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1. Full Text Superoxide Dismutase Inhibits the Expression of Vascular Cell Adhesion Molecule-1 and Intracellular Cell Adhesion Molecule-1 Induced by Tumor Necrosis Factor-α in Human Endothelial Cells Through the JNK/p38 Pathways
by Lin, Shing-Jong and Shyue, Song-Kun and Hung, Ya-Yun and Chen, Yung-Hsiang and Ku, Hung-Hai and Chen, Jaw-Wen and Tam, Ka-Bik and Chen, Yuh-Lien
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2005, Volume 25, Issue 2, pp. 334 - 340
Superoxide dismutase | Endothelial cell | Adhesion molecule | Atherosclerosis | MAPKs | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Cardiology. Vascular system | Blood vessels and receptors | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Medical sciences | Vertebrates: cardiovascular system | Blood and lymphatic vessels | Endothelium, Vascular - cytology | Neutrophils - cytology | Transcription, Genetic - drug effects | MAP Kinase Signaling System - physiology | Phosphorylation | Superoxide Dismutase - genetics | Humans | Recombinant Fusion Proteins - physiology | Depression, Chemical | Superoxide Dismutase - physiology | Intercellular Adhesion Molecule-1 - biosynthesis | Superoxides - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Flavonoids - pharmacology | NF-kappa B - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Transduction, Genetic | p38 Mitogen-Activated Protein Kinases - physiology | Endothelial Cells - metabolism | NADPH Oxidases - antagonists & inhibitors | Imidazoles - pharmacology | Inflammation | Cell Adhesion - drug effects | Anthracenes - pharmacology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Arteriosclerosis - prevention & control | Transcription Factor AP-1 - antagonists & inhibitors | MAP Kinase Signaling System - drug effects | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Protein Processing, Post-Translational | Pyridines - pharmacology | Aorta - cytology | Superoxide Dismutase-1 | Index Medicus
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2. Glucocorticoid-induced tumor necrosis factor receptor family-related ligand triggering upregulates vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 and promotes leukocyte adhesion
by Lacal, Pedro Miguel and Petrillo, Maria Grazia and Ruffini, Federica and Muzi, Alessia and Bianchini, Rodolfo and Ronchetti, Simona and Migliorati, Graziella and Riccardi, Carlo and Graziani, Grazia and Nocentini, Giuseppe
The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 10/2013, Volume 347, Issue 1, pp. 164 - 172
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Tumor Necrosis Factors - agonists | Humans | Glucocorticoid-Induced TNFR-Related Protein - metabolism | Tumor Necrosis Factors - metabolism | Glucocorticoid-Induced TNFR-Related Protein - agonists | Leukocytes - physiology | Mice, Knockout | Tumor Necrosis Factors - biosynthesis | Glucocorticoid-Induced TNFR-Related Protein - biosynthesis | Animals | Cell Adhesion - physiology | Intercellular Adhesion Molecule-1 - biosynthesis | Up-Regulation - physiology | HL-60 Cells | Ligands | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Leukocytes - metabolism | Cell Line, Transformed
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3. Full Text Ginsenoside Rb1 Inhibits Tumor Necrosis Factor-.ALPHA.-Induced Vascular Cell Adhesion Molecule-1 Expression in Human Endothelial Cells
by Chai, Hui and Wang, Qiuyan and Huang, Lifeng and Xie, Tian and Fu, Yan
Biological & pharmaceutical bulletin, ISSN 0918-6158, 2008, Volume 31, Issue 11, pp. 2050 - 2056
Superoxide anion | Endothelial cell | Tumor necrosis factor-alpha | Ginsenoside Rb1 | Vascular cell adhesion molecule-1 | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Cell Line | Vascular Cell Adhesion Molecule-1 - immunology | Phosphorylation | Endothelial Cells - metabolism | Humans | NF-kappa B - metabolism | Ginsenosides - pharmacology | Cell Adhesion - drug effects | Membrane Potential, Mitochondrial - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Endothelial Cells - immunology | Tumor Necrosis Factor-alpha - pharmacology | Superoxides - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
