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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2016, Volume 37, Issue 2, pp. 328 - 340
OBJECTIVE—The calcium composition of atherosclerotic plaque is thought to be associated with increased risk for cardiovascular events, but whether plaque... 
atherosclerosis | coronary artery disease | macrophages | inflammation | biomarkers | C-REACTIVE PROTEIN | CORONARY-ARTERY CALCIUM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | VULNERABLE PATIENT | RISK-ASSESSMENT STRATEGIES | COMPUTED-TOMOGRAPHY | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | NF-KAPPA-B | Up-Regulation | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | rac GTP-Binding Proteins - deficiency | rac GTP-Binding Proteins - metabolism | Male | Coronary Artery Disease - enzymology | Atherosclerosis - enzymology | Transfection | Interleukin-1beta - metabolism | rac GTP-Binding Proteins - genetics | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Aortic Diseases - prevention & control | Vascular Calcification - mortality | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aorta - enzymology | Aortic Diseases - pathology | Atherosclerosis - pathology | Coronary Vessels - pathology | Genetic Predisposition to Disease | Coronary Vessels - enzymology | Macrophages - pathology | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Neuropeptides - metabolism | Coronary Artery Disease - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Muscle, Smooth, Vascular - enzymology | Atherosclerosis - prevention & control | rac1 GTP-Binding Protein - metabolism | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2015, Volume 30, Issue 5, pp. 824 - 836
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2012, Volume 32, Issue 11, pp. 2580 - 2588
OBJECTIVE—Vascular calcification significantly increases cardiovascular morbidity and mortality. We recently reported that the deficiency of cartilage... 
Smooth muscle cell | Calcification | Animal model of human disease remodeling | MicroRNA | Protease | animal model of human disease remodeling | CELLS | smooth muscle cell | PATHOBIOLOGY | MECHANISMS | OLIGOMERIC MATRIX PROTEIN | protease | PHOSPHATE | IN-VITRO | ROLES | ARTERIAL CALCIFICATION | ANEURYSM DEVELOPMENT | microRNA | PERIPHERAL VASCULAR DISEASE | calcification | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | Up-Regulation | Carotid Artery Diseases - chemically induced | Humans | MicroRNAs - metabolism | Matrilin Proteins | Nephrectomy | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Transcription, Genetic | 3' Untranslated Regions | Real-Time Polymerase Chain Reaction | Organ Culture Techniques | Disease Models, Animal | Uremia - enzymology | Myocytes, Smooth Muscle - enzymology | Down-Regulation | Computational Biology | Rats | Vascular Calcification - pathology | Rats, Sprague-Dawley | Kidney Failure, Chronic - genetics | ADAM Proteins - metabolism | Aortic Diseases - genetics | Carotid Artery Diseases - enzymology | Carotid Artery, Common - enzymology | Carotid Artery Diseases - genetics | Kidney Failure, Chronic - complications | ADAMTS7 Protein | Carotid Artery, Common - pathology | ADAM Proteins - genetics | Muscle, Smooth, Vascular - enzymology | Vascular Calcification - genetics | Glycoproteins - metabolism | Myocytes, Smooth Muscle - pathology | Aortic Diseases - chemically induced | Transfection | Uremia - pathology | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Abdominal - pathology | Kidney Failure, Chronic - enzymology | Extracellular Matrix Proteins - metabolism | Aortic Diseases - pathology | Promoter Regions, Genetic | Calcium Chloride | Cartilage Oligomeric Matrix Protein | Cells, Cultured | Aorta, Abdominal - enzymology | Gene Expression Regulation, Enzymologic | Muscle, Smooth, Vascular - pathology | Animals | Vascular Calcification - chemically induced
Journal Article
Nature Communications, ISSN 2041-1723, 12/2015, Volume 6, Issue 1, p. 10006
Journal Article
Kidney International, ISSN 0085-2538, 10/2015, Volume 88, Issue 4, pp. 711 - 721
Vascular calcification (VC) is a major risk factor for cardiovascular mortality in chronic renal failure (CRF) patients, but the pathogenesis remains partially... 
