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Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 11/2014, Volume 58, Issue 11, pp. 6413 - 6423
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 09/2009, Volume 53, Issue 9, pp. 3675 - 3682
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, ISSN 0363-6135, 01/2006, Volume 290, Issue 1, pp. H406 - H415
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, pp. e16746 - e16746
PINK1 and Parkin mutations cause recessive Parkinson's disease (PD). In Drosophila and SH-SY5Y cells, Parkin is recruited by PINK1 to damaged mitochondria,... 
MUTANTS | FUSION | MITOCHONDRIAL-FUNCTION | DISEASE | PROTEASOME | BIOLOGY | DEGRADATION | COMPLEX I DEFICIENCY | MITOPHAGY | DROSOPHILA | MORPHOLOGY | Protein Kinases - genetics | Humans | Hydrogen Peroxide - pharmacology | Ubiquitin - metabolism | Mitochondria - metabolism | Protein Transport - drug effects | Mitochondria - drug effects | Mutation - genetics | Leupeptins - pharmacology | Mitochondrial Membrane Transport Proteins | GTP Phosphohydrolases - metabolism | Ubiquitination - drug effects | Mitochondrial Proteins - metabolism | Models, Biological | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Fibroblasts - drug effects | Macrolides - pharmacology | Membrane Transport Proteins - metabolism | Membrane Proteins - metabolism | Proteasome Endopeptidase Complex - metabolism | Oligopeptides - pharmacology | Ubiquitin-Protein Ligases - genetics | Valinomycin - pharmacology | Fibroblasts - metabolism | Ubiquitin | Genetic aspects | Research | Cell culture | Hydrogen peroxide | Parkinson's disease | Disease | Change detection | DNA damage | Genomes | Mitochondrial DNA | Kinases | Autophagy | Experiments | Fission | Proteins | Mitochondria | Ubiquitination | Bioenergetics | Rodents | Fibroblasts | Membrane potential | Movement disorders | Deoxyribonucleic acid--DNA | Neurodegenerative diseases | Drosophila | Studies | Neurology | Inhibitors | Insects | PTEN-induced putative kinase | Morphology | Quality control | Parkin protein | Mutation | proteasomes | ubiquitination | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, pp. e39569 - e39569
Background: Chloroquine (CQ)-resistant Plasmodium falciparum malaria has been a global health catastrophe, yet much about the CQ resistance (CQR) mechanism... 
MECHANISM | MULTIDISCIPLINARY SCIENCES | ANTIMALARIAL-DRUG RESISTANCE | SUSCEPTIBILITY | REVERSING AGENTS | TRANSMEMBRANE PROTEIN PFCRT | MUTATIONS | MALARIA PARASITES | DIGESTIVE VACUOLE | FERRIPROTOPORPHYRIN-IX | Chloroquine - metabolism | Cytoplasmic Vesicles - drug effects | Potassium - metabolism | Plasmodium falciparum - drug effects | Protozoan Proteins - genetics | Dictyostelium - drug effects | Chloroquine - pharmacology | Protozoan Proteins - metabolism | Membrane Transport Proteins - genetics | Adenosine Triphosphate - metabolism | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Plasmodium falciparum - genetics | Cytoplasmic Vesicles - metabolism | Macrolides - pharmacology | Membrane Transport Proteins - metabolism | Ionophores - pharmacology | Valinomycin - pharmacology | Membrane Potentials - drug effects | Ammonia - pharmacology | Verapamil - pharmacology | Antimalarials - pharmacology | Phenotype | Animals | Dictyostelium - genetics | Cell Line, Transformed | Hydrogen-Ion Concentration | Electrochemistry | Plasmodium falciparum | Malaria | Drug resistance | Ionophores | Electrochemical potential | Valinomycin | Laboratories | Chloroquine | Erythrocytes | Acidification | Parasites | Chemosensitization | pH effects | Accumulation | Genotype & phenotype | Vesicles | Red blood cells | Hydrogen ions | Spectrum analysis | Membrane potential | Public health | Vector-borne diseases | Antigens | Fatty acids | Studies | Chemotherapy | Infectious diseases | Verapamil | Mutation | Transporter | Vacuoles | Hyperpolarization | Index Medicus | pH
Journal Article
FASEB Journal, ISSN 0892-6638, 08/2004, Volume 18, Issue 11, pp. 1219 - 1227
It is generally considered that mitochondria regulate cardiac cell contractility by providing ATP for cellular ATPases and by participating in Ca2+... 
