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1. Full Text Molecular imaging of inflammation in atherosclerosis with targeted ultrasound detection of vascular cell adhesion molecule-1
by Kaufmann, Beat A and Sanders, John M and Davis, Christopher and Xie, Aris and Aldred, Patrick and Sarembock, Ian J and Lindner, Jonathan R
Circulation, ISSN 0009-7322, 07/2007, Volume 116, Issue 3, pp. 276 - 284
Background - The ability to image vascular inflammatory responses may allow early diagnosis and treatment of atherosclerosis. We hypothesized that molecular... 
Inflammation | Echocardiography | Imaging | Atherosclerosis | atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | echocardiography | MOUSE | RISK | imaging | PLAQUE | LESIONS | PREDICTION | STROKE | inflammation | ENDOTHELIUM | IN-VIVO | PERIPHERAL VASCULAR DISEASE | EXPRESSION | THICKNESS | Hypercholesterolemia - diagnostic imaging | Atherosclerosis - pathology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Mice, Inbred C57BL | Cells, Cultured | Male | Mice, Transgenic | Atherosclerosis - metabolism | Inflammation - metabolism | Animals | Atherosclerosis - diagnostic imaging | Hypercholesterolemia - pathology | Inflammation Mediators - analysis | Apolipoproteins E - genetics | Inflammation Mediators - metabolism | Microbubbles | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Hypercholesterolemia - genetics | Inflammation - diagnosis | Vascular Cell Adhesion Molecule-1 - analysis | Hypercholesterolemia - metabolism | Ultrasonography - methods | Diagnosis, Ultrasonic | Analysis | Diagnosis | Research | Methods | Cell adhesion molecules | Index Medicus | Abridged Index Medicus
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2. Full Text CD38/CD31, the CCL3 and CCL4 Chemokines, and CD49d/Vascular Cell Adhesion Molecule-1 Are Interchained by Sequential Events Sustaining Chronic Lymphocytic Leukemia Cell Survival
by Zucchetto, A and Benedetti, D and Tripodo, C and Bomben, R and Dal Bo, M and Marconi, D and Bossi, F and Lorenzon, D and Degan, M and Rossi, FM and Rossi, D and Bulian, P and Franco, V and Del Poeta, G and Deaglio, S and Gaidano, G and Tedesco, F and Malavasi, F and Gattei, V
CANCER RESEARCH, ISSN 0008-5472, 05/2009, Volume 69, Issue 9, pp. 4001 - 4009
CD38 and CD49d are associated negative prognosticators in chronic lymphocytic leukemia (CLL). Despite evidence that both molecules are involved in interactions... 
B-CELLS | CD49D EXPRESSION | ALPHA-4-BETA-1 INTEGRIN | NURSELIKE CELLS | ONCOLOGY | PROGRESSIVE DISEASE | ENDOTHELIAL-CELLS | LYMPH-NODES | BONE-MARROW | CD38 EXPRESSION | GENE-EXPRESSION SIGNATURES | Integrin alpha4 - biosynthesis | Antigens, CD - immunology | Up-Regulation | Chemokine CCL4 - biosynthesis | Chemokine CCL3 - immunology | Antigens, CD - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | Platelet Endothelial Cell Adhesion Molecule-1 - immunology | Chemokine CCL3 - biosynthesis | Bone Marrow Cells - immunology | ADP-ribosyl Cyclase 1 - immunology | Receptors, Chemokine - biosynthesis | Leukemia, Lymphocytic, Chronic, B-Cell - blood | Membrane Glycoproteins - immunology | Leukemia, Lymphocytic, Chronic, B-Cell - immunology | Macrophages - immunology | Cell Line | Vascular Cell Adhesion Molecule-1 - immunology | Receptors, Chemokine - immunology | Cell Survival - immunology | Integrin alpha4 - immunology | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Endothelial Cells - immunology | Apoptosis - immunology | Endothelial Cells - cytology | ADP-ribosyl Cyclase 1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - biosynthesis | Chemokine CCL4 - immunology | Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis | Index Medicus
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3. Full Text Glucocorticoid-induced tumor necrosis factor receptor family-related ligand triggering upregulates vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 and promotes leukocyte adhesion
by Lacal, Pedro Miguel and Petrillo, Maria Grazia and Ruffini, Federica and Muzi, Alessia and Bianchini, Rodolfo and Ronchetti, Simona and Migliorati, Graziella and Riccardi, Carlo and Graziani, Grazia and Nocentini, Giuseppe
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 10/2013, Volume 347, Issue 1, pp. 164 - 172
The interaction of glucocorticoid-induced tumor necrosis factor receptor-family related (GITR) protein with its ligand (GITRL) modulates different functions,... 
