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Journal of Clinical Investigation, ISSN 0021-9738, 09/2016, Volume 126, Issue 9, pp. 3263 - 3278
Journal Article
Obesity, ISSN 1930-7381, 01/2017, Volume 25, Issue 1, pp. 111 - 121
Objective To analyze whether a combination of quercetin (Q) and resveratrol (RSV) would induce a white adipose tissue (WAT) browning effect. Methods Thirty‐six... 
ENERGY | HOMEOSTASIS | ACTIVATION | NUTRITION & DIETETICS | C57BL/6J MICE | ADIPOCYTES | HIGH-FAT DIET | INSULIN-RESISTANCE | GLUCOSE | ENDOCRINOLOGY & METABOLISM | ADULT HUMANS | EXPRESSION | Body Weight | Bone Morphogenetic Protein 4 - genetics | Adipocytes - drug effects | Fatty Acid-Binding Proteins - metabolism | Stilbenes - pharmacology | RNA, Messenger - metabolism | Bone Morphogenetic Protein 4 - metabolism | Cyclooxygenase 2 - genetics | Adipose Tissue, White - physiology | Apoptosis Regulatory Proteins - genetics | Uncoupling Protein 1 - genetics | Uncoupling Protein 1 - metabolism | RNA, Messenger - genetics | Adipose Tissue, Brown - physiology | Rats | Amino Acid Transport Systems, Neutral - metabolism | Lipase - metabolism | Fatty Acid-Binding Proteins - genetics | Symporters - metabolism | Amino Acid Transport Systems, Neutral - genetics | Apoptosis Regulatory Proteins - metabolism | Animals | Diet | Quercetin - pharmacology | Adipocytes - metabolism | Symporters - genetics | Cyclooxygenase 2 - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, White - drug effects | Lipase - genetics | Obesity | Homeostasis | Mitochondrial DNA | Adipocytes | Gene expression | Fatty acids | Proteins | Studies | Thermogenesis | Rodents | Morphology | Polyphenols | Protein expression | Oxidation | Deoxyribonucleic acid--DNA
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1120 - 1130
Cachexia represents a fatal energy-wasting syndrome in a large number of patients with cancer that mostly results in a pathological loss of skeletal muscle and... 
MEDICINE, RESEARCH & EXPERIMENTAL | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | CELL-DEATH | CELL BIOLOGY | WHOLE-BODY LIPOLYSIS | OBESITY | ADIPOCYTE LIPOLYSIS | METABOLISM | ENERGY HOMEOSTASIS | AICAR | LIPID HOMEOSTASIS | Neoplasms - metabolism | Uncoupling Protein 1 - drug effects | AMP-Activated Protein Kinases - metabolism | Lipolysis - drug effects | AMP-Activated Protein Kinases - pharmacology | Uncoupling Protein 1 - metabolism | Cells, Cultured | Adipose Tissue, White - metabolism | Peptide Fragments - pharmacology | Apoptosis Regulatory Proteins - drug effects | Apoptosis Regulatory Proteins - metabolism | Neoplasms - complications | Adipocytes, White - drug effects | Animals | Lipogenesis - drug effects | Cachexia - etiology | Lipid Metabolism - drug effects | Mice | Thermogenesis - drug effects | In Vitro Techniques | Cachexia - metabolism | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects | Adipose tissues | Peptides | Cachexia | Genetic aspects | Research | Protein kinases | Health aspects | Type 2 diabetes | Muscles | Cancer | Phosphates | Adipose tissue | Deactivation | Preservation | AMP | Homeostasis | Adipocytes | Shielding | Kinases | Inactivation | AMP-activated protein kinase | Skeletal muscle | Proteins | Thermogenesis | Energy | Uncoupling protein 1 | Tumors | Biological Sciences | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinsk bioteknologi | Cell and Molecular Biology | Medical Biotechnology (with a focus on Cell Biology (including Stem Cell Biology), Molecular Biology, Microbiology, Biochemistry or Biopharmacy) | Medicinsk bioteknologi (med inriktning mot cellbiologi (inklusive stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci) | Medical Biotechnology | Naturvetenskap | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Natural Sciences | Cell- och molekylärbiologi
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 02/2018, Volume 496, Issue 2, pp. 287 - 293
Recent studies have highlighted recruiting and activating brite adipocytes in WAT (so-called “browning”) would be an attractive anti-obesity strategy. Zinc... 
