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Bone, ISSN 8756-3282, 2015, Volume 83, pp. 127 - 140
Abstract Lycopene supplementation decreases oxidative stress and exhibits beneficial effects on bone health, but the mechanisms through which it alters bone metabolism in vivo remain unclear... 
Orthopedics | Osteoclastogenesis | Oxidative stress | Bone turnover | Ovariectomy | Bone mass and microarchitecture | Lycopene | SYSTEM | RESORPTION | RAT MODEL | SUPPLEMENTATION | WOMEN | ENDOCRINOLOGY & METABOLISM | HYDROGEN-PEROXIDE | ESTROGEN-DEFICIENCY | OSTEOCLAST DIFFERENTIATION | Humans | Body Weight - drug effects | Femur - diagnostic imaging | Humerus - physiopathology | X-Ray Microtomography | Biomechanical Phenomena - drug effects | Carotenoids - blood | Hormones - blood | Lumbar Vertebrae - physiopathology | Tibia - physiopathology | Disease Models, Animal | Bone Resorption - physiopathology | Diaphyses - diagnostic imaging | Lumbar Vertebrae - diagnostic imaging | Bone Resorption - drug therapy | Tibia - diagnostic imaging | Lumbar Vertebrae - drug effects | Biomarkers - blood | Absorptiometry, Photon | Osteoporosis, Postmenopausal - drug therapy | Diaphyses - drug effects | Osteoporosis, Postmenopausal - blood | Diaphyses - physiopathology | Minerals - blood | Uterus - pathology | Bone and Bones - pathology | Deoxyguanosine - blood | Rats, Wistar | Uterus - drug effects | Bone Remodeling - drug effects | Carotenoids - pharmacology | Bone and Bones - drug effects | Humerus - drug effects | Carotenoids - therapeutic use | Bone and Bones - diagnostic imaging | Female | Deoxyguanosine - analogs & derivatives | Bone Resorption - blood | Femur - drug effects | Humerus - diagnostic imaging | Femur - physiopathology | Tibia - drug effects | Bone Resorption - diagnostic imaging | Bone Density - drug effects | Bone and Bones - physiopathology | Organ Size - drug effects | Animals | Osteoporosis, Postmenopausal - physiopathology | Enzymes - blood | Osteoporosis | Analysis | Bone densitometry | Physiological aspects | Superoxide | Postmenopausal women
Journal Article
Osteoporosis international, ISSN 0937-941X, 2012, Volume 24, Issue 4, pp. 1455 - 1470
Effects of cladrin treatment and withdrawal in osteopenic rats were studied. Cladrin improved trabecular microarchitecture, increased lumbar vertebral compressive strength, augmented coupled remodeling, and increased bone osteogenic genes... 
Phytoestrogen | Microarchitecture | Bone formation | Medicine & Public Health | Orthopedics | Gynecology | Treatment withdrawal | Rheumatology | Bone strength | Endocrinology | IPRIFLAVONE | BONE-MINERAL DENSITY | ULMUS-WALLICHIANA PLANCHON | TRABECULAR BONE | IN-VITRO | ENDOCRINOLOGY & METABOLISM | DOUBLE-BLIND | SOY ISOFLAVONES | HIP-FRACTURES | CONJUGATED ESTROGENS | POSTMENOPAUSAL WOMEN | Uterus - pathology | Isoflavones - pharmacology | Uterus - drug effects | Body Weight - drug effects | X-Ray Microtomography | Bone Diseases, Metabolic - drug therapy | Dose-Response Relationship, Drug | Compressive Strength - drug effects | Female | Isoflavones - administration & dosage | Lumbar Vertebrae - physiopathology | Osteogenesis - genetics | Tibia - physiopathology | Drug Evaluation, Preclinical - methods | Drug Administration Schedule | Ovariectomy | Femur - metabolism | Osteoblasts - drug effects | Osteogenesis - drug effects | RNA, Messenger - genetics | Bone Density Conservation Agents - therapeutic use | Rats | Femur - drug effects | Lumbar Vertebrae - drug effects | Biomarkers - blood | Bone Density Conservation Agents - administration & dosage | Femur - physiopathology | Tibia - drug effects | Osteoclasts - metabolism | Rats, Sprague-Dawley | Bone Density Conservation Agents - pharmacology | Gene Expression Regulation - drug effects | Organ Size - drug effects | Animals | Isoflavones - therapeutic use | Bone Diseases, Metabolic - physiopathology | Osteoblasts - metabolism | Osteoclasts - drug effects | Physiological aspects | Bones | Research | Drug therapy | Growth | Osteopenia | Rodents | Bone density | Phytochemicals | Compression | Vertebrae | Bone remodelling | Estrogens | Menopause | mRNA | Osteoblasts | 17 beta -Estradiol | Western blotting | Osteoporosis | Bone mass | Computed tomography | Uterus | Parathyroid hormone | Collagen | Bone loss | Enzyme-linked immunosorbent assay | Osteogenesis
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 50, pp. 82074 - 82084
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61537
.... Metformin is an oral anti-diabetic drug of the biguanide family used for treatment of type 2 diabetes... 