MEDIATED INDUCTION | INTERFERON-GAMMA | PROTEIN GITR | IFN-GAMMA | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | MULTIPLE ORGAN FAILURE | TNF-ALPHA | PHARMACOLOGICAL INHIBITION | T-CELLS | GENE GITR | Tumor Necrosis Factors - agonists | Humans | Glucocorticoid-Induced TNFR-Related Protein - metabolism | Tumor Necrosis Factors - metabolism | Glucocorticoid-Induced TNFR-Related Protein - agonists | Leukocytes - physiology | Mice, Knockout | Tumor Necrosis Factors - biosynthesis | Glucocorticoid-Induced TNFR-Related Protein - biosynthesis | Animals | Cell Adhesion - physiology | Intercellular Adhesion Molecule-1 - biosynthesis | Up-Regulation - physiology | HL-60 Cells | Ligands | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Leukocytes - metabolism | Cell Line, Transformed | Index Medicus
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4. Full Text Superoxide Dismutase Inhibits the Expression of Vascular Cell Adhesion Molecule-1 and Intracellular Cell Adhesion Molecule-1 Induced by Tumor Necrosis Factor-α in Human Endothelial Cells Through the JNK/p38 Pathways
by Lin, Shing-Jong and Shyue, Song-Kun and Hung, Ya-Yun and Chen, Yung-Hsiang and Ku, Hung-Hai and Chen, Jaw-Wen and Tam, Ka-Bik and Chen, Yuh-Lien
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2005, Volume 25, Issue 2, pp. 334 - 340
OBJECTIVE—Expression of adhesion molecules on endothelial cells and subsequent leukocyte recruitment are critical early events in the development of... 
Superoxide dismutase | Endothelial cell | Adhesion molecule | Atherosclerosis | MAPKs | atherosclerosis | ACTIVATION | INDUCED APOPTOSIS | endothelial cell | adhesion molecule | MAP KINASES | INDUCTION | AP-1 | SIGNAL-TRANSDUCTION | IN-VITRO | superoxide dismutase | VCAM-1 EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | NF-KAPPA-B | Endothelium, Vascular - cytology | Neutrophils - cytology | Transcription, Genetic - drug effects | MAP Kinase Signaling System - physiology | Phosphorylation | Superoxide Dismutase - genetics | Humans | Recombinant Fusion Proteins - physiology | Depression, Chemical | Superoxide Dismutase - physiology | Intercellular Adhesion Molecule-1 - biosynthesis | Superoxides - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Flavonoids - pharmacology | NF-kappa B - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Transduction, Genetic | p38 Mitogen-Activated Protein Kinases - physiology | Endothelial Cells - metabolism | NADPH Oxidases - antagonists & inhibitors | Imidazoles - pharmacology | Inflammation | Cell Adhesion - drug effects | Anthracenes - pharmacology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Arteriosclerosis - prevention & control | Transcription Factor AP-1 - antagonists & inhibitors | MAP Kinase Signaling System - drug effects | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Protein Processing, Post-Translational | Pyridines - pharmacology | Aorta - cytology | Superoxide Dismutase-1 | Index Medicus
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5. Full Text An inflammation-induced mechanism for leukocyte transmigration across lymphatic vessel endothelium
