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JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2015, Volume 65, Issue 13, pp. 1273 - 1282
Abstract Background Statins can regress coronary atheroma and lower clinical events. Although pre-clinical studies suggest procalcific effects of statins... 
Cardiovascular | Internal Medicine | atherosclerosis | intravascular ultrasound | calcium | statins | CARDIAC & CARDIOVASCULAR SYSTEMS | CLINICAL-TRIAL | VASCULAR CALCIFICATION | RANDOMIZED CONTROLLED-TRIAL | BEAM COMPUTED-TOMOGRAPHY | LIPID-LOWERING THERAPY | ARTERY CALCIFICATION | IN-VITRO | CALCIFIED PLAQUE BURDEN | SMOOTH-MUSCLE-CELLS | Coronary Vessels - diagnostic imaging | Plaque, Atherosclerotic - diagnostic imaging | Coronary Vessels - drug effects | Coronary Vessels - pathology | Prospective Studies | Humans | Middle Aged | Ultrasonography, Interventional | Vascular Calcification - diagnostic imaging | Male | Coronary Artery Disease - drug therapy | Vascular Calcification - drug therapy | Disease Progression | Calcium - blood | Coronary Artery Disease - diagnostic imaging | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Coronary Artery Disease - pathology | Female | Aged | Plaque, Atherosclerotic - drug therapy | Plaque, Atherosclerotic - pathology | Calcification | Pharmaceutical industry | Atherosclerotic plaque | Statins | Cardiovascular agents | Medical research | Analysis | Cardiac patients | Medicine, Experimental | Coronary heart disease | Proteins | Lipoproteins | Medical imaging | Laboratories | Coronary vessels | Atherosclerosis | Cardiovascular disease | Lipids | Triglycerides | Cholesterol
Journal Article
Circulation, ISSN 0009-7322, 06/2013, Volume 127, Issue 24, pp. 2442 - 2451
BACKGROUND—Progerin is a mutant form of lamin A responsible for Hutchinson-Gilford progeria syndrome (HGPS), a premature aging disorder characterized by... 
Tissue-non specific alkaline phosphatase | Progerin | Pyrophosphate | Smooth | Vascular calcification | Hutchinson-Gilford progeria syndrome | Muscle | ATP | CALCIUM-PHOSPHATE DEPOSITION | AORTIC CALCIFICATION | CARDIAC & CARDIOVASCULAR SYSTEMS | progerin | ALKALINE-PHOSPHATASE | vascular calcification | MITOCHONDRIA | pyrophosphate | MUSCLE-CELL CALCIFICATION | tissue-non specific alkaline phosphatase | LAMIN | SMOOTH-MUSCLE | BISPHOSPHONATES | muscle | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | ATP RELEASE | smooth | Diphosphates - pharmacology | Diphosphates - metabolism | Muscle, Smooth, Vascular - metabolism | Alkaline Phosphatase - metabolism | Male | Aorta - metabolism | Progeria - metabolism | Progeria - drug therapy | Vascular Calcification - metabolism | Mice, Mutant Strains | Adenosine Triphosphate - metabolism | Mitochondria, Muscle - physiology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Treatment Outcome | Lamin Type A - metabolism | Vascular Calcification - drug therapy | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Animals | Mice | Diphosphates - therapeutic use | Animal experimentation | Blood circulation disorders | Usage | Progeria | Drug therapy | Pyrophosphates | Health aspects | Risk factors
Journal Article
Vascular Pharmacology, ISSN 1537-1891, 2016, Volume 82, pp. 30 - 40
Abstract Vascular calcification (VC), a disorder that causes blood vessel hardening and dysfunction, is a significant risk factor for type-2 diabetes mellitus... 
Cardiovascular | Vascular calcification | RANKL | TRAIL | Type-2 diabetes mellitus | OPG | SERUM OSTEOPROTEGERIN | AORTIC CALCIFICATION | PLASMA OSTEOPROTEGERIN | APOPTOSIS-INDUCING LIGAND | CORONARY-ARTERY CALCIFICATION | RECEPTOR ACTIVATOR | OSTEOCLASTIC DIFFERENTIATION | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | KAPPA-B LIGAND | RANK Ligand - metabolism | Humans | Diabetic Angiopathies - etiology | Arteries - metabolism | Atherosclerosis - etiology | Vascular Calcification - metabolism | Diabetic Angiopathies - pathology | Vascular Calcification - etiology | Osteoprotegerin - therapeutic use | Diabetes Mellitus, Type 2 - complications | Diabetic Angiopathies - metabolism | Atherosclerosis - pathology | Osteoclasts - pathology | Atherosclerosis - drug therapy | Signal Transduction | Osteogenesis - drug effects | Arteries - drug effects | Arteries - pathology | Diabetic Angiopathies - drug therapy | Vascular Calcification - drug therapy | Plaque, Atherosclerotic | TNF-Related Apoptosis-Inducing Ligand - metabolism | Vascular Calcification - pathology | Osteoclasts - metabolism | TNF-Related Apoptosis-Inducing Ligand - therapeutic use | Atherosclerosis - metabolism | Cardiovascular Agents - therapeutic use | Osteoblasts - pathology | Animals | RANK Ligand - antagonists & inhibitors | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Type 2 diabetes | Analysis | Atherosclerosis | Venture capital | Physiological aspects | Calcification | Cardiovascular diseases | Health aspects
Journal Article
Current Osteoporosis Reports, ISSN 1544-1873, 8/2017, Volume 15, Issue 4, pp. 255 - 270
We give an update on the etiology and potential treatment options of rare inherited monogenic disorders associated with arterial calcification and calcific... 