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4. Full Text CD38/CD31, the CCL3 and CCL4 Chemokines, and CD49d/Vascular Cell Adhesion Molecule-1 Are Interchained by Sequential Events Sustaining Chronic Lymphocytic Leukemia Cell Survival
by Zucchetto, Antonella and Benedetti, Dania and Tripodo, Claudio and Bomben, Riccardo and Dal Bo, Michele and Marconi, Daniela and Bossi, Fleur and Lorenzon, Debora and Degan, Massimo and Rossi, Francesca Maria and Rossi, Davide and Bulian, Pietro and Franco, Vito and Del Poeta, Giovanni and Deaglio, Silvia and Gaidano, Gianluca and Tedesco, Francesco and Malavasi, Fabio and Gattei, Valter
Cancer research (Chicago, Ill.), ISSN 0008-5472, 05/2009, Volume 69, Issue 9, pp. 4001 - 4009
Life Sciences & Biomedicine | Oncology | Science & Technology | Hematologic and hematopoietic diseases | Pharmacology. Drug treatments | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Antineoplastic agents | Tumors | Integrin alpha4 - biosynthesis | Antigens, CD - immunology | Up-Regulation | Chemokine CCL4 - biosynthesis | Chemokine CCL3 - immunology | Antigens, CD - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | Platelet Endothelial Cell Adhesion Molecule-1 - immunology | Chemokine CCL3 - biosynthesis | Bone Marrow Cells - immunology | ADP-ribosyl Cyclase 1 - immunology | Receptors, Chemokine - biosynthesis | Leukemia, Lymphocytic, Chronic, B-Cell - blood | Membrane Glycoproteins - immunology | Leukemia, Lymphocytic, Chronic, B-Cell - immunology | Macrophages - immunology | Cell Line | Vascular Cell Adhesion Molecule-1 - immunology | Receptors, Chemokine - immunology | Cell Survival - immunology | Integrin alpha4 - immunology | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Endothelial Cells - immunology | Apoptosis - immunology | Endothelial Cells - cytology | ADP-ribosyl Cyclase 1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - biosynthesis | Chemokine CCL4 - immunology | Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis | Index Medicus
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5. Full Text Functional Mineralocorticoid Receptors in Human Vascular Endothelial Cells Regulate Intercellular Adhesion Molecule-1 Expression and Promote Leukocyte Adhesion
by Caprio, Massimiliano and Newfell, Brenna G and la Sala, Andrea and Baur, Wendy and Fabbri, Andrea and Rosano, Giuseppe and Mendelsohn, Michael E and Jaffe, Iris Z
Circulation research, ISSN 0009-7330, 06/2008, Volume 102, Issue 11, pp. 1359 - 1367
Endothelial cell | Spironolactone | Aldosterone | Mineralocorticoid receptor | ICAM1 | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Vertebrates: cardiovascular system | Cell Line | Receptors, Mineralocorticoid - genetics | Transcription, Genetic - drug effects | Endothelial Cells - metabolism | Receptors, Mineralocorticoid - metabolism | Humans | RNA, Small Interfering - pharmacology | Aldosterone - physiology | Aldosterone - pharmacology | Leukocytes - physiology | Cell Adhesion - drug effects | RNA, Messenger - biosynthesis | Receptors, Mineralocorticoid - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Spironolactone - pharmacology | Cell Adhesion - physiology | Coronary Vessels - cytology | Intercellular Adhesion Molecule-1 - genetics | Mineralocorticoid Receptor Antagonists - pharmacology | Aorta - cytology | Endothelial Cells - drug effects | Leukocytes - cytology | Index Medicus
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6. Full Text Protein kinase A-α directly phosphorylates FoxO1 in vascular endothelial cells to regulate expression of vascular cellular adhesion molecule-1 mRNA
by Lee, Ji-Won and Chen, Hui and Pullikotil, Philomena and Quon, Michael J
The Journal of biological chemistry, ISSN 0021-9258, 02/2011, Volume 286, Issue 8, pp. 6423 - 6432
