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Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2014, Volume 34, Issue 4, pp. 790 - 800
OBJECTIVE—Cardiovascular disease (CVD), the most common morbidity resulting from atherosclerosis, remains a frequent cause of death. Efforts to develop... 
atherosclerosis | endothelium-derived vasoconstrictor factors | cardiovascular diseases | inflammation | METAANALYSIS | METASTASIS | INJURY | RISK | MECHANISMS | PREDICTION | INHIBITION | ANGIOPOIETIN-LIKE PROTEIN-2 | DISEASE | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | Chemotaxis, Leukocyte | Vasculitis - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Vasculitis - prevention & control | Vasodilation | Vasculitis - genetics | Time Factors | Diet, High-Fat | Bone Marrow Transplantation | Aged, 80 and over | Disease Models, Animal | Endothelium, Vascular - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Signal Transduction | Endothelial Cells - metabolism | Angiopoietins - deficiency | Obesity - physiopathology | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Macrophages - metabolism | Endothelium, Vascular - metabolism | Dyslipidemias - metabolism | Endothelium, Vascular - pathology | Mice | Endothelial Cells - pathology | Integrin alpha5beta1 - metabolism | Vasculitis - pathology | Monocytes - metabolism | Vasculitis - immunology | Angiopoietin-like Proteins | Inflammation Mediators - metabolism | Female | Macrophages - immunology | Angiopoietins - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Cells, Cultured | Endothelium, Vascular - physiopathology | Obesity - metabolism | Endothelial Cells - immunology | Animals | Apolipoproteins E - genetics | Angiopoietins - genetics | Nitric Oxide - metabolism | Atherosclerosis - prevention & control | Dyslipidemias - physiopathology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children. We have previously shown that both... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | Index Medicus | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
Circulation, ISSN 0009-7322, 09/2011, Volume 124, Issue 12, pp. 1370 - 1381
Background-Angiotensin II (ATII), a potent vasoconstrictor, causes hypertension, promotes infiltration of myelomonocytic cells into the vessel wall, and... 
blood vessels | inflammation | hypertension | angiotensin II | oxidative stress | MACROPHAGE INFILTRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | SUPEROXIDE-PRODUCTION | MITOCHONDRIAL ALDEHYDE DEHYDROGENASE | CELL-LINEAGE-ABLATION | BLOOD-PRESSURE | IN-VITRO | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | MICE | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Oxidative Stress - physiology | Muscle, Smooth, Vascular - immunology | Male | Monocytes - metabolism | Monocytes - immunology | Muramidase - immunology | Vasculitis - immunology | Respiratory Burst - physiology | Hypertension - chemically induced | Gene Expression - immunology | Neutrophils - metabolism | Vasculitis - chemically induced | Macrophages - immunology | Endothelium, Vascular - immunology | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Mice, Inbred C57BL | Muramidase - genetics | Receptors, Chemokine - metabolism | Neutrophils - immunology | Mice, Transgenic | Hypertension - immunology | Muramidase - metabolism | Hypertension - metabolism | Macrophages - metabolism | Animals | Endothelium, Vascular - metabolism | Mice | CD11b Antigen - metabolism | Nitric Oxide - metabolism | Hypertension | Blood circulation disorders | Oxidative stress | Angiotensin | Physiological aspects | Development and progression | Research | Lysozyme | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2013, Volume 33, Issue 8, pp. 1986 - 1993
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4190 - 4202
Macrophages in atherosclerotic plaques drive inflammatory responses, degrade lipoproteins, and phagocytose dead cells. MicroRNAs (miRs) control the... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-SYSTEM | REVERSE CHOLESTEROL TRANSPORT | ACTIVATED-RECEPTOR-GAMMA | LIPID UPTAKE | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | DEFICIENT MICE | INFLAMMATORY RESPONSE | FOAM CELLS | APOLIPOPROTEIN-E | Vasculitis - metabolism | Plaque, Atherosclerotic - genetics | Atherosclerosis - genetics | Vasculitis - pathology | MicroRNAs - metabolism | NF-kappa B - metabolism | Interferon-gamma - metabolism | Lipoproteins, LDL - genetics | Apolipoproteins E - metabolism | DNA-Binding Proteins - metabolism | Vasculitis - genetics | Chemokine CCL2 - metabolism | Lipoproteins, LDL - metabolism | Interferon-gamma - genetics | Plaque, Atherosclerotic - pathology | Atherosclerosis - pathology | DNA-Binding Proteins - antagonists & inhibitors | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Gene Expression Regulation - genetics | Chemokine CCL2 - genetics | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | NF-kappa B - genetics | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-bcl-6 | MicroRNA | Atherosclerosis | Development and progression | Genetic aspects | Research | Properties | Macrophages | Gene expression | Index Medicus | Abridged Index Medicus | Atherogenesis | Lipoproteins (low density) | Transcription factors | Immune response | Data processing | Inflammation | Lipoproteins | Monocytes | NF- Kappa B protein | Arteriosclerosis | gamma -Interferon | miRNA | Differentiation | Chemokines | Monocyte chemoattractant protein 1 | Plaques | Bcl-6 protein
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 102, pp. 10 - 21
Abstract We recently reported that increased NADPH oxidase 4 (NOX4) expression and activity during aging results in enhanced cellular and mitochondrial... 