by Johnson, Louise A and Clasper, Steven and Holt, Andrew P and Lalor, Patricia F and Baban, Dilair and Jackson, David G
Journal of Experimental Medicine, ISSN 0022-1007, 11/2006, Volume 203, Issue 12, pp. 2763 - 2777
The exit of antigen-presenting cells and lymphocytes from inflamed skin to afferent lymph is vital for the initiation and maintenance of dermal immune... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | CHEMOKINE TRANSPORT | AFFERENT LYMPHATICS | INTERCELLULAR-ADHESION MOLECULE-1 | TRANSENDOTHELIAL MIGRATION | CONTACT HYPERSENSITIVITY | MATURE DENDRITIC CELLS | MONOCLONAL-ANTIBODY | PERIPHERAL-TISSUES | IMMUNOLOGY | LANGERHANS CELLS | Intercellular Adhesion Molecule-1 - physiology | Leukocytes - pathology | Dermatitis, Contact - pathology | Endothelium, Lymphatic - immunology | Dermatitis, Contact - metabolism | Humans | Cells, Cultured | Lymphatic Vessels - immunology | Male | Vascular Cell Adhesion Molecule-1 - physiology | Leukocytes - immunology | Dermatitis, Contact - immunology | Lymphatic Vessels - metabolism | Animals | Intercellular Adhesion Molecule-1 - biosynthesis | Lymphatic Vessels - pathology | Adult | Vascular Cell Adhesion Molecule-1 - biosynthesis | Endothelium, Lymphatic - metabolism | Mice | Mice, Inbred BALB C | Leukocytes - metabolism | Endothelium, Lymphatic - pathology | Index Medicus
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6. Full Text Angiotensin II induces vascular cell adhesion molecule-1 expression in rat vasculature: A potential link between the renin-angiotensin system and atherosclerosis
by Tummala, Pradyumna E and Chen, Xi-Lin and Sundell, Cynthia L and Laursen, Jørn Bech and Hammes, C. Patricia and Alexander, R. Wayne and Harrison, David G and Medford, Russell M
Circulation, ISSN 0009-7322, 09/1999, Volume 100, Issue 11, pp. 1223 - 1229
Background-Cardiovascular ischemic events may occur more frequently in hypertensive patients with activated renin-angiotensin systems. We tested the hypothesis... 
Hypertension | NF-κB | VCAM-1 | Angiotensin | CONVERTING ENZYME | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | RECEPTOR ANTAGONIST | NF-kappa B | CORONARY ATHEROSCLEROSIS | ENDOTHELIAL-CELLS | E-SELECTIN | angiotensin | HYPERTENSIVE RATS | PERIPHERAL VASCULAR DISEASE | LEUKOCYTE ADHESION | HEMATOLOGY | hypertension | NF-KAPPA-B | SMOOTH-MUSCLE CELLS | Index Medicus | Abridged Index Medicus
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7. Full Text Intermittent high glucose enhances ICAM-1, VCAM-1 and E-selectin expression in human umbilical vein endothelial cells in culture: The distinct role of protein kinase C and mitochondrial superoxide production
by Quagliaro, Lisa and Piconi, Ludovica and Assaloni, Roberta and Da Ros, Roberto and Maier, Amabile and Zuodar, Gianni and Ceriello, Antonio
Atherosclerosis, ISSN 0021-9150, 2005, Volume 183, Issue 2, pp. 259 - 267
In this study the effects of stable and intermittent high glucose concentrations on ICAM-1, VCAM-1 and E-selectin production, PKC activity and PKCβI, βII and δ... 
