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Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2009, Volume 183, Issue 8, pp. 5418 - 5427
IFN-beta-1a has been used over the past 15 years as a primary therapy for relapsing-remitting multiple sclerosis (MS). However, the immunomodulatory mechanisms... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | T(H)17 CELLS | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | MESSENGER-RNA EXPRESSION | CENTRAL-NERVOUS-SYSTEM | INTERFERON-BETA | IMMUNOLOGY | UP-REGULATION | T-CELLS | Receptors, CCR6 - immunology | Interleukin-12 Subunit p35 - metabolism | Interleukin-23 Subunit p19 - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Receptors, CCR6 - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin - metabolism | Interleukin-23 Subunit p19 - metabolism | STAT1 Transcription Factor - metabolism | Interleukin-1beta - metabolism | Receptors, Retinoic Acid - immunology | Phosphorylation - immunology | Phosphorylation - drug effects | Receptors, Thyroid Hormone - immunology | Interleukin-1beta - antagonists & inhibitors | Nuclear Receptor Subfamily 1, Group F, Member 3 | Receptors, Interleukin - immunology | Receptors, Interleukin - antagonists & inhibitors | Receptors, Retinoic Acid - metabolism | Cell Differentiation - immunology | Interferon beta-1a | Up-Regulation - immunology | Interleukin-23 Subunit p19 - immunology | STAT3 Transcription Factor - immunology | Interleukin-10 - immunology | Interleukin-17 - immunology | STAT1 Transcription Factor - drug effects | T-Lymphocytes, Helper-Inducer - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Receptors, CCR6 - metabolism | Interleukin-10 - metabolism | Dendritic Cells - drug effects | Interleukin-10 - agonists | STAT3 Transcription Factor - metabolism | Cells, Cultured | Interleukin-1beta - immunology | Interleukin-12 Subunit p35 - agonists | Down-Regulation - drug effects | STAT1 Transcription Factor - immunology | Receptors, Thyroid Hormone - metabolism | Up-Regulation - drug effects | Interferon-beta - pharmacology | Interleukin-12 Subunit p35 - immunology | Cell Differentiation - drug effects | Down-Regulation - immunology | STAT3 Transcription Factor - drug effects | Multiple Sclerosis - immunology | CD4-Positive T-Lymphocytes - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
Science, ISSN 0036-8075, 05/2016, Volume 352, Issue 6286, pp. 712 - 716
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 07/2008, Volume 205, Issue 7, pp. 1517 - 1522
Some immunologists have characterized T helper (Th)17 T cells as the master mediators of tissue damage in a variety of pathological conditions. New data now... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | ROR-GAMMA-T | MULTIPLE-SCLEROSIS | IFN-GAMMA | EFFECTOR RESPONSE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | BIOLOGICAL-ACTIVITIES | IMMUNOLOGY | T-H-17 CELLS | T-HELPER-CELLS | Dendritic Cells - immunology | Humans | Central Nervous System - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Interleukin-23 - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Disease Susceptibility | Macrophages - pathology | Interleukin-17 - genetics | Interleukin-12 Subunit p35 - genetics | Interleukin-23 - immunology | Mice, Knockout | Models, Immunological | Up-Regulation - immunology | Uveitis - immunology | Mice | Uveitis - genetics | Proteins - antagonists & inhibitors | Eye - pathology | Chemokines, CXC - immunology | Interleukin-17 - immunology | Autoimmunity - genetics | Neutralization Tests | Adoptive Transfer | Paralysis - pathology | Th1 Cells - immunology | Central Nervous System - immunology | Eye - immunology | Nitric Oxide Synthase Type II - immunology | Female | Interferon-gamma - genetics | Macrophages - immunology | Neutrophils - pathology | Inflammation Mediators - immunology | Cell Line | Encephalomyelitis, Autoimmune, Experimental - pathology | Myelin Sheath - immunology | Myelin Sheath - pathology | Gene Expression Regulation - immunology | Uveitis - prevention & control | Mice, Inbred C57BL | Neutrophils - immunology | Paralysis - immunology | Up-Regulation - genetics | Proteins - immunology | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Animals | Interleukin-12 Subunit p35 - immunology | Interleukin-17 - antagonists & inhibitors | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Antigens - immunology | Autoimmunity - drug effects | Uveitis - pathology | Index Medicus | Commentaries
Journal Article
Journal Article
Blood, ISSN 0006-4971, 03/2005, Volume 105, Issue 6, pp. 2473 - 2479
The tyrosine kinase inhibitor imatinib (imatinib, ST1571, Glivec, and Gleevec) is increasingly used in patients undergoing allogeneic transplantation for... 
