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Journal of Clinical Investigation, ISSN 0021-9738, 10/2008, Volume 118, Issue 10, pp. 3343 - 3354
Journal Article
Life Sciences, ISSN 0024-3205, 2008, Volume 83, Issue 7, pp. 264 - 271
We have previously demonstrated the participation of reactive oxygen species (ROS) in the positive inotropic effect of a physiological concentration of... 
Reactive oxygen species | Inotropic effect | Cardiac myocytes | Endothelin-1 | Sodium–hydrogen exchanger | Sodium-hydrogen exchanger | MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN-KINASE-C | OXIDATIVE STRESS | K-ATP CHANNELS | NA+-H+ EXCHANGER | ANGIOTENSIN-II | inotropic effect | cardiac myocytes | NA+/CA2+ EXCHANGER | RABBIT VENTRICULAR MYOCYTES | reactive oxygen species | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | HYDROGEN-PEROXIDE | endothelin-1 | sodium-hydrogen exchanger | Endothelin-1 - antagonists & inhibitors | Free Radical Scavengers - pharmacology | Glycine - analogs & derivatives | Heart Ventricles - cytology | Mitochondria, Heart - metabolism | Sulfhydryl Compounds - pharmacology | Endothelin-1 - pharmacology | NADPH Oxidases - metabolism | Myocardial Contraction - drug effects | Sodium-Hydrogen Exchangers - metabolism | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Superoxides - metabolism | Superoxides - antagonists & inhibitors | Potassium Channel Blockers - pharmacology | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Cardiotonic Agents - antagonists & inhibitors | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Cats | Myocytes, Cardiac - cytology | Vasoconstrictor Agents - antagonists & inhibitors | Sarcomeres - metabolism | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Angiotensin II - antagonists & inhibitors | Cardiotonic Agents - pharmacology | Animals | Glycine - pharmacology | Myocytes, Cardiac - metabolism | Heart Ventricles - metabolism
Journal Article
Journal Article
EUROPEAN JOURNAL OF PHARMACOLOGY, ISSN 0014-2999, 05/2012, Volume 683, Issue 1-3, pp. 204 - 210
During a migraine attack capsaicin-sensitive trigeminal sensory nerves release calcitonin gene-related peptide (CGRP), producing cranial vasodilatation and... 
B-HT 933 | SUBTYPES | Migraine | JP-1302 | External carotid artery | MEDIATE | ERGOTAMINE | CLONIDINE | ALPHA(2A/2C)-ADRENOCEPTORS | BRL44408 | PROFILE | PHARMACOLOGY & PHARMACY | CGRP | VASOCONSTRICTION | 5-HT1B RECEPTORS | Spinal Cord - drug effects | Spinal Cord - metabolism | Infusions, Spinal | Receptors, Adrenergic, alpha-2 - metabolism | Male | Acetylcholine - metabolism | Receptors, Adrenergic, alpha-2 - chemistry | Migraine Disorders - metabolism | Migraine Disorders - chemically induced | Protein Isoforms - metabolism | Regional Blood Flow - drug effects | Adrenergic alpha-2 Receptor Antagonists - administration & dosage | Calcitonin Gene-Related Peptide - metabolism | Disease Models, Animal | Adrenergic alpha-2 Receptor Antagonists - chemistry | Vasoconstrictor Agents - therapeutic use | Analgesics, Non-Narcotic - antagonists & inhibitors | Cervical Vertebrae | Vasoconstrictor Agents - administration & dosage | Vasoconstrictor Agents - antagonists & inhibitors | Vasodilator Agents - antagonists & inhibitors | Capsaicin - toxicity | Carotid Artery, External - physiology | Adrenergic alpha-2 Receptor Antagonists - therapeutic use | Analgesics, Non-Narcotic - administration & dosage | Analgesics, Non-Narcotic - therapeutic use | Migraine Disorders - drug therapy | Animals | Vasodilator Agents - toxicity | Dogs | Hemodynamics - drug effects | Capsaicin - antagonists & inhibitors | Carotid Artery, External - drug effects | Protein Isoforms - antagonists & inhibitors
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 07/2013, Volume 346, Issue 1, pp. 38 - 47
Extracellular calcium (Ca-e(2+))-induced relaxation of isolated, phenylephrine (PE)-contracted mesenteric arteries is dependent on an intact perivascular... 
