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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles in the acute hemostatic cascade... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Metabolic syndrome and related disorders, ISSN 1540-4196, 2003
Journal
Nature (London), ISSN 1476-4687, 2011, Volume 472, Issue 7341, pp. 57 - 63
Metabolomics studies hold promise for the discovery of pathways linked to disease processes... 
CHLAMYDIA-PNEUMONIAE | TRIMETHYLAMINE-N-OXIDE | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | MURINE MACROPHAGES | MOUSE | GENE-EXPRESSION | ATHEROSCLEROSIS | MICE | CORONARY-HEART-DISEASE | MASS-SPECTROMETRY | Choline - metabolism | Dietary Fats - metabolism | Metabolomics | Liver - enzymology | Diet - adverse effects | Atherosclerosis - genetics | Betaine - metabolism | Humans | Phosphatidylcholines - administration & dosage | Phosphatidylcholines - metabolism | Choline - blood | Dietary Fats - pharmacology | Oxygenases - metabolism | Cardiovascular Diseases - blood | Female | Atherosclerosis - microbiology | Betaine - blood | Methylamines - pharmacology | Cardiovascular Diseases - diagnosis | Methylamines - metabolism | Receptors, Scavenger - metabolism | Biomarkers - metabolism | Phosphatidylcholines - blood | Cardiovascular Diseases - metabolism | Risk Assessment | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Phosphatidylcholines - pharmacology | Gene Expression Regulation | Methylamines - blood | Gastrointestinal Tract - microbiology | Biomarkers - blood | Dietary Fats - blood | Atherosclerosis - metabolism | Gastrointestinal Tract - metabolism | Cardiovascular Diseases - microbiology | Macrophages - metabolism | Phenotype | Animals | Choline - administration & dosage | Choline - pharmacology | Germ-Free Life | Cholesterol, HDL - blood | Mice | Oxygenases - genetics | Lecithin | Microbiota (Symbiotic organisms) | Physiological aspects | Research | Cardiovascular diseases | Metabolism | Health aspects | Risk factors | Plasma | Nuclear magnetic resonance--NMR | Heart attacks | Pathogenesis | Lipids | Cardiovascular disease | Probiotics | Metabolites | Diet | Atherosclerosis | Bacteria | Insulin resistance | Mass spectrometry | atherosclerosis | intestinal microbiota | choline | diet | phospholipid | metabolomics
Journal Article
Clinical and experimental pharmacology & physiology, ISSN 1440-1681, 09/2007, Volume 34, Issue 9, pp. 906 - 911
... (NO) synthesis and the development of vascular disease. • Arginase inhibits the production of NO via several potential mechanisms, including competition with NO synthase (NOS... 
smooth muscle cell proliferation | arginine | nitric oxide synthase | diabetes | endothelial dysfunction | hypertension | arginase | Hypertension | Arginine | Endothelial dysfunction | Smooth muscle cell proliferation | Arginase | Nitric oxide synthase | Diabetes | PHYSIOLOGY | MEDIATED DILATION | I EXPRESSION | TRANSLATIONAL CONTROL | AMINO-ACID TRANSPORTER | INTIMAL HYPERPLASIA | SYNTHASE ACTIVITY | MOUSE MODEL | L-ARGININE TRANSPORT | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | Cardiovascular Diseases - physiopathology | Cell Proliferation | Reactive Oxygen Species - metabolism | Cardiovascular Diseases - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Arginase - metabolism | Endothelium, Vascular - physiopathology | Homeostasis | Cardiovascular Diseases - enzymology | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Arginine - analogs & derivatives | Collagen - metabolism | Animals | Endothelium, Vascular - metabolism | Urea - metabolism | Nitric Oxide Synthase Type III - metabolism | Nitric Oxide - metabolism | Muscle, Smooth, Vascular - enzymology | Arginine - metabolism | Muscle, Smooth, Vascular, metabolism | Collagen, metabolism | Endothelium, Vascular, physiopathology | Arginine, analogs and derivatives | Nitric Oxide, metabolism | Endothelium, Vascular, metabolism | Urea, metabolism | Nitric Oxide Synthase Type III, metabolism | Arginine, metabolism | Cardiovascular Diseases, enzymology | Endothelium, Vascular, enzymology | Muscle, Smooth, Vascular, physiopathology | Cardiovascular Diseases, physiopathology | Cardiovascular Diseases, metabolism | Arginase, metabolism | Muscle, Smooth, Vascular, enzymology | Reactive Oxygen Species, metabolism | Deicing chemicals | Physiological aspects | Endothelium-derived relaxing factors | Nitric oxide
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
The journal of clinical endocrinology and metabolism, ISSN 0021-972X, 08/2017, Volume 102, Issue 8, pp. 2905 - 2913
Journal Article