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1. Full Text Piezo1 expression increases in rat bladder after partial bladder outlet obstruction
by Michishita, Mai and Yano, Kazuo and Tomita, Ken-ichi and Matsuzaki, Osamu and Kasahara, Ken-ichi
Life Sciences, ISSN 0024-3205, 12/2016, Volume 166, pp. 1 - 7
For patients with benign prostatic hyperplasia (BPH), storage symptoms due to bladder dysfunction are bothersome, and that mechanism elucidation is needed.... 
Rats | Smooth muscle | Piezo1 | Suburothelium | Bladder outlet obstruction | Urothelium | MEDICINE, RESEARCH & EXPERIMENTAL | DETRUSOR MUSCLE | INSTABILITY | RECEPTOR | MODEL | OVERACTIVITY | URINARY-TRACT | PHARMACOLOGY & PHARMACY | 5-HT2A | Up-Regulation | Urinary Bladder Neck Obstruction - pathology | Animals | Urinary Bladder - metabolism | Membrane Proteins - genetics | RNA, Messenger - genetics | Female | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder - pathology | Rats, Sprague-Dawley | Real-Time Polymerase Chain Reaction | Membrane Proteins - analysis
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2. Full Text The inflammatory cytokine IL‐1β is involved in bladder remodeling after bladder outlet obstruction in mice
by Kanno, Yukiko and Mitsui, Takahiko and Kitta, Takeya and Moriya, Kimihiko and Tsukiyama, Tadasuke and Hatakeyama, Shigetsugu and Nonomura, Katsuya
Neurourology and Urodynamics, ISSN 0733-2467, 03/2016, Volume 35, Issue 3, pp. 377 - 381
Aims We investigated the relationship between IL‐1β and morphological and functional changes following partial bladder outlet obstruction (pBOO). Methods... 
IGF‐1 | bladder outlet obstruction | IL‐1β | remodeling | hypertrophy | inflammation | IGF-1 | IL-1β | Urinary Bladder Neck Obstruction - pathology | Urinary Bladder Neck Obstruction - physiopathology | Urinary Bladder Neck Obstruction - genetics | Insulin-Like Growth Factor I - genetics | Urinary Bladder Neck Obstruction - metabolism | Interleukin-1beta - genetics | Urinary Bladder - pathology | Interleukin-1beta - deficiency | Time Factors | Interleukin-1beta - metabolism | Inflammation Mediators - metabolism | Insulin-Like Growth Factor I - administration & dosage | Female | Disease Models, Animal | Genetic Predisposition to Disease | Signal Transduction | Urinary Bladder - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Pressure | Mice, Knockout | Urodynamics | Phenotype | Animals | Transforming Growth Factor beta - genetics | Urinary Bladder - physiopathology | Urinary Bladder - drug effects | Cell Proliferation - drug effects | Urination | Transforming Growth Factor beta - metabolism | Insulin-Like Growth Factor I - metabolism | Hypertrophy
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3. Full Text Effect of silodosin, a selective alpha(1A)-adrenoceptor antagonist, on voiding behavior and bladder blood flow in a rat model of bladder outlet obstruction
by Goi, Y and Tomiyama, Y and Yokoyama, A and Tatemichi, S and Maruyama, K and Kobayashi, M and Yamaguchi, O
EUROPEAN JOURNAL OF PHARMACOLOGY, ISSN 0014-2999, 10/2015, Volume 764, pp. 489 - 496
This study was performed to investigate the effects of silodosin (selective alpha(1A)-adrenoceptor antagonist) on bladder blood flow (BBF) and bladder function... 