inorganic phosphate | aging | vascular smooth muscle cell | chronic renal failure | LIFE-SPAN | STEM-CELLS | ACTIVATION | PROTEIN | OSTEOBLASTIC DIFFERENTIATION | MUSCLE-CELL CALCIFICATION | PHOSPHATE | UROLOGY & NEPHROLOGY | CHRONIC KIDNEY-DISEASE | EXPRESSION | CHRONIC-RENAL-FAILURE | Muscle, Smooth, Vascular - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Vascular Calcification - genetics | Glucuronidase - metabolism | Myocytes, Smooth Muscle - pathology | Aorta, Abdominal - drug effects | Glucuronidase - deficiency | TOR Serine-Threonine Kinases - antagonists & inhibitors | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Kidney Failure, Chronic - pathology | Tumor Suppressor Proteins - genetics | Aortic Diseases - prevention & control | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Kidney Failure, Chronic - enzymology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Genetic Predisposition to Disease | Tumor Suppressor Proteins - metabolism | Kidney Failure, Chronic - drug therapy | Mice, Inbred C57BL | Osteogenesis - drug effects | Cells, Cultured | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aorta, Abdominal - enzymology | Sirolimus - pharmacology | Mice, Knockout | Aorta, Thoracic - enzymology | Aortic Diseases - genetics | Phenotype | Animals | Glucuronidase - genetics | Signal Transduction - drug effects | Protein Kinase Inhibitors - pharmacology | Muscle, Smooth, Vascular - enzymology
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 10/2016, Volume 31, Issue 10, pp. 1865 - 1876
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2017, Volume 113, Issue 13, pp. 1639 - 1652
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2016, Volume 36, Issue 1, pp. 108 - 121
OBJECTIVE—Chronic administration of selective cyclooxygenase-2 (COX-2) inhibitors leads to an increased risk of adverse cardiovascular events, including... 
calcium | phosphates | adenine | myocardial infarction | vascular calcification | Phosphates | Humans | Vascular Calcification - genetics | Calcitriol | Myocytes, Smooth Muscle - pathology | Male | Aortic Diseases - chemically induced | Nephrectomy | Time Factors | Cyclooxygenase 2 - genetics | Kidney Failure, Chronic - chemically induced | Aortic Diseases - prevention & control | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Abdominal - pathology | Kidney Failure, Chronic - enzymology | Intramolecular Oxidoreductases - metabolism | Myocytes, Smooth Muscle - drug effects | Cyclooxygenase 2 Inhibitors - toxicity | Aorta, Thoracic - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Adenine | Vascular Calcification - prevention & control | Intramolecular Oxidoreductases - deficiency | Aorta, Abdominal - enzymology | Metabolomics - methods | Rats, Sprague-Dawley | Mice, Knockout | Dinoprostone - metabolism | Kidney Failure, Chronic - genetics | Aorta, Thoracic - enzymology | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Animals | Intramolecular Oxidoreductases - genetics | Prostaglandin-Endoperoxide Synthases - metabolism | Prostaglandin-E Synthases | Cyclooxygenase 2 - metabolism | Vascular Calcification - chemically induced | Cyclooxygenase 2 - deficiency | Muscle, Smooth, Vascular - enzymology
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 243, Issue 2, pp. 357 - 363
Abstract Reactive oxygen species (ROS)-induced osteogenic differentiation of vascular smooth muscle cells (VSMCs) is associated with the pathogenesis of... 
Cardiovascular | Vascular calcification | Oxidized low-density lipoprotein | Vascular peroxidase 1 | Osteogenic differentiation | Vascular smooth muscle cells | Hemeproteins - genetics | Alkaline Phosphatase - metabolism | NADPH Oxidases - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Core Binding Factor Alpha 1 Subunit - metabolism | RNA Interference | Time Factors | Aortic Diseases - prevention & control | Peroxidases - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Muscle, Smooth, Vascular - drug effects | Myocytes, Smooth Muscle - enzymology | NADPH Oxidases - antagonists & inhibitors | Vascular Calcification - pathology | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Aorta, Thoracic - enzymology | Aortic Diseases - genetics | Lipoproteins, LDL - toxicity | Signal Transduction - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Muscle, Smooth, Vascular - enzymology | Phosphorylation | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Aortic Diseases - chemically induced | Dose-Response Relationship, Drug | Transfection | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Thoracic - drug effects | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Hemeproteins - metabolism | Osteogenesis - drug effects | Cells, Cultured | Vascular Calcification - prevention & control | Muscle, Smooth, Vascular - pathology | Animals | Cell Differentiation - drug effects | Vascular Calcification - chemically induced | Protein Kinase Inhibitors - pharmacology | Peroxidases - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Hypochlorous Acid - metabolism | Oxidases | Hydrogen peroxide | Low density lipoproteins | Calcification | Smooth muscle | Peroxidase | Cardiovascular agents
Journal Article
Atherosclerosis, ISSN 0021-9150, 10/2017, Volume 265, pp. 78 - 86
Vascular calcification is a common health problem related to oxidative stress, inflammation, and circulating calciprotein particles (CPP). Hydrogen sulfide is... 
