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1. Full Text Human Induced Pluripotent Stem‐Derived Retinal Pigment Epithelium (RPE) Cells Exhibit Ion Transport, Membrane Potential, Polarized Vascular Endothelial Growth Factor Secretion, and Gene Expression Pattern Similar to Native RPE
by Kokkinaki, Maria and Sahibzada, Niaz and Golestaneh, Nady
STEM CELLS, ISSN 1066-5099, 05/2011, Volume 29, Issue 5, pp. 825 - 835
Age‐related macular degeneration (AMD) is one of the major causes of blindness in aging population that progresses with death of retinal pigment epithelium... 
Telomere shortening | Human induced pluripotent stem cells | Ion transport | Membrane potential | Growth arrest | Gene expression | Retinal pigment epithelium | TRANSLOCATION | MOUSE | MACULAR DEGENERATION | GRAFT | INDUCTION | FIBROBLASTS | TRANSPLANTATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | MOLECULAR SIGNATURE | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENERATION | HEMATOLOGY | Retinal Pigment Epithelium - metabolism | Membrane Potentials - genetics | Humans | Phagocytosis - physiology | Electrophysiology | Immunoblotting | Vascular Endothelial Growth Factor A - metabolism | Phagocytosis - genetics | Membrane Potentials - physiology | Vascular Endothelial Growth Factor A - secretion | Polymerase Chain Reaction | DNA Damage - genetics | Fibroblasts - cytology | Induced Pluripotent Stem Cells - cytology | Ion Transport - physiology | Retinal Pigment Epithelium - cytology | Fibroblasts - metabolism | Ion Transport - genetics | Macular degeneration | Retina | Vascular endothelial growth factor | Stem cells
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2. Full Text Priming Dental Pulp Stem Cells With Fibroblast Growth Factor‐2 Increases Angiogenesis of Implanted Tissue‐Engineered Constructs Through Hepatocyte Growth Factor and Vascular Endothelial Growth Factor Secretion
by Gorin, Caroline and Rochefort, Gael Y and Bascetin, Rumeyza and Ying, Hanru and Lesieur, Julie and Sadoine, Jérémy and Beckouche, Nathan and Berndt, Sarah and Novais, Anita and Lesage, Matthieu and Hosten, Benoit and Vercellino, Laetitia and Merlet, Pascal and Le-Denmat, Dominique and Marchiol, Carmen and Letourneur, Didier and Nicoletti, Antonino and Vital, Sibylle Opsahl and Poliard, Anne and Salmon, Benjamin and Muller, Laurent and Chaussain, Catherine and Germain, Stéphane
STEM CELLS Translational Medicine, ISSN 2157-6564, 03/2016, Volume 5, Issue 3, pp. 392 - 404
Tissue engineering strategies based on implanting cellularized biomaterials are promising therapeutic approaches for the reconstruction of large tissue... 
Angiogenesis | Pulp engineering | Dynamic vascular imaging | Hepatocyte growth factor | Hypoxia | Vascular endothelial growth factor | Mesenchymal stem cells | COLLAGEN | VIVO | EXFOLIATED DECIDUOUS TEETH | SIDE POPULATION CELLS | LIMB ISCHEMIA | VEGF | DIFFERENTIATE | CELL & TISSUE ENGINEERING | IN-VITRO | STROMAL CELLS | Hepatocyte Growth Factor - biosynthesis | Mesenchymal Stromal Cells - drug effects | Vascular Endothelial Growth Factor A - biosynthesis | Cell Hypoxia - drug effects | Hepatocyte Growth Factor - secretion | Neovascularization, Physiologic - genetics | Humans | Fibroblast Growth Factor 2 - biosynthesis | Dental Pulp - cytology | Mesenchymal Stromal Cells - metabolism | Fibroblast Growth Factor 2 - administration & dosage | Vascular Endothelial Growth Factor A - secretion | Mesenchymal Stem Cell Transplantation | Tissue Engineering | Cell culture | Fibroblast growth factor | Hydrogels | Mesenchyme | Biomaterials | Vascularization | Penicillin | Tomography | Fibroblasts | Extracellular matrix | Nutrients | Reconstruction | Data analysis | Cytokines | Tissue engineering | Secretion | Teeth | Grants | Progenitor cells | Endothelial cells | Perfusion | Stem cells | Dental pulp | Life Sciences | Bioengineering | Tissue Engineering and Regenerative Medicine
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3. Full Text Akt3 controls vascular endothelial growth factor secretion and angiogenesis in ovarian cancer cells
by Liby, Tiera A and Spyropoulos, Perry and Buff Lindner, Haley and Eldridge, Juanita and Beeson, Craig and Hsu, Tien and Muise‐Helmericks, Robin C
International Journal of Cancer, ISSN 0020-7136, 02/2012, Volume 130, Issue 3, pp. 532 - 543
The PI3 kinase/Akt pathway is commonly deregulated in human cancers, functioning in such processes as proliferation, glucose metabolism, survival and motility.... 