Bladder blood flow | ADRENOCEPTOR SUBTYPES | CHRONIC ISCHEMIA | Nerve growth factor | SYMPTOMS | silodosin | LOWER URINARY-TRACT | BENIGN PROSTATIC HYPERPLASIA | OVERACTIVE BLADDER | Ischemia | MEN | alpha(1A)-Adrenoceptor | PHARMACOLOGY & PHARMACY | DYSFUNCTION | EXPRESSION | ASSOCIATION | Bladder outlet obstruction | Immunohistochemistry | Receptors, Adrenergic, alpha-1 - metabolism | Ureteral Obstruction - complications | Urological Agents - pharmacology | Urinary Bladder Neck Obstruction - physiopathology | Urodynamics - drug effects | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - metabolism | Microcirculation - drug effects | Urinary Bladder - innervation | In Situ Hybridization | Time Factors | Adrenergic alpha-1 Receptor Antagonists - pharmacology | Urination - drug effects | Female | Indoles - pharmacology | Deoxyguanosine - analogs & derivatives | Disease Models, Animal | Deoxyguanosine - urine | Urinary Bladder - metabolism | Receptors, Adrenergic, alpha-1 - drug effects | Urinary Bladder Neck Obstruction - etiology | Laser-Doppler Flowmetry | Rats, Sprague-Dawley | Regional Blood Flow | Animals | Receptors, Adrenergic, alpha-1 - genetics | Blood Flow Velocity | Urinary Bladder - blood supply | Urinary Bladder - drug effects | Nerve Growth Factor - urine | Urinary Bladder Neck Obstruction - drug therapy
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4. Full Text Congenital lower urinary tract obstruction: a population‐based epidemiological study
by Malin, G and Tonks, AM and Morris, RK and Gardosi, J and Kilby, MD
BJOG: An International Journal of Obstetrics & Gynaecology, ISSN 1470-0328, 11/2012, Volume 119, Issue 12, pp. 1455 - 1464
Please cite this paper as: Malin G, Tonks A, Morris R, Gardosi J, Kilby M. Congenital lower urinary tract obstruction: a population‐based epidemiological... 
prevalence | Epidemiology | lower urinary tract obstruction (LUTO) | DIAGNOSIS | ULTRASOUND | POSTNATAL RENAL-FUNCTION | POSTERIOR URETHRAL VALVES | INTERVENTION | ENGLAND | ACCURACY | OBSTETRICS & GYNECOLOGY | Ultrasonography, Prenatal - statistics & numerical data | Urogenital Abnormalities - diagnostic imaging | Urinary Bladder Neck Obstruction - diagnostic imaging | Prevalence | Urogenital Abnormalities - epidemiology | Infant Mortality | Humans | Male | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - congenital | False Positive Reactions | Abortion, Spontaneous - epidemiology | Urethra - diagnostic imaging | England - epidemiology | Sensitivity and Specificity | Abnormalities, Multiple - epidemiology | Adult | Female | Registries | Retrospective Studies | Abnormalities, Multiple - genetics | Infant, Newborn | Abnormalities, Multiple - diagnostic imaging | Urinary Bladder Neck Obstruction - epidemiology | Pregnancy | Urethra - abnormalities | Chromosome Aberrations | Stillbirth - epidemiology | Abortion, Eugenic - statistics & numerical data | Urogenital Abnormalities - genetics | Medical colleges | Genetic disorders | Pregnant women | Health aspects | Prevalence studies (Epidemiology) | Congenital diseases | Fetuses | Urinary tract diseases
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5. Full Text Mir-29 repression in bladder outlet obstruction contributes to matrix remodeling and altered stiffness
by Ekman, Mari and Bhattachariya, Anirban and Dahan, Diana and Uvelius, Bengt and Albinsson, Sebastian and Swärd, Karl and Urology and Lund University and Kärlfysiologi and Cellulär biomekanik and Cellular Biomechanics and Urologi and Vascular Physiology and Lunds universitet
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e82308
Recent work has uncovered a role of the microRNA (miRNA) miR-29 in remodeling of the extracellular matrix. Partial bladder outlet obstruction is a prevalent... 
