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Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Angiogenesis, ISSN 0969-6970, 6/2012, Volume 15, Issue 2, pp. 171 - 185
Pharmacological inhibition of VEGF-A has proven to be effective in inhibiting angiogenesis and vascular leak associated with cancers and various eye diseases.... 
Biomedicine | Age-related macular degeneration | Cancer Research | Oncology | Affinity | Ophthalmology | VEGF receptor | Cardiology | Aflibercept | Placental growth factor | Biomedicine general | Cell Biology | EYE DISEASE | MACULAR DEGENERATION | PHASE-II | CHOROIDAL NEOVASCULARIZATION | FACTOR THERAPY | IN-VITRO | PROLIFERATIVE DIABETIC-RETINOPATHY | PATHOLOGICAL CONDITIONS | RETINAL DISEASES | PERIPHERAL VASCULAR DISEASE | INTRAVITREAL BEVACIZUMAB | Angiogenesis Inhibitors - immunology | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor B - antagonists & inhibitors | Recombinant Fusion Proteins - pharmacology | Endothelial Cells - metabolism | Humans | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor B - immunology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor A - immunology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - immunology | Enzyme Activation - drug effects | Bevacizumab | Endothelial Cells - immunology | Vascular Endothelial Growth Factor B - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | HEK293 Cells | Ranibizumab | Vascular Endothelial Growth Factor Receptor-1 - immunology | Antibodies, Monoclonal, Humanized - immunology | Index Medicus | Original Paper
Journal Article
Nature, ISSN 0028-0836, 04/2010, Volume 464, Issue 7290, pp. 917 - 921
The vascular endothelial growth factors (VEGFs) are major angiogenic regulators and are involved in several aspects of endothelial cell physiology. However,... 
MYOCARDIUM | SKELETAL-MUSCLE | LIPID-METABOLISM | ANGIOGENESIS | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | MOUSE | RECEPTOR | DEFICIENT MICE | VEGF-B | TRANSPORT PROTEIN | Neuropilin-1 - genetics | Vascular Endothelial Growth Factor B - deficiency | Oligonucleotide Array Sequence Analysis | Neuropilin-1 - metabolism | Humans | Adipose Tissue, White - metabolism | Male | Mitochondrial Proteins - genetics | Biological Transport | Mitochondria - genetics | Mitochondrial Proteins - metabolism | Myocardium - metabolism | Transcription, Genetic | Muscles - metabolism | Fatty Acids - metabolism | Cell Line | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Mitochondria - metabolism | Organ Specificity | Endothelium - cytology | Animals | Cell Nucleus - genetics | Endothelium - metabolism | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor B - genetics | Adipose Tissue, Brown - metabolism | Mice | Fatty Acid Transport Proteins - genetics | Care and treatment | Physiological aspects | Genetic aspects | Diabetes | Research | Cardiovascular diseases | Vascular endothelial growth factor | Proteins | Signal transduction | Rodents | Binding sites | Index Medicus | Basic Medicine | Medical and Health Sciences | Internal medicine | Medicin och hälsovetenskap | Molekylär medicin (genetik och patologi) | Molecular medicine (genetics and pathology) | MEDICINE | Dermatology and venerology,clinical genetics, internal medicine | Medical Genetics | Medicinsk genetik | MEDICIN | Medicinska och farmaceutiska grundvetenskaper | Invärtesmedicin | Dermatologi och venerologi, klinisk genetik, invärtesmedicin
Journal Article
Circulation Research, ISSN 0009-7330, 07/2011, Volume 109, Issue 3, pp. e14 - e26
RATIONALE:Systemic sclerosis (SSc) is characterized by widespread microangiopathy, fibrosis, and autoimmunity. Despite the lack of angiogenesis, the expression... 