CHRONIC MYELOGENOUS LEUKEMIA | RHEUMATOID-ARTHRITIS | LYMPHOCYTE RESPONSES | DENDRITIC CELLS | VIRUS INFECTION | TYROSINE KINASE | HEALTHY-INDIVIDUALS | STEM-CELL | HEMATOLOGY | ANTIGEN RECEPTOR | CHRONIC MYELOID-LEUKEMIA | Immunodominant Epitopes - immunology | CD8-Positive T-Lymphocytes - cytology | Humans | Graft vs Host Disease - immunology | Stem Cell Transplantation | Lymphocyte Activation - immunology | Protein Kinase Inhibitors - immunology | Adaptor Proteins, Signal Transducing - immunology | Receptors, Antigen, T-Cell - immunology | Phosphorylation - drug effects | Protein Processing, Post-Translational - immunology | Jurkat Cells | Lectins, C-Type | Leukemia - therapy | Piperazines - therapeutic use | Pyrimidines - pharmacology | Piperazines - pharmacology | Lymphocyte Specific Protein Tyrosine Kinase p56(lck) - antagonists & inhibitors | Signal Transduction - drug effects | Up-Regulation - immunology | Leukemia - immunology | ZAP-70 Protein-Tyrosine Kinase - immunology | Graft vs Leukemia Effect - immunology | CD8-Positive T-Lymphocytes - immunology | Antigens, CD - immunology | Pyrimidines - immunology | Lymphocyte Specific Protein Tyrosine Kinase p56(lck) - immunology | Transplantation, Homologous | Dose-Response Relationship, Drug | Signal Transduction - immunology | Interleukin-2 - immunology | Protein Processing, Post-Translational - drug effects | Antigens, Differentiation, T-Lymphocyte - immunology | Herpesvirus 4, Human - immunology | Membrane Proteins - immunology | Clinical Trials as Topic | Imatinib Mesylate | Receptors, Interleukin-2 - immunology | Sequence Homology, Nucleic Acid | Up-Regulation - drug effects | Cytomegalovirus - immunology | Lymphocyte Activation - drug effects | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Piperazines - immunology | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Benzamides | Genome, Human - immunology | Protein Structure, Tertiary - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2006, Volume 36, Issue 2, pp. 361 - 370
Toll-like receptors (TLR) represent an ancient front-line defence system that enables the host organism to sense the presence of microbial components within... 
Human | Lipopolysaccharide | Inflammation | Monocytes/macrophages | TLR | CELLS | ACTIVATION | CYTOKINES | PROTEIN | monocytes/macrophages | FACTOR-KAPPA-B | IMMUNOLOGY | 1-ALPHA,25-DIHYDROXYVITAMIN D-3 | TOLL-LIKE RECEPTOR-2 | inflammation | FLOW-CYTOMETRY | CD14 | lipopolysaccharide | human | BINDING | Inflammation - pathology | Cholecalciferol - immunology | Toll-Like Receptor 4 - biosynthesis | Humans | Monocytes - metabolism | Monocytes - immunology | Lipopolysaccharides - immunology | Dose-Response Relationship, Drug | Calcitriol - analogs & derivatives | Signal Transduction - immunology | Inflammation - metabolism | Teichoic Acids - pharmacology | Monocytes - pathology | Transcription Factor RelA - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Lipopolysaccharide Receptors - biosynthesis | Receptors, Calcitriol - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - immunology | Macrophage Activation - immunology | Cells, Cultured | Lipopolysaccharide Receptors - immunology | Inflammation - immunology | Teichoic Acids - immunology | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Receptors, Calcitriol - metabolism | Down-Regulation - drug effects | Toll-Like Receptor 2 - biosynthesis | Up-Regulation - drug effects | Receptors, Calcitriol - immunology | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Up-Regulation - immunology | Down-Regulation - immunology | Active Transport, Cell Nucleus - drug effects | Calcitriol - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Active Transport, Cell Nucleus - immunology | Macrophage Activation - drug effects | Cholecalciferol - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2013, Volume 190, Issue 3, pp. 1276 - 1284
Pseudomonas aeruginosa persists in patients with cystic fibrosis (CF) and drives CF lung disease progression. P. aeruginosa potently activates the innate... 