DETRUSOR | SMAD1 | GROWTH-FACTOR-BETA | ACTIVATION | MULTIDISCIPLINARY SCIENCES | ANEURYSM DEVELOPMENT | DISEASE | KAPPA-B | MICRORNAS | RAT URINARY-BLADDER | EXPRESSION | Urinary Bladder Neck Obstruction - pathology | Humans | Extracellular Matrix - metabolism | Rats | Male | MicroRNAs - biosynthesis | NF-kappa B - metabolism | Urinary Bladder Neck Obstruction - genetics | Smad3 Protein - metabolism | Urinary Bladder Neck Obstruction - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Extracellular Matrix - genetics | Smad3 Protein - genetics | Animals | NF-kappa B - genetics | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-myc - genetics | Extracellular Matrix - pathology | Protein biosynthesis | DNA microarrays | MicroRNA | Collagen | Analysis | Phosphorylation | Senescence | c-Myc protein | Bladder | Science | Smooth muscle | Smad3 protein | mRNA | Activation | Myc protein | Urinary tract | Kinases | Remodeling | Proteins | Transfection | Clonal deletion | Urinary bladder | Rodents | Osteonectin | Deletion | Extracellular matrix | Basal lamina | Stiffness | Collagen (type IV) | Growth factors | Enlargement | NF-κB protein | MiRNA | Mechanical properties | Muscles | Tropoelastin | Tamoxifen | Ribonucleic acids | Protein synthesis | MicroRNAs | Prostate | Cancer | Basic Medicine | Physiology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi
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6. Full Text Increased expression of TREK-1 K+ channel in the dorsal root ganglion of rats with detrusor overactivity after partial bladder outlet obstruction
by Zhang, Junlong and Cao, Mingxin and Chen, Yu and Wan, Zi and Wang, Hua and Lin, Huanyi and Liang, Weijie and Liang, Yueyou
Medical Science Monitor, ISSN 1234-1010, 02/2018, Volume 24, pp. 1064 - 1071
Background: Changes in expression and activity of ion channels are important pathophysiological mechanisms underlying detrusor overactivity (DO) in partial... 
Urinary bladder neck obstruction | Tandem pore domain | Overactive | Potassium channels | Urinary bladder | Potassium Channels | Tandem Pore Domain | BENIGN PROSTATIC HYPERPLASIA | MEDICINE, RESEARCH & EXPERIMENTAL | POSITIVE RESPONSE | Urinary Bladder Neck Obstruction | SPINAL-CORD | Urinary Bladder Overactive | ACTIVATED K | T-TYPE | ICE WATER TEST | LOWER URINARY-TRACT | Potassium Channels, Tandem Pore Domain - metabolism | Up-Regulation | Spinal Cord - metabolism | Urinary Bladder - metabolism | RNA, Messenger - genetics | Potassium Channels, Tandem Pore Domain - genetics | Rats | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder, Overactive - metabolism | Urinary Bladder Neck Obstruction - metabolism | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Animals | Urinary Bladder, Overactive - genetics | Female | Potassium Channels, Tandem Pore Domain - biosynthesis | Ganglia, Spinal - metabolism
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7. Full Text Muscarinic Acetylcholine Receptor M3 Mutation Causes Urinary Bladder Disease and a Prune-Belly-like Syndrome
by Weber, Stefanie and Thiele, Holger and Mir, Sevgi and Toliat, Mohammad Reza and Sozeri, Betül and Reutter, Heiko and Draaken, Markus and Ludwig, Michael and Altmüller, Janine and Frommolt, Peter and Stuart, Helen M and Ranjzad, Parisa and Hanley, Neil A and Jennings, Rachel and Newman, William G and Wilcox, Duncan T and Thiel, Uwe and Schlingmann, Karl Peter and Beetz, Rolf and Hoyer, Peter F and Konrad, Martin and Schaefer, Franz and Nürnberg, Peter and Woolf, Adrian S
The American Journal of Human Genetics, ISSN 0002-9297, 11/2011, Volume 89, Issue 5, pp. 668 - 674
Urinary bladder malformations associated with bladder outlet obstruction are a frequent cause of progressive renal failure in children. We here describe a... 