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Phosphorylation - physiology | RNA, Small Interfering - genetics | Humans | Transcriptional Activation - drug effects | Monocytes - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Isoquinolines - pharmacology | Cattle | Forkhead Transcription Factors - metabolism | HEK293 Cells | Vascular Cell Adhesion Molecule-1 - genetics | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Endothelial Cells - metabolism | Cell Adhesion - drug effects | Forkhead Transcription Factors - genetics | Sulfonamides - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Cell Adhesion - physiology | Endothelial Cells - cytology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Forkhead Box Protein O1 | Protein Kinase Inhibitors - pharmacology | Transcriptional Activation - physiology | Mutation | Protein kinase A | Alanine | Phosphorylation | Transcription factors | Antibodies | AKT protein | siRNA | Insulin | Endothelial cells | Promoters | 1-Phosphatidylinositol 3-kinase | Monocytes | Prostaglandins | FOXO1 protein | Aorta | Forkhead protein | Tumor necrosis factor- alpha | Antigen-antibody complexes | Index Medicus | Protein Kinase A (PKA) | VCAM-1 | Signal Transduction | Transcription Factors | Akt PKB | Endothelium | Protein-Protein Interactions | FoxO1
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7. Full Text Hydrogen Sulfide Suppresses High Glucose–Induced Expression of Intercellular Adhesion Molecule-1 in Endothelial Cells
by Guan, Qingbo and Wang, Xiaolei and Gao, Ling and Chen, Jicui and Liu, Yuantao and Yu, Chunxiao and Zhang, Nan and Zhang, Xu and Zhao, Jiajun
Journal of cardiovascular pharmacology, ISSN 0160-2446, 09/2013, Volume 62, Issue 3, pp. 278 - 284
Hydrogen sulfide | Human umbilical vein endothelial cells | Nuclear factor-κB | Intercellular adhesion molecule-1 | High glucose | Cardiac & Cardiovascular Systems | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiovascular system | Biological and medical sciences | Medical sciences | Pharmacology. Drug treatments | Cell Nucleus - immunology | Endothelium, Vascular - cytology | Hydrogen Sulfide - metabolism | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Transcription Factor RelA - antagonists & inhibitors | Humans | Human Umbilical Vein Endothelial Cells - immunology | Endothelium, Vascular - drug effects | NF-kappa B - metabolism | Protein Transport - drug effects | RNA, Messenger - metabolism | Reactive Oxygen Species - agonists | Cell Nucleus - metabolism | Cell Nucleus - pathology | Human Umbilical Vein Endothelial Cells - cytology | Intercellular Adhesion Molecule-1 - biosynthesis | Hyperglycemia - pathology | Electrophoretic Mobility Shift Assay | Endothelium, Vascular - immunology | NF-kappa B - agonists | Diabetic Angiopathies - metabolism | NF-kappa B - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - drug effects | Cells, Cultured | Hyperglycemia - immunology | Antioxidants - pharmacology | Diabetic Angiopathies - blood | Down-Regulation - drug effects | Hyperglycemia - metabolism | Up-Regulation - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Transcription Factor RelA - metabolism | Endothelium, Vascular - metabolism | Transcription Factor RelA - agonists | Diabetic Angiopathies - immunology | Intercellular Adhesion Molecule-1 - genetics | Kinetics | Cell Nucleus - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
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8. Full Text Motorcycle exhaust particles up-regulate expression of vascular adhesion molecule-1 and intercellular adhesion molecule-1 in human umbilical vein endothelial cells
by Lee, Chen-Chen and Huang, Shih-Hsuan and Yang, Ya-Ting and Cheng, Yu-Wen and Li, Ching-Hao and Kang, Jaw-Jou
Toxicology in vitro, ISSN 0887-2333, 06/2012, Volume 26, Issue 4, pp. 552 - 560
Motorcycle exhaust particle | Oxidative stress | NF-κB | Polycyclic aromatic hydrocarbons | Cell adhesion molecule | Life Sciences & Biomedicine | Toxicology | Science & Technology | Up-Regulation | Vehicle Emissions - toxicity | Human Umbilical Vein Endothelial Cells - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Oxidative Stress | Polycyclic Aromatic Hydrocarbons - toxicity | Humans | Soot - toxicity | NF-kappa B - metabolism | Cell Adhesion - drug effects | RNA, Messenger - biosynthesis | Intercellular Adhesion Molecule-1 - biosynthesis | Intercellular Adhesion Molecule-1 - genetics | Motorcycles | Cell Line, Tumor | Vascular Cell Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Air Pollutants - toxicity | Endothelium | Index Medicus
