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Rheumatology, ISSN 1462-0324, 04/2018, Volume 57, Issue suppl_3
Journal Article
Journal Article
Rheumatology (Oxford, England), ISSN 1462-0332, 01/2017, Volume 56, Issue 1, pp. 77 - 86
Objectives. Neuropsychiatric (NP) involvement is a poorly understood manifestation of SLE. We studied post-mortem histopathology in relation to clinical NPSLE... 
systemic lupus erythematosus | histology | complement | neuropsychiatric lupus | Life Sciences & Biomedicine | Rheumatology | Science & Technology | Immunohistochemistry | Brain - diagnostic imaging | Complement Membrane Attack Complex - metabolism | Complement C5b - immunology | Humans | Middle Aged | Brain Infarction - metabolism | Complement System Proteins - immunology | Lupus Erythematosus, Systemic - metabolism | Lupus Vasculitis, Central Nervous System - pathology | Brain Infarction - pathology | Male | Complement C5b - metabolism | Complement Membrane Attack Complex - immunology | Case-Control Studies | Complement System Proteins - metabolism | Vasculitis, Central Nervous System - diagnostic imaging | Brain - metabolism | Autopsy | Peptide Fragments - immunology | Vasculitis, Central Nervous System - metabolism | Adult | Female | Complement Pathway, Mannose-Binding Lectin | Complement Pathway, Classical | Complement C4b - immunology | Intracranial Thrombosis - pathology | Peptide Fragments - metabolism | Lupus Vasculitis, Central Nervous System - diagnostic imaging | Lupus Vasculitis, Central Nervous System - complications | Intracranial Thrombosis - metabolism | Brain Infarction - etiology | Magnetic Resonance Imaging | Autoantibodies - immunology | Intracranial Thrombosis - etiology | Complement C4b - metabolism | Vasculitis, Central Nervous System - pathology | Brain - pathology | Aged | Lupus Vasculitis, Central Nervous System - metabolism | Vasculitis, Central Nervous System - etiology | Lupus Erythematosus, Systemic - pathology | Brain Infarction - diagnostic imaging | Intracranial Thrombosis - diagnostic imaging | Index Medicus | Abridged Index Medicus
Journal Article
The New England journal of medicine, ISSN 0028-4793, 07/2012, Volume 367, Issue 3, pp. 214 - 223
This study confirms the presence of a genetic component of the pathogenesis of antineutrophil cytoplasmic antibody (ANCA)–associated vasculitis and genetic... 
WEGENERS-GRANULOMATOSIS | PATHOGENESIS | SYSTEMIC VASCULITIS | ANTIBODY-ASSOCIATED VASCULITIS | DISEASE | ANTINEUTROPHIL CYTOPLASMIC AUTOANTIBODIES | PROTEINASE-3 | NEUTROPHILS | SMALL-VESSEL VASCULITIS | UMCG Approved | GENOME-WIDE ASSOCIATION | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Genetic Predisposition to Disease | Genome-Wide Association Study | alpha 1-Antitrypsin - genetics | Major Histocompatibility Complex - genetics | Humans | Risk Factors | Male | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - genetics | Genotyping Techniques | Myeloblastin - genetics | Case-Control Studies | Granulomatosis with Polyangiitis - genetics | HLA-DP Antigens - genetics | Female | Polymorphism, Single Nucleotide | Microscopic Polyangiitis - genetics | Care and treatment | Autoantibodies | Vasculitis | Causes of | Genetic aspects | Diagnosis | Health aspects | Medical research | Statistical analysis | Disease | Pathogenesis | Proteinase | Granulomatosis | Major histocompatibility complex | Quality control | Antineutrophil cytoplasmic antibodies | Peroxidase | Histocompatibility antigen HLA | Wegener's granulomatosis | Proteinase 3 | Index Medicus | Abridged Index Medicus | Urologi och njurmedicin | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Urology and Nephrology
Journal Article
Circulation research, ISSN 0009-7330, 07/2010, Volume 107, Issue 2, pp. 263 - 270
RATIONALE:We have previously found that T lymphocytes are essential for development of angiotensin II–induced hypertension; however, the mechanisms responsible... 
vascular inflammation | T cells | hypertension | central nervous system | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Blood Pressure | Vasculitis - metabolism | Third Ventricle - injuries | Vasculitis - physiopathology | Homeodomain Proteins - metabolism | Antihypertensive Agents - administration & dosage | Receptors, Antigen, T-Cell, alpha-beta - immunology | Adoptive Transfer | Vasculitis - prevention & control | Genes, T-Cell Receptor beta | Third Ventricle - physiopathology | T-Lymphocytes - transplantation | Vasculitis - immunology | Vasculitis - genetics | Time Factors | T-Lymphocytes - drug effects | Hypertension - chemically induced | Superoxides - metabolism | Hypertension - prevention & control | Hypertension - genetics | Angiotensin II | Vasculitis - chemically induced | Disease Models, Animal | Administration, Oral | Mice, Inbred C57BL | Mice, Transgenic | Hypertension - immunology | Homeodomain Proteins - genetics | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Hydralazine - administration & dosage | Animals | Lymphocyte Activation - drug effects | Third Ventricle - immunology | Genes, T-Cell Receptor alpha | T-Lymphocytes - immunology | Mice | Norepinephrine | Index Medicus | Hypertension
Journal Article