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Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1109 - 1123
Imatinib mesylate (IM), a potent inhibitor of the BCR/ABL tyrosine kinase, has become standard first-line therapy for patients with chronic myeloid leukemia... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 04/2018, Volume 1864, Issue 4, pp. 1454 - 1460
Cholangiocarcinoma (CCA) represents a diverse group of epithelial cancers associated with the biliary tract, and can best be stratified anatomically into... 
Molecular targets | Microenvironment | Driver mutations | Biliary tract cancer | Cholangiocarcinoma | BILIARY-TRACT CANCER | INTRAHEPATIC CHOLANGIOCARCINOMA | CANCER-ASSOCIATED FIBROBLASTS | SOLID TUMORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHASE-II | CELL-PROLIFERATION | I DOSE-ESCALATION | BIOPHYSICS | SIGNALING PATHWAY | MUTATIONS | INHIBITOR | Epithelial Cells - metabolism | Apoptosis - drug effects | Cancer-Associated Fibroblasts - metabolism | Epithelial Cells - drug effects | Humans | Cancer-Associated Fibroblasts - drug effects | Apoptosis - genetics | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Bile Ducts - cytology | Bile Duct Neoplasms - immunology | Tumor Microenvironment - genetics | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Biomarkers, Tumor - metabolism | Costimulatory and Inhibitory T-Cell Receptors - antagonists & inhibitors | Bile Duct Neoplasms - genetics | Biomarkers, Tumor - antagonists & inhibitors | Costimulatory and Inhibitory T-Cell Receptors - immunology | Molecular Targeted Therapy - methods | Tumor Microenvironment - drug effects | Cholangiocarcinoma - therapy | Cholangiocarcinoma - immunology | Epithelial Cells - pathology | Bile Ducts - surgery | Chemoradiotherapy, Adjuvant - methods | Bile Duct Neoplasms - therapy | Cancer-Associated Fibroblasts - pathology | Cholangiocarcinoma - pathology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Neoadjuvant Therapy - methods | Costimulatory and Inhibitory T-Cell Receptors - metabolism | Cholangiocarcinoma - genetics | T-Lymphocytes - immunology | Biomarkers, Tumor - genetics | Bile Duct Neoplasms - pathology | Mutation | Bile Ducts - pathology
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 08/2016, Volume 235, pp. 245 - 258
The traditional drug delivery systems always suffer from the unexpected drug release during circulation and the sluggish release of drug in target site. To... 
One-demand drug release | Photodynamic-chemotherapy | Fullerene (C60) | Nanomedicine | ROS-manipulated drug release | Fullerene (C | SYSTEM | CELLS | RNAI | DRUG-DELIVERY | DOXORUBICIN | PHOTODYNAMIC ACTIVITY | Fullerene (C-60) | RELEASE | CHEMISTRY, MULTIDISCIPLINARY | PHOTOTHERMAL THERAPY | PHARMACOLOGY & PHARMACY | C-60 | COPOLYMER | Neoplasms - metabolism | Doxorubicin - therapeutic use | Reactive Oxygen Species - metabolism | Phosphatidylethanolamines - therapeutic use | Delayed-Action Preparations - chemistry | Polyethylene Glycols - chemistry | Doxorubicin - chemistry | Polyethylene Glycols - therapeutic use | Delayed-Action Preparations - administration & dosage | Drug Delivery Systems | Antibiotics, Antineoplastic - chemistry | Delayed-Action Preparations - pharmacokinetics | Phosphatidylethanolamines - pharmacokinetics | Female | Fullerenes - chemistry | Antibiotics, Antineoplastic - pharmacokinetics | Fullerenes - administration & dosage | Doxorubicin - administration & dosage | Cell Survival - drug effects | Fullerenes - therapeutic use | Polyethylene Glycols - pharmacokinetics | Phosphatidylethanolamines - administration & dosage | Doxorubicin - pharmacokinetics | Polyethylene Glycols - administration & dosage | Neoplasms - drug therapy | Antibiotics, Antineoplastic - administration & dosage | Animals | Antibiotics, Antineoplastic - therapeutic use | Tumor Burden - drug effects | Cell Line, Tumor | Delayed-Action Preparations - therapeutic use | Mice | Mice, Inbred BALB C | Neoplasms - pathology | Fullerenes - pharmacokinetics | Phosphatidylethanolamines - chemistry | Drugs | Chemotherapy | Drug delivery systems | Drug therapy | Cancer | Vehicles | Anthracyclines | Lasers | Photochemotherapy | Index Medicus
Journal Article