M-2 | GENE | RAT | EXPRESSION | MICE LACKING | GENETICS & HEREDITY | Immunohistochemistry | Urinary Bladder Neck Obstruction - pathology | Humans | Models, Molecular | INDEL Mutation - genetics | Male | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder - pathology | Receptor, Muscarinic M3 - genetics | Metabolism, Inborn Errors - genetics | Sequence Homology, Nucleic Acid | Urinary Bladder - embryology | Mice, Knockout | Prune Belly Syndrome - genetics | Animals | Prune Belly Syndrome - pathology | Base Sequence | Sex Factors | Female | Consanguinity | Mice | Receptor, Muscarinic M3 - deficiency | Frameshift Mutation - genetics | Bladder diseases | Gene mutations | Prune belly syndrome | Causes of | Genetic aspects | Research | Muscarinic receptors | Health aspects | Acetylcholine receptors (muscarinic) | Urinary bladder | Congenital defects | Renal failure | Frameshift mutation | Muscles | Children | Kidney | Report
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8. Full Text Detrusor induction of mir-132/212 following bladder outlet obstruction: Association with MeCP2 repression and cell viability
by Sadegh, Mardjaneh Karbalaei and Ekman, Mari and Krawczyk, Katarzyna and Svensson, Daniel and Göransson, Olga and Dahan, Diana and Nilsson, Bengt-Olof and Albinsson, Sebastian and Uvelius, Bengt and Swärd, Karl and Urology and Lund University and Protein Phosphorylation and Kärlfysiologi and Cellulär biomekanik and Proteinfosforylering and Cellular Biomechanics and Urologi and Vascular Physiology and Lunds universitet
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0116784
The microRNAs (miRNAs) miR-132 and miR-212 have been found to regulate synaptic plasticity and cholinergic signaling and recent work has demonstrated roles... 
HYPERTROPHY | COMPLEX | CREB | CIRCADIAN-CLOCK | RAT | MULTIDISCIPLINARY SCIENCES | INDUCED MICRORNA | DIFFERENTIATION | HYPERPLASIA | EXPRESSION | PLASTICITY | Urinary Bladder Neck Obstruction - pathology | Humans | RNA, Messenger - genetics | Cell Survival - genetics | Rats | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - metabolism | Muscle, Smooth - metabolism | RNA, Messenger - metabolism | Rats, Sprague-Dawley | GPI-Linked Proteins - metabolism | Methyl-CpG-Binding Protein 2 - deficiency | Methyl-CpG-Binding Protein 2 - genetics | Animals | Synapses - metabolism | Muscle Proteins - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Female | MicroRNAs - genetics | Muscle, Smooth - pathology | Acetylcholinesterase - metabolism | Gene expression | MicroRNA | Phosphorylation | Correlation | Protein kinase C | Transcription factors | Target recognition | Central nervous system | Bladder | Science | Smooth muscle | Synaptic density | Nuclei | Ethics | Reduction | Cell growth | Transfection | Urethra | Methyl-CpG binding protein | Urinary bladder | Rodents | DNA methylation | MiRNA | Muscles | Cyclic AMP response element-binding protein | Ribonucleic acid--RNA | Signaling | Plasticity (synaptic) | MeCP2 protein | MicroRNAs | Viability | Binding sites | Basic Medicine | Physiology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi | RNA | Ribonucleic acid
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9. Full Text Mechanical stretch upregulates proteins involved in Ca2+ sensitization in urinary bladder smooth muscle hypertrophy
by Boopathi, E and Gomes, C and Zderic, SA and Malkowicz, B and Chakrabarti, R and Patel, DP and Wein, AJ and Chacko, S
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY, ISSN 0363-6143, 09/2014, Volume 307, Issue 6, pp. C542 - C553
Partial bladder outlet obstruction (pBOO)-induced remodeling of bladder detrusor smooth muscle (DSM) is associated with the modulation of cell signals... 