AIRWAY EPITHELIAL-CELLS | TRANSMEMBRANE CONDUCTANCE REGULATOR | IMMUNE-RESPONSE | TOLL-LIKE RECEPTOR-5 | ANTIINFLAMMATORY TARGET | INFLAMMATION | ALLERGIC BRONCHOPULMONARY ASPERGILLOSIS | MICE | IMMUNOLOGY | TLR5 | LYMPHOCYTES | Pseudomonas aeruginosa - immunology | T-Lymphocyte Subsets - immunology | Flagella - physiology | Lung - microbiology | Pseudomonas Infections - etiology | Pseudomonas aeruginosa - isolation & purification | Humans | Pneumonia, Bacterial - etiology | Culture Media, Conditioned - pharmacology | Male | Flagellin - pharmacology | Young Adult | Pseudomonas aeruginosa - pathogenicity | Immune Tolerance - immunology | Myeloid Cells - immunology | Flagellin - genetics | Adult | Female | Flagella - immunology | Myeloid Cells - drug effects | Receptors, CXCR4 - genetics | Cells, Cultured - immunology | Flagellin - immunology | Pneumonia, Bacterial - microbiology | Disease Susceptibility | Cystic Fibrosis - microbiology | Gene Expression Regulation - immunology | Bacterial Proteins - genetics | Cystic Fibrosis - complications | Immunity, Innate | Pseudomonas Infections - microbiology | Pseudomonas Infections - immunology | Toll-Like Receptor 5 - immunology | Pseudomonas aeruginosa - genetics | Receptors, CXCR4 - biosynthesis | Receptors, CXCR4 - immunology | Up-Regulation - immunology | Adolescent | Th17 Cells - immunology | Pneumonia, Bacterial - immunology | Immune Evasion - immunology | Myelopoiesis - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 02/2015, Volume 42, Issue 2, pp. 321 - 331
T helper 1 (Th1) cell-associated immunity exacerbates ileitis induced by oral infection. We show here that attenuated ileitis observed in interleukin-22... 
ORAL INFECTION | INTERFERON-GAMMA | IFN-GAMMA | IL-18 | INFLAMMATION | HOST-DEFENSE | TOXOPLASMA-GONDII | MICE | TNF-ALPHA | IMMUNOLOGY | T-CELLS | Toxoplasma - immunology | Up-Regulation | Interleukin-18 - immunology | Toxoplasmosis - immunology | Ileum - parasitology | Ileum - immunology | Th1 Cells - immunology | Toxoplasmosis - parasitology | Enterobacteriaceae Infections - immunology | Interleukins - genetics | RNA, Messenger - biosynthesis | Intestinal Mucosa - immunology | Interleukins - immunology | Organ Culture Techniques | Neutrophil Infiltration - immunology | Citrobacter rodentium - immunology | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Inflammation - immunology | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-18 - biosynthesis | Animals | Ileitis - parasitology | Ileitis - immunology | Epithelial Cells - immunology | Ileitis - microbiology | Mice | Ileum - microbiology | Interferon-gamma - biosynthesis | Intestinal Mucosa - parasitology | RNA | Interleukins | Health aspects | Gastrointestinal diseases | Medical research | Microbiology | Medicine, Experimental | Automation | Data analysis | Cytokines | Cysts | Rodents | Gangrene | Infections | Gene expression | Small intestine | Experiments | Immune system | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2014, Volume 193, Issue 7, pp. 3664 - 3675
Trehalose-6,6-dimycolate (TDM), the mycobacterial cord factor, and its synthetic analog Trehalose-6,6-dibehenate (TDB) bind to the C-type lectin receptors... 
