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Journal Article
BBA - Reviews on Cancer, ISSN 0304-419X, 2010, Volume 1806, Issue 1, pp. 18 - 28
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2015, Volume 19, Issue 2, pp. 315 - 326
Journal Article
Cancer Science, ISSN 1347-9032, 03/2017, Volume 108, Issue 3, pp. 478 - 487
Pancreatic ductal adenocarcinoma ( PDAC ) is one of the most aggressive human malignancies. The Yes‐associated protein‐1 ( YAP ) plays a critical role in cell... 
Angiogenesis | apoptosis | pancreatic cancer | vasculogenic mimicry | verteporfin | HUMAN CHOLANGIOCARCINOMA | HIPPO PATHWAY | DOWN-REGULATION | ANGIOPOIETIN-2 | CANCER | MOLECULAR PATHWAYS | HEPATOCELLULAR-CARCINOMA | IN-VITRO | ONCOLOGY | POOR-PROGNOSIS | CHEMORESISTANCE | Human Umbilical Vein Endothelial Cells | Cyclin D1 - metabolism | Apoptosis - drug effects | Humans | Male | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Proto-Oncogene Proteins c-bcl-2 - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Cell Survival - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Matrix Metalloproteinase 2 - metabolism | Pancreatic Neoplasms - pathology | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Poly (ADP-Ribose) Polymerase-1 - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Carcinoma, Pancreatic Ductal - pathology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Cadherins - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | Antigens, CD | Adenocarcinoma | Proteins | Photochemotherapy | Analysis | Pancreatic cancer | Cancer | Cell proliferation | Metastasis | Cancer therapies | Mimicry | Cell adhesion & migration | Macular degeneration | Cell cycle | Xenografts | Conflicts of interest | G1 phase | Cell survival | Photodynamic therapy | Poly(ADP-ribose) polymerase | Gene expression | Cadherin | Gelatinase A | Yes-associated protein | Microscopy | Medical prognosis | Stem cells | Hypoxia | Apoptosis | Tumors | Original
Journal Article
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2014, Volume 133, Issue 3, pp. 575 - 583
Abstract Objectives The functions of hypoxia and subsequent hypoxia-inducible factor-1α (HIF-1α) activation in vasculogenic mimicry (VM) are currently unclear.... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Hypoxia | Epithelial–mesenchymal transition | HIF-1α | Ovarian cancer | Vasculogenic mimicry | Epithelial-mesenchymal transition | HIP-1 alpha | STEM-CELLS | METASTASIS | E-CADHERIN | OBSTETRICS & GYNECOLOGY | TUMOR-CELL PLASTICITY | HEPATOCELLULAR-CARCINOMA | COOPERATIVE INTERACTIONS | MELANOMA | ONCOLOGY | IN-VIVO | EXPRESSION | FUNCTIONAL-SIGNIFICANCE | Cadherins - metabolism | Neoplasms, Glandular and Epithelial - blood supply | Vimentin - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Middle Aged | Neoplasms, Glandular and Epithelial - metabolism | RNA, Messenger - analysis | Hypoxia - metabolism | Ovarian Neoplasms - genetics | Young Adult | Neoplasms, Glandular and Epithelial - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Vimentin - genetics | Aged, 80 and over | Adult | Female | Neovascularization, Pathologic - physiopathology | Ovarian Neoplasms - metabolism | Ovarian Neoplasms - blood supply | Cadherins - genetics | Snail Family Transcription Factors | Nuclear Proteins - metabolism | Transcription Factors - metabolism | Hypoxia - genetics | Carcinoma, Ovarian Epithelial | Cell Line, Tumor | Hypoxia - physiopathology | Neovascularization, Pathologic - genetics | Aged | Neovascularization, Pathologic - metabolism | Twist-Related Protein 1 - metabolism | Stem cells
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3741 - 3751
Matrix metalloproteinases ( MMP s) have critical functions in tumour vasculogenic mimicry ( VM ). This study explored the mechanisms underlying MMP ‐13 and MMP... 
large cell lung cancer | MMP | 13 | vasculogenic mimicry | laminin5 | MMP-13 | MMP-2 | MEDICINE, RESEARCH & EXPERIMENTAL | METASTASIS | ANGIOGENESIS | EGF | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | MELANOMA | LAMININ-5 | ACTIN-CYTOSKELETON | EXTRACELLULAR-MATRIX | TUMOR-GROWTH | METALLOPROTEINASE-13 | Receptor, Epidermal Growth Factor - genetics | Cell Adhesion Molecules - genetics | Carcinoma, Large Cell - pathology | Humans | Lung Neoplasms - metabolism | Actins - metabolism | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Actins - genetics | Neovascularization, Pathologic - pathology | Molecular Mimicry | Receptor, Epidermal Growth Factor - metabolism | Transfection | Cell Adhesion Molecules - pharmacology | Neovascularization, Pathologic - prevention & control | Carcinoma, Large Cell - genetics | Cell Culture Techniques | Lung Neoplasms - genetics | Carcinoma, Large Cell - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Matrix Metalloproteinase 13 - genetics | Lung Neoplasms - therapy | Tumor Burden | Cell Adhesion Molecules - metabolism | Matrix Metalloproteinase 13 - metabolism | Xenograft Model Antitumor Assays | Matrix Metalloproteinase 2 - genetics | Animals | Mice, Nude | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Mice, Inbred BALB C | Neovascularization, Pathologic - metabolism | Carcinoma, Large Cell - therapy | Muscle proteins | Lung cancer | Cell culture | Matrix metalloproteinases | Epidermal growth factor receptors | Fragments | Blood vessels | Cultures | Tissues | Collagenase 3 | Endothelium | Mimicry | Gelatinase A | Fragmentation | Actin | Cell lines | Xenografts | Protein structure | Cancer | Tumors | Recombinant | MMP‐13 | Original | MMP‐2
Journal Article