CELLS | PHYSIOLOGY | cell stretch | RHO-KINASE | partial bladder outlet obstruction | CELL BIOLOGY | OUTLET OBSTRUCTION | SIGNAL-TRANSDUCTION | human bladder | IN-VITRO | GATA-6 | calcium sensitization | NONMUSCLE MYOSIN-II | CARDIAC-HYPERTROPHY | GENE-EXPRESSION | TRANSCRIPTION FACTOR | benign prostatic hyperplasia | Urinary Bladder Neck Obstruction - pathology | Cell Proliferation | Urinary Bladder Neck Obstruction - physiopathology | Humans | Middle Aged | Phosphoprotein Phosphatases - metabolism | Myocytes, Smooth Muscle - pathology | Male | NF-kappa B - metabolism | Urinary Bladder Neck Obstruction - genetics | rhoA GTP-Binding Protein - metabolism | Urinary Bladder Neck Obstruction - metabolism | Urinary Bladder - pathology | rhoA GTP-Binding Protein - genetics | RNA, Messenger - metabolism | Prostatic Hyperplasia - complications | Time Factors | rho-Associated Kinases - metabolism | Transcription, Genetic | Binding Sites | Myocytes, Smooth Muscle - metabolism | Calcium Signaling | Disease Models, Animal | Promoter Regions, Genetic | Urinary Bladder - metabolism | Urinary Bladder Neck Obstruction - etiology | rho GTP-Binding Proteins - genetics | Cells, Cultured | Gene Expression Regulation | rho-Associated Kinases - genetics | Muscle, Smooth - metabolism | GATA6 Transcription Factor - metabolism | Mechanotransduction, Cellular | Animals | Muscle Contraction | Phosphoprotein Phosphatases - genetics | rho GTP-Binding Proteins - metabolism | Muscle, Smooth - physiopathology | Urinary Bladder - physiopathology | Aged | Mice | Muscle, Smooth - pathology | Hypertrophy
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10. Full Text Chronic Cyclic Bladder Over Distention Up-Regulates Hypoxia Dependent Pathways
by Stephany, Heidi A and Strand, Douglas W and Ching, Christina B and Tanaka, Stacy T and Milne, Ginger L and Cajaiba, Mariana M and Thomas, John C and Pope, John C and Adams, Mark C and Brock, John W and Hayward, Simon W and Matusik, Robert J and Clayton, Douglass B
Journal of Urology, The, ISSN 0022-5347, 2013, Volume 190, Issue 4, pp. 1603 - 1609
Purpose Bladder over distention secondary to anatomical or functional obstruction can eventually lead to pathological changes, including decreased elasticity... 
Urology | urinary bladder neck obstruction | urinary bladder | anoxia | oxidative stress | gene expression | oxidative stress anoxia gene expression urinary bladder neck obstruction | UROLOGY & NEPHROLOGY | Up-Regulation | Urinary Bladder Neck Obstruction - pathology | Oxidative Stress | Urinary Bladder - metabolism | Mice, Inbred C57BL | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - metabolism | Urinary Bladder - pathology | Hypoxia - metabolism | RNA - genetics | Hypoxia - genetics | Animals | Glucose Transporter Type 1 - biosynthesis | Glucose Transporter Type 1 - genetics | Hypoxia - pathology | Urinary Bladder - physiopathology | Female | Mice | Urination | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Medical colleges | Analysis
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11. Full Text Non-uniform changes in membrane receptors in the rat urinary bladder following outlet obstruction
by Zeng, Jianwen and Ekman, Mari and Jiang, Chonghe and Uvelius, Bengt and Swärd, Karl and Cellular Biomechanics and Urology and Urologi and Lund University and Lunds universitet and Cellulär biomekanik
European Journal of Pharmacology, ISSN 0014-2999, 09/2015, Volume 762, pp. 82 - 88
The aim of the present study was to investigate the expression and distribution of membrane receptors after bladder outlet obstruction (BOO). Partial bladder... 