CALCIUM-SENSING RECEPTOR | NERVE CA2+ RECEPTOR | HYPERPOLARIZING FACTOR | RESISTANCE ARTERIES | INTERSTITIAL CA2 | ENDOTHELIAL-CELLS | DEPENDENT CA2+-INDUCED RELAXATION | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | HYPERTENSIVE-RATS | BLOOD-PRESSURE | Up-Regulation | Mesenteric Arteries - drug effects | Nerve Net - enzymology | Nerve Net - drug effects | Receptors, Calcium-Sensing - metabolism | Nitric Oxide Synthase Type I - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Receptors, Calcium-Sensing - antagonists & inhibitors | Vasoconstrictor Agents - pharmacology | Nerve Tissue Proteins - antagonists & inhibitors | Vasodilator Agents - pharmacology | Vasoconstrictor Agents - antagonists & inhibitors | Nitric Oxide - antagonists & inhibitors | Down-Regulation | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Calcium Channel Blockers - pharmacology | Mesenteric Arteries - metabolism | Nerve Tissue Proteins - genetics | Nitric Oxide Synthase Type III - genetics | Vasoconstriction - drug effects | Receptors, Calcium-Sensing - agonists | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Nerve Tissue Proteins - metabolism | Animals | Calcium Signaling - drug effects | Nitric Oxide Synthase Type I - metabolism | Nerve Net - metabolism | Mesenteric Arteries - innervation | Mice | Nitric Oxide Synthase Type I - genetics | Vasodilation - drug effects | In Vitro Techniques | Nitric Oxide - metabolism
Journal Article
Journal of Physiology and Biochemistry, ISSN 1138-7548, 6/2016, Volume 72, Issue 2, pp. 245 - 253
We previously observed that sarcoendoplasmic reticulum Ca2+ ATPase (SERCA) blockade by cyclopiazonic acid (CPA) significantly potentiates serotonin... 
Animal Physiology | Biomedicine | Dexamethasone | Human Physiology | Serotonin | CPA | SOC | PKC | Biomedicine general | CA2+ ENTRY | PHYSIOLOGY | PULMONARY-HYPERTENSION | RAT | INTRACELLULAR CALCIUM | ATPASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | GENE | STORE | RECEPTORS | EXPRESSION | Dexamethasone - antagonists & inhibitors | Muscle, Smooth, Vascular - metabolism | Methysergide - pharmacology | Serotonin - chemistry | Serotonin Receptor Agonists - pharmacology | Dexamethasone - pharmacology | Protein Kinase C - metabolism | Enzyme Inhibitors - chemistry | Indoles - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - antagonists & inhibitors | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Cell Line | Ketanserin - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Vasodilator Agents - pharmacology | Vasoconstrictor Agents - antagonists & inhibitors | Anti-Inflammatory Agents - pharmacology | Serotonin Receptor Agonists - chemistry | Vasodilator Agents - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Protein Kinase C - antagonists & inhibitors | Calcium Channel Blockers - pharmacology | Serotonin Antagonists - pharmacology | Animals | Anti-Inflammatory Agents - chemistry | Calcium Signaling - drug effects | Serotonin - metabolism | Protein Kinase Inhibitors - pharmacology | Indoles - antagonists & inhibitors | Analysis | Pharmacy | Central nervous system depressants | Sphingosine | Phenols | Drugstores | Smooth muscle | Protein kinases | Adenosine triphosphatase
Journal Article
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 06/2001, Volume 88, Issue 11, pp. 1102 - 1104
The aim of this work was to investigate the coupling of human urotensin II (hU-II) to RhoA activation and regulation of RhoA-dependent functions. The use of... 
