, ISSN 1001-0602, 2008, Volume 18, Issue 3, pp. 350 - 359
Journal Article
Clinica Chimica Acta, ISSN 0009-8981, 06/2010, Volume 411, Issue 11-12, pp. 846 - 852
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2015, Volume 17, Issue 1, pp. 95 - 103
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 34, pp. 29003 - 29020
The processes by which cells sense and respond to ambient oxygen concentration are fundamental to cell survival and function, and they commonly target gene... 
CELLS | REGULATORY ELEMENT DATABASE | INDUCIBLE FACTOR-I | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | RECEPTOR | MESSENGER-RNA STABILITY | TUMOR ANGIOGENESIS | MICRORNAS | ENDOTHELIAL GROWTH-FACTOR | PROMOTES ANGIOGENESIS | Endothelium, Vascular - cytology | Human Umbilical Vein Endothelial Cells | Vascular Endothelial Growth Factor A - biosynthesis | Humans | Gene Expression Regulation, Enzymologic - physiology | Endothelium, Vascular - enzymology | Proto-Oncogene Proteins - biosynthesis | Vascular Endothelial Growth Factor A - genetics | Oxygen - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Cell Hypoxia | DEAD-box RNA Helicases - biosynthesis | Glucose Transporter Type 1 - biosynthesis | Tumor Suppressor Proteins - genetics | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Vascular Endothelial Growth Factor Receptor-1 - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Ribonuclease III - genetics | Membrane Proteins - genetics | Adaptation, Physiological - physiology | MicroRNAs - biosynthesis | Proto-Oncogene Proteins - genetics | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Ribonuclease III - biosynthesis | Hep G2 Cells | DEAD-box RNA Helicases - genetics | Membrane Proteins - biosynthesis | Glucose Transporter Type 1 - genetics | MicroRNAs - genetics | Tumor Suppressor Proteins - biosynthesis | Index Medicus | Gene Regulation | MicroRNA | Molecular Biology | Dicer | Molecular Cell Biology | Hypoxia | Hypoxia-inducible Factor (HIF) | Endothelial Cell | Endothelium
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2013, Volume 19, Issue 12, pp. 3259 - 3267
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 11/2013, Volume 18, Issue 11, pp. 1225 - 1234
Genome-wide association studies (GWAS) have identified a region upstream the BIN1 gene as the most important genetic susceptibility locus in Alzheimer's... 
Brain | Tau | BIN1 | Alzheimer | Drosophila | COMMON VARIANTS | PHOSPHORYLATION | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | brain | ENDOCYTOSIS | NEUROSCIENCES | IDENTIFIES VARIANTS | DISEASE | AMPHIPHYSIN-II | GENOME-WIDE ASSOCIATION | Endophenotypes | Gene Expression - genetics | Humans | Transcription Factors - deficiency | Nerve Degeneration - genetics | tau Proteins - metabolism | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Case-Control Studies | Alzheimer Disease - pathology | Brain - metabolism | Drosophila melanogaster - metabolism | Synaptosomes - pathology | Nuclear Proteins - biosynthesis | Tumor Suppressor Proteins - genetics | Nuclear Proteins - genetics | tau Proteins - antagonists & inhibitors | Genetic Predisposition to Disease - genetics | Plaque, Amyloid - pathology | Cells, Cultured | Transcription Factors - genetics | Nerve Degeneration - pathology | Transcription Factors - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Adaptor Proteins, Signal Transducing - genetics | Alzheimer Disease - metabolism | Drosophila Proteins - deficiency | Brain - pathology | Polymorphism, Single Nucleotide - genetics | Adaptor Proteins, Signal Transducing - biosynthesis | Mice | Drosophila Proteins - genetics | Alzheimer Disease - genetics | Tumor Suppressor Proteins - biosynthesis | Physiological aspects | Complications and side effects | Genetic aspects | Alzheimer's disease | Risk factors | Amyloid beta-protein | Index Medicus | Original
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2012, Volume 122, Issue 10, pp. 3563 - 3578
The strength and duration of NF-kappa B signaling are tightly controlled by multiple negative feedback mechanisms. However, in cancer cells, these feedback... 