BREAST-CANCER | TARGETED THERAPY | MULTIDISCIPLINARY SCIENCES | ENDOMETRIAL CANCER-CELLS | RESISTANCE | RISK | RECEPTOR GENE | MODEL | EXPRESSION | CHEMOTHERAPY | GROWTH-FACTOR-I | Cell Cycle - genetics | Receptor, IGF Type 1 - metabolism | Insulin-Like Growth Factor I - pharmacology | Metformin - therapeutic use | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Uterine Neoplasms - pathology | Humans | Cell Survival - genetics | Apoptosis - genetics | Glycogen Synthase Kinase 3 beta | Cystadenocarcinoma, Serous - pathology | Cystadenocarcinoma, Serous - drug therapy | Promoter Regions, Genetic - genetics | Uterine Neoplasms - enzymology | Receptor, Insulin - genetics | Adenylate Kinase - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Cell Survival - drug effects | Metformin - pharmacology | Tumor Suppressor Protein p53 - metabolism | Down-Regulation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Receptor, IGF Type 1 - genetics | Cystadenocarcinoma, Serous - enzymology | Cell Movement - drug effects | Insulin - metabolism | Signal Transduction - drug effects | Uterine Neoplasms - drug therapy | Cell Line, Tumor | Endometrial Neoplasms - pathology | Receptor, Insulin - metabolism | Cell Proliferation - drug effects | Forkhead Box Protein O1 | Cell Cycle - drug effects | Forkhead Transcription Factors | Insulin-Like Growth Factor I - metabolism | Type 2 diabetes | Obesity | Carcinoma | Endometrial cancer | Insulin resistance | Metformin | Tumor proteins | Health aspects | Epidemiology | Diabetes therapy | Cancer | Cell proliferation | Hyperinsulinemia | Insulin-like growth factor I | p53 Protein | Insulin-like growth factors | Kinases | Cancer therapies | Ovarian cancer | Signal transduction | Cell growth | Pathways | Uterus | Growth factors | Endometrium | Diabetes mellitus | Health risks | Insulin | Signaling | Cell migration | Apoptosis
Journal Article
Human reproduction update, ISSN 1460-2369, 2010, Volume 17, Issue 1, pp. 76 - 95
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2016, Volume 83, pp. 942 - 957
Abstract Objective This study aims to evaluate the skeletal effects of dalbergin (DBN... 