Adhesion molecules | PKC | Mitochondrial superoxide | Intermittent glucose | Endothelium | endothelium | OXIDATIVE STRESS | ACTIVATION | CIRCULATING ADHESION MOLECULES | BLOOD-GLUCOSE | ATHEROSCLEROSIS | MECHANISMS | intermittent glucose | DIABETES-MELLITUS | mitochondrial superoxide | HYPERGLYCEMIA | adhesion molecules | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | COMPLICATIONS | Endothelium, Vascular - cytology | Mitochondria - enzymology | Vascular Cell Adhesion Molecule-1 - drug effects | Gene Expression - drug effects | Humans | Glucose - administration & dosage | Dose-Response Relationship, Drug | RNA, Messenger - biosynthesis | E-Selectin - genetics | Protein Kinase C - metabolism | Intercellular Adhesion Molecule-1 - biosynthesis | E-Selectin - biosynthesis | Superoxides - metabolism | Intercellular Adhesion Molecule-1 - drug effects | Vascular Cell Adhesion Molecule-1 - genetics | E-Selectin - drug effects | Umbilical Veins - cytology | Deoxyguanosine - analogs & derivatives | Infant, Newborn | Enzyme-Linked Immunosorbent Assay | Protein Kinase C - drug effects | RNA, Messenger - genetics | Cells, Cultured | Blotting, Western | Blotting, Northern | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | Deoxyguanosine - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Oxidative Stress - drug effects | In Vitro Techniques | Sweetening Agents - administration & dosage | Umbilical Veins - metabolism | Messenger RNA | Atherosclerosis | Superoxide | Glucose | Protein kinases | Dextrose | Porphyrins | Index Medicus
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8. Full Text Protein kinase A-α directly phosphorylates FoxO1 in vascular endothelial cells to regulate expression of vascular cellular adhesion molecule-1 mRNA
by Lee, Ji-Won and Chen, Hui and Pullikotil, Philomena and Quon, Michael J
Journal of Biological Chemistry, ISSN 0021-9258, 02/2011, Volume 286, Issue 8, pp. 6423 - 6432
FoxO1, a forkhead box O class transcription factor, is abundant in insulin-responsive tissues. Akt, downstream from phosphatidylinositol 3-kinase in insulin... 
STIMULATES PHOSPHORYLATION | ACTIVATION | ANGIOGENESIS | METABOLISM | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PKA | INVOLVEMENT | MECHANISMS | CAMP | FORKHEAD TRANSCRIPTION FACTORS | Phosphorylation - physiology | RNA, Small Interfering - genetics | Humans | Transcriptional Activation - drug effects | Monocytes - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Isoquinolines - pharmacology | Cattle | Forkhead Transcription Factors - metabolism | HEK293 Cells | Vascular Cell Adhesion Molecule-1 - genetics | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Endothelial Cells - metabolism | Cell Adhesion - drug effects | Forkhead Transcription Factors - genetics | Sulfonamides - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Cell Adhesion - physiology | Endothelial Cells - cytology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Forkhead Box Protein O1 | Protein Kinase Inhibitors - pharmacology | Transcriptional Activation - physiology | Mutation | Protein kinase A | Alanine | Phosphorylation | Transcription factors | Antibodies | AKT protein | siRNA | Insulin | Endothelial cells | Promoters | 1-Phosphatidylinositol 3-kinase | Monocytes | Prostaglandins | FOXO1 protein | Aorta | Forkhead protein | Tumor necrosis factor- alpha | Antigen-antibody complexes | Index Medicus | Protein Kinase A (PKA) | VCAM-1 | Signal Transduction | Transcription Factors | Akt PKB | Endothelium | Protein-Protein Interactions | FoxO1
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9. Full Text Motorcycle exhaust particles up-regulate expression of vascular adhesion molecule-1 and intercellular adhesion molecule-1 in human umbilical vein endothelial cells
by Lee, Chen-Chen and Huang, Shih-Hsuan and Yang, Ya-Ting and Cheng, Yu-Wen and Li, Ching-Hao and Kang, Jaw-Jou
Toxicology in Vitro, ISSN 0887-2333, 06/2012, Volume 26, Issue 4, pp. 552 - 560
► The motorcycle exhaust particle (MEP) induced adhesion between cells of the THP-1 and HUVECs in a time-dependent manner. ► MEPs could induce ICAM-1 and... 