B-GAMMA | ONCOLOGY | EBAG9/RCAS1 | NITRIC-OXIDE | INDUCTION | ANTIGEN RCAS1 | HYPOXIA | PHOSPHOINOSITIDE 3-KINASE | EXPRESSION | P42/P44 MAP KINASE | SIGNALING ANGIOGENESIS | Humans | Gene Expression Regulation, Neoplastic | Endoplasmic Reticulum - metabolism | Vascular Endothelial Growth Factor A - metabolism | Proto-Oncogene Proteins c-akt - genetics | Vascular Endothelial Growth Factor A - secretion | Ovarian Neoplasms - genetics | Mitochondria - genetics | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Female | Tumor Burden - genetics | Ovarian Neoplasms - blood supply | Proto-Oncogene Proteins c-akt - metabolism | Signal Transduction | Gene Silencing | PTEN Phosphohydrolase - metabolism | Mitochondria - metabolism | Mice, SCID | Ovarian Neoplasms - enzymology | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Kinases | Gene expression | Vascular endothelial growth factor | Ovarian cancer
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4. Full Text The stimulation of osteogenic differentiation of mesenchymal stem cells and vascular endothelial growth factor secretion of endothelial cells by β‐CaSiO3/β‐Ca3(PO4)2 scaffolds
by Wang, Chen and Lin, Kaili and Chang, Jiang and Sun, Jiao
Journal of Biomedical Materials Research Part A, ISSN 1549-3296, 07/2014, Volume 102, Issue 7, pp. 2096 - 2104
Porous β‐CaSiO3/β‐Ca3(PO4)2 (β‐CS/β‐TCP) composite scaffolds have been previously shown to promote bone formation in vivo. However, the mechanisms underlying... 
bioactive ions | AMPK | calcium silicate | tricalcium phosphate | osteogenic differentiation | Endothelium, Vascular - cytology | Human Umbilical Vein Endothelial Cells | Calcium Compounds | Humans | Rats | Silicates | DNA Primers | Rats, Sprague-Dawley | Vascular Endothelial Growth Factor A - secretion | Tissue Scaffolds | Calcium Phosphates | Animals | Endothelium, Vascular - secretion | Adenylate Kinase - metabolism | Base Sequence | Cell Differentiation | Real-Time Polymerase Chain Reaction | Bone and Bones - cytology
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5. Full Text Exogenous miRNA-146a enhances the therapeutic efficacy of human mesenchymal stem cells by increasing vascular endothelial growth factor secretion in the ischemia/reperfusion-injured heart
by Seo, Hyang-Hee and Lee, Se-Yeon and Lee, Chang Youn and Kim, Ran and Kim, Pilseog and Oh, Sekyung and Lee, Hojin and Lee, Min Young and Kim, Jongmin and Kim, Lark Kyun and Hwang, Ki-Chul and Chang, Woochul
Journal of Vascular Research, ISSN 1018-1172, 05/2017, Volume 54, Issue 2, pp. 100 - 108
Adult stem cells have been studied as a promising therapeutic modality for the functional restoration of the damaged heart. In the present study, a strategy... 