Internal Medicine | Medical Education | Neo-flavonoids DBN (dalbergin) | Trabecular micro-architecture | Ovariectomy | Postmenopausal osteoporosis | ESTROGENS | MEDICINE, RESEARCH & EXPERIMENTAL | MANAGEMENT | PREVENTION | PHYTOESTROGENS | ISOFLAVONES | BONE LOSS | DIET | PHARMACOLOGY & PHARMACY | DIFFERENTIATION | EXPRESSION | CONSTITUENTS | Liver - pathology | Alkaline Phosphatase - metabolism | Body Weight - drug effects | Cancellous Bone - drug effects | Protective Agents - chemistry | X-Ray Microtomography | Osteocalcin - blood | Liver - drug effects | Osteoporosis - diagnostic imaging | Biomechanical Phenomena - drug effects | Osteoporosis - genetics | Disease Models, Animal | Biomarkers - metabolism | Osteoclasts - pathology | Calcification, Physiologic - drug effects | Administration, Oral | Protective Agents - pharmacokinetics | Biomarkers - blood | Flavonoids - pharmacokinetics | Osteoporosis - pathology | Dalbergia - chemistry | Mice, Inbred BALB C | Flavonoids - chemistry | Uterus - pathology | Femur - pathology | Uterus - drug effects | Coumarins - chemistry | Osteoporosis - drug therapy | Protective Agents - therapeutic use | Bone Remodeling - drug effects | Flavonoids - therapeutic use | Cancellous Bone - pathology | Flavonoids - administration & dosage | Bone Marrow Cells - drug effects | Female | Osteoblasts - drug effects | Coumarins - administration & dosage | Coumarins - pharmacokinetics | Femur - drug effects | Femur - physiopathology | Osteoclasts - metabolism | Coumarins - therapeutic use | Gene Expression Regulation - drug effects | Osteoblasts - pathology | Animals | Protective Agents - administration & dosage | Osteoblasts - metabolism | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | Osteoporosis | Postmenopausal women | Research institutes | Analysis | Animal experimentation | Bone morphogenetic proteins | Rankings | Gene expression | Health aspects | Estradiol | Isoflavones
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e17877
To our knowledge, there is no report on long-term reproductive and developmental side effects in the offspring of mothers treated with a widely used chemotherapeutic drug such as doxorubicin (DXR... 
APOPTOSIS | CHILDHOOD-CANCER | GENE | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | DOXORUBICIN | FEMALE SURVIVORS | ANTICANCER DRUGS | PREMATURE OVARIAN FAILURE | RING-SHAPED NUCLEI | FERTILITY | Uterus - pathology | Doxorubicin - therapeutic use | Myometrium - drug effects | Chromosomes, Mammalian - genetics | Humans | Uterus - drug effects | Male | Antineoplastic Agents - therapeutic use | Anxiety - drug therapy | Antineoplastic Agents - administration & dosage | Atrophy | Lysophospholipids - therapeutic use | Ovarian Follicle - drug effects | Antineoplastic Agents - adverse effects | Lysophospholipids - pharmacology | Oocytes - drug effects | Behavior, Animal - drug effects | Female | Antineoplastic Agents - pharmacology | Ovarian Follicle - transplantation | Ovulation - drug effects | bcl-2-Associated X Protein - deficiency | Doxorubicin - administration & dosage | Chromosome Deletion | Myometrium - pathology | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | Heredity - drug effects | Spermatozoa - drug effects | Reproduction - drug effects | Sphingosine - pharmacology | Phenotype | Sphingosine - analogs & derivatives | Animals | Heredity - genetics | Heredity - physiology | Sphingosine - therapeutic use | Mice | Ovarian Follicle - pathology | Doxorubicin - adverse effects | Doxorubicin - pharmacology | Chemotherapy | Infants | Analysis | Patient outcomes | Cancer | Neonates | Motility | Laboratories | Sperm | Hydrocarbons | Mental depression | Experiments | Cancer therapies | Doxorubicin | Oocytes | Rodents | Animal tissues | Chromosome 10 | Physiology | Anxiety | Age | Complications | Abnormalities | Animal sciences | Injection | Gene expression | Progeny | Offspring | Side effects | Toxicosis | Females | Long-term effects | Apoptosis | Drugs | toxicosis | Animal models | Data processing | Depression | Chromosome deletion | Follicles | chromosome 10
Journal Article