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index medicus (121) 121
humans (97) 97
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permeability (80) 80
inflammation (59) 59
proteins (57) 57
inflammatory bowel disease (50) 50
cytokines (49) 49
tight junctions (49) 49
intestine (47) 47
tumor necrosis factor-tnf (40) 40
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cell biology (37) 37
epithelial cells (37) 37
intestinal mucosa - metabolism (36) 36
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rodents (33) 33
tumor-necrosis-factor (33) 33
small intestine (32) 32
tumor necrosis factor-α (32) 32
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expression (28) 28
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intestinal permeability (22) 22
tumor necrosis factor-alpha - pharmacology (22) 22
analysis (21) 21
cells (21) 21
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gastroenterology and hepatology (20) 20
mucosa (20) 20
necrosis-factor-alpha (20) 20
review (20) 20
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activation (19) 19
interferon-gamma (19) 19
intestinal mucosa - pathology (19) 19
medicine, research & experimental (19) 19
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mice, inbred c57bl (18) 18
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zonula occludens-1 protein (17) 17
antigens (16) 16
article (16) 16
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electric impedance (13) 13
rats (13) 13
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Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e61944
Intestinal barrier dysfunction occurs in many intestinal diseases, in which proinflammatory cytokines play critical roles. However, researchers are still on... 
INDUCED INCREASE | IN-VITRO | INTERFERON-GAMMA | COLITIS | INFLAMMATORY RESPONSES | MULTIDISCIPLINARY SCIENCES | KINASE | NECROSIS-FACTOR-ALPHA | HIF-1-ALPHA | EXPRESSION | TIGHT JUNCTION PERMEABILITY | Caco-2 Cells | Tight Junctions - metabolism | Myosin-Light-Chain Kinase - genetics | Phosphorylation | Gene Expression - drug effects | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Myosin-Light-Chain Kinase - metabolism | Epithelial Cells - drug effects | Humans | Interferon-gamma - physiology | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | NF-kappa B - metabolism | Permeability | Intestinal Mucosa - cytology | Berberine - pharmacology | Epithelial Cells - physiology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Tumor Necrosis Factor-alpha - physiology | Myosin Light Chains - metabolism | Protein Processing, Post-Translational | Tight Junctions - drug effects | Epithelial cells | Gastroenteritis | Kinases | Small intestine | Myosin-light-chain kinase | Proteins | Signal transduction | Intestine | Rodents | Myosin | Tumor necrosis factor-TNF | Inhibition | Hypoxia-inducible factors | Translocation | Berberine | NF-κB protein | Cytokines | Diarrhea | Inflammation | Tumor necrosis factor-α | Nuclear transport | Zonula occludens-1 protein | Inflammatory bowel disease | Signaling | γ-Interferon | Monolayers | Hypoxia | Interferon | Immunofluorescence
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 03/2018, Volume 141, Issue 3, pp. 951 - 963.e8
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, p. e16139
Background: Guanylyl Cyclase C (GCC) signaling via uroguanylin (UGN) and guanylin activation is a critical mediator of intestinal fluid homeostasis, intestinal... 
LIGHT-CHAIN KINASE | INFLAMMATORY-BOWEL-DISEASE | EPITHELIAL-CELLS | INTERFERON-GAMMA | IFN-GAMMA | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | IN-VIVO | TIGHT JUNCTION DYSFUNCTION | TRANSCRIPTION 5B | ENTEROTOXIN RECEPTOR | Intestinal Mucosa - metabolism | Signal Transduction | Receptors, Peptide - deficiency | Receptors, Guanylate Cyclase-Coupled - deficiency | Cells, Cultured | Intestines - metabolism | Permeability | Receptors, Enterotoxin | Receptors, Guanylate Cyclase-Coupled - metabolism | Mice, Knockout | Animals | Peptides - metabolism | Tight Junctions | Mice | Intestines - physiopathology | Intestines - enzymology | Receptors, Peptide - metabolism | RNA | Muscle proteins | Mitogens | Myosin | Cell proliferation | Pediatrics | Phosphorylation | Epithelial cells | Mucosa | Colorectal cancer | Homeostasis | Chains | Activation | Uroguanylin | Kinases | Guanylate cyclase | Small intestine | Myosin-light-chain kinase | Lipopolysaccharides | Proteins | Ischemia | Intestine | Rodents | Hepatology | Gastroenterology | Cell cycle | Tumor necrosis factor-TNF | Fluid filters | Physiology | Tumorigenesis | Localization | Stat1 protein | Light levels | Cytokines | Nutrition | RNA-mediated interference | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Children & youth | Crohns disease | Inflammatory bowel disease | Signaling | Hospitals | γ-Interferon | Irritable bowel syndrome | Monolayers | Dismantling | Apoptosis | Ribonucleic acid
Journal Article
Scandinavian Journal of Gastroenterology, ISSN 0036-5521, 2009, Volume 44, Issue 10, pp. 1226 - 1235
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2008, Volume 12, Issue 4, pp. 1331 - 1346
The patients with Crohn's disease (CD) have a ‘leaky gut’ manifested by an increase in intestinal epithelial tight junction (TJ) permeability. Tumour necrosis... 
intestinal permeability | myosin light chain kinase | tumour necrosis factor‐α | tight junction | NF‐κB subunit | Tight junction | NF-κB subunit | Tumour necrosis factor-α | Myosin light chain kinase | Intestinal permeability | MEDICINE, RESEARCH & EXPERIMENTAL | BARRIER DYSFUNCTION | CROHNS-DISEASE | DNA-BINDING SUBUNIT | CELL BIOLOGY | INFLAMMATORY-BOWEL-DISEASE | INTESTINAL EPITHELIAL MONOLAYERS | DIARRHEA IN-VIVO | MONOCLONAL-ANTIBODY CA2 | TNF-ALPHA | NF-KAPPA-B | TIGHT JUNCTION PERMEABILITY | tumour necrosis factor-alpha | NF-kappa B subunit | Caco-2 Cells | Gene Expression Regulation, Enzymologic - drug effects | Myosin-Light-Chain Kinase - genetics | Humans | Tumor Suppressor Protein p53 - metabolism | Molecular Sequence Data | Mutation - genetics | Promoter Regions, Genetic - genetics | Genome, Human - genetics | Tumor Necrosis Factor-alpha - pharmacology | Transfection | Enterocytes - drug effects | Transcription Factor RelA - metabolism | Base Sequence | Transcription Initiation Site | Enterocytes - enzymology | Binding Sites | Dimerization | RNA, Small Interfering - metabolism | Inflammatory bowel diseases | Epithelial cells | p53 Protein | Chains | Gene deletion | Kinases | Myosin-light-chain kinase | Proteins | Clonal deletion | Intestine | Modulation | Myosin | Penicillin | Tumor necrosis factor-TNF | Antigens | Cytokines | Cloning | Inflammation | Crohn's disease | Permeability | Gene expression | Molecular modelling | Mutagenesis | Tumor necrosis factor | Protein expression | Dimers | Binding sites | Tumors | tumour necrosis factor-α
Journal Article