Motorcycle exhaust particle | Oxidative stress | NF-κB | Polycyclic aromatic hydrocarbons | Cell adhesion molecule | LUNG | KAPPA-B ACTIVATION | ATHEROSCLEROSIS | AIR-POLLUTION | NF-kappa B | EPITHELIAL-CELLS | IN-VIVO | CARDIOVASCULAR-DISEASE | ULTRAFINE PARTICLES | CYCLOOXYGENASE-2 EXPRESSION | TOXICOLOGY | CORONARY-ARTERY-DISEASE | Up-Regulation | Vehicle Emissions - toxicity | Human Umbilical Vein Endothelial Cells - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Oxidative Stress | Polycyclic Aromatic Hydrocarbons - toxicity | Humans | Soot - toxicity | NF-kappa B - metabolism | Cell Adhesion - drug effects | RNA, Messenger - biosynthesis | Intercellular Adhesion Molecule-1 - biosynthesis | Intercellular Adhesion Molecule-1 - genetics | Motorcycles | Cell Line, Tumor | Vascular Cell Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Air Pollutants - toxicity | Endothelium | Index Medicus
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10. Full Text Ginsenoside Rb1 Inhibits Tumor Necrosis Factor-.ALPHA.-Induced Vascular Cell Adhesion Molecule-1 Expression in Human Endothelial Cells
by Chai, Hui and Wang, Qiuyan and Huang, Lifeng and Xie, Tian and Fu, Yan
Biological and Pharmaceutical Bulletin, ISSN 0918-6158, 2008, Volume 31, Issue 11, pp. 2050 - 2056
This article was retracted. Please see the retraction notice. 
Superoxide anion | Endothelial cell | Tumor necrosis factor-alpha | Ginsenoside Rb1 | Vascular cell adhesion molecule-1 | ACTIVATION | endothelial cell | ATHEROSCLEROSIS | COMPOUND-K | vascular cell adhesion molecule-1 | ginsenoside Rb1 | tumor necrosis factor-alpha | IN-VITRO | VCAM-1 | DISEASES | PATHWAY | INFLAMMATION | MONOCYTE ADHESION | PHARMACOLOGY & PHARMACY | superoxide anion | NF-KAPPA-B | Cell Line | Vascular Cell Adhesion Molecule-1 - immunology | Phosphorylation | Endothelial Cells - metabolism | Humans | NF-kappa B - metabolism | Ginsenosides - pharmacology | Cell Adhesion - drug effects | Membrane Potential, Mitochondrial - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Endothelial Cells - immunology | Tumor Necrosis Factor-alpha - pharmacology | Superoxides - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
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11. Full Text Novel modulator for endothelial adhesion molecules: Adipocyte-derived plasma protein adiponectin
by Ouchi, Noriyuki and Kihara, Shinji and Arita, Yukio and Maeda, Kazuhisa and Kuriyama, Hiroshi and Okamoto, Yoshihisa and Hotta, Kikuko and Nishida, Makoto and Takahashi, Masahiko and Nakamura, Tadashi and Yamashita, Shizuya and Funahashi, Tohru and Matsuzawa, Yuji
Circulation, ISSN 0009-7322, 12/1999, Volume 100, Issue 25, pp. 2473 - 2476
Background-Among the many adipocyte-derived endocrine factors, we recently found an adipocyte-specific secretory protein, adiponectin, which was decreased in... 
Coronary disease | Adiponectin | Cell adhesion molecules | Endothelium | endothelium | adiponectin | cell adhesion molecules | VISCERAL FAT | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | SELECTIN | coronary disease | OBESITY | GENE | CLONING | DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | ADIPOSE | Index Medicus | Abridged Index Medicus
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12. Full Text Shear-sensitive microRNA-34a modulates flow-dependent regulation of endothelial inflammation
by Fan, Wendong and Fang, Rong and Wu, Xiaoyuan and Liu, Jia and Feng, Mingzhe and Dai, Gang and Chen, Guojun and Wu, Guifu
Journal of Cell Science, ISSN 0021-9533, 2015, Volume 128, Issue 1, pp. 70 - 80
Although many studies have described the roles of microRNAs (miRNAs) in the modulation of the endothelial response to shear stress, the mechanisms remain... 
