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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 313, Issue 3, pp. H491 - H507
Preeclampsia is a pregnancy-related disorder manifested as maternal hypertension in pregnancy (HTN-Preg) and fetal growth restriction. Placental ischemia could... 
Aorta | Placenta | Tumor necrosis factor-α | Uterus | Remodeling | Preeclampsia | ENDOTHELIN RECEPTOR | remodeling | CHRONIC REDUCTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | VENOUS DILATION | preeclampsia | MESENTERIC MICROVESSELS | VASOACTIVE FACTORS | tumor necrosis factor-alpha | SYNTHASE MESSENGER-RNA | uterus | ARTERIAL-PRESSURE | placenta | PERIPHERAL VASCULAR DISEASE | aorta | UTERINE PERFUSION-PRESSURE | PLACENTAL ISCHEMIA | INFLAMMATORY CYTOKINES | Blood Pressure | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Uterus - drug effects | Ischemia - enzymology | Matrix Metalloproteinase 7 - metabolism | Placental Circulation | Female | Vascular Remodeling - drug effects | Uterus - blood supply | Aorta - enzymology | Organ Culture Techniques | Disease Models, Animal | Hypertension, Pregnancy-Induced - enzymology | Collagen Type I - metabolism | Placentation - drug effects | Signal Transduction | Hypertension, Pregnancy-Induced - physiopathology | Placenta - enzymology | Ischemia - physiopathology | Gestational Age | Rats, Sprague-Dawley | Placenta - drug effects | Pregnancy | Placenta - blood supply | Tumor Necrosis Factor-alpha - pharmacology | Animals | Immunoglobulin Fc Fragments - pharmacology | Uterus - enzymology | Hypertension, Pregnancy-Induced - etiology | Matrix Metalloproteinase 1 - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Ischemia - etiology | tumor necrosis factor-α
Journal Article
British Journal of Cancer, ISSN 0007-0920, 02/2013, Volume 108, Issue 3, pp. 613 - 620
Background: Lobular endocervical glandular hyperplasia (LEGH) is a rare lesion of the uterine cervix. It has been proposed that LEGH may represent a precursor... 
PAPANICOLAOU SMEARS | ADENOMA MALIGNUM | H-HPV | MINIMAL DEVIATION ADENOCARCINOMA | MN ANTIGEN | HUMAN-PAPILLOMAVIRUS | CELL CARCINOMA | CA-IX | UTERINE CERVIX | MN/CA9 PROTEIN | gastric phenotype | GASTRIC MORPHOLOGY | ONCOLOGY | lobular endocervical glandular hyperplasia | DIAGNOSTIC BIOMARKER | minimal deviation | conventional adenocarcinoma | Carcinoma, Lobular - pathology | Prognosis | Adenocarcinoma, Mucinous - pathology | Humans | Middle Aged | Adenocarcinoma, Mucinous - virology | Male | Stomach Neoplasms - pathology | Uterine Cervical Neoplasms - pathology | Carcinoma, Lobular - enzymology | Immunoenzyme Techniques | Papillomaviridae - genetics | Carbonic Anhydrases - metabolism | Papillomavirus Infections - pathology | Adenocarcinoma, Mucinous - enzymology | Neoplasms, Glandular and Epithelial - virology | Polymerase Chain Reaction | Aged, 80 and over | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Adult | Female | Papillomavirus Infections - virology | Carbonic Anhydrase IX | Stomach Neoplasms - enzymology | Neoplasms, Glandular and Epithelial - pathology | Uterine Cervical Neoplasms - enzymology | Stomach Neoplasms - virology | Papillomavirus Infections - enzymology | Hyperplasia - virology | Carcinoma, Lobular - virology | Hyperplasia - pathology | Neoplasms, Glandular and Epithelial - enzymology | Hyperplasia - enzymology | Uterine Cervical Neoplasms - virology | Aged | DNA, Viral - genetics | Index Medicus | Molecular Diagnostics
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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 19, pp. 7890 - 7895
Transporters at the hepatic canalicular membrane are essential for the formation of bile and the prevention of cholestatic liver disease. One such example is... 
Cholic acids | Bile acids | Hyperbilirubinemia | Hepatocytes | B lymphocytes | Liver | Mice | Genetic mutation | Blood plasma | Bile | Jaundice | Gallbladder | Uterus | Enterohepatic circulation | Bile acid | HOMOZYGOUS DISRUPTION | HOMEOSTASIS | gallbladder | ACID | BILE-SALTS | bile acid | MULTIDISCIPLINARY SCIENCES | jaundice | IDENTIFICATION | DEFICIENCY | uterus | DISEASE | MICE | P-GLYCOPROTEIN GENE | enterohepatic circulation | FAMILIAL INTRAHEPATIC CHOLESTASIS | Genetic Diseases, X-Linked - enzymology | Male | DNA Primers - genetics | Lymphopenia - enzymology | Lymphopenia - pathology | Mitochondrial Proton-Translocating ATPases - genetics | Genes, X-Linked | Mice, Mutant Strains | Base Sequence | Female | Genetic Diseases, X-Linked - genetics | B-Lymphocytes - pathology | Cholic Acid - toxicity | Disease Models, Animal | Cholestasis, Intrahepatic - genetics | Dystocia - genetics | Hyperbilirubinemia, Hereditary - genetics | Cholestasis, Intrahepatic - pathology | Mice, Inbred C57BL | Molecular Chaperones - genetics | Molecular Chaperones - physiology | Cholestasis, Intrahepatic - enzymology | Dystocia - enzymology | Pregnancy | Homozygote | Hyperbilirubinemia, Hereditary - enzymology | Phenotype | Animals | Mitochondrial Proton-Translocating ATPases - physiology | Mutation | Lymphopenia - genetics | Cholic Acid - administration & dosage | Care and treatment | Jaundice, Obstructive | Genetic aspects | Gene mutations | Identification and classification | Cholestasis | Biological Sciences
Journal Article
Cancer Research, ISSN 0008-5472, 09/2008, Volume 68, Issue 18, pp. 7386 - 7393
Journal Article
Biochemistry and Cell Biology, ISSN 0829-8211, 04/2006, Volume 84, Issue 2, pp. 215 - 231
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