SYK KINASE | MYCOBACTERIUM-TUBERCULOSIS | TARGETED DISRUPTION | CANDIDA-ALBICANS | C-TYPE LECTIN | INDUCIBLE FACTOR 1-ALPHA | GENE-EXPRESSION | NLRP3 INFLAMMASOME | IMMUNOLOGY | NF-KAPPA-B | INNATE IMMUNITY | Dendritic Cells - immunology | Lectins, C-Type - immunology | Protein Biosynthesis - immunology | Intracellular Signaling Peptides and Proteins - immunology | Lectins, C-Type - genetics | Enzyme Activation - immunology | Protein-Tyrosine Kinases - immunology | beta-Glucans - chemistry | Phosphorylation - genetics | Protein-Tyrosine Kinases - genetics | Nitric Oxide Synthase Type II - immunology | Early Growth Response Protein 1 - genetics | Myeloid Differentiation Factor 88 - immunology | Mycobacterium tuberculosis - chemistry | Phosphorylation - immunology | Protein Biosynthesis - genetics | Cord Factors - pharmacology | Phosphorylation - drug effects | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Macrophages - immunology | Hypoxia-Inducible Factor 1, alpha Subunit - immunology | Nitric Oxide - immunology | Early Growth Response Protein 1 - immunology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Membrane Proteins - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Cord Factors - chemistry | Toll-Like Receptors - immunology | Myeloid Differentiation Factor 88 - genetics | Membrane Proteins - immunology | Up-Regulation - genetics | Macrophages - cytology | beta-Glucans - pharmacology | Enzyme Activation - drug effects | Mice, Knockout | Up-Regulation - drug effects | Animals | Nitric Oxide Synthase Type II - genetics | Up-Regulation - immunology | CCAAT-Enhancer-Binding Protein-beta - immunology | Nitric Oxide - genetics | Toll-Like Receptors - genetics | Protein Biosynthesis - drug effects | Dendritic Cells - cytology | Mice | Granulocyte Colony-Stimulating Factor - immunology | Enzyme Activation - genetics | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Infectious Diseases, ISSN 0022-1899, 02/2015, Volume 211, Issue 3, pp. 416 - 425
Na-ASP-2 is an efficacious hookworm vaccine antigen. However, despite elucidation of its crystal structure and studies addressing its immunobiology, the... 
host'pathogen interaction | B cell | CD79A | Na-ASP-2 | hookworm | protein microarray | antigen receptor | Necator americanus | SCP/TAPS | host-pathogen interaction | INFECTIOUS DISEASES | VACCINE | MICROBIOLOGY | ALLERGEN | IMMUNOLOGY | NECATOR-AMERICANUS | RNA-SEQ | PURIFICATION | INFECTION | DIFFERENTIATION | EXPRESSION | ANTIGEN | RNA, Messenger - immunology | Proteome - genetics | CD79 Antigens - immunology | Humans | Middle Aged | Pre-B Cell Receptors - immunology | Phosphatidylinositol 3-Kinases - immunology | Protein Array Analysis - methods | Host-Pathogen Interactions - immunology | Signal Transduction - immunology | Ancylostomatoidea - immunology | Leukocytes, Mononuclear - immunology | Proteome - immunology | Adult | Hookworm Infections - immunology | RNA, Messenger - genetics | Cells, Cultured | Up-Regulation - genetics | Signal Transduction - genetics | src-Family Kinases - immunology | Down-Regulation - genetics | Phosphatidylinositol 3-Kinases - genetics | Antigens, Helminth - immunology | Animals | B-Lymphocytes - immunology | Host-Pathogen Interactions - genetics | Recombinant Proteins - immunology | Up-Regulation - immunology | Down-Regulation - immunology | Helminth Proteins - immunology | Transcription, Genetic - immunology | Transcription, Genetic - genetics | src-Family Kinases - genetics | Hookworm disease | Protein microarrays | Antigens | Usage | Research | B cells | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2015, Volume 212, Issue 1, pp. 23 - 35
A major function of T helper (Th) 17 cells is to induce the production of factors that activate and mobilize neutrophils. Although Th17 cells have been... 