Sulfide | NQO1 | H2S/HS | Calciprotein particles | Vascular smooth muscle cells | S/HS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | KEAP1 | THIOSULFATE | IN-VITRO | NRF2 | DISEASE | HEMODIALYSIS-PATIENTS | PERIPHERAL VASCULAR DISEASE | EXPRESSION | FETUIN-A | Hydrogen Sulfide - metabolism | Calcium - metabolism | Humans | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | NAD(P)H Dehydrogenase (Quinone) - genetics | Dose-Response Relationship, Drug | Kelch-Like ECH-Associated Protein 1 - genetics | Transfection | RNA Interference | NF-E2-Related Factor 2 - genetics | Vascular Calcification - enzymology | Sulfides - metabolism | Myocytes, Smooth Muscle - drug effects | Muscle, Smooth, Vascular - drug effects | Sulfides - pharmacology | Cytokines - metabolism | Myocytes, Smooth Muscle - enzymology | Cells, Cultured | Vascular Calcification - prevention & control | Gene Expression Profiling - methods | Kelch-Like ECH-Associated Protein 1 - metabolism | Vascular Calcification - pathology | Muscle, Smooth, Vascular - pathology | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | High-Throughput Nucleotide Sequencing | Enzyme Activation | Oxidative Stress - drug effects | Muscle, Smooth, Vascular - enzymology | Hydrogen sulfide | Calcification | Smooth muscle | Index Medicus
Journal Article
Science Signaling, ISSN 1945-0877, 12/2016, Volume 9, Issue 458, pp. ra121 - ra121
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 01/2013, Volume 28, Issue 1, pp. 81 - 91
Medial vascular calcification (MVC) is common in patients with chronic kidney disease, obesity, and aging. MVC is an actively regulated process that resembles... 
HIGH‐THROUGHPUT SCREENING | KINETIC STUDIES | SMALL‐MOLECULES | PHARMACOLOGICAL INHIBITORS | ALKALINE PHOSPHATASE | HIGH-THROUGHPUT SCREENING | SMALL-MOLECULES | AORTIC CALCIFICATION | MATRIX VESICLES | MODEL | IDENTIFICATION | PYROPHOSPHATE | NONSPECIFIC ALKALINE-PHOSPHATASE | ARTERIAL CALCIFICATION | ENDOCRINOLOGY & METABOLISM | CHRONIC KIDNEY-DISEASE | SKELETAL MINERALIZATION | EADOCK DSS | Alkaline Phosphatase - metabolism | Myocytes, Smooth Muscle - pathology | Phosphoric Monoester Hydrolases - deficiency | 2-Pyridinylmethylsulfinylbenzimidazoles - pharmacology | RNA, Messenger - metabolism | Computer Simulation | Enzyme Inhibitors - chemistry | Inhibitory Concentration 50 | Vascular Calcification - enzymology | Myocytes, Smooth Muscle - drug effects | Phosphoric Monoester Hydrolases - antagonists & inhibitors | Catalytic Domain | Calcification, Physiologic - drug effects | Myocytes, Smooth Muscle - enzymology | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Models, Molecular | Vascular Calcification - pathology | Gene Expression Regulation - drug effects | Muscle, Smooth, Vascular - pathology | Alkaline Phosphatase - antagonists & inhibitors | Animals | Substrate Specificity - drug effects | Mice | Phosphoric Monoester Hydrolases - metabolism | Lansoprazole | Phosphoric Monoester Hydrolases - chemistry | Phosphates | Enzymes | Phosphatases | Genes | Smooth muscle | Gene expression | Chronic kidney failure | Analysis | Calcification | Universities and colleges | Research institutes | Children | Health aspects | Calcification (ectopic) | Alkaline phosphatase | Cell culture | Obesity | Transformation | high-throughput screening | Ossification | Mineralization | Phosphate | Aging | Kidney diseases | Bone (endochondral) | pharmacological inhibitors | High-throughput screening | small-molecules | alkaline phosphatase | kinetic studies
Journal Article