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Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | TNF-alpha | BONE MORPHOGENETIC PROTEIN-2 | MATRIX GLA PROTEIN | VASCULAR CALCIFICATION | aortic calcification | DEFICIENT MICE | metabolic syndrome | INFLAMMATION | IN-VIVO | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | HEMATOLOGY | diabetes | EXPRESSION | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2017, Volume 37, Issue 2, pp. 328 - 340
... outcomes remains highly controversial. Inflammation is likely a key mediator of vascular calcification, but immune signaling mechanisms that promote this process are minimally understood... 
atherosclerosis | coronary artery disease | macrophages | inflammation | biomarkers | C-REACTIVE PROTEIN | CORONARY-ARTERY CALCIUM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | VULNERABLE PATIENT | RECEPTOR | RISK-ASSESSMENT STRATEGIES | PLAQUE | SMOOTH-MUSCLE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Up-Regulation | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | rac GTP-Binding Proteins - deficiency | rac GTP-Binding Proteins - metabolism | Male | Coronary Artery Disease - enzymology | Atherosclerosis - enzymology | Transfection | Interleukin-1beta - metabolism | rac GTP-Binding Proteins - genetics | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Aortic Diseases - prevention & control | Vascular Calcification - mortality | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aorta - enzymology | Aortic Diseases - pathology | Atherosclerosis - pathology | Coronary Vessels - pathology | Genetic Predisposition to Disease | Coronary Vessels - enzymology | Macrophages - pathology | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Neuropeptides - metabolism | Coronary Artery Disease - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Muscle, Smooth, Vascular - enzymology | Atherosclerosis - prevention & control | rac1 GTP-Binding Protein - metabolism
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2015, Volume 65, Issue 13, pp. 1273 - 1282
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-9071, 2019, Volume 76, Issue 11, pp. 2077 - 2091
Medial vascular calcification has emerged as a putative key factor contributing to the excessive cardiovascular mortality of patients with chronic kidney disease (CKD... 
Life Sciences | Biomedicine, general | Biochemistry, general | Life Sciences, general | Phosphate | Vascular calcification | CKD | Osteogenic signaling | Vascular smooth muscle cells | Cell Biology | OXIDATIVE-STRESS | BONE MORPHOGENETIC PROTEIN-2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHOSPHATE-INDUCED CALCIFICATION | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | ARTERIAL MEDIAL CALCIFICATION | IN-VITRO | CALCIUM-SENSING RECEPTOR | WNT/BETA-CATENIN | GROWTH-FACTOR 23 | CHRONIC KIDNEY-DISEASE | RANK Ligand - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Hyperphosphatemia - metabolism | NF-kappa B - metabolism | Renal Insufficiency, Chronic - complications | Hyperphosphatemia - pathology | Renal Insufficiency, Chronic - metabolism | Vascular Calcification - metabolism | Renal Insufficiency, Chronic - genetics | Vascular Calcification - complications | Cell Transdifferentiation | Receptor Activator of Nuclear Factor-kappa B - metabolism | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | Hyperphosphatemia - complications | Chondrocytes - pathology | Signal Transduction | Gene Expression Regulation | Calcium Phosphates - chemistry | Vascular Calcification - pathology | Renal Insufficiency, Chronic - pathology | RANK Ligand - genetics | Osteoblasts - pathology | Muscle, Smooth, Vascular - pathology | Animals | NF-kappa B - genetics | Receptor Activator of Nuclear Factor-kappa B - genetics | Hyperphosphatemia - genetics | Osteoblasts - metabolism | Calcium Phosphates - metabolism | Chronic kidney failure | Mortality | Cardiac patients | Physiological aspects | Calcification | Calcium phosphate | Smooth muscle | Phosphorus imbalance | Precipitation (Meteorology) | Cardiology | Calcification (ectopic) | Phenotypes | Calcium | Chondroblasts | Crystals | Muscles | Intracellular signalling | Arteries | Calcium phosphates | Signal transduction | Biomedical materials | Pathways | Hyperphosphatemia | Biocompatibility | Kidney diseases | Intracellular | Cardiovascular diseases | Calcium (extracellular) | Review
Journal Article