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Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1037 - 1048
Resolution of inflammation is now recognized as a biosynthetically active process involving pro-resolving mediators. Here, we show in zymosan-initiated... 
CIRCUITS | MEDICINE, RESEARCH & EXPERIMENTAL | PROTECTINS | CHEMOTAXIS | INHIBITS LEUKOCYTE MIGRATION | NETRIN-1 | ATHEROSCLEROSIS | MECHANISMS | IMMUNOLOGY | RECEPTORS | HUMAN PHAGOCYTES | PRORESOLVING LIPID MEDIATORS | Netrin-1 | Humans | Lipids - immunology | Monocytes - metabolism | Monocytes - immunology | Nerve Growth Factors - metabolism | Docosahexaenoic Acids - immunology | Inflammation - metabolism | Lipids - chemistry | Tumor Suppressor Proteins - genetics | Inflammation Mediators - metabolism | Chemotaxis - immunology | Neutrophils - metabolism | Macrophages - immunology | Inflammation Mediators - immunology | Recombinant Proteins - metabolism | Zymosan | Tumor Suppressor Proteins - metabolism | Vagus Nerve - metabolism | Mice, Inbred C57BL | Neutrophils - drug effects | Peritonitis - metabolism | Cells, Cultured | Lipoxins - immunology | Neutrophils - immunology | Vagus Nerve - surgery | Inflammation - immunology | Nerve Growth Factors - immunology | Peritonitis - chemically induced | Chemotaxis - drug effects | Peritonitis - immunology | Recombinant Proteins - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Tumor Suppressor Proteins - immunology | Vagotomy | Blotting, Western | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Nerve Growth Factors - genetics | Vagus Nerve - immunology | Inflammation - genetics | Mice | Lipoxins - metabolism | Microscopy, Fluorescence | Docosahexaenoic Acids - metabolism | Index Medicus | 303
Journal Article
Gut, ISSN 0017-5749, 06/2014, Volume 63, Issue 6, pp. 938 - 948
Journal Article
Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 07/2013, Volume 72, Issue 7, pp. 614 - 623
Journal Article
Neuroscience Letters, ISSN 0304-3940, 2006, Volume 396, Issue 1, pp. 67 - 72
The progressive degenerative process associated with sporadic Parkinson's disease (sPD) is characterized by formation of α-synuclein-containing inclusion... 
Neurotropic pathogen | Enteric nervous system | Retrograde axonal transport | Lewy neurites/bodies | Sporadic Parkinson's disease | retrograde axonal transport | HELICOBACTER-PYLORI INFECTION | ENTERIC NERVOUS-SYSTEM | GASTROINTESTINAL DYSFUNCTION | ALZHEIMERS-DISEASE | NEURONS | enteric nervous system | sporadic Parkinson's disease | NEUROSCIENCES | neurotropic pathogen | LEWY BODIES | Myenteric Plexus - pathology | Neurons - pathology | Humans | Middle Aged | Male | Myenteric Plexus - metabolism | Neural Pathways - pathology | Brain - metabolism | Aged, 80 and over | Female | Gastric Mucosa - physiopathology | Neurons - metabolism | Parkinson Disease - metabolism | Disease Transmission, Infectious | Neural Pathways - physiopathology | Submucous Plexus - metabolism | Inclusion Bodies - metabolism | Nerve Net - physiopathology | Prions - metabolism | Parkinson Disease - pathology | Submucous Plexus - physiopathology | Vagus Nerve - metabolism | Brain - physiopathology | Submucous Plexus - pathology | Axonal Transport - physiology | Gastric Mucosa - innervation | Parkinson Disease - physiopathology | Nerve Net - pathology | Protein Folding | Vagus Nerve - physiopathology | Myenteric Plexus - physiopathology | Nerve Net - metabolism | Brain - pathology | Inclusion Bodies - pathology | Neural Pathways - metabolism | Aged | Models, Neurological | alpha-Synuclein - metabolism
Journal Article
Journal Article
CARDIOVASCULAR RESEARCH, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Ischaemia | Brain | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | Vagus nerve | Preconditioning | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Ischaemia/reperfusion injury | Original
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2014, Volume 124, Issue 10, pp. 4473 - 4488
Journal Article