Signal transduction | Vascular | G protein | SIGNAL-TRANSDUCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | SENSITIZATION | PROTEIN-KINASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | VASOCONSTRICTION | Norepinephrine - antagonists & inhibitors | Norepinephrine - pharmacology | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Recombinant Fusion Proteins - pharmacology | Urotensins - pharmacology | Humans | rhoA GTP-Binding Protein - metabolism | Recombinant Fusion Proteins - metabolism | Botulinum Toxins | Dose-Response Relationship, Drug | Vasoconstriction - physiology | Urotensins - metabolism | Potassium Chloride - pharmacology | rho-Associated Kinases | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - metabolism | ADP Ribose Transferases - metabolism | Muscle, Smooth, Vascular - drug effects | Amides - pharmacology | Vasoconstrictor Agents - pharmacology | Vasoconstrictor Agents - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Aorta, Thoracic - metabolism | Intracellular Signaling Peptides and Proteins | Rats | rhoA GTP-Binding Protein - antagonists & inhibitors | Muscle, Smooth, Vascular - cytology | Vasoconstriction - drug effects | Cell Division - drug effects | ADP Ribose Transferases - genetics | Aorta, Thoracic - cytology | Animals | Signal Transduction - drug effects | ADP Ribose Transferases - pharmacology | Cytoskeleton - metabolism | Recombinant Fusion Proteins - genetics | Signal Transduction - physiology | Pyridines - pharmacology | In Vitro Techniques | Enzyme Activation - physiology | Cytoskeleton - drug effects
Journal Article
Journal of Cardiovascular Pharmacology, ISSN 0160-2446, 09/2005, Volume 46, Issue 3, pp. 274 - 279
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2000, Volume 97, Issue 17, pp. 9753 - 9758
A major feature of septic shock is the development of a vascular crisis characterized by nonresponsiveness to sympathetic vasoconstrictor agents and the... 
Enzymes | Molecules | Biological Sciences | DNA damage | Superoxides | Catecholamines | Septic shock | Norepinephrine | Blood pressure | Hypotension | Blood plasma | TUMOR-NECROSIS-FACTOR | SYNTHASE MESSENGER-RNA | MUTANT MICE | METABOLITES | CIRCULATORY SHOCK | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISMUTASE | NITRIC-OXIDE | PEROXYNITRITE FORMATION | EPINEPHRINE | Hypotension - pathology | Norepinephrine - antagonists & inhibitors | Norepinephrine - pharmacology | Catecholamines - metabolism | Male | Epinephrine - antagonists & inhibitors | Shock, Septic - metabolism | Dose-Response Relationship, Drug | Adrenochrome - blood | Shock, Septic - blood | Superoxides - metabolism | Blood Pressure - drug effects | Superoxides - antagonists & inhibitors | Hypotension - blood | Superoxide Dismutase - metabolism | Disease Models, Animal | Vasoconstrictor Agents - pharmacology | Shock, Septic - pathology | Shock, Septic - chemically induced | Vasoconstrictor Agents - antagonists & inhibitors | Superoxides - pharmacology | Rats | Catecholamines - blood | Hypotension - chemically induced | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Catecholamines - antagonists & inhibitors | Norepinephrine - blood | Animals | Hypotension - metabolism | Norepinephrine - metabolism | Vasoconstrictor Agents - blood | Vasoconstrictor Agents - metabolism | Epinephrine - blood | Lipopolysaccharides - pharmacology | Kinetics | Catecholamines - pharmacology | Epinephrine - metabolism | Organometallic Compounds - pharmacology | Manganese | Physiological aspects | Superoxide | Research | Catecholamine metabolism
Journal Article