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PLoS Pathogens, ISSN 1553-7366, 05/2015, Volume 11, Issue 5, p. e1004901
Varicella zoster virus (VZV) causes chickenpox in humans and, subsequently, establishes latency in the sensory ganglia from where it reactivates to cause... 
FRAME-66 PROTEIN-KINASE | IFN-LAMBDA | IMMUNE-RESPONSE | II INTERFERON | ALPHA | MICROBIOLOGY | SIMIAN VARICELLA | ZOSTER-VIRUS | VIROLOGY | DNA-SEQUENCE | GENE-EXPRESSION | REGULATORY PROTEIN | PARASITOLOGY | Phosphorylation | Humans | STAT Transcription Factors - metabolism | Herpesvirus 3, Human - immunology | Janus Kinases - metabolism | Proteasome Endopeptidase Complex | Chickenpox - immunology | Herpesviridae Infections - pathology | Immediate-Early Proteins - metabolism | Varicellovirus - immunology | Cercopithecinae | Proteolysis | Chickenpox - virology | Herpesviridae Infections - metabolism | Interferon Type I - metabolism | Varicellovirus - physiology | Herpesvirus 3, Human - physiology | Recombinant Proteins - metabolism | Cell Line | Signal Transduction | Chickenpox - pathology | Chickenpox - metabolism | Herpesviridae Infections - virology | Immunity, Innate | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Host-Pathogen Interactions | STAT Transcription Factors - genetics | Animals | Immediate-Early Proteins - genetics | Gene Expression Regulation, Viral | Protein Processing, Post-Translational | DNA, Recombinant - metabolism | Interferon-Stimulated Gene Factor 3, gamma Subunit - antagonists & inhibitors | Herpesviridae Infections - immunology | Interferon Type I - antagonists & inhibitors | Colleges & universities | Viruses | Infections | Grants | Gene expression | Proteins | Rodents | Adenoviruses | Deoxyribonucleic acid | DNA | Interferon | Dihydrofolate reductase | Viral infections | Immune system
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2005, Volume 102, Issue 14, pp. 5144 - 5149
Detection and elimination of virus-infected cells by cytotoxic T lymphocytes depends on recognition of virus-derived peptides presented by MHC class I... 
T lymphocytes | Proteins | Molecules | Antigen presentation | Biological Sciences | Cellular metabolism | Viral proteins | Herpesviridae | Cell lines | Viruses | Immunity | EQUINE HERPESVIRUS-1 | MOLECULAR-MECHANISM | VIRUS PROTEIN ICP47 | GLYCOPROTEIN-M | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | CLASS-I EXPRESSION | PEPTIDE-LOADING COMPLEX | TRANSMEMBRANE DOMAINS | DISULFIDE-LINKED COMPLEX | ENDOPLASMIC-RETICULUM | T-Lymphocytes, Cytotoxic - immunology | Amino Acid Sequence | Cell Line | Viral Envelope Proteins - physiology | Varicellovirus - genetics | Viral Envelope Proteins - genetics | Varicellovirus - pathogenicity | Humans | Endoplasmic Reticulum - metabolism | Molecular Sequence Data | Recombinant Fusion Proteins - metabolism | ATP-Binding Cassette, Sub-Family B, Member 3 | Histocompatibility Antigens Class I - metabolism | Recombinant Fusion Proteins - antagonists & inhibitors | Varicellovirus - immunology | Biological Transport, Active | Animals | Adenosine Triphosphate - metabolism | Protein Binding | Recombinant Fusion Proteins - genetics | Viral Envelope Proteins - immunology | Proteasome Endopeptidase Complex - metabolism | ATP-Binding Cassette Sub-Family B Member 2 | ATP-Binding Cassette Transporters - antagonists & inhibitors | Research | Varicella-zoster virus | T cells | glycoprotein-m | peptide-loading complex | transmembrane domains | class-i expression | equine herpesvirus-1 | virus protein icp47 | molecular-mechanism | down-regulation | disulfide-linked complex | endoplasmic-reticulum
Journal Article
Journal Article