Neurofibromin 2 | Vascular endothelial growth factor secretion | Angiogenesis | Micro-RNA-146a | PHYSIOLOGY | PROTEIN | VEGF | REGENERATION | TUMOR-SUPPRESSOR GENE | MICRORNAS | CANCER | SIGNALING PATHWAY | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | NEUROFIBROMATOSIS TYPE-2 | Myocardial Infarction - genetics | Up-Regulation | Neurofibromin 2 - genetics | Humans | rac GTP-Binding Proteins - metabolism | Male | MicroRNAs - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Recovery of Function | Myocardial Reperfusion Injury - surgery | Vascular Endothelial Growth Factor A - secretion | Myocardial Reperfusion Injury - pathology | Transfection | Myocardial Infarction - pathology | Myocardium - metabolism | 3' Untranslated Regions | Binding Sites | Myocardial Reperfusion Injury - genetics | Neurofibromin 2 - metabolism | Disease Models, Animal | Myocardial Infarction - surgery | Signal Transduction | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Myocardium - pathology | Mesenchymal Stromal Cells - secretion | Myocardial Infarction - metabolism | p21-Activated Kinases - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - metabolism | Regeneration | Animals | Fibrosis | MicroRNAs - genetics | Mesenchymal Stem Cell Transplantation | Neovascularization, Physiologic
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6. Xenoestrogens modulate vascular endothelial growth factor secretion in breast cancer cells through an estrogen receptor-dependent mechanism
by Buteau-Lozano, Hélène and Velasco, Guillaume and Cristofari, Monique and Balaguer, Patrick and Perrot-Applanat, Martine
Journal of Endocrinology, ISSN 0022-0795, 02/2008, Volume 196, Issue 2, pp. 399 - 412
Environmental chemicals may affect human health by disrupting endocrine function. Their possible role in the mammary gland and breast tumors is still unknown.... 
MENSTRUAL-CYCLE | FACTOR VEGF | ENDOCRINE DISRUPTORS | PERMEABILITY FACTOR EXPRESSION | IN-UTERO EXPOSURE | ENDOCRINOLOGY & METABOLISM | BISPHENOL-A ALTERS | GENE-EXPRESSION | MAMMARY-CANCER | ENVIRONMENTAL ESTROGENS | ORGANOCHLORINE PESTICIDES | Phosphotransferases - metabolism | Cell Survival - drug effects | Up-Regulation | Estrogens - pharmacology | Humans | Breast Neoplasms - physiopathology | Xenobiotics - pharmacology | Breast Neoplasms - metabolism | Vascular Endothelial Growth Factor A - secretion | Breast Neoplasms - pathology | Genistein - pharmacology | Estrogen Receptor alpha - metabolism | Female | Phytoestrogens - pharmacology
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7. Full Text Nicotine promotes vascular endothelial growth factor secretion by human trophoblast cells under hypoxic conditions and improves the proliferation and tube formation capacity of human umbilical endothelial cells
by Zhao, Hongbo and Wu, Lanxiang and Wang, Yahui and Zhou, Jiayi and Li, Ruixia and Zhou, Jiabing and Wang, Zehua and Xu, Congjian
Reproductive BioMedicine Online, ISSN 1472-6483, 2017, Volume 34, Issue 4, pp. 406 - 413
Abstract Pre-eclampsia, characterized as defective uteroplacental vascularization, remains the major cause of maternal and fetal mortality and morbidity.... 
Obstetrics and Gynecology | pre-eclampsia | trophoblast cells | VEGF | Nicotine | RISK | PREECLAMPSIA | CANCER | PREGNANCY | OBSTETRICS & GYNECOLOGY | CIGARETTE-SMOKING | REPRODUCTIVE BIOLOGY | LETHALITY | GENE | ENDOMETRIAL | EXPRESSION | Human Umbilical Vein Endothelial Cells | Vascular Endothelial Growth Factor A - biosynthesis | Vascular Endothelial Growth Factor Receptor-1 - secretion | Cell Proliferation | Trophoblasts - metabolism | Humans | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Nicotine - pharmacology | Vascular Endothelial Growth Factor A - secretion | Cell Hypoxia | Trophoblasts - drug effects | Female | Trophoblasts - cytology
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8. Full Text Vascular Endothelial Growth Factor Secretion by Nonmyocytes Modulates Connexin-43 Levels in Cardiac Organoids
by Iyer, Rohin K and Odedra, Devang and Chiu, Loraine L.Y and Vunjak-Novakovic, Gordana and Radisic, Milica
Tissue Engineering Part A, ISSN 1937-3341, 09/2012, Volume 18, Issue 17-18, pp. 1771 - 1783
We previously showed that the sequential, but not simultaneous, culture of endothelial cells (ECs), fibroblasts (FBs), and cardiomyocytes (CMs) resulted in... 
