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Angiogenesis, ISSN 0969-6970, 6/2012, Volume 15, Issue 2, pp. 171 - 185
Pharmacological inhibition of VEGF-A has proven to be effective in inhibiting angiogenesis and vascular leak associated with cancers and various eye diseases.... 
Biomedicine | Age-related macular degeneration | Cancer Research | Oncology | Affinity | Ophthalmology | VEGF receptor | Cardiology | Aflibercept | Placental growth factor | Biomedicine general | Cell Biology | ANGIOGENESIS | EFFICACY | MACULAR DEGENERATION | PHASE-II | ANTIBODY | FACTOR THERAPY | EYE | IN-VITRO | PEGAPTANIB | RETINAL DISEASES | PERIPHERAL VASCULAR DISEASE | Angiogenesis Inhibitors - immunology | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor B - antagonists & inhibitors | Recombinant Fusion Proteins - pharmacology | Endothelial Cells - metabolism | Humans | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor B - immunology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor A - immunology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - immunology | Enzyme Activation - drug effects | Bevacizumab | Endothelial Cells - immunology | Vascular Endothelial Growth Factor B - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | HEK293 Cells | Ranibizumab | Vascular Endothelial Growth Factor Receptor-1 - immunology | Antibodies, Monoclonal, Humanized - immunology | Original Paper
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 01/2010, Volume 207, Issue 1, pp. 101 - 115
Inflammatory lymphangiogenesis plays a crucial role in the development of inflammation and transplant rejection. The mechanisms of inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | HERPES-SIMPLEX-VIRUS | INFLAMMATORY LYMPHANGIOGENESIS | STROMAL KERATITIS | ADENOSINE A(2A) RECEPTORS | FACTOR-C | IMMUNE PRIVILEGE | TRANSCRIPTION FACTOR SP1 | IMMUNOLOGY | NF-KAPPA-B | TYPE-1 INFECTION | ENDOTHELIAL GROWTH-FACTOR | Corneal Neovascularization - immunology | Inflammation - pathology | Vascular Endothelial Growth Factor A - biosynthesis | Antigens, Viral - metabolism | Cornea - immunology | Corneal Neovascularization - genetics | Vascular Endothelial Growth Factor Receptor-2 - immunology | Vascular Endothelial Growth Factor A - genetics | Herpes Simplex - genetics | Cell Movement - genetics | Vascular Endothelial Growth Factor Receptor-3 - immunology | Vascular Endothelial Growth Factor Receptor-2 - genetics | Cell Movement - immunology | Signal Transduction - immunology | Inflammation - metabolism | Lymphatic Vessels - metabolism | Herpesvirus 1, Human - immunology | Cornea - pathology | Corneal Neovascularization - virology | Macrophages - immunology | Cornea - virology | Herpesvirus 1, Human - metabolism | Macrophages - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Inflammation - virology | Lymphatic Vessels - immunology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mice, Transgenic | Vascular Endothelial Growth Factor A - immunology | Inflammation - immunology | Signal Transduction - genetics | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Herpes Simplex - immunology | Herpes Simplex - metabolism | Antigens, Viral - immunology | Cornea - metabolism | Macrophages - metabolism | Animals | Corneal Neovascularization - metabolism | Herpes Simplex - pathology | Lymphatic Vessels - pathology | Inflammation - genetics | Mice
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 08/2010, Volume 70, Issue 15, pp. 6171 - 6180
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 5, pp. 1339 - 1350.e1
Background & Aims Vascular endothelial growth factor (VEGF)−induced angiogenesis is implicated in fibrogenesis and portal hypertension. However, the function... 