Obesity | Browning | Mitochondrial biogenesis | Zinc alpha2 glycoprotein | WHITE | ACTIVATION | LIPID-METABOLISM | BETA-ADRENERGIC-RECEPTORS | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | BIOPHYSICS | FAT-LIKE PHENOTYPE | 3T3-L1 | ADIPOSE-TISSUE | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Carnitine O-Palmitoyltransferase - genetics | Adipocytes, Brown - metabolism | Glycoproteins - metabolism | Nuclear Respiratory Factor 1 - genetics | DNA-Binding Proteins - metabolism | Carbon-Carbon Ligases - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Isoquinolines - pharmacology | Carnitine O-Palmitoyltransferase - metabolism | Nuclear Respiratory Factor 1 - metabolism | Apoptosis Regulatory Proteins - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Lipid Metabolism - genetics | NF-E2-Related Factor 2 - genetics | Carbon-Carbon Ligases - genetics | Cyclic AMP-Dependent Protein Kinases - metabolism | Glycoproteins - genetics | Uncoupling Protein 1 - genetics | Signal Transduction | Adipocytes, Brown - drug effects | Uncoupling Protein 1 - metabolism | Gene Expression Regulation | Imidazoles - pharmacology | Lipase - metabolism | Transcription Factors - genetics | 3T3-L1 Cells | DNA-Binding Proteins - genetics | Sulfonamides - pharmacology | Apoptosis Regulatory Proteins - metabolism | Transcription Factors - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | NF-E2-Related Factor 2 - metabolism | Mice | Pyridines - pharmacology | Adipocytes, Brown - cytology | Lipase - genetics | Lipids | Biosynthesis | Lipase | Body weight | Stem cells
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 11/2016, Volume 6, Issue 1, p. 34747
Nesfatin-1, an 82 amino acid gastric peptide, is involved in regulation of food uptake and in multiple metabolic activities. Whether nesfatin-1 modulates the... 
THERMOGENESIS | TARGET | LIPID-METABOLISM | WHITE ADIPOSE-TISSUES | FOOD-INTAKE | MULTIDISCIPLINARY SCIENCES | MOUSE | DIFFERENTIATION | DIET-INDUCED OBESITY | MODULATION | TRANSDIFFERENTIATION | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Leucine - pharmacology | Lipolysis - drug effects | Adipocytes, Brown - metabolism | TOR Serine-Threonine Kinases - metabolism | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Lipolysis - genetics | Mitochondrial Proton-Translocating ATPases - genetics | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | TOR Serine-Threonine Kinases - genetics | Gene Deletion | Tumor Suppressor Proteins - genetics | Nerve Tissue Proteins - pharmacology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Phosphorylation - drug effects | Calcium-Binding Proteins - metabolism | DNA-Binding Proteins - pharmacology | Tumor Suppressor Proteins - metabolism | Uncoupling Protein 1 - genetics | Adipocytes, Brown - drug effects | Uncoupling Protein 1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Mice, Transgenic | Signal Transduction - genetics | Lipase - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Mitochondrial Proton-Translocating ATPases - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Ribosomal Protein S6 Kinases, 90-kDa - genetics | Tuberous Sclerosis Complex 1 Protein | Mice | Calcium-Binding Proteins - pharmacology | Primary Cell Culture | Adipocytes, Brown - cytology | Calcium-Binding Proteins - genetics | Lipase - genetics | TOR protein | Phosphorylation | Clonal deletion | Amino acids | mRNA | Lipid metabolism | Leucine | Adipocytes | TSC1 protein | Lipolysis | Food
Journal Article
FASEB Journal, ISSN 0892-6638, 01/2017, Volume 31, Issue 1, pp. 333 - 345
Increasing energy expenditure through activation of brown adipose tissue (BAT) is a critical approach to treating obesity and diabetes. In this study, rutin, a... 
Energy expenditure | Obesity | Brown adipose tissue | Mitochondria | Rutin | RESVERATROL | mitochondria | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT HUMANS | rutin | energy expenditure | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | SIRT1 | CELL BIOLOGY | GLUCOSE-HOMEOSTASIS | WHITE FAT | brown adipose tissue | METABOLISM | BIOLOGY | MICE | ADIPOSE-TISSUE THERMOGENESIS | obesity | Sirtuin 1 - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Cold Temperature | Glucose Tolerance Test | Uncoupling Protein 1 - genetics | Body Temperature Regulation - physiology | High Mobility Group Proteins - metabolism | Obesity - drug therapy | Uncoupling Protein 1 - metabolism | Humans | Rutin - pharmacology | Adipose Tissue, Brown - physiology | Diet, High-Fat - adverse effects | DNA-Binding Proteins - genetics | Sirtuin 1 - genetics | DNA-Binding Proteins - metabolism | Animals | HEK293 Cells | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Body Temperature Regulation - drug effects | Mice, Obese | Mice | Energy Metabolism - drug effects | High Mobility Group Proteins - genetics | Energy metabolism | Animal models | Adipose tissue | Diabetes mellitus | Homeostasis | Metabolism | SIRT1 protein | Side effects | Rodents | Adipose tissue (brown) | Metabolic disorders
Journal Article
Journal Article
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 10/2016, Volume 24, Issue 4, pp. 555 - 565
Journal Article