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Circulation Research, ISSN 0009-7330, 09/2013, Volume 113, Issue 8, pp. 986 - 996
RATIONALE:Endothelial dysfunction inflicted by inflammation is found in a host of cardiovascular pathologies. One hallmark event in this process is the... 
Endothelial dysfunction | Epigenetics | Inflammation | Transcription | Brg1 | Atherosclerosis | atherosclerosis | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | transcription | MUSCLE DEVELOPMENT | SWI/SNF COMPLEXES | TRANSENDOTHELIAL MIGRATION | MECHANISMS | endothelial dysfunction | epigenetics | inflammation | REMODELING COMPLEXES | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Cell Adhesion Molecules - genetics | Vascular System Injuries - immunology | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Vascular System Injuries - genetics | Endothelium, Vascular - injuries | Epigenesis, Genetic | Humans | Transcriptional Activation | Human Umbilical Vein Endothelial Cells - immunology | Endothelium, Vascular - drug effects | NF-kappa B - metabolism | Vascular System Injuries - pathology | Transfection | RNA Interference | Time Factors | HEK293 Cells | Inflammation Mediators - metabolism | Nuclear Proteins - genetics | Binding Sites | DNA Helicases - genetics | Estradiol - pharmacology | Disease Models, Animal | Endothelium, Vascular - immunology | Promoter Regions, Genetic | Signal Transduction | Endothelial Cells - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Cell Adhesion Molecules - metabolism | Mice, Knockout | Endothelial Cells - immunology | Transcription Factors - metabolism | DNA Helicases - metabolism | Vascular System Injuries - metabolism | Animals | Endothelium, Vascular - metabolism | Apolipoproteins E - genetics | Cell Line, Tumor | Endothelium, Vascular - pathology | Human Umbilical Vein Endothelial Cells - pathology | Mice | Endothelial Cells - pathology | Endothelial Cells - drug effects
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2017, Volume 113, Issue 14, pp. 1753 - 1762
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 09/2018, Volume 115, Issue 37, pp. E8660 - E8667
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2579 - 2593
OBJECTIVE—Endoluminal vascular interventions such as angioplasty initiate a sterile inflammatory response resulting from local tissue damage. This response... 
hyperplasia | macrophages | carotid artery injuries | Angioplasty | HMGBI protein | STERILE INFLAMMATION | ISCHEMIA-REPERFUSION INJURY | TOLL-LIKE RECEPTOR-4 | THERAPY | PROTEIN HMG1 | IN-VIVO | RESTENOSIS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | MICE | HEMATOLOGY | angioplasty | BOX 1 HMGB1 | Carotid Arteries - metabolism | Chemotaxis, Leukocyte | Vasculitis - metabolism | Cell Proliferation | Vascular System Injuries - genetics | Humans | Hyperplasia | Vasculitis - pathology | Myocytes, Smooth Muscle - pathology | Adaptor Proteins, Vesicular Transport - metabolism | HMGB1 Protein - genetics | Vascular System Injuries - pathology | Vasculitis - genetics | Neointima | Time Factors | Toll-Like Receptor 4 - deficiency | HMGB1 Protein - metabolism | Inflammation Mediators - metabolism | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | HMGB1 Protein - deficiency | Cytokines - metabolism | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Vascular System Injuries - metabolism | Animals | Carotid Arteries - pathology | Macrophages, Peritoneal - metabolism | Myeloid Differentiation Factor 88 - metabolism | Cell Movement
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2016, Volume 36, Issue 5, pp. 874 - 885
OBJECTIVE—Restenosis after percutaneous coronary intervention remains to be a serious medical problem. Although mineralocorticoid receptor (MR) has been... 
hyperplasia | macrophage | myeloid cells | mineralocorticoid receptor | femoral artery | Inflammation - pathology | Receptors, Mineralocorticoid - genetics | Vascular Remodeling | Cell Proliferation | Vascular System Injuries - genetics | Coculture Techniques | Hyperplasia | Myocytes, Smooth Muscle - pathology | Vascular System Injuries - enzymology | Male | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Femoral Artery - enzymology | Vascular System Injuries - pathology | Femoral Artery - injuries | Immediate-Early Proteins - metabolism | Neointima | Receptors, Mineralocorticoid - deficiency | Transfection | RNA Interference | Time Factors | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Genetic Predisposition to Disease | Paracrine Communication | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Vascular System Injuries - prevention & control | Macrophages - enzymology | Mice, Knockout | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Immediate-Early Proteins - genetics | Femoral Artery - metabolism | Inflammation - genetics | RAW 264.7 Cells | Inflammation - prevention & control | Mice | Inflammation - enzymology | Muscle, Smooth, Vascular - enzymology | Cell Movement
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2017, Volume 37, Issue 12, pp. 2311 - 2321
OBJECTIVE—Drug-eluting stent delivery of mTORC1 (mammalian target of rapamycin complex 1) inhibitors is highly effective in preventing intimal hyperplasia... 
Gene expression | Muscle, smooth | Hyperplasia | Drug-eluting stents | Therapeutics | MAMMALIAN TARGET | RAPAMYCIN | MIGRATION | MYOCARDIN | therapeutics | drug-eluting stents | MICE LACKING | INSULIN | muscle | IN-VIVO | hyperplasia | CELL DIFFERENTIATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | gene expression | smooth | REVEALS | Vascular System Injuries - genetics | Humans | Proto-Oncogene Proteins c-akt - deficiency | Myocytes, Smooth Muscle - pathology | Vascular System Injuries - enzymology | RNA, Messenger - metabolism | Proto-Oncogene Proteins c-akt - genetics | Vascular System Injuries - pathology | Neointima | Transfection | RNA Interference | Time Factors | Trans-Activators - genetics | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Binding Sites | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | RNA, Messenger - genetics | Cells, Cultured | Gene Expression Regulation | Nuclear Proteins - metabolism | Transcription Factors - genetics | Vascular System Injuries - prevention & control | Sirolimus - pharmacology | Mice, Knockout | Transcription Factors - metabolism | Cell Movement - drug effects | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Trans-Activators - metabolism | Cell Proliferation - drug effects | Muscle, Smooth, Vascular - enzymology | differentiation | signal transduction | basic science research | vascular biology | Akt | FOXO4 | neointimal hyperplasia | cell signaling | smooth muscle proliferation and differentiation | myocardin | mTORC1 | mechanisms | rapamycin
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 07/2016, Volume 96, pp. 223 - 233
Journal Article
Circulation, ISSN 0009-7322, 04/2018, Volume 137, Issue 17, pp. 1824 - 1841
Journal Article
Journal Article