Original Articles | CELLS | ACTIVATED PROTEIN-KINASE | STIMULATION | MYOCYTES | ANGIOGENESIS | VEGF | MUSCLE | CYCLICAL MECHANICAL STRETCH | FIBROBLASTS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENE-EXPRESSION | Cell Count | Myocardial Contraction - drug effects | Protein Transport - drug effects | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - secretion | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Myocardium - metabolism | Organoids - metabolism | Cell Death - drug effects | Vascular Endothelial Growth Factor A - pharmacology | Connexin 43 - genetics | Fibroblasts - metabolism | Animals, Newborn | Connexin 43 - metabolism | Myocytes, Cardiac - cytology | Endothelial Cells - metabolism | RNA, Messenger - genetics | Cells, Cultured | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Antibodies, Neutralizing - pharmacology | Up-Regulation - genetics | Down-Regulation - drug effects | Down-Regulation - genetics | Up-Regulation - drug effects | Animals | Myocytes, Cardiac - drug effects | Endothelial Cells - cytology | Fibroblasts - drug effects | Organoids - drug effects | Myocytes, Cardiac - metabolism | Ligands | Fibroblasts - cytology | Electrophysiological Phenomena - drug effects | Endothelial Cells - drug effects | Heart | Cell culture | Cardiology | Tissue engineering | Vascular endothelial growth factor | Indexing in process | Original
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9. Full Text Secretion of Vascular Endothelial Growth Factor by Primary Human Fibroblasts at Senescence
by Jean-Philippe Coppà and Katalin Kauser and Judith Campisi and Christian M. Beauséjour
Journal of Biological Chemistry, ISSN 0021-9258, 10/2006, Volume 281, Issue 40, pp. 29568 - 29574
Cellular senescence prevents the proliferation of cells at risk for neoplastic transformation. Nonetheless, the senescence response is thought to be... 
ANGIOGENESIS | HUMAN-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | TUMOR-SUPPRESSOR | STROMAL FIBROBLASTS | REPLICATIVE LIFE-SPAN | CELLULAR SENESCENCE | CANCER | EXPRESSION | P53 | Vascular Endothelial Growth Factor A - biosynthesis | Fibroblasts - secretion | Humans | Cells, Cultured | Cellular Senescence - physiology | Cell Transformation, Neoplastic - metabolism | Neovascularization, Pathologic - etiology | Vascular Endothelial Growth Factor A - secretion | Culture Media, Conditioned | Animals | Mice, Nude | Neoplasm Invasiveness - pathology | Mammary Neoplasms, Experimental - pathology | Cell Line, Tumor | Adult | Mammary Neoplasms, Experimental - blood supply | Mice | Neovascularization, Pathologic - metabolism | Cell Transformation, Neoplastic - pathology | Mammary Neoplasms, Experimental - etiology
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10. Full Text Cabergoline reduces cell viability in non functioning pituitary adenomas by inhibiting vascular endothelial growth factor secretion
by Gagliano, Teresa and Gagliano, Teresa and Filieri, Carlo and Filieri, Carlo and Minoia, Mariella and Minoia, Mariella and Buratto, Mattia and Buratto, Mattia and Tagliati, Federico and Tagliati, Federico and Ambrosio, Maria Rosaria and Ambrosio, Maria Rosaria and Lapparelli, Marcello and Lapparelli, Marcello and Zoli, Matteo and Zoli, Matteo and Frank, Giorgio and Frank, Giorgio and degli Uberti, Ettore and degli Uberti, Ettore and Zatelli, Maria Chiara and Zatelli, Maria Chiara
Pituitary, ISSN 1386-341X, 3/2013, Volume 16, Issue 1, pp. 91 - 100
Dopamine (DA) therapy of non-functioning pituitary adenomas (NFA) can result in tumor stabilization and shrinkage. However, the mechanism of action is still... 
Diabetes | Medicine & Public Health | Human Physiology | Non functioning pituitary adenomas | Dopamine receptor type 2 | Vascular endothelial growth factor | FACTOR-A | BROMOCRIPTINE | TUMOR CELLS | MACROADENOMAS | AGONISTS | RAT PITUITARY | THERAPY | ENDOCRINOLOGY & METABOLISM | ESTROGEN | DOPAMINE-RECEPTORS | EXPRESSION | Cell Survival - drug effects | Humans | Middle Aged | Male | Vascular Endothelial Growth Factor A - secretion | Pituitary Neoplasms - secretion | Female | Aged | Tumor Cells, Cultured | Receptors, Dopamine - genetics | Vascular Endothelial Growth Factor A - pharmacology | Ergolines - pharmacology | Microscopy, Fluorescence | Cell proliferation | Cell culture | Dopamine | Secretion | Brain tumors | Data processing | Adenoma | Atrophy | Pituitary | Dopamine receptors | Tumors | sulpiride
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11. Full Text Dopamine receptor 2 activation inhibits ovarian vascular endothelial growth factor secretion in an ovarian hyperstimulation syndrome (OHSS) animal model: implications for treatment of OHSS with dopamine receptor 2 agonists
by Ferrero, Hortensia, Ph.D and García-Pascual, Carmen M., Ph.D and Gaytán, María, Ph.D and Morales, Concepción, Ph.D and Simón, Carlos, M.D and Gaytán, Francisco, M.D and Pellicer, Antonio, M.D and Gómez, Raúl, Ph.D
Fertility and Sterility, ISSN 0015-0282, 2014, Volume 102, Issue 5, pp. 1468 - 1476.e1
Objective To explore whether a dopamine receptor 2 agonist (D2-ag) can prevent ovarian hyperstimulation syndrome (OHSS) in a rat model by decreasing ovarian... 