Mitochondria | Potassium | Myocardial contractility | Myofibrils | FORCE PRODUCTION | potassium | CATION-TRANSPORT | mitochondria | BIOCHEMISTRY & MOLECULAR BIOLOGY | INORGANIC-PHOSPHATE | SARCOPLASMIC-RETICULUM | MUSCLE-CELLS | PERMEABILITY TRANSITION | CELL BIOLOGY | BIOLOGY | myofibrils | myocardial contractility | RAT VENTRICULAR MYOCYTES | CYTOCHROME-C RELEASE | K+ CHANNELS | CREATINE-KINASE | Mitochondria, Heart - ultrastructure | Potassium - metabolism | Tetraethylammonium - pharmacology | Electron Transport - drug effects | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Stress, Mechanical | Male | Potassium-Hydrogen Antiporters - metabolism | Mitochondria, Heart - drug effects | Sarcoplasmic Reticulum - enzymology | Cell Compartmentation | Oligomycins - pharmacology | Adenosine Triphosphate - metabolism | Quinine - pharmacology | Ion Transport - drug effects | Sodium-Calcium Exchanger - antagonists & inhibitors | Valinomycin - pharmacology | Pinacidil - pharmacology | Creatine Kinase - deficiency | Clonazepam - analogs & derivatives | Creatine Kinase, MM Form | Isoenzymes - genetics | Sarcomeres - drug effects | Creatine Kinase, Mitochondrial Form | Mice, Inbred C57BL | Clonazepam - pharmacology | Rats | Bongkrekic Acid - pharmacology | Thiazepines - pharmacology | Isoenzymes - deficiency | Myofibrils - physiology | Mice, Knockout | Creatine Kinase - genetics | Sarcomeres - ultrastructure | Animals | Ruthenium Red - pharmacology | Calcium Signaling - drug effects | Mitochondria, Heart - physiology | Thapsigargin - pharmacology | Benzimidazoles - pharmacology | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Mice | Nigericin - pharmacology | Sodium Azide - pharmacology | Energy Metabolism - drug effects | Index Medicus
Journal Article
Journal Article
Hearing Research, ISSN 0378-5955, 2005, Volume 204, Issue 1, pp. 170 - 182
Transient receptor potential (TRP) receptors are, typically, calcium-permeant cation channels that transduce environmental stimuli. Both kidney epithelial and... 
Cytoplasmic drug uptake | TRP channel | Aminoglycosides | Non-endocytotic uptake | GANGLIA NEURONS | SACCULAR HAIR-CELLS | AUDIOLOGY & SPEECH-LANGUAGE PATHOLOGY | CATION CHANNEL | VANILLOID RECEPTOR VR1 | FUNCTIONAL-CHARACTERIZATION | NEUROSCIENCES | AMINOGLYCOSIDE ANTIBIOTICS | CAPSAICIN RECEPTOR | FREEZE-FRACTURE | NOCICEPTIVE NEURONS | OTORHINOLARYNGOLOGY | ION-CHANNEL | aminoglycosides | cytoplasmic drug uptake | non-endocytotic uptake | Gadolinium - pharmacology | Calcium - metabolism | Lanthanum - pharmacology | Polyunsaturated Alkamides | Kidney Tubules, Distal - cytology | Gentamicins - metabolism | TRPV Cation Channels - antagonists & inhibitors | Ruthenium Red | Anti-Bacterial Agents - adverse effects | Endocannabinoids | Ionophores - pharmacology | Valinomycin - pharmacology | Diterpenes - pharmacology | Membrane Potentials - drug effects | Cell Line | TRPV Cation Channels - physiology | Gentamicins - adverse effects | TRPV Cation Channels - agonists | Anti-Bacterial Agents - metabolism | Calcium Channel Blockers - pharmacology | Cinnamates - pharmacology | Arachidonic Acids - pharmacology | Xanthenes | Animals | Kidney Tubules, Distal - drug effects | Anilides - pharmacology | Dogs | Indicators and Reagents | Fluorescent Dyes | Hydrogen-Ion Concentration | Kidney Tubules, Distal - metabolism | Index Medicus
Journal Article
BioMetals, ISSN 0966-0844, 10/2017, Volume 30, Issue 5, pp. 747 - 755
AQP9 is an aquaglyceroporin with a very broad substrate spectrum. In addition to its orthodox nutrient substrates, AQP9 also transports multiple neutral and... 
Biochemistry, general | Water | Microbiology | Lactate | Liver | AQP9 | Glycerol | Plant Physiology | Monomethyselenic acid (MSeA) | Medicine/Public Health, general | Cell Biology | Life Sciences | Selenite | Arsenic trioxide (As III ) | Pharmacology/Toxicology | ) | Arsenic trioxide (As | MOLECULAR CHARACTERIZATION | SODIUM-SELENITE | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | NEUTRAL SOLUTE CHANNEL | Arsenic trioxide (As-III) | HUMAN PROSTATE-CANCER | METHYLSELENINIC ACID | IN-VIVO | CELL-CYCLE | EXPRESSION | Humans | Lactic Acid - analogs & derivatives | Substrate Specificity | Selenious Acid - metabolism | Selenious Acid - antagonists & inhibitors | Aquaporins - genetics | Oocytes - cytology | Arsenicals - metabolism | Phloretin - pharmacology | Oocytes - drug effects | Biological Transport - drug effects | Lactic Acid - pharmacology | Transgenes | Cacodylic Acid - metabolism | Oxides - metabolism | Valinomycin - pharmacology | Gene Expression | Oocytes - metabolism | Xenopus laevis | Aquaporins - metabolism | Organoselenium Compounds - antagonists & inhibitors | Aquaporins - antagonists & inhibitors | Animals | Organometallic Compounds - metabolism | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Nigericin - pharmacology | Kinetics | Organoselenium Compounds - metabolism | Hydrogen-Ion Concentration | Selenium compounds | Glycerin | Lactates | Medical colleges | Arsenic | Gypsum | Selenium | Translocation | Valinomycin | Substrate inhibition | Arsenic trioxide | Biochemistry | pH effects | Substrates | Nigericin | Proteins | Acids | Hydrogen ions | Microenvironments | Lactic acid | Transport | Arsenite | Cancer | Index Medicus | liver | monomethyselenic acid (MSeA) | lactate | water | selenite | glycerol | arsenic trioxide (AsIII)
Journal Article