Cardiovascular | Nanoparticles | Reactive oxygen species | Inflammation | Cytokines | Atherosclerosis | MITOCHONDRIAL OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | MELITTIN-DERIVED PEPTIDES | TGF-BETA | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CELL BIOLOGY | GROWTH-FACTOR-BETA | SIRNA TRANSFECTION | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | CORONARY-ARTERY-DISEASE | Carotid Arteries - metabolism | RNA, Small Interfering - genetics | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Humans | Vasculitis - etiology | Vasculitis - pathology | NADPH Oxidases - metabolism | Cell Survival - genetics | Male | NF-kappa B - metabolism | Gene Knockdown Techniques | Aging - genetics | RNA Interference | Inflammation Mediators - metabolism | NADPH Oxidases - genetics | Cytokines - genetics | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Cytokines - metabolism | MAP Kinase Kinase Kinases - metabolism | Mitochondria - metabolism | NADPH Oxidase 4 | Atherosclerosis - metabolism | Hydrogen Peroxide - metabolism | Mice, Knockout | Animals | Biomarkers | Carotid Arteries - pathology | Mice | Aging - metabolism | Oxidases | RNA | Genes | Transforming growth factors | Gene expression | Medical colleges | Anopheles | Peptides | Peroxides | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
CELL-ADHESION MOLECULE-1 | TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | LEUKOCYTE RECRUITMENT | HUMAN-ENDOTHELIAL-CELLS | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION PROFILES | SIGNAL-TRANSDUCTION PATHWAY | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 10/2012, Volume 111, Issue 10, pp. 1274 - 1285
RATIONALE:Atherosclerosis is a chronic inflammatory disease of the arterial wall. Several proinflammatory cytokines are known to promote atherosclerosis, but... 
atherosclerosis | vascular inflammation | cytokines | chemokines | inflammation | leukocyte recruitment | CARDIAC & CARDIOVASCULAR SYSTEMS | IL-27 | NECROSIS-FACTOR-ALPHA | T-HELPER-CELLS | IMMUNE | REDUCES ATHEROSCLEROSIS | WSX-1 | MONOCYTE | PERIPHERAL VASCULAR DISEASE | INFECTION | CHRONIC INFLAMMATION | HEMATOLOGY | E-DEFICIENT MICE | Myeloid Cells - cytology | Tumor Necrosis Factor-alpha - metabolism | Male | Aorta - metabolism | Th1 Cells - immunology | Interleukins - metabolism | CD11c Antigen - metabolism | CD4-Positive T-Lymphocytes - immunology | Th1 Cells - metabolism | Signal Transduction - immunology | Th17 Cells - metabolism | Mice, Mutant Strains | Myeloid Cells - immunology | Female | Th17 Cells - cytology | Chemokine CCL2 - metabolism | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - metabolism | Receptors, Cytokine - metabolism | Receptors, LDL - genetics | Atherosclerosis - immunology | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Receptors, LDL - metabolism | Aorta - immunology | Atherosclerosis - metabolism | Interleukin-17 - metabolism | Animals | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - immunology | Myeloid Cells - metabolism | Th17 Cells - immunology | Mice | Th1 Cells - cytology | Aorta - cytology | CD11b Antigen - metabolism | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 15, pp. 2757 - 2764
Journal Article
Circulation, ISSN 0009-7322, 02/2013, Volume 127, Issue 7, pp. 842 - 853
Background-Revealing the mechanisms underlying the functional integrity of the vascular system could make available novel therapeutic approaches. We previously... 
endothelium | vasculature | arteriosclerosis | aging | edema | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | HYPOTENSIVE PEPTIDE | ENDOGENOUS PEPTIDE | BARRIER FUNCTION | ATHEROSCLEROSIS | CELL SENESCENCE | ISCHEMIA | GENE | PLASMA-LEVELS | PERIPHERAL VASCULAR DISEASE | RECEPTOR-LIKE RECEPTOR | Vasculitis - metabolism | Age Factors | Cadherins - metabolism | Kidney - pathology | Vasculitis - physiopathology | Oxidative Stress - physiology | Vasculitis - pathology | Fibrosis - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Kidney - metabolism | Glomerulosclerosis, Focal Segmental - physiopathology | Cadherins - genetics | Kidney - physiopathology | Disease Models, Animal | Fibrosis - physiopathology | Arteriosclerosis - physiopathology | Endothelium, Vascular - physiopathology | Receptor Activity-Modifying Protein 2 - metabolism | Adrenomedullin - metabolism | Glomerulosclerosis, Focal Segmental - metabolism | Mice, Knockout | Edema - metabolism | Animals | Endothelium, Vascular - metabolism | Homeostasis - physiology | Receptor Activity-Modifying Protein 2 - genetics | Endothelium, Vascular - pathology | Arteriosclerosis - metabolism | Edema - physiopathology | Mice | Fibrosis - pathology | Arteriosclerosis - pathology | Edema - pathology | Leukocytes - metabolism | Glomerulosclerosis, Focal Segmental - pathology | Aging - metabolism | Homeostasis | Physiological aspects | Vascular endothelium | Research | Cellular control mechanisms | Index Medicus | Abridged Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 01/2012, Volume 13, Issue 1, pp. 67 - 76
Journal Article