Purinergic receptors | P2×5 | Tachykinin receptor 1 | Endothelin type A receptor | Neuromedin B receptor | Membrane receptor | V1a receptor | Thrombin receptor | Detrusor | Glycine receptor | Lgr5 | AVP | Lgr6 | Bladder outlet obstruction | β3-Adrenoceptor | Endothelin type Areceptor | V 1a receptor | P 2×5 | RABBIT BLADDER | beta-Adrenoceptor | BETA-ADRENOCEPTOR AGONIST MIRABEGRON | BOMBESIN RECEPTORS | ADRENERGIC-RECEPTORS | TRACT SYMPTOMS | P-2x5 | IN-VITRO | OVERACTIVE BLADDER | OUTFLOW OBSTRUCTION | PHARMACOLOGY & PHARMACY | V-1a receptor | BLOOD-FLOW | ARGININE-VASOPRESSIN | Urinary Bladder - metabolism | Membrane Proteins - genetics | RNA, Messenger - genetics | Gene Expression Regulation | Rats | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - metabolism | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Protein Transport | Animals | Female | Membrane Proteins - metabolism | Immunohistochemistry | Physiological aspects | Pituitary hormones | RNA | Glycine | Basic Medicine | Farmakologi och toxikologi | Medical and Health Sciences | Pharmacology and Toxicology | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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12. Full Text Functional and molecular evidence for Kv7 channel subtypes in human detrusor from patients with and without bladder outflow obstruction
by Svalø, Julie and Sheykhzade, Majid and Nordling, Jørgen and Matras, Christina and Bouchelouche, Pierre
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 2, p. e0117350
The aim of the study was to investigate whether Kv7 channels and their ancillary β-subunits, KCNE, are functionally expressed in the human urinary bladder. Kv7... 
Urinary Bladder Neck Obstruction - pathology | Urinary Bladder Neck Obstruction - physiopathology | Humans | Middle Aged | Male | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder Neck Obstruction - metabolism | Urinary Bladder - pathology | Muscle, Smooth - drug effects | Female | Muscle, Smooth - physiology | Receptors, Adrenergic, beta - metabolism | Urinary Bladder - metabolism | Colforsin - pharmacology | Muscle, Smooth - metabolism | Gene Expression Regulation - drug effects | KCNQ Potassium Channels - metabolism | Muscle Contraction - drug effects | Isoproterenol - pharmacology | Muscle, Smooth - physiopathology | Urinary Bladder - physiopathology | Aged | Muscle Relaxation - drug effects | Urinary Bladder - physiology | KCNQ Potassium Channels - genetics | Phosphorylation | Bladder | Smooth muscle | Biochemistry | Kinases | Urology | Channels | Modulators | Proteins | Urinary bladder | Rodents | Outflow | Potassium channels (voltage-gated) | Urine | Pulmonary arteries | KCNQ5 protein | Gene expression | Patients | Molecular chains | KCNQ3 protein | Carbachol | Human performance | Forskolin | KCNQ1 protein | Adrenergic receptors | Potassium | Receptors (physiology) | Veins & arteries
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13. Full Text GATA-6 and NF-κB Activate CPI-17 Gene Transcription and Regulate Ca2+ Sensitization of Smooth Muscle Contraction
by Ettickan Boopathi and Joseph A. Hypolite and Stephen A. Zderic and Cristiano Mendes Gomes and Bruce Malkowicz and Hsiou-Chi Liou and Alan J. Wein and Samuel Chacko
Molecular and Cellular Biology, ISSN 0270-7306, 03/2013, Volume 33, Issue 5, pp. 1085 - 1102
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
SIGNAL-TRANSDUCTION | PROTEIN-KINASE-C | INHIBITORY PROTEIN | NONMUSCLE MYOSIN-II | URINARY-BLADDER | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIAC-HYPERTROPHY | RHO-KINASE | BLADDER OUTLET OBSTRUCTION | UP-REGULATION | BINDING-PROTEINS | CELL BIOLOGY | Up-Regulation | Urinary Bladder Neck Obstruction - pathology | Calcium - metabolism | Humans | Male | NF-kappa B - metabolism | Urinary Bladder Neck Obstruction - genetics | Urinary Bladder - pathology | Hypertrophy - pathology | RNA Interference | Base Sequence | Hypertrophy - genetics | Conserved Sequence | Muscle, Smooth - physiology | Urinary Bladder - cytology | Promoter Regions, Genetic | Urinary Bladder - metabolism | Cells, Cultured | Phosphoproteins - genetics | Gene Knockout Techniques | GATA6 Transcription Factor - metabolism | Mice, Knockout | Muscle Proteins - genetics | Animals | Muscle Contraction | Phosphoprotein Phosphatases - genetics | NF-kappa B - genetics | Mice | GATA6 Transcription Factor - genetics
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