GACI | Medicine & Public Health | Orthopedics | Phosphate | ABCC6 | Pyrophosphate | ENPP1 | PXE | Epidemiology | Arterial calcification | FAMILIAL TUMORAL CALCINOSIS | OF-FUNCTION MUTATION | SINGLETON-MERTEN SYNDROME | KLOTHO-DEFICIENT MICE | BASAL GANGLIA CALCIFICATION | MATRIX GLA PROTEIN | CONNECTIVE-TISSUE MINERALIZATION | CAUSE PSEUDOXANTHOMA ELASTICUM | HUTCHINSON-GILFORD-PROGERIA | ENDOCRINOLOGY & METABOLISM | SMOOTH-MUSCLE-CELLS | Humans | Dental Enamel Hypoplasia - drug therapy | Metacarpus - abnormalities | Basal Ganglia Diseases - drug therapy | Aortic Diseases - metabolism | Hyperostosis, Cortical, Congenital - metabolism | Vascular Calcification - metabolism | Cartilage Diseases - genetics | Osteoporosis - metabolism | Muscular Diseases - drug therapy | Osteoporosis - genetics | Abnormalities, Multiple - genetics | Enzyme Replacement Therapy | Calcinosis - drug therapy | Muscular Diseases - metabolism | Vascular Calcification - drug therapy | Hyperostosis, Cortical, Congenital - genetics | Aortic Diseases - genetics | Phosphates - metabolism | Basal Ganglia Diseases - metabolism | Hyperostosis, Cortical, Congenital - drug therapy | Pseudoxanthoma Elasticum - genetics | Interferons - metabolism | Hyperphosphatemia - drug therapy | Hyperphosphatemia - genetics | Pulmonary Valve Stenosis - metabolism | Dental Enamel Hypoplasia - genetics | Cartilage Diseases - metabolism | Abnormalities, Multiple - metabolism | Calcinosis - genetics | Metacarpus - metabolism | Diphosphates - metabolism | Odontodysplasia - metabolism | Vascular Calcification - genetics | Hyperphosphatemia - metabolism | Osteoporosis - drug therapy | Abnormalities, Multiple - drug therapy | Progeria - metabolism | Cartilage Diseases - drug therapy | Progeria - drug therapy | Basal Ganglia Diseases - genetics | Hand Deformities, Congenital - metabolism | Dental Enamel Hypoplasia - metabolism | Calcinosis - metabolism | Gaucher Disease - drug therapy | Pseudoxanthoma Elasticum - metabolism | Pulmonary Valve Stenosis - genetics | Gaucher Disease - metabolism | Pseudoxanthoma Elasticum - drug therapy | Progeria - genetics | Hand Deformities, Congenital - drug therapy | Aortic Diseases - drug therapy | Gaucher Disease - genetics | Hand Deformities, Congenital - genetics | Odontodysplasia - drug therapy | Odontodysplasia - genetics | Muscular Diseases - genetics | Pulmonary Valve Stenosis - drug therapy | Phosphates | Calcification | Medicine, Experimental | Medical research | Interferon
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 02/2012, Volume 27, Issue 2, pp. 514 - 521
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2018, Volume 71, Issue 10, pp. 1117 - 1126
In pseudoxanthoma elasticum (PXE), low pyrophosphate levels may cause ectopic mineralization, leading to skin changes, visual impairment, and peripheral... 
bisphosphonates | PXE | etidronate | arterial calcification | AORTIC CALCIFICATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ABCC6 GENE | VASCULAR CALCIFICATION | PYROPHOSPHATE | DISEASE | GENERALIZED ARTERIAL CALCIFICATION | MUTATIONS | CARDIOVASCULAR RISK | INFANCY | Peripheral Arterial Disease - etiology | Calcium - metabolism | Pseudoxanthoma Elasticum - physiopathology | Humans | Middle Aged | Tomography, X-Ray Computed - methods | Male | Pseudoxanthoma Elasticum - complications | Drug Monitoring - methods | Femur - diagnostic imaging | Calcium - blood | Etidronic Acid - pharmacokinetics | Etidronic Acid - adverse effects | Vascular Calcification - etiology | Peripheral Arterial Disease - blood | Phosphates - blood | Female | Vascular Calcification - diagnosis | Bone Density Conservation Agents - pharmacokinetics | Etidronic Acid - administration & dosage | Bone Density Conservation Agents - adverse effects | Pseudoxanthoma Elasticum - drug therapy | Treatment Outcome | Bone Density Conservation Agents - administration & dosage | Positron-Emission Tomography - methods | Vascular Calcification - drug therapy | Peripheral Arterial Disease - prevention & control | Bone Density - drug effects | Peripheral Arterial Disease - diagnosis | Pseudoxanthoma Elasticum - diagnosis | Aged | Prevention | Phosphates | Care and treatment | Phosphonates | Bones | Pseudoxanthoma elasticum | Density | Calcification (ectopic) | Femur | Calcium | Systematic review | Vascularization | Calcium (blood) | Computed tomography | Mineralization | Atherosclerosis | Skin diseases | Bone density | Safety | Drug therapy | Hypocalcemia | Positron emission | Vitamin deficiency | Preventive medicine | Patients | Calcium phosphates | Side effects | Sodium | Visual impairment | Etidronic acid | Arteriosclerosis | Calcification | Hyperphosphatemia | Skin | Adults | Positron emission tomography | Veins & arteries
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 02/2014, Volume 29, Issue 2, pp. 450 - 457
Journal Article