UBIQUITINATION | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | KINASE | BREAST-CANCER PROGRESSION | PROGNOSTIC MARKER | ALPHA | CELL-PROLIFERATION | PATIENT SURVIVAL | EXPRESSION | A20 | Phosphorylation | MicroRNAs - antagonists & inhibitors | Deubiquitinating Enzyme CYLD | Humans | Brain Neoplasms - pathology | NF-kappa B - metabolism | Cell Line, Tumor - drug effects | Cell Line, Tumor - transplantation | Neoplasm Proteins - metabolism | Brain Neoplasms - metabolism | Glioma - metabolism | RNA - genetics | Glioma - genetics | Smad Proteins - biosynthesis | Ubiquitination - drug effects | I-kappa B Kinase - metabolism | Glioma - pathology | Tumor Suppressor Proteins - genetics | Neovascularization, Pathologic - physiopathology | Transcription, Genetic | Neoplasm Proteins - genetics | Cell Line, Tumor - metabolism | Genes, Reporter | NF-KappaB Inhibitor alpha | RNA, Neoplasm - physiology | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | Transforming Growth Factor beta - physiology | Brain Neoplasms - genetics | MicroRNAs - biosynthesis | Smad Proteins - genetics | RNA - biosynthesis | Transforming Growth Factor beta - pharmacology | Tumor Suppressor Proteins - physiology | Animals | RNA, Neoplasm - biosynthesis | Signal Transduction - drug effects | Mice, Nude | RNA, Neoplasm - genetics | Mice | MicroRNAs - genetics | Protein Processing, Post-Translational | MicroRNAs - physiology | Tumor Suppressor Proteins - biosynthesis | I-kappa B Proteins - deficiency | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 03/2016, Volume 133, Issue 11, pp. 1081 - 1092
BACKGROUND—Adult mammalian cardiomyocytes (CMs) have the potential to proliferate, but this is not sufficient to generate adequate CMs after myocardial... 
Myocardial infarction | Regeneration | Myocytes, cardiac | Molecular biology | Cell cycle | molecular biology | RENEWAL | CARDIAC & CARDIOVASCULAR SYSTEMS | cell cycle | regeneration | STAGE | myocardial infarction | GENES | YAP | PERIPHERAL VASCULAR DISEASE | myocytes | cardiac | Single-Blind Method | T-Box Domain Proteins - physiology | RNA, Small Interfering - genetics | T-Box Domain Proteins - biosynthesis | Muscle Proteins - biosynthesis | RNA, Messenger - biosynthesis | RNA Interference | Cell Division | Myocardial Infarction - pathology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Electrocardiography | Female | Myocardial Infarction - physiopathology | Genes, Reporter | Heart - physiopathology | Immediate-Early Proteins - physiology | Cell Size - drug effects | Immediate-Early Proteins - biosynthesis | RNA, Messenger - genetics | Genes, cdc - drug effects | Mice, Transgenic | Myocardium - pathology | Cell Cycle Proteins - biosynthesis | Myocardial Infarction - metabolism | Random Allocation | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Tumor Suppressor Proteins - physiology | Organ Size - drug effects | Animals | Immediate-Early Proteins - genetics | Signal Transduction - drug effects | Tamoxifen - pharmacology | Myocytes, Cardiac - metabolism | Fetal Proteins - genetics | Mice | Tumor Suppressor Proteins - biosynthesis | Cell proliferation | Care and treatment | Transcription factors | Research | Heart attack | Heart cells | Index Medicus | Abridged Index Medicus | remodeling
Journal Article
Oncology Reports, ISSN 1021-335X, 4/2015, Volume 33, Issue 4, pp. 1976 - 1984
There has been increasing evidence that numerous bioactive dietary agents can hamper the process of carcino-genesis by targeting epigenetic alterations... 
DNMT3B | dietary agents | reactivation | HDAC1 | epigenetic | EGCG | Dietary agents | DNMT3β | Reactivation | Epigenetic | TEA POLYPHENOL | METHYLATION | GREEN TEA | LUNG-CANCER | OVEREXPRESSION | IN-VITRO | RETINOIC ACID | ONCOLOGY | EPIGENETIC REGULATION | HUMAN-MELANOMA | DNA (Cytosine-5-)-Methyltransferases - physiology | Humans | Uterine Cervical Neoplasms - pathology | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Chromatin Assembly and Disassembly - drug effects | Genes, Tumor Suppressor - drug effects | RNA, Messenger - biosynthesis | Tumor Suppressor Proteins - genetics | Enzyme Induction - drug effects | Female | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Catechin - pharmacology | Neoplasm Proteins - genetics | Anticarcinogenic Agents - pharmacology | Histone Deacetylase 1 - genetics | DNA (Cytosine-5-)-Methyltransferases - biosynthesis | Neoplasm Proteins - biosynthesis | Models, Molecular | Histone Deacetylase 1 - biosynthesis | Uterine Cervical Neoplasms - genetics | DNA (Cytosine-5-)-Methyltransferases - genetics | RNA, Neoplasm - biosynthesis | Histone Deacetylase Inhibitors - pharmacology | Protein Conformation | Catechin - analogs & derivatives | Molecular Docking Simulation | HeLa Cells | Histone Deacetylases - physiology | Tumor Suppressor Proteins - biosynthesis | DNA Methylation - drug effects | Pharmacology, Experimental | Oncology, Experimental | Development and progression | Tumor suppressor genes | Drug targeting | Genetic aspects | Research | Health aspects | Cervical cancer | Cancer | Studies | Proteins | Enzymes | Cell cycle | DNA methylation | Polyphenols | Epigenetics | Ligands | Gene expression | Kinases | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article