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ANTIMICROBIAL AGENTS AND CHEMOTHERAPY, ISSN 0066-4804, 06/2018, Volume 62, Issue 6, p. e00029-18
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 2013, Volume 304, Issue 6, pp. C508 - C518
Younce CW, Burmeister MA, Ayala JE. Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and... 
Heart | Glucagon-like peptide-1 | Hyperglycemia | CARDIAC-FUNCTION | PEPTIDE-1 RECEPTOR | OXIDATIVE STRESS | PHYSIOLOGY | GLP-1 | CONTRACTILE DYSFUNCTION | ER STRESS | glucagon-like peptide-1 | DIABETIC CARDIOMYOPATHY | heart | CELL BIOLOGY | hyperglycemia | ENDOTHELIAL-CELLS | ZINC-FINGER PROTEIN | Receptors, Glucagon - antagonists & inhibitors | Diabetic Cardiomyopathies - metabolism | Apoptosis - drug effects | Peptide Fragments - pharmacology | Exenatide | RNA, Messenger - biosynthesis | Isoquinolines - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - antagonists & inhibitors | Phosphorylation - drug effects | Transcription Factor CHOP - biosynthesis | Calcium-Binding Proteins - metabolism | Receptors, Glucagon - agonists | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Endoplasmic Reticulum Stress - drug effects | Colforsin - pharmacology | Heat-Shock Proteins - metabolism | Cells, Cultured | Hydrogen Peroxide - pharmacology | Rats | Receptors, Glucagon - metabolism | Sulfonamides - pharmacology | Unfolded Protein Response | Hypoglycemic Agents - pharmacology | Peptides - pharmacology | Hyperglycemia - metabolism | Membrane Proteins | Animals | Glucagon-Like Peptide-1 Receptor | Myocytes, Cardiac - physiology | Tunicamycin - pharmacology | Glucose - metabolism | Thapsigargin - pharmacology | Protein Kinase Inhibitors - pharmacology | Venoms - pharmacology | Enzyme Activation | Oxidative Stress - drug effects | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Transcription Factor CHOP - metabolism | HSP70 Heat-Shock Proteins | Care and treatment | Heart cells | Physiological aspects | Stress (Physiology) | Dosage and administration | Health aspects | Endoplasmic reticulum | Apoptosis | Molecules | Phosphorylation | Glucose | Enzyme kinetics | Rodents | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2013, Volume 33, Issue 3, pp. 595 - 604
Journal Article
Investigative ophthalmology & visual science, ISSN 0146-0404, 01/2019, Volume 60, Issue 1, pp. 265 - 273
Journal Article
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2008, Volume 28, Issue 52, pp. 14056 - 14061
Mutations in the hypoxia-inducible factor angiogenin (ANG) have been identified in Amyotrophic Lateral Sclerosis (ALS) patients, but the potential role of ANG... 
ALS | Neuroprotection | Hypoxia | Motoneuron | Angiogenin | SOD1 | hypoxia | PROTEIN | PROLIFERATION | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | NUCLEAR TRANSLOCATION | angiogenin | MOTOR-NEURON DEGENERATION | neuroprotection | ENDOTHELIAL-CELLS | motoneuron | MICE | MUTATIONS | PROGRESSION | RNA, Small Interfering - genetics | Insulin-Like Growth Factor I - pharmacology | Superoxide Dismutase - genetics | Gene Expression - drug effects | Embryo, Mammalian | Male | Mutagenesis, Site-Directed - methods | Amyotrophic Lateral Sclerosis - drug therapy | Neuroprotective Agents - pharmacology | Transfection - methods | Ribonuclease, Pancreatic - pharmacology | Time Factors | Female | Spinal Cord - cytology | Oncogene Protein v-akt - metabolism | Motor Neurons - drug effects | Disease Models, Animal | Ribonuclease, Pancreatic - therapeutic use | Cell Survival - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Ribonuclease, Pancreatic - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Excitatory Amino Acid Agonists - pharmacology | Gene Expression Regulation - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Animals | Androstadienes - pharmacology | Ribonuclease, Pancreatic - metabolism | Tunicamycin - pharmacology | Wortmannin | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Mice | RNA, Small Interfering - metabolism | Index Medicus | Brief Communications
Journal Article
Journal Article
Diabetologia, ISSN 0012-186X, 11/2012, Volume 55, Issue 11, pp. 2999 - 3009
Pancreatic beta cell destruction in type 1 diabetes may be mediated by cytokines such as IL-1β, IFN-γ and TNF-α. Endoplasmic reticulum (ER) stress and nuclear... 