antiangiogenic agents | systemic sclerosis | peripheral vascular diseases | vascular endothelial growth factor A | angiogenesis | scleroderma | Phosphorylation - physiology | Gene Expression - drug effects | Humans | Transforming Growth Factor beta1 - metabolism | Culture Media, Conditioned - pharmacology | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | RNA-Binding Proteins | Phosphoproteins - metabolism | Vascular Endothelial Growth Factor A - genetics | Gene Expression - physiology | Scleroderma, Systemic - physiopathology | Dermis - blood supply | Neovascularization, Pathologic - physiopathology | Phosphorylation - drug effects | Nuclear Proteins - genetics | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Transforming Growth Factor beta1 - pharmacology | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Ischemia - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Alternative Splicing - physiology | Nuclear Proteins - metabolism | Transforming Growth Factor beta1 - genetics | Ischemia - physiopathology | Phosphoproteins - genetics | Vascular Endothelial Growth Factor B - pharmacology | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Endothelial Cells - cytology | Serine-Arginine Splicing Factors | Neovascularization, Pathologic - genetics | Signal Transduction - physiology | Vascular Endothelial Growth Factor B - genetics | Neovascularization, Pathologic - metabolism | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2018, Volume 557, Issue 7707, pp. 724 - 728
Microglia and astrocytes modulate inflammation and neurodegeneration in the central nervous system (CNS)(1-3). Microglia modulate pro-inflammatory and... 
ACTIVATION | ARYL-HYDROCARBON RECEPTOR | MULTIDISCIPLINARY SCIENCES | TGF-ALPHA | CENTRAL-NERVOUS-SYSTEM | SPINAL-CORD-INJURY | DIVERSITY | VEGF-B | EXPRESSION | CNS INFLAMMATION | ENDOTHELIAL GROWTH-FACTOR | Inflammation - pathology | Microglia - metabolism | Central Nervous System - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Astrocytes - pathology | Central Nervous System - pathology | Vascular Endothelial Growth Factor B - biosynthesis | Tryptophan - metabolism | Lipopolysaccharide Receptors - metabolism | Inflammation - metabolism | Tryptophan - deficiency | Microglia - pathology | Receptors, Aryl Hydrocarbon - metabolism | Female | Transforming Growth Factor alpha - metabolism | Central Nervous System - microbiology | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - microbiology | Multiple Sclerosis - metabolism | Transforming Growth Factor alpha - biosynthesis | Symbiosis | Inflammation - microbiology | Encephalomyelitis, Autoimmune, Experimental - pathology | ErbB Receptors - metabolism | Mice, Inbred C57BL | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Animals | Vascular Endothelial Growth Factor B - metabolism | Encephalomyelitis, Autoimmune, Experimental - prevention & control | Multiple Sclerosis - pathology | Inflammation - prevention & control | Mice | Astrocytes - metabolism | Physiological aspects | Microbiological research | Metabolites | Transforming growth factors | Astrocytes | Vascular endothelial growth factor | Regulators | Multiple sclerosis | Encephalomyelitis | Transcription | Central nervous system | Hydrocarbons | Nervous system | CD14 antigen | Recruitment | Angiogenesis | Signal transduction | Neurotoxicity | Microorganisms | Neurodegeneration | Aromatic compounds | Bone marrow | ErbB-1 protein | Tryptophan | Inflammation | Gene expression | Experimental allergic encephalomyelitis | Microglia | Neurological diseases | Diet | Lymphocytes B | Flora | Spinal cord injuries | Alzheimers disease | Index Medicus | Life Sciences | Immunology
Journal Article
Nature, ISSN 0028-0836, 10/2012, Volume 490, Issue 7420, pp. 426 - 430
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2016, Volume 101, pp. 1 - 9
The objective was to examine the protective effect of resveratrol (RSV) on myocardial ischemia/reperfusion (IR) injury and whether the mechanism was related to... 
