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Lancet Oncology, The, ISSN 1470-2045, 2012, Volume 13, Issue 8, pp. 782 - 789
Summary Background MEK is a member of the MAPK signalling cascade that is commonly activated in melanoma. Direct inhibition of MEK blocks cell proliferation... 
Hematology, Oncology and Palliative Medicine | SURVIVAL | BRAF | ONCOLOGY | RAF | SOMATIC MUTATIONS | PROGRESSION | Skin Neoplasms - drug therapy | United States | Humans | Middle Aged | Melanoma - enzymology | Male | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Molecular Targeted Therapy | Uveal Neoplasms - enzymology | Young Adult | Pyridones - administration & dosage | Skin Neoplasms - enzymology | Time Factors | Antineoplastic Agents - adverse effects | DNA Mutational Analysis | Melanoma - genetics | Skin Neoplasms - mortality | Adult | Female | Skin Neoplasms - pathology | MAP Kinase Kinase 1 - antagonists & inhibitors | Uveal Neoplasms - genetics | Uveal Neoplasms - pathology | Drug Administration Schedule | Administration, Oral | Pyrimidinones - adverse effects | Kaplan-Meier Estimate | MAP Kinase Kinase 1 - metabolism | Treatment Outcome | MAP Kinase Kinase 2 - metabolism | Melanoma - pathology | Uveal Neoplasms - drug therapy | Disease-Free Survival | Protein Kinase Inhibitors - administration & dosage | MAP Kinase Kinase 2 - antagonists & inhibitors | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Skin Neoplasms - genetics | Uveal Neoplasms - mortality | Aged | Pyrimidinones - administration & dosage | Mutation | Pyridones - adverse effects | Melanoma - mortality | Prevention | Care and treatment | Melanoma | Cancer | Index Medicus
Journal Article
Journal of Medical Genetics, ISSN 0022-2593, 12/2011, Volume 48, Issue 12, pp. 856 - 859
ObjectiveTo investigate the potential contribution of germline sequence alterations in the BAP1 gene in uveal melanoma (UM) patients with possible... 
BREAST-CANCER | GENE | HIGH-RISK | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | BRCA1 | UBIQUITIN HYDROLASE | BRCA1-ASSOCIATED PROTEIN-1 | Adenocarcinoma - pathology | Genetic Testing | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Genetic Diseases, Inborn - genetics | Lung Neoplasms - pathology | Male | Meningioma - genetics | Genetic Diseases, Inborn - pathology | Loss of Heterozygosity | Adenocarcinoma - metabolism | DNA Mutational Analysis | Melanoma - genetics | Tumor Suppressor Proteins - genetics | Germ-Line Mutation | Ubiquitin Thiolesterase - metabolism | Adult | Female | Adenocarcinoma - genetics | Genetic Diseases, Inborn - metabolism | Uveal Neoplasms - metabolism | Microsatellite Repeats | Lung Neoplasms - genetics | Melanoma - metabolism | Uveal Neoplasms - genetics | Genetic Predisposition to Disease - genetics | Tumor Suppressor Proteins - metabolism | Meningeal Neoplasms - genetics | Meningioma - metabolism | Uveal Neoplasms - pathology | Gene Silencing | Chromosomes, Human, Pair 3 - metabolism | Melanoma - pathology | Ubiquitin Thiolesterase - genetics | Meningioma - pathology | Pedigree | Meningeal Neoplasms - metabolism | Meningeal Neoplasms - pathology | Chromosomes, Human, Pair 3 - genetics | Genetic aspects | Research | BRCA mutations | Cancer | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7229, pp. 599 - 602
BRAF and NRAS are common targets for somatic mutations in benign and malignant neoplasms that arise from melanocytes situated in epithelial structures, and... 
CELLS | ALPHA-SUBUNIT | PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE-C | BRAF | CANCER | TUMORS | MELANOCYTOSIS | Cell Proliferation | GTP-Binding Protein alpha Subunits - chemistry | Humans | Melanoma - enzymology | Nevus, Blue - pathology | Uveal Neoplasms - enzymology | MAP Kinase Signaling System | Skin Neoplasms - enzymology | Nevus, Blue - genetics | DNA Mutational Analysis | Melanoma - genetics | ras Proteins - chemistry | Melanocytes - pathology | Codon - genetics | Protein Structure, Tertiary | Skin Neoplasms - pathology | Oncogenes - genetics | Uveal Neoplasms - genetics | Melanocytes - enzymology | GTP-Binding Protein alpha Subunits - metabolism | Uveal Neoplasms - pathology | Cells, Cultured | Nevus, Blue - enzymology | GTP-Binding Protein alpha Subunits - genetics | Melanoma - pathology | Mutation - genetics | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Biopsy | Cell Transformation, Neoplastic | Skin Neoplasms - genetics | GTP-Binding Protein alpha Subunits - deficiency | Enzyme Activation | Genes, Dominant - genetics | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Antibody diversity | Evaluation | Gene mutations | Uvea | Melanoma | Genetic aspects | Research | Health aspects | Cancer | Proteins | Studies | Enzymes | Cell cycle | Mutation | Kinases | Risk factors | Skin cancer | Index Medicus
Journal Article
Cancer, ISSN 0008-543X, 11/2016, Volume 122, Issue 21, pp. 3354 - 3362
Journal Article
Journal Article
Journal Article
Cancer, ISSN 0008-543X, 11/2016, Volume 122, Issue 21, pp. 3344 - 3353
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 03/2016, Volume 40, Issue 3, pp. 368 - 377
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 08/2013, Volume 31, Issue 22, pp. 2825 - 2831
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 03/2016, Volume 22, Issue 5, pp. 1234 - 1242
Purpose: Uveal melanoma (UM) can be classified by gene expression profiling (GEP) into Class 1 (low metastatic risk) and Class 2 (high metastatic risk), the... 
ASSAY | ONCOLOGY | CHROMOSOME-3 | GNAQ | TRANSCRIPTION | VALIDATION |