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Nature (London), ISSN 1476-4687, 05/2006, Volume 441, Issue 7092, pp. 523 - 527
Journal Article
Nature (London), ISSN 0028-0836, 08/2009, Volume 460, Issue 7257, pp. 904 - 908
Journal Article
Nature (London), ISSN 1476-4687, 03/2011, Volume 471, Issue 7336, pp. 119 - 123
...). Defects in DNA damage response factors such as ataxia telangiectasia mutated (ATM) protein and combined deficiencies in classical non-homologous end joining and p53 predispose to RAG-initiated genomic rearrangements and lymphomagenesis(2-11... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Genomic Instability | Protein-Serine-Threonine Kinases - deficiency | Gene Rearrangement, T-Lymphocyte - genetics | Chromosomes, Mammalian - genetics | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Recombination, Genetic - genetics | Lymphoma - pathology | Chromosome Deletion | Tumor Suppressor Proteins - metabolism | Kaplan-Meier Estimate | Protein-Serine-Threonine Kinases - genetics | Thymus Gland - cytology | Lymphoma - genetics | In Situ Hybridization, Fluorescence | Ataxia Telangiectasia Mutated Proteins | Genes, p53 - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Disease Progression | Animals | Genes, Immunoglobulin Heavy Chain - genetics | Mice | Receptors, Antigen, T-Cell - genetics | Translocation, Genetic - genetics | Causes of | Tumor suppressor genes | Lymphomas | Genetic aspects | Abnormalities | Software | Chromosomes | Deoxyribonucleic acid--DNA | Cell cycle | Apoptosis | Tumors | Index Medicus | Thymus Gland | Translocation, Genetic | Protein-Serine-Threonine Kinases | Gene Rearrangement, T-Lymphocyte | Receptors, Antigen, T-Cell | Lymphoma | Chromosomes, Mammalian | Genes, Immunoglobulin Heavy Chain | Life Sciences | Genes, p53 | Immunology | Recombination, Genetic | Cell Cycle Proteins | Tumor Suppressor Proteins | DNA-Binding Proteins
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 10/2012, Volume 122, Issue 10, pp. 3742 - 3754
.... Surprisingly, we found that loss of the tumor suppressor gene neurofibromatosis type 1 (Nf1) increased HSF1 levels and triggered its activation in mouse embryonic fibroblasts... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | NIH 3T3 Cells | Phosphorylation | Macrolides - toxicity | Humans | Neoplasm Proteins - physiology | Transcription Factors - deficiency | Withanolides - toxicity | Gene Expression Regulation, Neoplastic | Neurofibromin 1 - deficiency | Genes, Neurofibromatosis 1 | DNA-Binding Proteins - deficiency | Gene Knockdown Techniques | Heat Shock Transcription Factors | MAP Kinase Signaling System | RNA, Messenger - biosynthesis | Cell Transformation, Neoplastic - genetics | Nerve Sheath Neoplasms - pathology | Active Transport, Cell Nucleus | Neoplasm Proteins - genetics | Cell Line, Tumor - metabolism | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Neoplasm Proteins - biosynthesis | Mice, Inbred C57BL | Neurofibromin 1 - physiology | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Transcription Factors - biosynthesis | Hot Temperature | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | RNA, Neoplasm - biosynthesis | RNA, Neoplasm - genetics | Mice | Mice, Inbred BALB C | Neoplasm Proteins - deficiency | Protein Processing, Post-Translational | DNA-Binding Proteins - biosynthesis | Leupeptins - toxicity | Heat shock proteins | Physiological aspects | Tumor suppressor genes | Genetic aspects | Research | Carcinogenesis | Health aspects | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 5110 - 13
.... Here we propose that miR-224/-520c-dependent TUSC3 deficiency enhances the metastatic potential of NSCLC through the alteration of three unfolded protein response pathways and HRD1-dependent ERAD... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Endoribonucleases - genetics | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | MicroRNAs - metabolism | Activating Transcription Factor 6 - genetics | Tumor Suppressor Protein p53 - genetics | In Situ Hybridization | Tumor Suppressor Proteins - genetics | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - physiology | Gene Expression Regulation, Neoplastic - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Cell Proliferation - genetics | Tumor Suppressor Proteins - metabolism | Endoribonucleases - metabolism | Unfolded Protein Response - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Membrane Proteins - genetics | Endoplasmic Reticulum-Associated Degradation - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Cell Proliferation - physiology | Tumor Suppressor Protein p53 - metabolism | Ubiquitin-Protein Ligases - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Xenograft Model Antitumor Assays | Activating Transcription Factor 6 - metabolism | Animals | Endoplasmic Reticulum-Associated Degradation - physiology | Cell Line, Tumor | Signal Transduction - physiology | Mice | Ubiquitin-Protein Ligases - genetics | Unfolded Protein Response - physiology | Small cell lung carcinoma | p53 Protein | Lung cancer | Non-small cell lung carcinoma | Lung carcinoma | Metastasis | Substrates | Metastases | Proteins | Protein folding | Chromosome 8 | Tumor suppressor genes | miRNA | Solid tumors | Tumors | Cancer | Index Medicus
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 6, pp. 2640 - 2650
Journal Article
Nature cell biology, ISSN 1465-7392, 09/2017, Volume 19, Issue 10, pp. 1226 - 1236
.... Anti-apoptotic BCL-2 proteins engage a BCL-2 homology (BH) domain sequence found in SUFU (suppressor of fused... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | NIH 3T3 Cells | Transcription, Genetic - drug effects | Cell Proliferation | Zinc Finger Protein GLI1 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Myeloid Cell Leukemia Sequence 1 Protein - metabolism | bcl-X Protein - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Molecular Mimicry | Transfection | Neoplasms - genetics | RNA Interference | Time Factors | Tumor Suppressor Proteins - genetics | HEK293 Cells | Female | Antineoplastic Agents - pharmacology | Myeloid Cell Leukemia Sequence 1 Protein - deficiency | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Peptide Fragments - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Cell Survival | Zinc Finger Protein GLI1 - genetics | Repressor Proteins - genetics | Genotype | Myeloid Cell Leukemia Sequence 1 Protein - genetics | Mice, Knockout | Neoplasms - drug therapy | Phenotype | Animals | Mice, Nude | CRISPR-Cas Systems | Mice | Neoplasms - pathology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Genes | Genetic aspects | Genetic transcription | Drug resistance | Binding proteins | Tumors | Protein binding | Regulators | Cell survival | Bcl-2 protein | Transcription | Decision making | Homology | Gene expression | Mcl-1 protein | Proteins | Signaling | Mitochondria | DNA-binding protein | Bcl-x protein | Cell death | Feedforward | Deoxyribonucleic acid--DNA | Cancer | Index Medicus
Journal Article