Myocardial ischemia reperfusion injury | VEGF-B | Resveratrol | ROS | Apoptosis | CANCER-CELLS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | ISCHEMIA | RECEPTOR | HEART | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | CARDIOPROTECTION | Myocardial Infarction - genetics | RNA, Small Interfering - genetics | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | Vascular Endothelial Growth Factor B - antagonists & inhibitors | Apoptosis - drug effects | Glycogen Synthase Kinase 3 beta - genetics | Creatine Kinase, MB Form - secretion | Male | Stilbenes - pharmacology | Proto-Oncogene Proteins c-akt - genetics | Myocardial Reperfusion Injury - pathology | Myocardial Infarction - pathology | Myocardium - metabolism | Vascular Endothelial Growth Factor B - agonists | Ventricular Function, Left - physiology | Myocardial Reperfusion Injury - drug therapy | Proto-Oncogene Proteins c-akt - metabolism | Myocardial Reperfusion Injury - genetics | Organ Culture Techniques | Superoxide Dismutase - metabolism | Cell Line | Signal Transduction | Ventricular Function, Left - drug effects | Gene Expression Regulation | Rats | Glycogen Synthase Kinase 3 beta - antagonists & inhibitors | Myocardium - pathology | Creatine Kinase, MB Form - antagonists & inhibitors | Antioxidants - pharmacology | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Vascular Endothelial Growth Factor B - metabolism | Proto-Oncogene Proteins c-akt - agonists | Myocytes, Cardiac - metabolism | Vascular Endothelial Growth Factor B - genetics | RNA, Small Interfering - metabolism | Medical colleges | RNA | Cell death | Vascular endothelial growth factor | Analysis | Endothelial growth factors | Index Medicus
Journal Article
Cancer, ISSN 0008-543X, 04/2009, Volume 115, Issue 8, pp. 1701 - 1712
BACKGROUND: Lung cancer is the leading cause of cancer death in the world. The objective of this study was to investigate the expression of vascular... 
Angiogenesis-inducing agents | Survival analysis | Vascular endothelial growth factor | Lung neoplasms | Vascular endothelial growth factor receptors | angiogenesis-inducing agents | lung neoplasms | GASTRIC ADENOCARCINOMA | FOLLOW-UP | VEGF-C | TUMOR-CELLS | BREAST-CANCER | vascular endothelial growth factor receptors | survival analysis | LYMPH-NODE METASTASIS | MESSENGER-RNA | ONCOLOGY | VESSEL DENSITY | vascular endothelial growth factor | MALIGNANT MESOTHELIOMA | CLINICAL-SIGNIFICANCE | Immunohistochemistry | Prognosis | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Middle Aged | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Lung Neoplasms - pathology | Male | Vascular Endothelial Growth Factor A - metabolism | Small Cell Lung Carcinoma - metabolism | Small Cell Lung Carcinoma - pathology | Vascular Endothelial Growth Factor D - metabolism | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Aged, 80 and over | Female | Small Cell Lung Carcinoma - mortality | Vascular Endothelial Growth Factors - metabolism | Aged | Vascular Endothelial Growth Factor Receptor-3 - genetics | Receptors, Growth Factor - metabolism | Vascular Endothelial Growth Factor Receptor-1 - genetics | Development and progression | Genetic aspects | Research | Lung cancer, Non-small cell | Gene expression | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Science Signaling, ISSN 1945-0877, 07/2013, Volume 6, Issue 282, pp. ra52 - ra52
Vascular endothelial growth factors (VEGFs) regulate blood and lymphatic vessel development through VEGF receptors (VEGFRs). The VEGFR immunoglobulin homology... 
SIGNAL-TRANSDUCTION | ACTIVATION | IMMUNOGLOBULIN-LIKE DOMAIN | FACTOR-B | STRUCTURAL BASIS | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | FACTOR VEGF FAMILY | PATHOLOGICAL ANGIOGENESIS | BINDING | CELL BIOLOGY | NIH 3T3 Cells | Pregnancy Proteins - chemistry | Phosphorylation | Humans | Pregnancy Proteins - genetics | Molecular Sequence Data | Pregnancy Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor Receptor-2 - genetics | Vascular Endothelial Growth Factor A - chemistry | Vascular Endothelial Growth Factor B - chemistry | Female | Vascular Endothelial Growth Factor Receptor-1 - genetics | Binding, Competitive | Protein Structure, Tertiary | Amino Acid Sequence | Cell Line | Signal Transduction | Cells, Cultured | Models, Molecular | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Binding Sites - genetics | Vascular Endothelial Growth Factor Receptor-1 - chemistry | Blotting, Western | Sequence Homology, Amino Acid | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Animals | Vascular Endothelial Growth Factor B - metabolism | Placenta Growth Factor | Protein Binding | Ligands | Vascular Endothelial Growth Factor B - genetics | Mice | Index Medicus
Journal Article