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1. Full Text Tumor necrosis factor alpha: A link between neuroinflammation and excitotoxicity
by Olmos, Gabriel and Lladó, Jerònia
Mediators of Inflammation, ISSN 0962-9351, 2014, Volume 2014, pp. 861231 - 12
Tumor necrosis factor alpha (TNF-alpha) is a proinflammatory cytokine that exerts both homeostatic and pathophysiological roles in the central nervous system.... 
NERVOUS-SYSTEM | OXIDATIVE STRESS | INTERFERON-GAMMA | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | AMPA-RECEPTOR TRAFFICKING | POTENTIATES GLUTAMATE NEUROTOXICITY | TNF-ALPHA | IMMUNOLOGY | CEREBROSPINAL-FLUID | NF-KAPPA-B | CELL BIOLOGY | Astrocytes - cytology | Receptors, Glutamate - metabolism | Inflammation - pathology | Neurons - pathology | Cytokines - metabolism | Signal Transduction | Calcium - metabolism | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Analgesics, Opioid - pharmacology | Synaptic Transmission | Neuroglia - cytology | Animals | Neuronal Plasticity | Tumor Necrosis Factor-alpha - physiology | Glutamic Acid - metabolism | Receptors, GABA-A - metabolism | Neuralgia | Receptors, AMPA - metabolism | Physiological aspects | GABA | Inflammation | Tumor necrosis factor | Health aspects | Review
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2. Full Text Tumor necrosis factor alpha in mycobacterial infection
by Dorhoi, Anca and Kaufmann, Stefan H.E
Seminars in Immunology, ISSN 1044-5323, 2014, Volume 26, Issue 3, pp. 203 - 209
Highlights • In TB, TNF-α is produced primarily by myeloid cells and also by lymphocytes and its availability is regulated by cytokines, enzymes, lipid... 
Allergy and Immunology | Granuloma | Inflammation | Mycobacterium tuberculosis | Cell death | Immunotherapy | IMMUNE-RESPONSE | NONPATHOGENIC MYCOBACTERIA | SOLUBLE ANTIGEN | IMMUNOLOGY | DRUG-RESISTANT TUBERCULOSIS | CYTOKINE PRODUCTION | PULMONARY TUBERCULOSIS | ALVEOLAR MACROPHAGES | TNF-ALPHA | DELAYED HYPERSENSITIVITY | INDUCED GRANULOMA-FORMATION | Tuberculosis - immunology | Tumor Necrosis Factor-alpha - metabolism | Animals | Humans | Tuberculosis - drug therapy | Mycobacterium tuberculosis - physiology | Tumor Necrosis Factor-alpha - immunology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Bacterial infections | Tumor necrosis factor | Health aspects | Tumors
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3. Full Text Epigenetic Regulation of Tumor Necrosis Factor Alpha
by K. E. Sullivan and A. B. M. Reddy and K. Dietzmann and A. R. Suriano and V. P. Kocieda and M. Stewart and M. Bhatia
Molecular and Cellular Biology, ISSN 0270-7306, 07/2007, Volume 27, Issue 14, pp. 5147 - 5160
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
HUMAN MONOCYTIC CELLS | INTERCELLULAR-ADHESION MOLECULE-1 | HISTONE H3 | IFN-GAMMA PROMOTER | BIOCHEMISTRY & MOLECULAR BIOLOGY | EMBRYONIC STEM-CELLS | CREB-BINDING-PROTEIN | TNF-ALPHA | NF-KAPPA-B | ACTIVE GENES | BETA-GLOBIN LOCUS | CELL BIOLOGY | Sulfites | Embryonic Stem Cells - cytology | Humans | Tumor Necrosis Factor-alpha - genetics | Thionucleosides - pharmacology | In Situ Hybridization, Fluorescence | Protein Transport - drug effects | Adenosine - pharmacology | Euchromatin - metabolism | Acetylation - drug effects | Animals | Cell Nucleus - metabolism | Embryonic Stem Cells - drug effects | Cell Differentiation - drug effects | Adenosine - analogs & derivatives | Cell Line, Tumor | Epigenesis, Genetic - drug effects | Mice | Models, Genetic | Histones - metabolism | Cell Nucleus - drug effects | DNA Methylation - drug effects
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4. Full Text Tumor necrosis factor alpha in sleep regulation
by Rockstrom, Matthew D and Chen, Liangyu and Taishi, Ping and Nguyen, Joseph T and Gibbons, Cody M and Veasey, Sigrid C and Krueger, James M
Sleep Medicine Reviews, ISSN 1087-0792, 08/2018, Volume 40, pp. 69 - 78
This review details tumor necrosis factor alpha (TNF) biology and its role in sleep, and describes how TNF medications influence sleep/wake activity.... 
