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Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 6, pp. 2026 - 2038
Journal Article
Cell death & disease, ISSN 2041-4889, 2012, Volume 3, Issue 2, pp. e271 - e271
...://www.nature.com/cddis Web End =www.nature.com/cddis Inhibition of Casein kinase-2 induces p53-dependent cell cycle arrest and sensitizes glioblastoma cells to tumor necrosis... 
NF-κB | TNFα | Casein kinase-2 | Glioblastoma | p53 | GLIOMA-CELLS | ACTIVATION | TNF alpha | PROTEIN-KINASE | DOWN-REGULATION | CK2 | INDUCTION | CANCER | CELL BIOLOGY | glioblastoma | NF-kappa B | GROWTH ARREST | NF-KAPPA-B | casein kinase-2 | RNA, Small Interfering - genetics | Protein Binding - genetics | Proto-Oncogene Proteins c-mdm2 - genetics | Apigenin - therapeutic use | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | NF-kappa B - metabolism | Casein Kinase II - genetics | Dichlororibofuranosylbenzimidazole - pharmacology | Sirtuin 1 - genetics | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Brain - metabolism | Telomerase - genetics | Transfection | Glioblastoma - genetics | Glioblastoma - metabolism | Telomerase - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Signal Transduction | Casein Kinase II - antagonists & inhibitors | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | Sirtuin 1 - antagonists & inhibitors | Brain - drug effects | Tumor Necrosis Factor-alpha - pharmacology | NF-kappa B - genetics | Cell Cycle Checkpoints - drug effects | Dichlororibofuranosylbenzimidazole - therapeutic use | Glioblastoma - pathology | Brain - pathology | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Apigenin - pharmacology | Apoptosis | Original
Journal Article
Journal Article
International journal of cancer, ISSN 0020-7136, 10/2011, Volume 129, Issue 8, pp. 2032 - 2037
Journal Article
American Journal of Epidemiology, ISSN 0002-9262, 2/2010, Volume 171, Issue 3, pp. 267 - 276
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 12, p. e52298
Tumor Necrosis Factor (TNF)-alpha, is a paracrine inhibitor of melanocytes, which plays a critical role in the pathogenesis of several autoimmune diseases including vitiligo, as abnormal immune responses have frequently... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | LINKAGE DISEQUILIBRIUM | GENERALIZED VITILIGO | GENETIC ASSOCIATION | GUJARAT POPULATION | AUTOIMMUNE-DISEASES | ANTIOXIDANT STATUS | MULTIDISCIPLINARY SCIENCES | MONOCLONAL-ANTIBODY | TNF-ALPHA POLYMORPHISMS | CLASS-II | Haplotypes | Tumor Necrosis Factor-alpha - metabolism | Promoter Regions, Genetic | Genetic Predisposition to Disease | Genetic Association Studies | Gene Frequency | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Vitiligo - immunology | Gene Expression Regulation | Male | Disease Progression | Polymorphism, Genetic | Vitiligo - pathology | Age of Onset | Adult | Female | Vitiligo - genetics | Disease | Transcription | Pathogenesis | Genes | Paracrine signalling | Melanocytes | Biochemistry | Risk factors | Tropical diseases | Vitiligo | Control | Tumor necrosis factor-TNF | Enzyme-linked immunosorbent assay | Deoxyribonucleic acid--DNA | Lupus | Immune response | Psoriasis | Cytokines | Anemia | Tumor necrosis factor-α | Risk analysis | Disease control | Patients | Polymerase chain reaction | Studies | Restriction fragment length polymorphism | Genotyping | Gene frequency | Correlation analysis | Rheumatoid arthritis | TNF inhibitors | Diabetes | Autoimmune diseases | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2011, Volume 286, Issue 22, pp. 19297 - 19310
Journal Article
Frontiers in immunology, ISSN 1664-3224, 2018, Volume 9, p. 1274
...). The expression of tumor necrosis factor receptor-2 (TNFR2) on Tregs could be used to identify the maximally suppressive Treg population, and INF-alpha furtherly promoted the expansion and function of Tregs via TNFR2 in mice... 
T helper cells | Tumor necrosis factor receptor-2 | Tumor necrosis factor-α | Regulatory T cells | Acute myeloid leukemia | ACUTE MYELOGENOUS LEUKEMIA | TREG | TNFR2 | ALPHA | tumor necrosis factor receptor-2 | IMMUNOLOGY | SUPPRESSION | tumor necrosis factor-alpha | regulatory T cells | acute myeloid leukemia | EXPRESSION | MYELODYSPLASTIC SYNDROMES | INCREASED FREQUENCY | tumor necrosis factor-α
Journal Article