Shear stress | Inflammation | MicroRNA | SIRT1 | Endothelial cells | CELLS | ATHEROSCLEROSIS | CELL BIOLOGY | DISTURBED FLOW | BIOGENESIS | IN-VIVO | GENE-EXPRESSION | SENESCENCE | STRESS | NF-KAPPA-B | CORONARY-ARTERY-DISEASE | Inflammation - pathology | Cell Adhesion - genetics | Shear Strength | Human Umbilical Vein Endothelial Cells - metabolism | Signal Transduction | Humans | Gene Expression Regulation | MicroRNAs - metabolism | Monocytes - metabolism | Transcription Factor RelA - genetics | Inflammation - metabolism | Transcription Factor RelA - metabolism | Intercellular Adhesion Molecule-1 - biosynthesis | Intercellular Adhesion Molecule-1 - genetics | Monocytes - pathology | Inflammation - genetics | Human Umbilical Vein Endothelial Cells - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | MicroRNAs - genetics | Index Medicus
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13. Full Text Molecular magnetic resonance imaging of acute vascular cell adhesion molecule-1 expression in a mouse model of cerebral ischemia
by Hoyte, Lisa C and Brooks, Keith J and Nagel, Simon and Akhtar, Asim and Chen, Ruoli and Mardiguian, Sylvie and McAteer, Martina A and Anthony, Daniel C and Choudhury, Robin P and Buchan, Alastair M and Sibson, Nicola R
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 06/2010, Volume 30, Issue 6, pp. 1178 - 1187
The pathogenesis of stroke is multifactorial, and inflammation is thought to have a critical function in lesion progression at early time points. Detection of... 
Focal ischemia | Molecular imaging | Adhesion molecules | Inflammation | MRI | ARTERY OCCLUSION | RAT | PROTECTION | molecular imaging | NEUROSCIENCES | MURINE STROKE | IRON-OXIDE | BRAIN INFLAMMATION | adhesion molecules | inflammation | ENDOCRINOLOGY & METABOLISM | focal ischemia | MICE | HEMATOLOGY | RECEPTOR-BINDING | INFLAMMATORY MEDIATORS | Humans | Gene Expression Regulation | Infarction, Middle Cerebral Artery - metabolism | Radiography | Ischemic Preconditioning - methods | Magnetic Resonance Imaging | Inflammation - metabolism | Animals | Endothelium, Vascular - diagnostic imaging | Endothelium, Vascular - metabolism | Monitoring, Physiologic - methods | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Inflammation - diagnostic imaging | Infarction, Middle Cerebral Artery - diagnostic imaging | Disease Models, Animal | Index Medicus | Original
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14. Full Text Dihydrotestosterone Decreases Tumor Necrosis Factor-α and Lipopolysaccharide-Induced Inflammatory Response in Human Endothelial Cells
by Norata, Giuseppe Danilo and Tibolla, Gianpaolo and Seccomandi, Paul Maria and Poletti, Angelo and Catapano, Alberico Luigi
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 02/2006, Volume 91, Issue 2, pp. 546 - 554
Context: An increasing body of evidence suggests that testosterone may exert beneficial effects on the development of atherosclerosis. It was suggested that... 
ACTIVATION | CAROTID ATHEROSCLEROSIS | EPITHELIAL-CELLS | TESTOSTERONE | ENDOCRINOLOGY & METABOLISM | CYCLOOXYGENASE-2 EXPRESSION | MECHANISMS | DEPENDENT PATHWAY | ELDERLY-MEN | CORONARY-ARTERY-DISEASE | ADHESION MOLECULE-1 EXPRESSION | Androgen Receptor Antagonists | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Humans | Immunoblotting | NF-kappa B - metabolism | Cyclooxygenase 2 - biosynthesis | RNA, Messenger - biosynthesis | Inflammation - metabolism | Drug Interactions | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Inflammation - drug therapy | Intercellular Adhesion Molecule-1 - biosynthesis | E-Selectin - biosynthesis | Dihydrotestosterone - pharmacology | Nitriles | Tosyl Compounds | Vascular Cell Adhesion Molecule-1 - genetics | Endothelial Cells - metabolism | RNA, Messenger - genetics | Androgen Antagonists - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Endothelial Cells - immunology | Receptors, Androgen - immunology | Tumor Necrosis Factor-alpha - pharmacology | Intercellular Adhesion Molecule-1 - genetics | Anilides - pharmacology | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Vascular Cell Adhesion Molecule-1 - biosynthesis | Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis | Endothelial Cells - drug effects | Index Medicus | Abridged Index Medicus
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15. Full Text High Glucose Increases Angiopoietin-2 Transcription in Microvascular Endothelial Cells through Methylglyoxal Modification of mSin3A
by Dachun Yao and Tetsuya Taguchi and Takeshi Matsumura and Richard Pestell and Diane Edelstein and Ida Giardino and Guntram Suske and Naila Rabbani and Paul J. Thornalley and Vijay P. Sarthy and Hans-Peter Hammes and Michael Brownlee
Journal of Biological Chemistry, ISSN 0021-9258, 10/2007, Volume 282, Issue 42, pp. 31038 - 31045
Methylglyoxal is a highly reactive dicarbonyl degradation product formed from triose phosphates during glycolysis. Methylglyoxal forms stable adducts primarily... 