Autoimmunity | Sleep regulation | Tumor necrosis factor alpha | Brain organization of sleep | Plasticity | Sleep function | RHEUMATOID-ARTHRITIS | SLOW-WAVE SLEEP | ANKYLOSING-SPONDYLITIS | PSORIATIC-ARTHRITIS | FACTOR RECEPTOR | NEUROSCIENCES | CLINICAL NEUROLOGY | PATIENT-REPORTED OUTCOMES | G308A POLYMORPHISM | TNF-ALPHA | TRANSCRIPTION FACTOR | REM-SLEEP | Animals | Neuronal Plasticity | Humans | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - physiology | Sleep - physiology | Brain - physiology | Medical research | Medical colleges | Neurosciences | Sleep | Tumor necrosis factor | Medicine, Experimental | Tumors | Necrosis | plasticity | sleep regulation | autoimmunity | brain organization of sleep | sleep function
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5. Full Text Childhood trauma and adulthood inflammation: A meta-analysis of peripheral C-reactive protein, interleukin-6 and tumour necrosis factor-α
by Baumeister, D and Akhtar, R and Ciufolini, S and Pariante, C.M and Mondelli, V
Molecular Psychiatry, ISSN 1359-4184, 05/2016, Volume 21, Issue 5, pp. 642 - 649
Childhood trauma confers higher risk of adulthood physical and mental illness; however, the biological mechanism mediating this association remains largely... 
POSTTRAUMATIC-STRESS-DISORDER | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPRESSION PROFILE | NEUROSCIENCES | NATIONAL COMORBIDITY SURVEY | HEALTHY-ADULTS | GLUCOCORTICOID-RECEPTOR | EPIGENETIC REGULATION | ARTERY RISK DEVELOPMENT | MAJOR DEPRESSIVE DISORDER | EARLY-LIFE ADVERSITY | 1ST-EPISODE PSYCHOSIS | C-Reactive Protein - metabolism | Interleukin-6 - blood | Humans | Tumor Necrosis Factor-alpha - blood | Adult Survivors of Child Abuse | Inflammation - etiology | Inflammation - blood | Original
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6. Full Text A metal-based inhibitor of tumor necrosis factor-α
by Leung, Chung-Hang and Zhong, Hai-Jing and Yang, Hui and Cheng, Zhen and Chan, Daniel Shiu-Hin and Ma, Victor Pui-Yan and Abagyan, Ruben and Wong, Chun-Yuen and Ma, Dik-Lung
Angewandte Chemie - International Edition, ISSN 1433-7851, 09/2012, Volume 51, Issue 36, pp. 9010 - 9014
Staying in the pocket: A cyclometalated iridium(III) biquinoline complex targets the protein-protein interface (see picture; C yellow, N blue, Ir dark green)... 
inhibitors | metal-based inhibitors | tumor necrosis factor | iridium(III) complexes | protein-protein interactions | ENZYME | DNA | COMPLEXES | KINASE | THERAPIES | BINDING | CHEMISTRY, MULTIDISCIPLINARY | Protein Structure, Tertiary | Tumor Necrosis Factor-alpha - metabolism | Stereoisomerism | Humans | Crystallography, X-Ray | Immobilized Proteins - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type I - antagonists & inhibitors | Coordination Complexes - chemistry | Hep G2 Cells | Protein Interaction Maps | Immobilized Proteins - metabolism | Binding Sites | Iridium - chemistry | Tumor Necrosis Factor-alpha - antagonists & inhibitors
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7. Full Text Interleukin (IL)-6, tumour necrosis factor alpha (TNF-α) and soluble interleukin-2 receptors (sIL-2R) are elevated in patients with major depressive disorder: A meta-analysis and meta-regression
by Liu, Yang and Ho, Roger Chun-Man and Mak, Anselm
Journal of Affective Disorders, ISSN 0165-0327, 2011, Volume 139, Issue 3, pp. 230 - 239
Abstract Background Many studies have explored the association between soluble interleukin-2 receptor (sIL-2R), cytokines and major depressive disorder (MDD).... 
