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Journal of translational medicine, ISSN 1479-5876, 04/2019, Volume 17, Issue 1, pp. 127 - 127
...) especially in those with HF at baseline. These might indicate a potent direct cardioprotective effect, which is currently incompletely understood... 
Sodium-glucose cotransporter-2 inhibitor | Cardioprotection | Canagliflozin | Myocardial ischemia-reperfusion injury | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Apoptosis - drug effects | Canagliflozin - pharmacology | Myocardial Reperfusion Injury - complications | Systole - drug effects | Male | Aldehydes - metabolism | Liver - physiopathology | Cardiotonic Agents - therapeutic use | Liver - drug effects | Canagliflozin - therapeutic use | Diastole - drug effects | Aorta - physiopathology | Myocardial Reperfusion Injury - drug therapy | Phosphorylation - drug effects | Kidney - physiopathology | Biomarkers - metabolism | Endothelium - pathology | Glycosuria - physiopathology | Kidney - drug effects | Aorta - drug effects | Ventricular Function, Left - drug effects | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glycosuria - complications | Myocardial Reperfusion Injury - physiopathology | Endothelium - physiopathology | Aorta - pathology | Endothelium - drug effects | Animals | Signal Transduction - drug effects | Oxidative Stress - drug effects | Vasodilation - drug effects | Blood Glucose - metabolism | Myocardial Reperfusion Injury - prevention & control | Nitrosative Stress - drug effects | Heart failure | Diabetics | Rats as laboratory animals | Analysis | Clinical trials | Research | Drug therapy | Health aspects | Occlusion | Myocardial infarction | Heart | Oxidative stress | Phosphorylation | Drug delivery systems | Intravenous administration | Heart attacks | Bax protein | 4-Hydroxynonenal | Bcl-2 protein | AKT protein | Myocardial ischemia | mRNA | Size determination | Kinases | Vasodilation | Proteins | Reperfusion | Ischemia | Rodents | Calcium-binding protein | Aorta | Heart diseases | Injury analysis | Medical research | Adenosine monophosphate | AMP | Diabetes mellitus | Coronary artery | Gene expression | Nitric-oxide synthase | Endothelium | Sodium | Protein kinase | Adenosine kinase | Nitric oxide | Ventricle | Diabetes | Laboratory animals | Apoptosis | Index Medicus | Myocardial ischemia–reperfusion injury | Sodium–glucose cotransporter-2 inhibitor
Journal Article
Circulation research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 342 - 353
A large body of evidence has accrued indicating that voltage-gated Ca channel subtypes, including L-, T-, N-, and P/Q-type, are present within renal vascular... 
Renal microcirculation | Mibefradil | channels | Afferent arteriole | channel blockers | Efferent arteriole | Renal disease | Voltage-dependent Ca | Efonidipine | Ca | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Arterioles - physiology | Kidney - physiology | Protein Subunits | Antihypertensive Agents - pharmacology | Kidney - blood supply | Humans | Calcium Channel Blockers - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - classification | Calcium Channels - physiology | Calcium Channels, T-Type - chemistry | Neurotransmitter Agents - secretion | Calcium Channels - drug effects | Calcium Channels, N-Type - drug effects | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Calcium Channels - classification | Rats | Calcium Channels, L-Type - physiology | Antihypertensive Agents - therapeutic use | Arterioles - drug effects | Disease Progression | Antihypertensive Agents - adverse effects | Mice, Knockout | Calcium Signaling - drug effects | Diabetes Mellitus - physiopathology | Models, Biological | Calcium Channels - chemistry | Calcium Channels, T-Type - drug effects | Mice | Vasodilation - drug effects | Hydronephrosis - physiopathology | Calcium Channel Blockers - adverse effects | Calcium Signaling - physiology | Cardiovascular Diseases - drug therapy | Renal Circulation - physiology | Microcirculation - drug effects | Microcirculation - physiology | Renal Circulation - drug effects | Blood Pressure - drug effects | Calcium Channels, L-Type - chemistry | Kidney Diseases - metabolism | Calcium Channels, T-Type - physiology | Renin - secretion | Aldosterone - physiology | Kidney Diseases - drug therapy | Renin-Angiotensin System - physiology | Calcium Channel Blockers - pharmacology | Hypertension - physiopathology | Animals | Calcium Channels, L-Type - drug effects | Calcium Channels, N-Type - physiology | Calcium Channels, N-Type - chemistry | Index Medicus
Journal Article
Journal of internal medicine, ISSN 0954-6820, 04/2016, Volume 279, Issue 4, pp. 315 - 336
Nitric oxide (NO) is generated endogenously by NO synthases to regulate a number of physiological processes including cardiovascular and metabolic functions. A... 
blood pressure | vasodilatation | ischaemia | nitric oxide | beetroot | Beetroot | Ischaemia | Vasodilatation | Blood pressure | Nitric oxide | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Nitric Oxide - biosynthesis | Cardiovascular Diseases - drug therapy | Cardiovascular Diseases - prevention & control | Humans | Reperfusion Injury - drug therapy | Nitrites - metabolism | Biological Availability | Metabolic Diseases - drug therapy | Cardiovascular System - drug effects | Myocardial Infarction - metabolism | Heart Failure - drug therapy | Nitrites - therapeutic use | Nitrates - metabolism | Nitrates - therapeutic use | Animals | Myocardial Infarction - drug therapy | Leukocytes - drug effects | Nitric Oxide Synthase - biosynthesis | Blood Pressure - drug effects | Hypertension, Pulmonary - drug therapy | Metabolic Diseases - prevention & control | Platelet Activation - drug effects | Vasodilation - drug effects | Disease Models, Animal | Hypertension | Analysis | Atherosclerosis | Physiological aspects | Health aspects | Heart attack | Myocardial infarction | Reactive oxygen species | Animal models | Vegetables | Nitrites | Clinical trials | Cardiovascular disease | Nitrates | Vasodilation | Anions | Mitochondria | Reperfusion | Ischemia | Biocompatibility | Oxidation | Medical research | Diabetes mellitus | Lung diseases | Bioavailability | Substrates | Pulmonary artery | Biological activity | Arteriosclerosis | Infarction | Cardiovascular diseases | Metabolic disorders | Index Medicus
Journal Article
Aging cell, ISSN 1474-9718, 02/2017, Volume 16, Issue 1, pp. 17 - 26
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 176, Issue 3, pp. 1306 - 1315
Journal Article