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9. Full Text Acute hypoxia induces HIF-independent monocyte adhesion to endothelial cells through increased intercellular adhesion molecule-1 expression: The role of hypoxic inhibition of prolyl hydroxylase activity for the induction of NF-κB
by Winning, Sandra and Splettstoesser, Frank and Fandrey, Joachim and Frede, Stilla
The Journal of immunology (1950), ISSN 0022-1767, 08/2010, Volume 185, Issue 3, pp. 1786 - 1793
Life Sciences & Biomedicine | Immunology | Science & Technology | Endothelium, Vascular - cytology | Cell Hypoxia - immunology | Monocytes - cytology | Coculture Techniques | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Monocytes - immunology | Amino Acids, Dicarboxylic - pharmacology | Endothelium, Vascular - enzymology | Enzyme Activation - immunology | Procollagen-Proline Dioxygenase - metabolism | RNA, Messenger - biosynthesis | Intercellular Adhesion Molecule-1 - biosynthesis | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Endothelium, Vascular - immunology | Cell Line | Intercellular Adhesion Molecule-1 - physiology | Acute Disease | Basic Helix-Loop-Helix Transcription Factors - physiology | Rats | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Enzyme Activation - drug effects | Cell Adhesion - immunology | Monocytes - enzymology | Animals | Up-Regulation - immunology | NF-kappa B - biosynthesis | Intercellular Adhesion Molecule-1 - genetics | Cell Line, Tumor | Index Medicus | Abridged Index Medicus
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10. Full Text Intercellular adhesion molecule-1 (ICAM-1) expression and cell signaling cascades
by Hubbard, Andrea K and Rothlein, Robert
Free radical biology & medicine, ISSN 0891-5849, 2000, Volume 28, Issue 9, pp. 1379 - 1386
Cell signaling pathways | ICAM-1 | Free radical | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | CD18 Antigens - physiology | MAP Kinase Signaling System - physiology | Multigene Family | Phosphorylation | Reactive Oxygen Species | Oxidative Stress | Humans | Monocytes - metabolism | Intercellular Adhesion Molecule-1 - biosynthesis | Lymphocytes - metabolism | Intercellular Adhesion Molecule-1 - physiology | Gene Expression Regulation - physiology | Cell Adhesion | Gene Expression Regulation - drug effects | Monocytes - drug effects | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | Lymphocytes - drug effects | Transcription Factor AP-1 - physiology | Signal Transduction - physiology | Protein Processing, Post-Translational | Antigen Presentation - physiology | Cytokines - biosynthesis | Cytokines - pharmacology | Index Medicus
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11. Full Text Decoy receptor 3 increases monocyte adhesion to endothelial cells via NF-κB-dependent up-regulation of intercellular adhesion molecule-1, VCAM-1, and IL-8 expression
by Yang, Chia-Ron and Hsieh, Shie-Liang and Ho, Feng-Ming and Lin, Wan-Wan
The Journal of immunology (1950), ISSN 0022-1767, 02/2005, Volume 174, Issue 3, pp. 1647 - 1656
Life Sciences & Biomedicine | Immunology | Science & Technology | Endothelium, Vascular - cytology | Up-Regulation | Humans | Molecular Sequence Data | Interleukin-8 - physiology | Monocytes - metabolism | Monocytes - immunology | Aorta - metabolism | Membrane Glycoproteins - physiology | U937 Cells | Intercellular Adhesion Molecule-1 - biosynthesis | Adult | Receptors, Cell Surface - physiology | Umbilical Veins - cytology | Endothelium, Vascular - immunology | Amino Acid Sequence | Intercellular Adhesion Molecule-1 - physiology | Interleukin-8 - biosynthesis | Cells, Cultured | Aorta - immunology | Vascular Cell Adhesion Molecule-1 - physiology | Cell Adhesion - immunology | Receptors, Tumor Necrosis Factor | Endothelium, Vascular - metabolism | Receptors, Tumor Necrosis Factor, Member 6b | NF-kappa B - physiology | Cell Line, Tumor | Vascular Cell Adhesion Molecule-1 - biosynthesis | Aorta - cytology | Index Medicus | Abridged Index Medicus
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