Psychiatry | Meta-regression | Depression | Inflammation | Cytokine | Meta-analysis | PSYCHIATRY | MARKERS | IL-6 | CLINICAL NEUROLOGY | TRANSFERRIN RECEPTOR | PLASMA-LEVELS | IMMUNE ACTIVATION | INFLAMMATORY RESPONSE | REUPTAKE INHIBITOR THERAPY | HPA-AXIS | BRAIN | Depressive Disorder, Major - blood | Humans | Middle Aged | Receptors, Interleukin-2 - blood | Logistic Models | Male | Depressive Disorder, Major - immunology | Tumor Necrosis Factor-alpha - analysis | Young Adult | Interleukin-6 - blood | Adolescent | Adult | Female | Aged | Child | Cytokines - blood | Receptors, Interleukin-2 - analysis
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8. Full Text Cumulative meta-analysis of interleukins 6 and 1β, tumour necrosis factor α and C-reactive protein in patients with major depressive disorder
by Haapakoski, Rita and Mathieu, Julia and Ebmeier, Klaus P and Alenius, Harri and Kivimäki, Mika
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 49, pp. 206 - 215
Highlights • This meta-analysis confirms a robust link between IL-6, CRP and major depression. • The role of TNF-α and IL-1β in major depression remains... 
Psychiatry | Allergy and Immunology | Tumour necrosis factor-α | C-reactive protein | Major depression | Interleukin-1β | Inflammation | Cumulative meta-analysis | Interleukin-6 | PSYCHIATRY | CELL-MEDIATED-IMMUNITY | CYTOKINE LEVELS | IMMUNOLOGY | NEUROSCIENCES | Interleukin-1 beta | INFLAMMATORY MARKERS | SOLUBLE INTERLEUKIN-2 | ACUTE-PHASE PROTEINS | IL-6 LEVELS | PLASMA-LEVELS | SERUM-LEVELS | TREATMENT RESPONSE | TNF-ALPHA | Tumour necrosis factor-alpha | Depressive Disorder, Major - blood | Inflammation Mediators - immunology | Humans | Tumor Necrosis Factor-alpha - blood | Inflammation Mediators - blood | Interleukin-1beta - immunology | Depressive Disorder, Major - complications | Male | Inflammation - immunology | Depressive Disorder, Major - immunology | Inflammation - blood | Interleukin-1beta - blood | Inflammation - complications | C-Reactive Protein - metabolism | Interleukin-6 - blood | Interleukin-6 - immunology | Tumor Necrosis Factor-alpha - immunology | Adult | Female | C-Reactive Protein - immunology | Index Medicus
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9. Full Text Tumor necrosis factor-α triggers a cytokine cascade yielding postoperative cognitive decline
by Niccolò Terrando and Claudia Monaco and Daqing Ma and Brian M. J. Foxwell and Marc Feldmann and Mervyn Maze
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 47, pp. 20518 - 20522
Cognitive decline following surgery in older individuals is a major clinical problem of uncertain mechanism; a similar cognitive decline also follows severe... 
Delirium | Cytokines | Memory | Rheumatoid arthritis | Antibodies | Tumor necrosis factors | Inflammation | Mice | Behavioral neuroscience | Microglia | RHEUMATOID-ARTHRITIS | CRITICAL ILLNESS | MULTIDISCIPLINARY SCIENCES | LETHAL BACTEREMIA | RECEPTOR 4 | MECHANICALLY VENTILATED PATIENTS | surgical complications | innate immunity | INTENSIVE-CARE-UNIT | INFLAMMATION | dementia | DYSFUNCTION | MONOCLONAL-ANTIBODIES | delirium | BRAIN | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Toll-Like Receptor 4 - genetics | Inflammation - etiology | Postoperative Complications - immunology | Mice, Knockout | Inflammation - complications | Signal Transduction - immunology | Animals | Cognition Disorders - etiology | Cytokines - toxicity | HMGB1 Protein - metabolism | Cognition Disorders - immunology | Myeloid Differentiation Factor 88 - metabolism | Biological Sciences
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10. Full Text Bioequivalence of biosimilar tumor necrosis factor-α inhibitors compared with their reference biologics: A systematic review
by Chingcuanco, Francine and Segal, Jodi B and Kim, Seoyoung C and Alexander, G. Caleb
Annals of Internal Medicine, ISSN 0003-4819, 10/2016, Volume 165, Issue 8, pp. 565 - 574
Background: Biosimilars are of growing clinical, regulatory, and commercial importance. Purpose: To summarize evidence about the bioequivalence between... 
MEDICINE, GENERAL & INTERNAL | INFLAMMATORY-BOWEL-DISEASE | INNOVATOR INFLIXIMAB | EFFICACY | SAFETY | PARALLEL-GROUP | DOUBLE-BLIND | COMPARATIVE PHARMACOKINETICS | ANKYLOSING-SPONDYLITIS | CT-P13 | CLINICAL-EXPERIENCE | Inflammatory Bowel Diseases - drug therapy | Biosimilar Pharmaceuticals - therapeutic use | Arthritis, Rheumatoid - drug therapy | Humans | Antibody Formation | Biosimilar Pharmaceuticals - pharmacokinetics | Biosimilar Pharmaceuticals - pharmacology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Biosimilar Pharmaceuticals - adverse effects | Therapeutic Equivalency
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