DIABETIC-RETINOPATHY | ACTIVATION | ADVANCED GLYCATION | SP1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | O-GLYCOSYLATION | MICE | 3 MAJOR PATHWAYS | EXPRESSION | HYPERGLYCEMIC DAMAGE | GLYOXALASE-I | Tumor Necrosis Factor-alpha - metabolism | Transcription, Genetic - drug effects | Kidney - pathology | Sweetening Agents - metabolism | Angiopoietin-2 - genetics | Protein Processing, Post-Translational - genetics | Diabetes Mellitus, Experimental - genetics | N-Acetylglucosaminyltransferases - genetics | Glycolysis - drug effects | Acetylglucosamine - metabolism | Glycolysis - genetics | Pyruvaldehyde - metabolism | Kidney - metabolism | Diabetic Angiopathies - pathology | Protein Processing, Post-Translational - drug effects | Arginine - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Response Elements - genetics | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Repressor Proteins - metabolism | Sp3 Transcription Factor - metabolism | Diabetic Angiopathies - metabolism | Sweetening Agents - pharmacology | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Sp3 Transcription Factor - genetics | Angiopoietin-2 - biosynthesis | Repressor Proteins - genetics | Glucose - pharmacology | Acetylglucosamine - genetics | N-Acetylglucosaminyltransferases - metabolism | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Intercellular Adhesion Molecule-1 - genetics | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Endothelial Cells - pathology | Arginine - metabolism | Cell Line, Transformed | Index Medicus
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16. Full Text α4-Integrin Antibody Treatment Blocks Monocyte/Macrophage Traffic to, Vascular Cell Adhesion Molecule-1 Expression in, and Pathology of the Dorsal Root Ganglia in an SIV Macaque Model of HIV-Peripheral Neuropathy
by Lakritz, Jessica R and Thibault, Derek M and Robinson, Jake A and Campbell, Jennifer H and Miller, Andrew D and Williams, Kenneth C and Burdo, Tricia H
American Journal of Pathology, The, ISSN 0002-9440, 2016, Volume 186, Issue 7, pp. 1754 - 1761
Traffic of activated monocytes into the dorsal root ganglia (DRG) is critical for pathology in HIV peripheral neuropathy. We have shown that accumulation of... 
Pathology | HIV Infections | Immunohistochemistry | Simian Acquired Immunodeficiency Syndrome - metabolism | Male | Monocytes - metabolism | Macaca mulatta | Monocytes - immunology | Simian Acquired Immunodeficiency Syndrome - complications | Peripheral Nervous System Diseases - metabolism | Peripheral Nervous System Diseases - virology | Macrophages - metabolism | Animals | Integrin alpha4 - metabolism | Chemotaxis, Leukocyte - drug effects | Natalizumab - pharmacology | Simian Acquired Immunodeficiency Syndrome - immunology | Ganglia, Spinal - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Peripheral Nervous System Diseases - pathology | Immunologic Factors - pharmacology | Macrophages - immunology | Index Medicus | Abridged Index Medicus | Short Communication
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