Medicine & Public Health | Human Physiology | Cytokines | Nuclear factor-kappa-B | Type 1 diabetes | Metabolic Diseases | Pancreatic beta cell | Internal Medicine | Islets | Apoptosis | Endoplasmic reticulum stress | Unfolded protein response | ACTIVATION | PHOSPHORYLATION | ER STRESS | MECHANISMS | ENDOPLASMIC-RETICULUM STRESS | DIABETES-MELLITUS | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | TYPE-1 | INITIATION-FACTOR-2 | Tumor Necrosis Factor-alpha - metabolism | Transcription Factor CHOP - genetics | Interleukin-1beta - pharmacology | Diabetes Mellitus, Type 1 - metabolism | NF-kappa B - metabolism | Interferon-gamma - metabolism | Insulin-Secreting Cells - metabolism | Interleukin-1beta - metabolism | Phenylbutyrates - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Pancreatic Neoplasms | Cell Survival - physiology | Cell Survival - drug effects | Cytokines - metabolism | Endoplasmic Reticulum Stress - drug effects | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Diabetes Mellitus, Type 1 - pathology | Enzyme Inhibitors - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Cell Death - physiology | Insulin-Secreting Cells - drug effects | Signal Transduction - drug effects | Tunicamycin - pharmacology | Cell Line, Tumor | Thapsigargin - pharmacology | Signal Transduction - physiology | Mice | Insulin-Secreting Cells - pathology | Transcription Factor CHOP - metabolism | Unfolded Protein Response - physiology | Cytokines - pharmacology | Interferon-gamma - pharmacology | Endoplasmic Reticulum Stress - physiology | Index Medicus | Splicing | Diabetes mellitus | Tunicamycin | Data processing | Chaperones | Gene expression | Stress | Western blotting | Beta cells | Polymerase chain reaction | Signal transduction | Cell death | Protein folding | Interleukin 1 | gamma -Interferon | Islets of Langerhans | Pancreas | Endoplasmic reticulum | thapsigargin | Tumor necrosis factor- alpha
Journal Article
Neuropharmacology, ISSN 0028-3908, 06/2018, Volume 135, pp. 242 - 252
Journal Article
Journals of Gerontology - Series A Biological Sciences and Medical Sciences, ISSN 1079-5006, 2008, Volume 63, Issue 3, pp. 232 - 241
Fibroblasts from long-lived mutant mice are resistant to many forms of lethal injury as well as to the metabolic effects of rotenone and low-glucose medium.... 
Naked mole-rat | Oxidation | Longevity | Endoplasmic reticulum (ER) stress | Stress resistance | Comparative biology | LIFE-SPAN | comparative biology | OXIDATIVE STRESS | ER STRESS | oxidation | ANTIOXIDANT DEFENSES | endoplasmic reticulum (ER) stress | LONGEST-LIVING RODENT | GERIATRICS & GERONTOLOGY | stress resistance | naked mole-rat | UNFOLDED PROTEIN RESPONSE | CAENORHABDITIS-ELEGANS | DWARF MICE | AGE-RELATED-CHANGES | GERONTOLOGY | longevity | HETEROCEPHALUS-GLABER | Species Specificity | Endoplasmic Reticulum - metabolism | Mole Rats | Male | Dose-Response Relationship, Drug | Ultraviolet Rays | Mice, Mutant Strains | Rotenone - pharmacology | Female | Mice, Inbred DBA | Culture Media, Serum-Free - pharmacology | Paraquat - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Hydrogen Peroxide - pharmacology | Cadmium - pharmacology | Glucose - pharmacology | Hot Temperature | Animals | Fibroblasts - radiation effects | Tunicamycin - pharmacology | Fibroblasts - drug effects | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | Mice, Inbred BALB C | Methyl Methanesulfonate - pharmacology | Fibroblasts | Aging | Research | African mole rats | Properties | Health aspects | Studies | Ultraviolet radiation | Rodents | Genetics | Growth hormones | Age | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 10/2003, Volume 10, Issue 10, pp. 1188 - 1203
Effective execution of apoptosis requires the activation of caspases. However, in many cases, broad-range caspase inhibitors such as Z-VAD.fmk do not inhibit... 
ER stress | Serine proteases | Caspase inhibition | IL-3 | Phagocytosis | Apoptosis | phagocytosis | CLEARANCE | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | MITOCHONDRIA | FAMILY PROTEASES | apoptosis | CLEAVAGE | caspase inhibition | serine proteases | CELL BIOLOGY | BCL-2 | TNF-ALPHA | CYTOCHROME-C RELEASE | GRANZYME-B | Apoptosis - drug effects | Microscopy, Phase-Contrast | Humans | Brefeldin A - pharmacology | Sulfones - pharmacology | Interleukin-3 - pharmacology | Proto-Oncogene Proteins c-bcl-2 - metabolism | fas Receptor - immunology | Caspases - metabolism | Flow Cytometry | Amino Acid Chloromethyl Ketones - pharmacology | U937 Cells | Amino Acid Chloromethyl Ketones - metabolism | Caspase 3 | Caspases - chemistry | Fibroblasts - metabolism | Phagocytosis - drug effects | Amino Acid Chloromethyl Ketones - chemistry | Antibodies, Monoclonal - pharmacology | Gene Expression Regulation | Phagocytosis - physiology | Rats | Interleukin-3 - deficiency | Cell Adhesion - drug effects | Caspase Inhibitors | Blotting, Western | Cycloheximide - pharmacology | Proto-Oncogene Proteins c-bcl-2 - pharmacology | Animals | Cell Adhesion - physiology | Models, Biological | Tunicamycin - pharmacology | Fibroblasts - drug effects | Cell Line, Tumor | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | Serine Endopeptidases - metabolism | Apoptosis - physiology | HeLa Cells | Proto-Oncogene Proteins c-bcl-2 - genetics | Microscopy, Fluorescence | Index Medicus
Journal Article