PROGRESSIVE MULTIPLE-SCLEROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | INTERLEUKIN-17 FAMILY-MEMBERS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | COLONY-STIMULATING FACTOR | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | ENDOTHELIAL-CELLS | CD4(+) T-CELLS | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | G-CSF | HEMATOPOIETIC STEM | Myelin-Oligodendrocyte Glycoprotein - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Multiple Sclerosis - blood | Humans | Granulocyte Colony-Stimulating Factor - blood | Encephalomyelitis, Autoimmune, Experimental - immunology | Male | Th17 Cells - transplantation | Adoptive Transfer | Chemokine CXCL5 - metabolism | Signal Transduction - immunology | Hematopoietic Stem Cells - immunology | Flow Cytometry | Th17 Cells - metabolism | Myeloid Cells - immunology | Peptide Fragments - immunology | Chemokine CXCL5 - blood | Female | Receptors, Granulocyte Colony-Stimulating Factor - metabolism | Chemokine CXCL1 - metabolism | Neutrophils - metabolism | Granulocyte Colony-Stimulating Factor - metabolism | Multiple Sclerosis - metabolism | Biomarkers - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Encephalomyelitis, Autoimmune, Experimental - blood | Mice, Transgenic | Chemokine CXCL1 - blood | Hematopoietic Stem Cells - metabolism | Biomarkers - blood | Mice, Knockout | Chemokine CXCL1 - immunology | Animals | Up-Regulation - immunology | Chemokine CXCL5 - immunology | Multiple Sclerosis - immunology | Myeloid Cells - metabolism | Th17 Cells - immunology | Granulocyte Colony-Stimulating Factor - immunology | Receptors, Granulocyte Colony-Stimulating Factor - immunology | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2016, Volume 17, Issue 2, pp. 140 - 149
Innate sensing of pathogens initiates inflammatory cytokine responses that need to be tightly controlled. We found here that after engagement of Toll-like... 
PATHWAYS | INTERFERON | ACTIVATION | SUMO | BETA-GENE | IFN-GAMMA | NEGATIVE REGULATION | ENHANCER | MICE | IMMUNOLOGY | UP-REGULATION | Chromatin - metabolism | Dendritic Cells - immunology | Gene Expression Profiling | Genetic Loci | Shock, Septic - immunology | Lipopolysaccharides - immunology | Shock, Septic - metabolism | Inflammation - metabolism | Inflammation Mediators - metabolism | Toll-Like Receptors - metabolism | Dendritic Cells - metabolism | Disease Models, Animal | Receptor, Interferon alpha-beta - metabolism | Shock, Septic - genetics | Sumoylation - genetics | Disease Resistance | Disease Susceptibility | Cytokines - metabolism | Signal Transduction | Immunomodulation | Gene Expression Regulation | Inflammation - virology | Inflammation - immunology | Immunity, Innate | Mice, Knockout | Animals | Enhancer Elements, Genetic | Interferon-beta - metabolism | Regulatory Elements, Transcriptional | Sumoylation - immunology | Inflammation - genetics | Protein Binding | Mice | SUMO-1 Protein - metabolism | Chromatin - genetics | Analysis | Influence | Interferon | Inflammation | Research | Biological response modifiers | Gene expression | Health aspects | Risk factors | Index Medicus | Shock, Septic/metabolism | Inflammation Mediators/metabolism | Sumoylation/immunology | Interferon-beta/metabolism | Dendritic Cells/metabolism | Inflammation/virology | Inflammation/immunology | Life Sciences | Immunology | Receptor, Interferon alpha-beta/metabolism | Inflammation/genetics | SUMO-1 Protein/metabolism | Cytokines/metabolism | Sumoylation/genetics | Chromatin/metabolism | Inflammation/metabolism | Toll-Like Receptors/metabolism | Lipopolysaccharides/immunology | Shock, Septic/immunology | Chromatin/genetics | Shock, Septic/genetics | Dendritic Cells/immunology
Journal Article