Gastroenterology and Hepatology | Extracellular Matrix | Sinusoidal Endothelial Cell | Liver Damage | Hepatic Sinusoid | Keywords | FACTOR VEGF | ANGIOGENESIS | MACROPHAGES | LIVER FIBROSIS | MECHANISMS | PATHOGENESIS | MATRIX METALLOPROTEINASES | HEPATIC STELLATE CELLS | INFLAMMATION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Antibodies, Neutralizing - administration & dosage | Liver - pathology | Capillary Permeability | Human Umbilical Vein Endothelial Cells - metabolism | Carbon Tetrachloride | Coculture Techniques | Humans | Monocytes - metabolism | Monocytes - immunology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | fas Receptor - metabolism | Liver Cirrhosis, Experimental - prevention & control | Liver - immunology | Ligation | Cholecystostomy | Adenoviridae - genetics | Liver Cirrhosis, Experimental - pathology | Macrophages - immunology | Tacrolimus Binding Protein 1A - genetics | Gene Transfer Techniques | Promoter Regions, Genetic | Liver Regeneration | Liver Cirrhosis, Experimental - immunology | Jejunostomy | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Receptor, Macrophage Colony-Stimulating Factor - genetics | Mice, Transgenic | Vascular Endothelial Growth Factor A - immunology | Bile Ducts - surgery | Liver Cirrhosis, Experimental - genetics | Remission Induction | Matrix Metalloproteinase 13 - metabolism | Injections | Macrophages - metabolism | Animals | Chemokine CXCL9 - metabolism | Liver Cirrhosis, Experimental - chemically induced | Tacrolimus Binding Protein 1A - metabolism | Mice | Genetic Vectors | Liver Cirrhosis, Experimental - metabolism | Apoptosis | Hypertension | Medical colleges | Vascular endothelial growth factor | Health aspects | Fibrosis
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2011, Volume 187, Issue 4, pp. 1970 - 1976
Macrophage secretion of vascular endothelial growth factor (VEGF) in response to the hypoxic tumor microenvironment contributes to tumor growth, angiogenesis,... 
CELLS | MELANOMA | HIF-2-ALPHA | VEGF | HIF-1-ALPHA | IMMUNOLOGY | EXPRESSION | CANCER | PROGRESSION | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Lung Neoplasms - metabolism | Neoplasm Proteins - immunology | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | Neoplasm Proteins - metabolism | Vascular Endothelial Growth Factor A - genetics | Oxygen - metabolism | Neovascularization, Pathologic - pathology | Neoplasm Metastasis | Basic Helix-Loop-Helix Transcription Factors - metabolism | Lung Neoplasms - secondary | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Melanoma - genetics | Vascular Endothelial Growth Factor Receptor-1 - immunology | Neovascularization, Pathologic - immunology | Neoplasm Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Macrophages - immunology | Vascular Endothelial Growth Factor Receptor-1 - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - immunology | Lung Neoplasms - genetics | Melanoma - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Macrophages - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation, Neoplastic - immunology | Vascular Endothelial Growth Factor A - immunology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Oxygen - immunology | Melanoma - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Mice, Knockout | Lung Neoplasms - immunology | Macrophages - metabolism | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Melanoma - immunology | Oxygen Consumption - genetics | Cell Line, Tumor | Basic Helix-Loop-Helix Transcription Factors - immunology | Neovascularization, Pathologic - genetics | Oxygen Consumption - immunology | Mice | Neovascularization, Pathologic - metabolism
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/2012, Volume 209, Issue 11, pp. 2001 - 2016
Infiltration of Foxp3(+) regulatory T (T reg) cells is considered to be a critical step during tumor development and progression. T reg cells supposedly... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | ANTI-VEGF | DENDRITIC CELLS | ANGIOGENESIS | AUTOIMMUNITY | RECEPTOR | SELECTIVE DEPLETION | MALIGNANT-MELANOMA | IMMUNOLOGY | EXPRESSION | TUMOR-IMMUNITY | Forkhead Transcription Factors - immunology | Immunohistochemistry | Neuropilin-1 - genetics | T-Lymphocytes, Regulatory - metabolism | Vascular Endothelial Growth Factor A - deficiency | Tumor Escape - genetics | Humans | Gene Expression Regulation, Neoplastic | Melanoma, Experimental - metabolism | Vascular Endothelial Growth Factor A - genetics | Cell Movement - genetics | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Melanoma, Experimental - immunology | Forkhead Transcription Factors - metabolism | CD8-Positive T-Lymphocytes - metabolism | Tumor Burden - genetics | Neuropilin-1 - immunology | Tumor Escape - immunology | Skin Neoplasms - immunology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Mice, Transgenic | Vascular Endothelial Growth Factor A - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Skin Neoplasms - metabolism | Animals | Melanoma, Experimental - genetics | Skin Neoplasms - genetics | Cell Line, Tumor | Mice | CD8-Positive T-Lymphocytes - immunology | Neuropilin-1 - deficiency | Tumor Burden - immunology
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 03/2008, Volume 358, Issue 11, pp. 1129 - 1136
Journal Article