Internal Medicine | Obstetrics and Gynecology | OHSS | animal model | VEGF | dopamine receptor 2 agonists | Dopamine receptor 2 agonists | Animal model | FACTOR VEGF | CELLS | PHOSPHORYLATION | HYPERPROLACTINEMIA | RATS | HUMAN GRANULOSA | OBSTETRICS & GYNECOLOGY | ASSISTED REPRODUCTION | REPRODUCTIVE BIOLOGY | CAPILLARY-PERMEABILITY | CABERGOLINE | EXPRESSION | Rats, Wistar | Humans | Rats | Treatment Outcome | Ovarian Hyperstimulation Syndrome - metabolism | Receptors, Dopamine D2 - metabolism | Dopamine Antagonists - administration & dosage | Vascular Endothelial Growth Factor A - secretion | Animals | Female | Ovary - drug effects | Ovarian Hyperstimulation Syndrome - drug therapy | Disease Models, Animal | Ovary - metabolism | Physiological aspects | Phenols | Dopamine | Vascular endothelial growth factor | Analysis | Cells
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12. Cigarette smoke induces nuclear translocation of heme oxygenase 1 (HO-1) in prostate cancer cells: Nuclear HO-1 promotes vascular endothelial growth factor secretion
by Birrane, Gabriel and Li, Huchun and Yang, Suping and Tachado, Souvenir D and Seng, Seyha
International Journal of Oncology, ISSN 1019-6439, 06/2013, Volume 42, Issue 6, pp. 1919 - 1928
Prostate cancer is the second leading cause of male-cancer related death in the United States. Despite a number of evidence-based studies which strongly... 
Prostate cancer | Vascular endothelial growth factor | Nuclear heme oxygenase 1 | Cigarette smoke | APOPTOSIS | nuclear heme oxygenase 1 | ANGIOGENESIS | VEGF | INDUCTION | cigarette smoke | ZINC PROTOPORPHYRIN | prostate cancer | INHIBITION | ONCOLOGY | vascular endothelial growth factor | NITRIC-OXIDE | HYDROGEN-PEROXIDE | TUMOR-GROWTH | EXPRESSION | Smoking - adverse effects | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | Heme Oxygenase-1 - metabolism | Humans | Molecular Sequence Data | Male | Protein Transport - drug effects | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor A - secretion | Heme Oxygenase-1 - genetics | Cell Nucleus - metabolism | Prostatic Neoplasms - genetics | Base Sequence | HEK293 Cells | Cell Line, Tumor | Cell Nucleus - drug effects | Prostatic Neoplasms - chemically induced
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13. Full Text Use of Culture Geometry to Control Hypoxia-Induced Vascular Endothelial Growth Factor Secretion from Adipose-Derived Stem Cells: Optimizing a Cell-Based Approach to Drive Vascular Growth
by Skiles, Matthew L and Sahai, Suchit and Rucker, Lindsay and Blanchette, James O
Tissue Engineering Part A, ISSN 1937-3341, 11/2013, Volume 19, Issue 21-22, pp. 233 - 2338
Adipose-derived stem cells (ADSCs) possess potent angiogenic properties and represent a source for cell-based approaches to delivery of bioactive factors to... 
Original Articles | ACTIVATION | SPHEROIDS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MECHANISMS | FIBROBLASTS | PROGRESSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Cell Hypoxia - physiology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Humans | Cells, Cultured | Adipocytes - cytology | Stem Cells - cytology | Cell Hypoxia - genetics | Stem Cells - metabolism | Cell Survival - physiology | Vascular Endothelial Growth Factor A - secretion | Cell culture | Angiogenesis | Body fat | Tissue engineering | Vascular endothelial growth factor | Stem cells | Original
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